標(biāo)題: Titlebook: C-Myc in B-Cell Neoplasia; 14th Workshop on Mec Michael Potter (Chief),Fritz Melchers (Director) Book 1997 The Editor(s) (if applicable) an [打印本頁] 作者: Filament 時(shí)間: 2025-3-21 17:13
書目名稱C-Myc in B-Cell Neoplasia影響因子(影響力)
書目名稱C-Myc in B-Cell Neoplasia影響因子(影響力)學(xué)科排名
書目名稱C-Myc in B-Cell Neoplasia網(wǎng)絡(luò)公開度
書目名稱C-Myc in B-Cell Neoplasia網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱C-Myc in B-Cell Neoplasia被引頻次
書目名稱C-Myc in B-Cell Neoplasia被引頻次學(xué)科排名
書目名稱C-Myc in B-Cell Neoplasia年度引用
書目名稱C-Myc in B-Cell Neoplasia年度引用學(xué)科排名
書目名稱C-Myc in B-Cell Neoplasia讀者反饋
書目名稱C-Myc in B-Cell Neoplasia讀者反饋學(xué)科排名
作者: VEN 時(shí)間: 2025-3-22 00:07
B-lymphocyte-lineage Cells from Early Precursors to Ig-secreting Plasma Cells: Targets of Regulation miz-1 (see papers in this volume, in particular Austen et al., Schneider et al., Schreiber-Agus et al.), is thought to control expression of genes involved in the proliferation and differentiation of cells. Very few lineages of cellular development have been studied . and . in comparable detail as 作者: follicle 時(shí)間: 2025-3-22 03:42
DNA Conformation, Topology, and the Regulation of , Expressionultitude of physiological processes and has been reported to be regulated by a long list of hormones, cytokines, lymphokines, nutritional status, development and differentiation. Myc levels can be pharmacologically perturbed, at least transiently, by a host of agents. Consistent with the notion that作者: Monocle 時(shí)間: 2025-3-22 07:56 作者: 修改 時(shí)間: 2025-3-22 11:35 作者: 狗舍 時(shí)間: 2025-3-22 14:27
Structure and Expression of the c-Myc/Pvt 1 Megagene Locusing Burkitt’s Lymphoma (BL), AIDs-NHL, mouse plasmacytoma (Pct) and possibly multiple myeloma (MM). There is a restriction associated with this Tx such that only the immunoglobulin (Ig) heavy chain gene is found juxtaposed to c-Myc and only the Ig light chain gene is found juxtaposed to Pvt 1. Over 作者: 狗舍 時(shí)間: 2025-3-22 18:49 作者: micturition 時(shí)間: 2025-3-22 21:16 作者: Nebulizer 時(shí)間: 2025-3-23 01:49
c-Myc Promotes Survival of WEHI 231 B Lymphoma Cells from Apoptosisssion or inappropriate time of expression of the c-. gene has been found to promote apoptosis. Cleveland and coworkers observed that addition of a vector expressing c-Myc protein accelerated apoptosis following IL-3 deprivation of the 32D Independent myeloid cell line [3]. Similarly Evan and coworke作者: ethereal 時(shí)間: 2025-3-23 08:15
Role of c-myc and p27 in Anti-IgM Induced B-Lymphoma Apoptosiso undetectable levels by 8–24 hours. These cells die soon thereafter via apoptosis. IgD receptor crosslinking also leads to an increase in c-myc expression, but it remains above baseline levels for more than 24 hours; these cells continue to proliferate and do not die. We previously reported that an作者: euphoria 時(shí)間: 2025-3-23 12:44
Mnt: A Novel Max-interacting Protein and Myc Antagonistx in vivo and functions as a transcriptional repressor of reporter genes containing promoter-proximal CACGTG sites. Mnt:Max complexes also efficiently suppress Myc-dependent activation from the same promoter. Transcription repression by Mnt maps to a 13 amino acid N-terminal region related to the Si作者: Harrowing 時(shí)間: 2025-3-23 17:26 作者: 秘方藥 時(shí)間: 2025-3-23 21:47
C-Myc Transrepression and Cell Transformationhe ability of c-Myc to transactivate through the E-box as the mechanism underlying all of these Myc functions. It is so widely accepted that c-Myc transactivation is necessary for cell transformation, that the authentication of putative target genes requires the presence of E-boxes within these gene作者: 有節(jié)制 時(shí)間: 2025-3-24 01:31 作者: 1FAWN 時(shí)間: 2025-3-24 04:34 作者: Glutinous 時(shí)間: 2025-3-24 08:48
Immobilized Biocatalyst Technology,part of the century with the observations of Wilhelm Eilermann and Olaf Bang in 1908–9 who demonstrated that erythroleukemias in chickens could be transmitted with cell free filtrates. At the time there was prevailing doubt about the neoplastic nature of this leukemic process; even Ellermann and Ban作者: keloid 時(shí)間: 2025-3-24 14:23 作者: 團(tuán)結(jié) 時(shí)間: 2025-3-24 17:51 作者: 黑豹 時(shí)間: 2025-3-24 19:45
