標(biāo)題: Titlebook: Cardiac Mechanobiology in Physiology and Disease; Markus Hecker,Dirk J. Duncker Book 2023 The Editor(s) (if applicable) and The Author(s), [打印本頁] 作者: enamel 時間: 2025-3-21 16:05
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書目名稱Cardiac Mechanobiology in Physiology and Disease網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cardiac Mechanobiology in Physiology and Disease被引頻次
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書目名稱Cardiac Mechanobiology in Physiology and Disease讀者反饋
書目名稱Cardiac Mechanobiology in Physiology and Disease讀者反饋學(xué)科排名
作者: 儀式 時間: 2025-3-22 00:14
https://doi.org/10.1007/978-3-658-11334-6nate into pathologies such as heart failure. In the present contribution, we will briefly discuss the role of mechanobiology in cardiac morphogenesis and the potential role of mechanical forces in cardiac disease.作者: RODE 時間: 2025-3-22 00:41
Nonverbale Kommunikation im Recruitingill help to develop a new generation of heart failure drugs that can slow down this process. The aim of this review is to describe the changes that are observed at the level of the cardiomyocyte mainly in hypertrophic and in dilated cardiomyopathy and to identify potential hot spots of mechanosignalling in the cells.作者: 網(wǎng)絡(luò)添麻煩 時間: 2025-3-22 06:01
https://doi.org/10.1007/978-3-642-46907-7s chapter, we highlight the key distinctions between iPSC-CMs and primary adult CMs and summarized the different biophysical strategies employed to improve the maturation of iPSC-CMs in terms of morphology, contractility, electrophysiology, and metabolism.作者: duplicate 時間: 2025-3-22 12:16
Mechanobiology of Cardiac Growth in Health and Disease,nate into pathologies such as heart failure. In the present contribution, we will briefly discuss the role of mechanobiology in cardiac morphogenesis and the potential role of mechanical forces in cardiac disease.作者: 窒息 時間: 2025-3-22 15:41 作者: 窒息 時間: 2025-3-22 17:50
Biophysical Stretch Induced Differentiation and Maturation of Induced Pluripotent Stem Cell-Deriveds chapter, we highlight the key distinctions between iPSC-CMs and primary adult CMs and summarized the different biophysical strategies employed to improve the maturation of iPSC-CMs in terms of morphology, contractility, electrophysiology, and metabolism.作者: Expand 時間: 2025-3-23 01:15 作者: 為敵 時間: 2025-3-23 02:23 作者: 喪失 時間: 2025-3-23 08:01 作者: 語言學(xué) 時間: 2025-3-23 09:49 作者: prick-test 時間: 2025-3-23 14:10 作者: indenture 時間: 2025-3-23 21:03
Therapeutic Innovations for Heart Failure,proaches aiming to reduce the severity of symptoms and improving cardiac function in the transition from compensated hypertrophy to myocardial failure. Moreover, we describe the main anti-inflammatory treatments analyzed in clinical trials and their outcomes.作者: 不易燃 時間: 2025-3-23 22:18 作者: COM 時間: 2025-3-24 04:08
Cardiac and Vascular Biologyhttp://image.papertrans.cn/c/image/221793.jpg作者: preservative 時間: 2025-3-24 09:46 作者: 評論者 時間: 2025-3-24 14:05 作者: Glycogen 時間: 2025-3-24 17:30 作者: Lineage 時間: 2025-3-24 21:13 作者: 善于 時間: 2025-3-25 00:08
Nonverbale Kommunikation beim Verkaufuires stimulation of an inflammatory reaction and subsequent activation of matrix-secreting myofibroblasts. Perturbed myofibroblast function results in defective healing and may cause rupture and adverse dilative remodeling, whereas prolonged, excessive, or unrestrained fibrogenic actions following 作者: 遣返回國 時間: 2025-3-25 05:04 作者: 圍裙 時間: 2025-3-25 08:43 作者: HARP 時間: 2025-3-25 12:56 作者: CRUMB 時間: 2025-3-25 18:56 作者: Rejuvenate 時間: 2025-3-25 21:49
https://doi.org/10.1007/978-3-642-61654-9ardiac remodeling, resulting from repetitive exposure to intense exercise, describes the process by which adaptations in myocardial structure and function occur. The athletic heart is typically characterized by either eccentric or concentric hypertrophy, depending on the type of exercise undertaken.作者: fiscal 時間: 2025-3-26 01:05 作者: Resistance 時間: 2025-3-26 07:31 作者: 鋼筆尖 時間: 2025-3-26 10:12
Nonverbale Kommunikation in der Politik, due to an electrical substrate pathology causing delayed left ventricular activation. However, 40–50% of patients do not respond to treatment. In this book chapter, we review cardiac computer models of the electrophysiology, electromechanics, and hemodynamics of the heart that have been used to inv作者: 下邊深陷 時間: 2025-3-26 14:05 作者: 小歌劇 時間: 2025-3-26 17:59 作者: 古老 時間: 2025-3-27 00:11 作者: sterilization 時間: 2025-3-27 02:23 作者: 厭倦嗎你 時間: 2025-3-27 07:52 作者: Fester 時間: 2025-3-27 11:29
Mechanobiology of Cardiac Growth in Health and Disease,rs in developmental biology, tissue homeostasis, and pathology programming. In fact, the evidence that cells are able to translate mechanical cues in the form of strain/stretch- or positional-/geometric-dependent information, adds a new complexity to the coordination of the multicellular regulation.作者: 敲詐 時間: 2025-3-27 16:47
The Role of Mechanosensitive Signaling Cascades in Repair and Fibrotic Remodeling of the Infarcted uires stimulation of an inflammatory reaction and subsequent activation of matrix-secreting myofibroblasts. Perturbed myofibroblast function results in defective healing and may cause rupture and adverse dilative remodeling, whereas prolonged, excessive, or unrestrained fibrogenic actions following 作者: ARBOR 時間: 2025-3-27 18:24
