標題: Titlebook: Calcium Signalling and Disease; Molecular pathology Ernesto Carafoli,Marisa Brini Book 2007 Springer Science+Business Media B.V. 2007 ATP. [打印本頁] 作者: 手或腳 時間: 2025-3-21 18:57
書目名稱Calcium Signalling and Disease影響因子(影響力)
書目名稱Calcium Signalling and Disease影響因子(影響力)學(xué)科排名
書目名稱Calcium Signalling and Disease網(wǎng)絡(luò)公開度
書目名稱Calcium Signalling and Disease網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Calcium Signalling and Disease被引頻次
書目名稱Calcium Signalling and Disease被引頻次學(xué)科排名
書目名稱Calcium Signalling and Disease年度引用
書目名稱Calcium Signalling and Disease年度引用學(xué)科排名
書目名稱Calcium Signalling and Disease讀者反饋
書目名稱Calcium Signalling and Disease讀者反饋學(xué)科排名
作者: Nonflammable 時間: 2025-3-21 21:47 作者: 共同時代 時間: 2025-3-22 01:39
https://doi.org/10.1007/978-1-4020-6191-2ATP; Calcium; bone; pathophysiology; physiology; proteins作者: emission 時間: 2025-3-22 05:09 作者: Freeze 時間: 2025-3-22 12:17
Ernesto Carafoli,Marisa BriniFirst book of this type.New, powerfully emerging topic.Interdisciplinary book, from biochemistry to clinics作者: integral 時間: 2025-3-22 14:09
Subcellular Biochemistryhttp://image.papertrans.cn/c/image/220790.jpg作者: integral 時間: 2025-3-22 20:33
Calcium Signalling and Disease978-1-4020-6191-2Series ISSN 0306-0225 Series E-ISSN 2542-8810 作者: 積云 時間: 2025-3-23 00:06
Neues Denken in der Unternehmensführungdiseases. This chapter discusses these diseases in terms of known mechanisms, controversies, and unanswered questions. We also compare the cardiac and skeletal muscle diseases to explore common mechanisms作者: Mortal 時間: 2025-3-23 02:38
Prozessketten in Organisation und Technikoss of one functional copy of the human SPCA1 gene (.) causes Hailey-Hailey disease, a rare skin disorder characterized by recurrent blisters and erosions in the flexural areas. Here, we will review the properties and functional role of the SPCAs. The relationship between Hailey-Hailey disease and its defective gene (.) will be adressed as well作者: callous 時間: 2025-3-23 09:35
Marc Pastowsky,Florence Hausen-Mabilone to elevated intracellular calcium. The family members (there are at least 12 in humans) have become specialized over evolutionary time and are involved in a diverse range of cellular functions both inside the cell and extracellularly Although a mutation in an annexin has never been categorically p作者: Mutter 時間: 2025-3-23 13:03
https://doi.org/10.1007/978-3-642-58996-6e conditions linked to dysfunctions of cellular Ca . homeostasis. The evidence supporting their involvement has traditionally been indirect and circumstantial, but recent work has added more solid evidence supporting the role of ubiquitous dimeric calpains in the process of neurodegeneration. The on作者: etidronate 時間: 2025-3-23 17:19 作者: 萬神殿 時間: 2025-3-23 20:15 作者: 轉(zhuǎn)換 時間: 2025-3-24 00:34 作者: 講個故事逗他 時間: 2025-3-24 05:29
Karl-Heinz Erdmann,Johanna Frommbergerel of Ca._o and to respond with changes in function that are directed at normalizing the blood calcium concentration. In addition to the parathyroid gland, the kidney is a key site for Ca._o-sensing that enables it to make physiologically relevant alterations in divalent cation and water metabolism.作者: Reverie 時間: 2025-3-24 09:39 作者: ZEST 時間: 2025-3-24 13:17
https://doi.org/10.1007/978-3-658-27206-7r contributions of voltage-gated calcium channels in multiple physiological processes. Moreover, we are beginning to comprehend their pathophysiological roles through both naturally occurring channelopathies in humans and mice and through targeted gene deletions. The data illustrate that small pertu作者: 無底 時間: 2025-3-24 15:33 作者: Statins 時間: 2025-3-24 20:40
Neues Denken in der Unternehmensführungdiseases. This chapter discusses these diseases in terms of known mechanisms, controversies, and unanswered questions. We also compare the cardiac and skeletal muscle diseases to explore common mechanisms作者: 陰郁 時間: 2025-3-25 02:23 作者: 挖掘 時間: 2025-3-25 06:31 作者: 淡紫色花 時間: 2025-3-25 08:02 作者: reject 時間: 2025-3-25 11:41
Prozessketten in Organisation und Technikoss of one functional copy of the human SPCA1 gene (.) causes Hailey-Hailey disease, a rare skin disorder characterized by recurrent blisters and erosions in the flexural areas. Here, we will review the properties and functional role of the SPCAs. The relationship between Hailey-Hailey disease and i作者: certitude 時間: 2025-3-25 19:10