Biofuel and Biorefinery Technologiesand death [1,2]. MYC normally initiates a cell cycle progression program culminating in S phase whilst enforced MYC expression can inhibit cellular differentiation programs and excessive levels of MYC lead to the induction of apoptosis (programmed cell death) [1–3]. The MYC polypeptide functions as 作者: 琺瑯 時(shí)間: 2025-3-25 00:13 作者: Morose 時(shí)間: 2025-3-25 04:11 作者: CARE 時(shí)間: 2025-3-25 09:12 作者: amnesia 時(shí)間: 2025-3-25 15:29 作者: anaerobic 時(shí)間: 2025-3-25 18:32
Microbial Extracellular Polymeric Substancesssion or inappropriate time of expression of the c-. gene has been found to promote apoptosis. Cleveland and coworkers observed that addition of a vector expressing c-Myc protein accelerated apoptosis following IL-3 deprivation of the 32D Independent myeloid cell line [3]. Similarly Evan and coworke作者: sulcus 時(shí)間: 2025-3-25 23:38
Microbial Extracellular Polymeric Substanceso undetectable levels by 8–24 hours. These cells die soon thereafter via apoptosis. IgD receptor crosslinking also leads to an increase in c-myc expression, but it remains above baseline levels for more than 24 hours; these cells continue to proliferate and do not die. We previously reported that an作者: pester 時(shí)間: 2025-3-26 03:47 作者: Vulnerary 時(shí)間: 2025-3-26 08:11
https://doi.org/10.1007/978-1-4899-3121-4e to interact directly or indirectly with c-Myc and/or Max have been described (for review see Marcu et al. 1992; Henriksson and Lüscher 1996). It has become evident that at the center of this network lies Max which on one hand forms heterodimers with the Myc family of proteins, including c-, N-, an作者: 討厭 時(shí)間: 2025-3-26 10:40
Microbiological control of food production,he ability of c-Myc to transactivate through the E-box as the mechanism underlying all of these Myc functions. It is so widely accepted that c-Myc transactivation is necessary for cell transformation, that the authentication of putative target genes requires the presence of E-boxes within these gene作者: obstruct 時(shí)間: 2025-3-26 13:57
https://doi.org/10.1007/978-1-4614-1177-2f expression of a number of genes and this repressive effect of Myc on gene expression may not be mediated by the Myc/Max complex. We recently isolated by two-hybrid cloning a novel zinc-finger protein that associates with the carboxy-terminus of Myc. We have termed this protein Miz-1 (.yc-mteractin作者: 裂隙 時(shí)間: 2025-3-26 17:16
P. Michael Davidson,Faith M. Critzerdimerize with Max through their respective helix loop helix (HLH) and leucine zipper (LZ) domains to cooperatively bind DNA in a sequence specific manner (reviewed in [15]). In contrast to the transactivating property of Myc/Max heterodimers [1, 18, 21], Max heterodimerization with Mad or Mxi1, foll作者: 鎮(zhèn)痛劑 時(shí)間: 2025-3-27 00:50
C-Myc in B-Cell Neoplasia978-3-642-60801-8Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: Lucubrate 時(shí)間: 2025-3-27 04:56
https://doi.org/10.1007/978-3-642-60801-8DNA; apoptosis; cell; cytokine; growth; immunoglobulin; interferon; lymphoma; pathogenesis; protein; proteins; 作者: COLON 時(shí)間: 2025-3-27 06:28 作者: Servile 時(shí)間: 2025-3-27 11:23
Current Topics in Microbiology and Immunologyhttp://image.papertrans.cn/c/image/220083.jpg作者: Spartan 時(shí)間: 2025-3-27 13:48 作者: Ventilator 時(shí)間: 2025-3-27 20:07
B-lymphocyte-lineage Cells from Early Precursors to Ig-secreting Plasma Cells: Targets of Regulationccurs as a consequence of chromosomal translocations to the IgH- and L-chain J segments which are frequently found in human Burkitt’s lymphomas, or to the IgH switch regions which are detected in mineral oil-induced mouse plasmacytomas [Potter and Wiener 1992, Shen-Ong et al 1982]. This signifies th作者: nonplus 時(shí)間: 2025-3-28 00:56