Mechanobiology of Cardiac Fibroblasts in Cardiac Remodeling,lular matrix (cECM) are the primary responsibility of cardiac fibroblasts (CFs). Long considered to be passive contributors to cardiac mechanics and function, CFs have undergone a major reevaluation in the past two decades and are now considered to be critical drivers of cardiac homeostasis. Primary作者: granite 時間: 2025-3-27 22:57 作者: 階層 時間: 2025-3-28 04:41
Biophysical Stretch Induced Differentiation and Maturation of Induced Pluripotent Stem Cell-Derivedrdiovascular disease, accelerating drug discovery, exploring the earliest stages of human development, and boosting potential regenerative therapeutics. While it is now feasible to produce human patient- and disease-specific iPSC-CMs at a scalable level with high purity, their fetal-like phenotypes 作者: 熱心 時間: 2025-3-28 08:03
Mechanical Considerations of Myocardial Tissue and Cardiac Regeneration,use in clinical practice, none has demonstrated great success in regenerating the cardiac tissue. Therefore, there are still significant challenges in repairing or regenerating cardiac tissue, which serves a predominantly biomechanical function. Furthermore, it is now evident that the mechanobiologi作者: Alpha-Cells 時間: 2025-3-28 11:09
Mechanobiology of Exercise-Induced Cardiac Remodeling in Health and Disease,ardiac remodeling, resulting from repetitive exposure to intense exercise, describes the process by which adaptations in myocardial structure and function occur. The athletic heart is typically characterized by either eccentric or concentric hypertrophy, depending on the type of exercise undertaken.作者: 兩棲動物 時間: 2025-3-28 17:18 作者: 有權(quán)威 時間: 2025-3-28 19:02
Cellular and Subcellular Mechanisms of Ventricular Mechano-Arrhythmogenesis,his includes the acute feedback of cardiac mechanics to electrics (‘mechano-electric coupling’, MEC), which is achieved primarily through cellular and subcellular elements, including mechano-sensitive ion channels, biophysical signal transmitters and mechano-sensitive biochemical signalling pathways作者: 摻和 時間: 2025-3-28 23:47
Computational Biomechanics of Ventricular Dyssynchrony and Resynchronization Therapy, due to an electrical substrate pathology causing delayed left ventricular activation. However, 40–50% of patients do not respond to treatment. In this book chapter, we review cardiac computer models of the electrophysiology, electromechanics, and hemodynamics of the heart that have been used to inv作者: 使服水土 時間: 2025-3-29 06:15
Therapeutic Innovations for Heart Failure,y high rates of hospitalization. Although there are specific, well-defined guidelines on pharmacological treatments, morbidity and mortality remain high. This chapter first summarizes fundamental basic mechanisms of cardiac hypertrophy due to increased afterload as well as heart failure as potential作者: Vulnerary 時間: 2025-3-29 07:17 作者: 神秘 時間: 2025-3-29 14:40
Mechanotransduction in Heart Development,art development. Here we discuss relatively well-studied examples of heart developmental processes that rely on transduction of the dynamic mechanical signals produced by heart function, heart looping and chamber expansion, the formation of the trabeculae, the formation of the valves, and the format作者: 情感 時間: 2025-3-29 18:32 作者: 憲法沒有 時間: 2025-3-29 22:12
Mechanobiology of Cardiac Fibroblasts in Cardiac Remodeling,nsiderations of their phenotype and mechanical behavior in vitro, which can diverge wildly from their in vivo behavior. With all this in mind, we also discuss some outstanding questions and challenges in CF research.作者: 容易懂得 時間: 2025-3-30 03:56
Mechanical Considerations of Myocardial Tissue and Cardiac Regeneration,rdiac injury. Therefore, the future cardiac regeneration strategies need to address the challenges of and questions on the role of biomechanics and mechanobiology in cardiac regeneration. This includes measurement and characterisation of multiscale mechanical properties of healthy and diseased cardi作者: 投票 時間: 2025-3-30 07:25
Cardiac Microvascular Endothelial Cells and Pressure Overload-Induced Cardiac Fibrosis,ast differentiation and thus excessive cardiac fibrosis and finally heart failure. Several factors have been shown to play a role in this endothelial cell dedifferentiation, such as transforming growth factor β1 and angiotensin II, and more are currently being assessed. This will pave the way for th作者: 連鎖 時間: 2025-3-30 11:58 作者: 騷擾 時間: 2025-3-30 14:43
Computational Biomechanics of Ventricular Dyssynchrony and Resynchronization Therapy,computational resources. These factors limit computational studies to one single heart or small patient numbers. Once these technical challenges are overcome, personalized models of the heart have the potential to help in HF diagnosis and treatment and to be incorporated into the clinical workflow.作者: Femine 時間: 2025-3-30 19:54
https://doi.org/10.1007/978-3-8349-6192-1her, these bona fide “cardiac stretch sensor” areas and complexes share common principles like involvement of specialized protein modules, containing domains serving as molecular springs and specific binding sites for protein-protein interactions or phosphorylation/dephosphorylation upon tension rel
