標(biāo)題: Titlebook: Brain Ischemia; Eugene Gusev,Veronica I. Skvortsova Book 2003 Springer Science+Business Media New York 2003 brain.death.depression.glutama [打印本頁(yè)] 作者: Mosquito 時(shí)間: 2025-3-21 19:25
書(shū)目名稱Brain Ischemia影響因子(影響力)
作者: cathartic 時(shí)間: 2025-3-21 21:48
Energy Failure Induced by Brain Ischemia a substrate for cytochrome oxidase, the terminal enzyme of the respiratory chain. The resulting state of hypoxia is the consequence of a complicated multi-stage process involving sequential changes in the properties of the mitochondrial enzyme complexes [.].作者: Nebulous 時(shí)間: 2025-3-22 00:35
Metabolic Acidosis and Ischemic Damagey activation of anaerobic glycolysis and to increased production of lactate and H. that causes the development of lactic acidosis. Modest increase in H. concentration in early stages of ischemia plays a compensatory and adaptive role as it promotes the improvement of perfusion in the penumbral area 作者: Fecal-Impaction 時(shí)間: 2025-3-22 07:21 作者: Thrombolysis 時(shí)間: 2025-3-22 09:30
Autoimmune Mechanisms of Trophic Dysfunction and Ischemic Brain Damagethey interact via mutual regulation involving neuromediators, neuropeptides, cytokines, trophic factors, and hormones and the corresponding receptor systems [.]. Mutual regulation of the nervous, endocrine, and immune systems determines the reliability of their common functioning. However, this enta作者: 我要沮喪 時(shí)間: 2025-3-22 16:50
Reaction of the Stress-Mediating Endocrine System in Response to Acute Brain Ischemiastem caused by the expression of immediate-early genes—by the synthesis of heat-shock (or stress) proteins (HSP) and cytokines, which are immune mediators [., ., ., .] (see Chapters 7 and 8; Fig. 8.1). The stress-mediating response provides the metabolie basis for compensatory and adaptive changes t作者: 尾隨 時(shí)間: 2025-3-22 20:52 作者: Condyle 時(shí)間: 2025-3-22 22:01
Reparative Therapyin tissue. Thus, from the first hours after stroke onset it is necessary to proceed with treatment aimed at the improvement of reparative and regenerative processes, plasticity of brain tissue, and the formation of new associative pathways, along with neuroprotection aimed at prevention of further b作者: 忙碌 時(shí)間: 2025-3-23 03:48
Vimal K. Shrivastava,Monoj K. Pradhanant to mention not only its therapeutic, but also preventive value. Correction of delayed consequences of ischemia can lead to retardation of vascular encephalopathy development in the post-stroke period.作者: Hyperopia 時(shí)間: 2025-3-23 08:20
Secondary Neuroprotectionant to mention not only its therapeutic, but also preventive value. Correction of delayed consequences of ischemia can lead to retardation of vascular encephalopathy development in the post-stroke period.作者: Fecal-Impaction 時(shí)間: 2025-3-23 13:20 作者: Derogate 時(shí)間: 2025-3-23 16:10 作者: Limpid 時(shí)間: 2025-3-23 18:52
ent blood is delivered to the brain. Many physiological processes occurring in the brain critically depend on the state of its energy metabolism. The state of brain energy metabolism in turn depends on the delivery of oxygen and glucose to the brain via the bloodstream. Although it comprises only 2%作者: 積極詞匯 時(shí)間: 2025-3-24 01:12 作者: 忘川河 時(shí)間: 2025-3-24 06:17 作者: ETCH 時(shí)間: 2025-3-24 08:48
Metabolic Acidosis and Ischemic DamageH. concentration in early stages of ischemia plays a compensatory and adaptive role as it promotes the improvement of perfusion in the penumbral area [.]. Significant elevation of lactate level within the first hours of ischemic stroke leads to decrease in pH to 6.4–6.7 [.] and appears to be an unfavorable prognostic sign [., ., ., .].作者: Collision 時(shí)間: 2025-3-24 10:40
Reparative Therapytive processes, plasticity of brain tissue, and the formation of new associative pathways, along with neuroprotection aimed at prevention of further brain damage and resulting enlargement of infarction volume.作者: 鞠躬 時(shí)間: 2025-3-24 15:02
A Brief Introduction on Kernels, the penumbra determine the final size of the brain infarction formed by the 3rd to 5th days after stroke onset. These changes must be studied to establish the basis for developing effective neuroprotective therapeutic approaches [., ., ., ., ., .].作者: 不感興趣 時(shí)間: 2025-3-24 21:28