Differential Regulation of the c-MYC P1 and P2 Promoters in the Absence of Functional Tumor Suppressw levels in normal proliferating cells [2,5]. Minor fluctuations in MYC protein and/or mRNA levels can have dramatic outcomes on cellular physiology. Consequently, mammalian MYC gene expression is subject to tight control at multiple levels.作者: 激怒 時(shí)間: 2025-3-28 03:53 作者: 我沒有強(qiáng)迫 時(shí)間: 2025-3-28 09:18
Murine and Human 3′ IgH Regulatory Sequencestory elements but also the acquisition of different regulatory influences associated with its new, immunoglobulin-associated, chromosomal environment. The focus of this paper is on the nature of IgH associated sequences that impact upon the c-myc oncogene in B cell malignancies.作者: CORD 時(shí)間: 2025-3-28 13:52
Role of c-myc and p27 in Anti-IgM Induced B-Lymphoma Apoptosiscause an increase in p27. Since dexamethasone causes a loss of myc and synergizes with the anti-IgM signal, we suggest that accelerated cell death with this steroid in the presence of anti-IgM is due to a more rapid degradation of this oncogene product. Finally, we propose that c-myc drives the tran作者: 小步走路 時(shí)間: 2025-3-28 15:20
Mnt: A Novel Max-interacting Protein and Myc Antagonistls. Likewise, Mnt protein is expressed in many proliferating cell types in culture where both Myc:Max and Mnt:Max complexes are detected. An exception is P19 embryonal carcinoma cells, where Mnt is expressed and in a complex with Max, but Myc proteins are not detected. Mnt is likely to be a key regu作者: 成份 時(shí)間: 2025-3-28 22:23
0070-217X nk the National Cancer Institute for sponsoring this workshop and the staff of Cygnus, Inc. , for their outstanding organizational assistance. The organizers ar978-3-642-64560-0978-3-642-60801-8Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: Indolent 時(shí)間: 2025-3-29 02:35
Book 1997h of this field. The organizers of the meeting have each contributed review chapters that summarize different aspects of the meeting. We thank the National Cancer Institute for sponsoring this workshop and the staff of Cygnus, Inc. , for their outstanding organizational assistance. The organizers ar作者: intangibility 時(shí)間: 2025-3-29 05:34 作者: 可轉(zhuǎn)變 時(shí)間: 2025-3-29 11:11 作者: thrombus 時(shí)間: 2025-3-29 11:44
Biofuel and Biorefinery Technologiesw levels in normal proliferating cells [2,5]. Minor fluctuations in MYC protein and/or mRNA levels can have dramatic outcomes on cellular physiology. Consequently, mammalian MYC gene expression is subject to tight control at multiple levels.作者: Myocarditis 時(shí)間: 2025-3-29 18:58
https://doi.org/10.1007/978-3-319-43679-1ice harboring a translocated Pvt 1/Ck segment in order to determine whether 1). these mice produce the Pvt 1/Ck fusion product 2). these mice are immunocompromised and 3). these mice develop tumors of a B cell origin.作者: octogenarian 時(shí)間: 2025-3-29 22:54
https://doi.org/10.1007/978-3-319-43679-1tory elements but also the acquisition of different regulatory influences associated with its new, immunoglobulin-associated, chromosomal environment. The focus of this paper is on the nature of IgH associated sequences that impact upon the c-myc oncogene in B cell malignancies.作者: 大方一點(diǎn) 時(shí)間: 2025-3-30 01:26
Microbial Extracellular Polymeric Substancescause an increase in p27. Since dexamethasone causes a loss of myc and synergizes with the anti-IgM signal, we suggest that accelerated cell death with this steroid in the presence of anti-IgM is due to a more rapid degradation of this oncogene product. Finally, we propose that c-myc drives the tran作者: 聲明 時(shí)間: 2025-3-30 06:14 作者: peptic-ulcer 時(shí)間: 2025-3-30 09:19 作者: Decline 時(shí)間: 2025-3-30 16:19
https://doi.org/10.1007/978-1-4614-1177-2d by two-hybrid cloning a novel zinc-finger protein that associates with the carboxy-terminus of Myc. We have termed this protein Miz-1 (.yc-mteracting .inc finger protein). Some of the properties of Miz-1 suggest that it may be involved in gene repression by Myc ..作者: GRAIN 時(shí)間: 2025-3-30 20:21 作者: 完成才會征服 時(shí)間: 2025-3-30 22:40 作者: SIT 時(shí)間: 2025-3-31 01:10 作者: Painstaking 時(shí)間: 2025-3-31 07:12
