標(biāo)題: Titlebook: Cardiac Metabolism in Health and Disease; Salah Abdel-Aleem,James E. Lowe Book 1998 Springer Science+Business Media Dordrecht 1998 ATP.Gly [打印本頁] 作者: 板條箱 時(shí)間: 2025-3-21 17:58
書目名稱Cardiac Metabolism in Health and Disease影響因子(影響力)
書目名稱Cardiac Metabolism in Health and Disease影響因子(影響力)學(xué)科排名
書目名稱Cardiac Metabolism in Health and Disease網(wǎng)絡(luò)公開度
書目名稱Cardiac Metabolism in Health and Disease網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cardiac Metabolism in Health and Disease被引頻次
書目名稱Cardiac Metabolism in Health and Disease被引頻次學(xué)科排名
書目名稱Cardiac Metabolism in Health and Disease年度引用
書目名稱Cardiac Metabolism in Health and Disease年度引用學(xué)科排名
書目名稱Cardiac Metabolism in Health and Disease讀者反饋
書目名稱Cardiac Metabolism in Health and Disease讀者反饋學(xué)科排名
作者: Cholagogue 時(shí)間: 2025-3-21 23:51 作者: Angiogenesis 時(shí)間: 2025-3-22 03:41
Carnitine deficiency-induced cardiomyopathy,e a major effect on the cardiac contractile function, although substrate oxidation may be altered. However, with longer durations of carnitine deficiency, alterations occur within the heart that may result in impaired contractile performance, particularly at high workloads.At this point, the mechani作者: 不合 時(shí)間: 2025-3-22 05:53
Fatty acid-binding proteins in the heart,unctioning. The low solubility of fatty acids in aqueous solutions impairs their cellular transport. However, cardiac tissue contains several proteins capable of binding fatty acids non-covalently. These fatty acid-binding proteins (FABPs) are thought to facilitate both cellular uptake and intracell作者: 變態(tài) 時(shí)間: 2025-3-22 08:59
Metabolic disturbances in diabetic cardiomyopathy,y cardiac myocytes could be the primary injury in the pathogenesis of this specific heart muscle disease. For example, in diabetes, glucose utilization is insignificant, and energy production is shifted almost exclusively towards β-oxidation of free fatty acids (FFA). FFA’s are supplied to cardiac c作者: GEN 時(shí)間: 2025-3-22 16:18 作者: GEN 時(shí)間: 2025-3-22 19:46
Phospholipase A2-mediated hydrolysis of cardiac phospholipids: The use of molecular and transgenic net degradation of phospholipids and the accumulation of degradation products, such as lysophospholipids and (non-esterified) fatty acids. The derangements in phospholipid metabolism are thought to be involved in the sequence of events leading to irreversible myocardial injury. The net degradation o作者: CROAK 時(shí)間: 2025-3-22 21:58 作者: 傳授知識(shí) 時(shí)間: 2025-3-23 03:25 作者: vanquish 時(shí)間: 2025-3-23 05:45
Regulation of carbohydrate and fatty acid utilization by L-carnitine during cardiac development andd intracellularly or by extracellular substrate availability. To determine this, the effect of exogenous L-carnitine on the regulation of carbohydrate and fatty acid metabolism was determined during cardiac stress (hypoxia) and during the postnatal period. The effect of L-carnitine on long chain (pa作者: Traumatic-Grief 時(shí)間: 2025-3-23 11:10
Identification of nucleoside transport binding sites in the human myocardium,kers. However, clinical application of nucleoside transport inhibitors remains to be demonstrated in humans. It is not known whether human heart has nucleoside transport similar to that of animals. The aim of this study is to pharmacologically identify the presence of nucleoside transport binding si作者: 堅(jiān)毅 時(shí)間: 2025-3-23 16:40
Mutagenesis and characterization of specific residues in fatty acid ethyl ester synthase: A gene fotion to site specific mutagenesis. In this study, three residues (Gly 32, Cys 39 and His 72) have been mutated to observe their role in enzyme activity. Gly to Gln, Cys to Trp and His to Ser mutations did not affect fatty acid ethyl ester synthase activity, but His to Ser mutant had less than 9% of 作者: duplicate 時(shí)間: 2025-3-23 18:23
Palmitate oxidation by the mitochondria from volume-overloaded rat hearts,aded rat hearts. The most significant data are the following: (1) The slowing down of long-chain fatty acid oxidation in severely hypertrophied hearts cannot be related to a feedback inhibition of carnitine palmitoyltransferase I from an excessive stimulation of glucose oxidation since, because of d作者: obligation 時(shí)間: 2025-3-24 01:21