Grundlagen der Organisationsgestaltungor signalling agents with spatial localization, magnitude and temporal characteristics of calcium signals ultimately determining cell’s fate. There are four primary compartments: extracellular space, cytoplasm, endoplasmic reticulum and mitochondria that participate in the cellular Ca. circulation. 作者: BRACE 時間: 2025-3-25 20:37
Management des Produktlebenslaufsis an important issue which has been addressed in several recent reviews (Alderton and Steinhardt, 2000a, Gailly, 2002, Allen et al., 2005). A key question when trying to understand the cellular basis of DMD is how the absence or low level of expression of dystrophin, a cytoskeletal protein, results作者: 共同生活 時間: 2025-3-26 01:07 作者: Evocative 時間: 2025-3-26 06:54 作者: myopia 時間: 2025-3-26 08:33 作者: 侵略者 時間: 2025-3-26 15:52
https://doi.org/10.1007/978-3-658-31565-8minant cone dystrophy, respectively. This report provides a recent account of the advances, challenges, and possible future prospects of studying this important step in visual transduction that transcends to other neuronal Ca . homeostasis processes作者: 門窗的側(cè)柱 時間: 2025-3-26 16:57 作者: meretricious 時間: 2025-3-27 00:32
Guanylate Cyclase-Activating Proteins and Retina Disease,minant cone dystrophy, respectively. This report provides a recent account of the advances, challenges, and possible future prospects of studying this important step in visual transduction that transcends to other neuronal Ca . homeostasis processes作者: Misgiving 時間: 2025-3-27 02:05 作者: Stagger 時間: 2025-3-27 07:56
Book 2007reditary amyloid polyneuropathy, malignant hyperthermia, cerebellar ataxia, atherothrombotic disease)...Clearly, the topic has now become not only very large, but also very stimulating. Its extensive critical coverage is likely to eventually stimulate fruitful interdisciplinary discussions..作者: 小溪 時間: 2025-3-27 11:52
https://doi.org/10.1007/978-3-658-27206-7and behavioral functions. At least in those instances wherein the channelopathies can be attributed to gain-of-function mechanisms, the data point towards new therapeutic strategies for developing highly selective calcium channel antagonists作者: GRAIN 時間: 2025-3-27 14:23 作者: Fecal-Impaction 時間: 2025-3-27 21:09
0306-0225 area of cellular calcium signaling: yet, it now spans the entire area, with discoveries that cover both genetic and acquired pathologies, even offering glimpses in the direction of therapy. ..Cellular calcium homeostasis, and thus calcium signalling, is mainly regulated by membrane intrinsic protei作者: elucidate 時間: 2025-3-27 22:53 作者: 噴出 時間: 2025-3-28 04:07
Transformation in eine agile Zukunft,activity of several families of Ca. channels and transporters differentially distributed between intracellular compartments. Cell damage induced by environmental insults or by overstimulation of physiological pathways results in pathological Ca. signals, which trigger necrotic or apoptotic cellular death作者: 色情 時間: 2025-3-28 08:19
Gelsolin and Diseases,n, cancer and amyloidosis. The tight regulation of gelsolin by calcium is crucial for its physiological role and constitutive activation leads to apoptosis. In the following we will give an overview on how gelsolin is regulated by calcium, and which clinical conditions have been linked to lack or misregulation of gelsolin作者: BALE 時間: 2025-3-28 11:38 作者: 秘密會議 時間: 2025-3-28 16:06
Book 2007acquired pathologies, even offering glimpses in the direction of therapy. ..Cellular calcium homeostasis, and thus calcium signalling, is mainly regulated by membrane intrinsic proteins and calcium sensor proteins. Both classes may be involved in pathological processes that affect both human and ani作者: STELL 時間: 2025-3-28 20:44
Marc Pastowsky,Florence Hausen-Mabilone antiphospholipid syndrome. In this chapter we will outline some of the more recent observations in regard to these conditions, and describe the involvement of annexins in some other major causes of human morbidity作者: 神化怪物 時間: 2025-3-28 23:59
https://doi.org/10.1007/978-3-642-58996-6pendent protease, calpain 10, appears to be involved in the etiology of type 2 diabetes. The involvement has been inferred essentially from genetic evidence. Also in the case of type 2 diabetes the molecular mechanisms that could link the disease to calpain 10 are unknown作者: CAGE 時間: 2025-3-29 04:48
Anwendungsbeispiel Applikation,ggested to be the active extracellular species required for receptor binding and activation through receptor multimerization .Most S100 genes are located in a gene cluster on human chromosome 1q21, a region frequently rearranged in human cancer .The functional diversification of S100 proteins is ach作者: remission 時間: 2025-3-29 08:31
