派博傳思國(guó)際中心

標(biāo)題: Titlebook: Brain-Iron Cross Talk; Wael Mohamed,Nicola Luigi Brogazzi,Richard M. Kost Book 2023 The Editor(s) (if applicable) and The Author(s), under [打印本頁]

作者: Awkward    時(shí)間: 2025-3-21 17:18
書目名稱Brain-Iron Cross Talk影響因子(影響力)




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書目名稱Brain-Iron Cross Talk被引頻次




書目名稱Brain-Iron Cross Talk被引頻次學(xué)科排名




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作者: tangle    時(shí)間: 2025-3-21 23:23
https://doi.org/10.1007/978-1-4614-1153-6y, small quantities of wine and red meat. The effect of the MD in the prevention of some chronic illness has stimulated the appetite of many researchers to study this diet in the last two decades. Previous studies have shown a significant relation between the adherence to MD and cognitive functions
作者: Cholagogue    時(shí)間: 2025-3-22 02:28
https://doi.org/10.1007/978-1-4614-4460-2the etiology of nigrostriatal damage. The exact mechanism behind iron metabolism problems in Parkinson’s disease are still unknown but it can be accredited to oxidative and nitrosative burden, inflammation, excitotoxicity, mitochondrial dysfunction, hampered proteolysis and lastly apoptotic cell dea
作者: Arteriography    時(shí)間: 2025-3-22 06:29
https://doi.org/10.1007/978-3-030-32205-2mine the facts surrounding a poorly known link between iron and calcium balance and connection, in which one’s deregulation has an affect on another, resulting in a negative feedback system that eventually leads to neurodegenerative disorders. We provides a brief account on background, homeostasis o
作者: 肉身    時(shí)間: 2025-3-22 11:14
https://doi.org/10.1007/978-3-030-32205-2AD development. It is concluded that iron could initiate and contribute to the pathology of AD, as it is significantly associated with key AD protein filaments of Aβ and τ hyperphosphorylation that leads to the accumulation of senile plaques and NFTs. Besides its ability to create free radicals, fer
作者: palpitate    時(shí)間: 2025-3-22 15:41
Oscar Bajo-Rubio,Carmen Díaz-Roldándicated by the use of antioxidants in reducing the development of iron-induced epileptiform discharges, oxidative stress-induced damage may be a significant factor in the emergence of persistent post-traumatic epilepsy caused by subcortical administration of ferric salts. The development of a variet
作者: Indebted    時(shí)間: 2025-3-22 19:59
https://doi.org/10.1057/978-1-137-57472-5AH i.e., acute seizures and hydrocephalus. DFX an iron chelator has shown promising results in the long term for the treatment of SAH. It has improved neurological function outcome in the patients. Several other therapeutic agents have also emerged in the experimental studies. Unfortunately, no drug
作者: Ordeal    時(shí)間: 2025-3-23 01:05

作者: 大洪水    時(shí)間: 2025-3-23 03:11
https://doi.org/10.1007/978-3-642-00146-8ency is the most common dietary problem worldwide, with numerous cognitive and neurological consequences. Not only iron deficiency, but also iron overload poses significant high risks to the development and function of the nervous system. Studies of completely developed infants that have been expose
作者: brachial-plexus    時(shí)間: 2025-3-23 08:46
https://doi.org/10.1007/978-3-642-00146-8in basal ganglia and iron patches can also be seen on the skin which is a cause of neuropathy. Iron deficiency could also induce neuropathy as iron is involved in myelination. Low-iron diet induces inflammatory responses which are involved in the onset of peripheral neuropathy. Low iron in the body
作者: 自然環(huán)境    時(shí)間: 2025-3-23 12:54
2730-6712 nd offers diagnosis and treatment of iron-related CNSdiseases. Finally, it relates dysregulated expression of iron-relatedgenes in brain tumors..??978-981-19-7329-1978-981-19-7327-7Series ISSN 2730-6712 Series E-ISSN 2730-6720
作者: depreciate    時(shí)間: 2025-3-23 17:24

作者: Isthmus    時(shí)間: 2025-3-23 21:01

作者: 連系    時(shí)間: 2025-3-24 00:05
The Critical Roleplay of Iron Neurochemistry in Progression of Parkinson’s Diseasethe etiology of nigrostriatal damage. The exact mechanism behind iron metabolism problems in Parkinson’s disease are still unknown but it can be accredited to oxidative and nitrosative burden, inflammation, excitotoxicity, mitochondrial dysfunction, hampered proteolysis and lastly apoptotic cell dea
作者: Congregate    時(shí)間: 2025-3-24 04:43