Effects of phosphodiesterase inhibitors on glucose utilization in isolated cardiac myocytes,oupled and inversely related in cardiac tissue to fatty acid oxidation, this study was designed to investigate enoximone’s effects on glucose metabolism in the heart. To determine if enoximone alters this reciprocal relationship, the effects of enoximone on [U-.C]glucose and [2-.C]pyruvate oxidation作者: 你敢命令 時(shí)間: 2025-3-24 03:29 作者: 感情脆弱 時(shí)間: 2025-3-24 07:13 作者: DENT 時(shí)間: 2025-3-24 11:27 作者: 我悲傷 時(shí)間: 2025-3-24 16:45
The liver isoform of carnitine palmitoyltransferase I is activated in neonatal rat cardiac myocytesy. Neonatal cardiac myocytes in culture respond to hypoxia/serum-and glucose-free medium by a slow decline in ATP which reverses upon oxygenation. This model was employed to examine whether carnitine palmitoyltransferase I (CPT-I) modulates high rates of β-oxidation following oxygen deprivation. Aft作者: adipose-tissue 時(shí)間: 2025-3-24 22:46
Human myocardial ATP content and , contractile function,ine nucleotides, and NAD were quantified in human myocardial biopsies using high performance liquid chromatography. Right ventricular endomyocardial biopsies were obtained from 43 patients with dilated cardiomyopathy, 6 with restrictive cardiomyopathy, 10 with normal systolic and diastolic function,作者: Initiative 時(shí)間: 2025-3-25 02:54
Developments in Molecular and Cellular Biochemistryhttp://image.papertrans.cn/c/image/221795.jpg作者: 下垂 時(shí)間: 2025-3-25 06:58
https://doi.org/10.1007/978-1-4615-5687-9ATP; Glycogen; Oxidation; Purine; biochemistry; cardiac function; cardiovascular; metabolism作者: 巨碩 時(shí)間: 2025-3-25 08:03 作者: Forage飼料 時(shí)間: 2025-3-25 14:46 作者: verdict 時(shí)間: 2025-3-25 18:49
Feed analyses and digestion methods,r cardiac muscle contraction. CPT-I catalyzes the transfer of long chain fatty acids into mitochondria and is recognized as the primary rate controlling step in fatty acid oxidation. Molecular cloning techniques have demonstrated that two CPT-I isoforms exist as genes encoding the ‘muscle’ and ‘live作者: 無法治愈 時(shí)間: 2025-3-25 21:01 作者: 鎮(zhèn)壓 時(shí)間: 2025-3-26 02:00
https://doi.org/10.1007/978-3-476-02693-4unctioning. The low solubility of fatty acids in aqueous solutions impairs their cellular transport. However, cardiac tissue contains several proteins capable of binding fatty acids non-covalently. These fatty acid-binding proteins (FABPs) are thought to facilitate both cellular uptake and intracell作者: blackout 時(shí)間: 2025-3-26 04:34 作者: 銀版照相 時(shí)間: 2025-3-26 11:35 作者: Keratectomy 時(shí)間: 2025-3-26 13:30 作者: heartburn 時(shí)間: 2025-3-26 17:19 作者: Habituate 時(shí)間: 2025-3-26 21:56 作者: 粗糙濫制 時(shí)間: 2025-3-27 03:34 作者: asthma 時(shí)間: 2025-3-27 06:46 作者: lipoatrophy 時(shí)間: 2025-3-27 12:01 作者: Scintigraphy 時(shí)間: 2025-3-27 14:23 作者: MOCK 時(shí)間: 2025-3-27 20:42
Tatiana Savoia Landini,Fran?ois Dépelteauoupled and inversely related in cardiac tissue to fatty acid oxidation, this study was designed to investigate enoximone’s effects on glucose metabolism in the heart. To determine if enoximone alters this reciprocal relationship, the effects of enoximone on [U-.C]glucose and [2-.C]pyruvate oxidation作者: HAIL 時(shí)間: 2025-3-27 23:30 作者: Galactogogue 時(shí)間: 2025-3-28 04:38 作者: 陶器 時(shí)間: 2025-3-28 09:50 作者: BLUSH 時(shí)間: 2025-3-28 12:49
Xavier Rousseaux,Quentin Verreyckeny. Neonatal cardiac myocytes in culture respond to hypoxia/serum-and glucose-free medium by a slow decline in ATP which reverses upon oxygenation. This model was employed to examine whether carnitine palmitoyltransferase I (CPT-I) modulates high rates of β-oxidation following oxygen deprivation. Aft作者: verdict 時(shí)間: 2025-3-28 16:58
Norbert Elias in Troubled Timesine nucleotides, and NAD were quantified in human myocardial biopsies using high performance liquid chromatography. Right ventricular endomyocardial biopsies were obtained from 43 patients with dilated cardiomyopathy, 6 with restrictive cardiomyopathy, 10 with normal systolic and diastolic function,作者: Affluence 時(shí)間: 2025-3-28 22:02 作者: Heretical 時(shí)間: 2025-3-28 23:35 作者: declamation 時(shí)間: 2025-3-29 05:44