Karl-Heinz Erdmann,Johanna Frommbergerarathyroidism (NSHPT). Activating mutations cause a hypocalcemic syndrome of varying severity, termed autosomal dominant hypocalcemia or hypoparathyroidism. Inactivating or activating antibodies directed at the CaR produce the expected hyper- or hypocalcemic syndromes, respectively. “Calcimimetic” C作者: 可能性 時間: 2025-3-29 12:41 作者: Anhydrous 時間: 2025-3-29 16:52
https://doi.org/10.1007/978-3-662-12572-4ogenesis of HDIn support of this idea, we demonstrated a connection between abnormal Ca. signaling and apoptosis of MSN cultured from YAC128 HD mouse model. These results indicate that InsP.R and other Ca. signaling proteins should be considered as potential therapeutic targets for treatment of HD作者: inflame 時間: 2025-3-29 21:38
Neue Organisationsformen im Unternehmenn heart failure in human and in mice models. Gene-targeting studies in mouse confirmed the expected function of these isoforms in some cases, but also resulted in unexpected phenotypes: SERCA1 null mutants die from respiratory failure, SERCA2 heterozygous mutant mice develop skin cancer with age and作者: Visual-Field 時間: 2025-3-30 02:18 作者: Buttress 時間: 2025-3-30 05:24 作者: sleep-spindles 時間: 2025-3-30 10:51 作者: intrude 時間: 2025-3-30 12:33 作者: 音樂學(xué)者 時間: 2025-3-30 18:01
Neue Perspektiven auf Basisarbeiting pores. . evidence has shown that Aβ impairs both LTP and cognition, whereas all of these ion channels cluster at the synapse and underlie synaptic transmission and hence cognition. Here we consider the evidence that Aβ causes cognitive deficits through altering calcium homeostasis at the synapse作者: DAMP 時間: 2025-3-30 22:33 作者: 權(quán)宜之計 時間: 2025-3-31 01:23
Calpains and Human Disease,pendent protease, calpain 10, appears to be involved in the etiology of type 2 diabetes. The involvement has been inferred essentially from genetic evidence. Also in the case of type 2 diabetes the molecular mechanisms that could link the disease to calpain 10 are unknown作者: Obscure 時間: 2025-3-31 08:45
Pathologies Involving the S100 Proteins and Rage,ggested to be the active extracellular species required for receptor binding and activation through receptor multimerization .Most S100 genes are located in a gene cluster on human chromosome 1q21, a region frequently rearranged in human cancer .The functional diversification of S100 proteins is ach作者: MEEK 時間: 2025-3-31 11:58 作者: 暴露他抗議 時間: 2025-3-31 13:58 作者: 有害處 時間: 2025-3-31 19:56 作者: olfction 時間: 2025-4-1 01:09
Serca pumps and human diseases,n heart failure in human and in mice models. Gene-targeting studies in mouse confirmed the expected function of these isoforms in some cases, but also resulted in unexpected phenotypes: SERCA1 null mutants die from respiratory failure, SERCA2 heterozygous mutant mice develop skin cancer with age and作者: oblique 時間: 2025-4-1 02:18
The plasma membrane calcium ATPase and disease,ns of the . and . genes in mouse are embryonic lethal and cause a sperm motility defect, respectively. These phenotypes point to complex human diseases like hearing loss, cardiac function and infertility. Changes in PMCA expression are associated with other diseases including cataract formation, car作者: Latency 時間: 2025-4-1 09:30 作者: Gene408 時間: 2025-4-1 12:45 作者: 珊瑚 時間: 2025-4-1 17:04
Calcium and Cell Death: The Mitochondrial Connection,ant for metabolic regulation, can become a death factor by inducing opening of the permeability transition pore (PTP), a high conductance inner membrane channel. Persistent PTP opening is followed by depolarization with Ca. release, cessation of oxidative phosphorylation, matrix swelling with inner 作者: 付出 時間: 2025-4-1 22:19 作者: 舊式步槍 時間: 2025-4-2 02:03
0306-0225 malignant hyperthermia, cerebellar ataxia, atherothrombotic disease)...Clearly, the topic has now become not only very large, but also very stimulating. Its extensive critical coverage is likely to eventually stimulate fruitful interdisciplinary discussions..978-90-481-7560-4978-1-4020-6191-2Series ISSN 0306-0225 Series E-ISSN 2542-8810 作者: 誰在削木頭 時間: 2025-4-2 06:53
Annexinopathies,e to elevated intracellular calcium. The family members (there are at least 12 in humans) have become specialized over evolutionary time and are involved in a diverse range of cellular functions both inside the cell and extracellularly Although a mutation in an annexin has never been categorically p作者: 水獺 時間: 2025-4-2 08:36