作者: CULP    時(shí)間: 2025-3-24 10:21

作者: BORE    時(shí)間: 2025-3-24 11:24

作者: 物種起源    時(shí)間: 2025-3-24 17:51

作者: 陪審團(tuán)每個(gè)人    時(shí)間: 2025-3-24 20:56
Iron Deficiency and Psychiatric Disordersartum depression and premenstrual syndrome. Mechanisms underlying the relationship between ID and psychiatric disorders remain little understood and under documented. Effects of iron supplementation in the improvement of a range of psychiatric disorders have been found to leading in most of the case
作者: 高度    時(shí)間: 2025-3-25 00:44

作者: chlorosis    時(shí)間: 2025-3-25 05:42
Iron and Neuropathiesin basal ganglia and iron patches can also be seen on the skin which is a cause of neuropathy. Iron deficiency could also induce neuropathy as iron is involved in myelination. Low-iron diet induces inflammatory responses which are involved in the onset of peripheral neuropathy. Low iron in the body
作者: MIRTH    時(shí)間: 2025-3-25 07:34

作者: Ataxia    時(shí)間: 2025-3-25 14:50
978-981-19-7329-1The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Singapor
作者: plasma    時(shí)間: 2025-3-25 19:38
Wael Mohamed,Nicola Luigi Brogazzi,Richard M. KostDiscusses the role of iron metabolism in normal brain development.Reviews implications of iron deficiency in neurodevelopmental and neurodegenerative diseases.Present iron chelators as potential thera
作者: SUE    時(shí)間: 2025-3-25 21:05

作者: 波動(dòng)    時(shí)間: 2025-3-26 00:41

作者: 不可救藥    時(shí)間: 2025-3-26 06:18
https://doi.org/10.1057/9780230280830essential sources, forms, and human requirements of iron were highlighted. The detailed information of iron’s bioactivity, metabolism, absorption, excretion, and regulation were also discussed. In addition, the proposed mechanisms behind the absorption, uptake, metabolism and transportation of iron
作者: neologism    時(shí)間: 2025-3-26 11:43

作者: auxiliary    時(shí)間: 2025-3-26 13:17
https://doi.org/10.1007/978-1-4614-1153-6heimer disease. Their prevention has become one the important and urgent action of public health, which conducts to the defiance of achieving optimal control and delaying its development. There is effort to assure a quality life of these vulnerable persons by non-pharmacological strategies. Therefor
作者: Nonthreatening    時(shí)間: 2025-3-26 18:42
https://doi.org/10.1007/978-1-4614-4460-2le part in different important biological processes that can include transportation of oxygen, oxidative phosphorylation, production of myelin, and in the production and utilization of various neurotransmitters. Due to iron’s potential to act as a redox agent, dysregulation of iron metabolism result
作者: 艱苦地移動(dòng)    時(shí)間: 2025-3-26 22:32
https://doi.org/10.1007/978-3-030-32205-2se, proliferation of axon and synapse formation, and govern the gene expression involved in memory formation, while, iron is essential for normal functioning of mitochondria, synapse formation, and the acquisition of cognitive skills. Latest research has discovered that cellular iron enhances calciu
作者: 咽下    時(shí)間: 2025-3-27 05:01
https://doi.org/10.1007/978-3-030-32205-2ts high redox potential can exacerbate bodily oxidative stress and toxicity under extreme concentrations. Thus, brain iron homeostasis is vital to brain health, as tight iron regulations in the CNS prevents abnormal iron localization and catastrophic oxidative damage to cellular structures and compo
作者: 令人發(fā)膩    時(shí)間: 2025-3-27 08:29

作者: PAN    時(shí)間: 2025-3-27 10:40

作者: 不可侵犯    時(shí)間: 2025-3-27 17:06

作者: commune    時(shí)間: 2025-3-27 21:19
https://doi.org/10.1057/978-1-137-57472-5rs such as dopamine, serotonin, and noradrenaline. As a result, iron deficiency (ID) may lead to a series of dysfunctions of brain systems underlying psychiatric psychopathology. Despite its promising clinical implications, the relationship between ID and psychiatric disorders has been the subject o
作者: 消散    時(shí)間: 2025-3-27 23:49
https://doi.org/10.1007/978-3-642-00146-8velopment. The cognitive, social, and emotional components of the brain remain to develop throughout the lifespan. The trajectories of the growth and development of the nervous system are non-homogenous across time. There is expanding evidence suggesting that nutrition especially during early life i
作者: CHAR    時(shí)間: 2025-3-28 02:32