Feed analyses and digestion methods, more sensitive to malonyl-CoA inhibition but has not been found to undergo any alteration of its sensitivity. Shortterm control of activity of muscle CPT-I is apparently regulated by malonyl-CoA concentration in response to fuel supply (glucose, lactate, pyruvate and ketone bodies). The liver isofo作者: DEFT 時(shí)間: 2025-3-29 08:32 作者: 青少年 時(shí)間: 2025-3-29 11:54
Norbert Elias and Empirical Researchhannel blockers are suitable agents, potassium channel openers cause myocardial preconditioning and decrease the infarct size in animal models, but they inhibit the insulin release after glucose administration in healthy subjects. Furthermore, potassium channel blockers abolish myocardial preconditi作者: Insatiable 時(shí)間: 2025-3-29 18:41 作者: 一小塊 時(shí)間: 2025-3-29 23:13 作者: Tempor 時(shí)間: 2025-3-30 01:30
https://doi.org/10.1057/9781137312112lycolysis and glucose oxidation using perfusate labelled with [5- .H/U-.C]-glucose. Rates of H. production were calculated by measuring the difference between glycolysis and glucose oxidation. The H. production throughout the perfusion was generally lower in diabetic rat hearts compared to control h作者: Arbitrary 時(shí)間: 2025-3-30 05:43
Elias, Freud, and the Human Science0.038 nmol/mg protein/30 min). Addition of L-carnitine to the incubation medium, which stimulates carnitine palmitoyl-transferase I (CPTI) accelerated palmitate oxidation 3 fold in NB and approximately 2 fold in HY and 2 week cells. In contrast, octanoate oxidation which was greater in new born myoc作者: obscurity 時(shí)間: 2025-3-30 08:57
Five Misunderstandings of Elias’s Thoughtmperature in the presence or absence of non-radioactive NBMPR or other nucleoside transport blockers such as p-nitrobenzylthioguanosine dipyridamole, lidoflazine, papaverin, adenosine and doxorubcine. From saturation curves and inhibition kinetics, we determined the relative maximal binding (B.) and作者: bypass 時(shí)間: 2025-3-30 12:37 作者: 細(xì)絲 時(shí)間: 2025-3-30 17:06 作者: champaign 時(shí)間: 2025-3-30 22:20
Florence Delmotte,Christophe Majastre 1 cardioplegia and 90 min ischemia at 25°C with infusions of cardioplegia every 30 min. The changes in ATP, phosphocreatine (PCr) and inorganic phosphate (Pi) were followed by .P nuclear magnetic resonance (NMR) spectroscopy. Systolic and diastolic function was assessed with an intraventricular bal作者: 幻想 時(shí)間: 2025-3-31 02:29 作者: labile 時(shí)間: 2025-3-31 08:42
Ludivine Damay,Florence Delmotteschemic function, cardiac power during reperfusion improved in groups PC5/10 and PC5/20, but was not different from control in groups PC5/5 and PC3 × 5/5. There was no correlation between preischemic glycogen levels and recovery of function during reperfusion. There was also no correlation between g作者: 暗指 時(shí)間: 2025-3-31 09:23 作者: lethal 時(shí)間: 2025-3-31 16:02
Norbert Elias in Troubled Timesssue samples from patients with dilated or restrictive cardiomyopathy. There was a significant correlation between ATP and pulmonary capillary wedge pressures but not ejection fractions. Declines in the sizes of myocardial ATP, adenine nucleotide, and pyridine nucleotide pools in the human myocardiu作者: 規(guī)章 時(shí)間: 2025-3-31 21:15
,Glucose and glycogen utilisation in myocardial ischemia — Changes in metabolism and consequences fomay explain some of the controversial findings reported in the literature. However, an optimal balance between the rate of ATP production and rate of accumulation of metabolites (determined by the glycolytic flux rate and the rate of coronary washout), may ensure optimal recovery. In addition, the e作者: 有法律效應(yīng) 時(shí)間: 2025-4-1 00:32 作者: 撫育 時(shí)間: 2025-4-1 04:45 作者: 植物群 時(shí)間: 2025-4-1 07:23
Diabetes mellitus and cardiac function,hannel blockers are suitable agents, potassium channel openers cause myocardial preconditioning and decrease the infarct size in animal models, but they inhibit the insulin release after glucose administration in healthy subjects. Furthermore, potassium channel blockers abolish myocardial preconditi作者: preeclampsia 時(shí)間: 2025-4-1 12:14