作者: 桉樹    時(shí)間: 2025-3-28 09:52

作者: –LOUS    時(shí)間: 2025-3-28 10:53
Iron and Neuro-Cognition one’s life expectancy. Nowadays, the magic of chemistry is being used to study the character and role of iron in the most challenging organ; the brain, which has been receiving tremendous attention recently..Iron, which is essential for almost every vital process in the body, must be present in a s
作者: Melodrama    時(shí)間: 2025-3-28 14:40
The Interplay between Iron and Oxidative Stress in Brain Neurodegenerative Diseasesessential sources, forms, and human requirements of iron were highlighted. The detailed information of iron’s bioactivity, metabolism, absorption, excretion, and regulation were also discussed. In addition, the proposed mechanisms behind the absorption, uptake, metabolism and transportation of iron
作者: VEIL    時(shí)間: 2025-3-28 20:25
Specific Nutritional Therapeutic Approaches Targeting Iron Overload and Other Hallmarks of Brain Deggenic iron chelators (lactoferrin) and natural antioxidants supplements (vitamins A and E). In addition, certain diet modifications are being delineated as potential management strategies for the prevention/ treatment of various neurogenerative disorders including diets rich in iron chelators, antio
作者: Volatile-Oils    時(shí)間: 2025-3-29 01:34

作者: legacy    時(shí)間: 2025-3-29 04:45

作者: 范例    時(shí)間: 2025-3-29 07:39
Iron-Calcium Crosstalk in Neurodegenerative Diseasesse, proliferation of axon and synapse formation, and govern the gene expression involved in memory formation, while, iron is essential for normal functioning of mitochondria, synapse formation, and the acquisition of cognitive skills. Latest research has discovered that cellular iron enhances calciu
作者: triptans    時(shí)間: 2025-3-29 14:50

作者: urethritis    時(shí)間: 2025-3-29 18:26

作者: Salivary-Gland    時(shí)間: 2025-3-29 23:20

作者: antiandrogen    時(shí)間: 2025-3-30 01:26
Iron and Subarachnoid Hemorrhageche which is also accounted by patients as “the worst headache of their life”. SAH is associated with neurological impairment, cognitive dysfunction and worse prognosis. There are two most important pathological mechanisms involved; early cerebral injury and cerebral vasospasm. Several pro inflammat
作者: remission    時(shí)間: 2025-3-30 05:27

作者: 帶來    時(shí)間: 2025-3-30 11:00
Iron and Neurodevelopmental Disordersvelopment. The cognitive, social, and emotional components of the brain remain to develop throughout the lifespan. The trajectories of the growth and development of the nervous system are non-homogenous across time. There is expanding evidence suggesting that nutrition especially during early life i
作者: Panther    時(shí)間: 2025-3-30 16:12
Iron and Neuropathiesherefore, balanced iron metabolism in body is an unmet need of the body to perform its various functions properly. Iron overload and deficiency both conditions can affect vital pathways of the living system which could lead to different types of neuropathies. Iron overload is associated with demyeli
作者: EXULT    時(shí)間: 2025-3-30 18:26

作者: FORGO    時(shí)間: 2025-3-30 21:04
https://doi.org/10.1057/9780230280830retion, and regulation were also discussed. In addition, the proposed mechanisms behind the absorption, uptake, metabolism and transportation of iron were delineated. This chapter also described the current available knowledge regarding selective cases of brain degenerative diseases and their interplay with iron-induced oxidative stress.
作者: SOB    時(shí)間: 2025-3-31 04:27
The Interplay between Iron and Oxidative Stress in Brain Neurodegenerative Diseasesretion, and regulation were also discussed. In addition, the proposed mechanisms behind the absorption, uptake, metabolism and transportation of iron were delineated. This chapter also described the current available knowledge regarding selective cases of brain degenerative diseases and their interplay with iron-induced oxidative stress.
作者: Adjourn    時(shí)間: 2025-3-31 07:51
Macroeconomic Analysis of Monetary Unionslvement of iron in exaggerating the immune cells in the central nervous system (CNS) had been studied. Magnetic resonance imaging has shown increased levels of iron in different parts of the CNS. Dysregulation of iron homeostasis causes worsening of the demyelination and lesions in MS.
作者: Heart-Rate    時(shí)間: 2025-3-31 10:03
Iron and Multiple Sclerosislvement of iron in exaggerating the immune cells in the central nervous system (CNS) had been studied. Magnetic resonance imaging has shown increased levels of iron in different parts of the CNS. Dysregulation of iron homeostasis causes worsening of the demyelination and lesions in MS.




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