https://doi.org/10.1007/978-3-658-40237-2ils the risk of systemic dysfunction when any one component of the whole system is affected. Such disorders can be defined as under-regulation pathology, the pathogenesis of which may be connected with primarily nervous or endocrine and/or immune mechanisms [.].作者: elucidate 時(shí)間: 2025-3-24 23:20
Fuwei Li,Lifeng Lai,Shuguang Cuihat to a considerable extent determine the features of the course of disease [., ., .]. This response is mediated by the sympathoadrenal, hypothalamic-pituitary-adrenal, renin-angiotensin, and thyroid hormonal axes [.].作者: 不能強(qiáng)迫我 時(shí)間: 2025-3-25 05:28
Book 2003is delivered to the brain. Many physiological processes occurring in the brain critically depend on the state of its energy metabolism. The state of brain energy metabolism in turn depends on the delivery of oxygen and glucose to the brain via the bloodstream. Although it comprises only 2% of the to作者: ADAGE 時(shí)間: 2025-3-25 11:29
Microglial Activation, Cytokine Production, and Local Inflammation in Focal Brain Ischemia the penumbra determine the final size of the brain infarction formed by the 3rd to 5th days after stroke onset. These changes must be studied to establish the basis for developing effective neuroprotective therapeutic approaches [., ., ., ., ., .].作者: 膽汁 時(shí)間: 2025-3-25 11:42
Autoimmune Mechanisms of Trophic Dysfunction and Ischemic Brain Damageils the risk of systemic dysfunction when any one component of the whole system is affected. Such disorders can be defined as under-regulation pathology, the pathogenesis of which may be connected with primarily nervous or endocrine and/or immune mechanisms [.].作者: HERTZ 時(shí)間: 2025-3-25 17:17 作者: 或者發(fā)神韻 時(shí)間: 2025-3-25 22:47
Kaizhu Huang,Haiqin Yang,Michael Lyuto the brain. Many physiological processes occurring in the brain critically depend on the state of its energy metabolism. The state of brain energy metabolism in turn depends on the delivery of oxygen and glucose to the brain via the blood stream.作者: Bouquet 時(shí)間: 2025-3-26 01:02 作者: 混合,攙雜 時(shí)間: 2025-3-26 07:38 作者: Gullible 時(shí)間: 2025-3-26 12:06 作者: –scent 時(shí)間: 2025-3-26 13:06 作者: 不近人情 時(shí)間: 2025-3-26 19:57 作者: Figate 時(shí)間: 2025-3-26 21:56 作者: antipsychotic 時(shí)間: 2025-3-27 01:59 作者: 吼叫 時(shí)間: 2025-3-27 08:31 作者: FECK 時(shí)間: 2025-3-27 10:59
Energy Failure Induced by Brain Ischemia a substrate for cytochrome oxidase, the terminal enzyme of the respiratory chain. The resulting state of hypoxia is the consequence of a complicated multi-stage process involving sequential changes in the properties of the mitochondrial enzyme complexes [.].作者: 咽下 時(shí)間: 2025-3-27 16:08 作者: 字的誤用 時(shí)間: 2025-3-27 20:35 作者: 毗鄰 時(shí)間: 2025-3-28 01:56
978-1-4613-4857-3Springer Science+Business Media New York 2003作者: 帶來(lái) 時(shí)間: 2025-3-28 03:45
Kaizhu Huang,Haiqin Yang,Michael Lyuto the brain. Many physiological processes occurring in the brain critically depend on the state of its energy metabolism. The state of brain energy metabolism in turn depends on the delivery of oxygen and glucose to the brain via the blood stream.作者: 使虛弱 時(shí)間: 2025-3-28 09:52
Acquisition of Proof Skills in Geometry a substrate for cytochrome oxidase, the terminal enzyme of the respiratory chain. The resulting state of hypoxia is the consequence of a complicated multi-stage process involving sequential changes in the properties of the mitochondrial enzyme complexes [.].作者: 描繪 時(shí)間: 2025-3-28 12:38 作者: 真實(shí)的人 時(shí)間: 2025-3-28 16:25
Thomas G. Dietterich,Ryszard S. MichalskiAcute focal brain ischemia triggers pathological biochemical reactions that take place in all neural tissue cell pools and cause neuronal dysfunction, astrocytosis, microglial activation, and accompanying changes in neutrophils, macrophages, and endothelial cells.作者: 剛毅 時(shí)間: 2025-3-28 21:42
An Overview of Machine LearningWhen glutamate is released from presynaptic terminals, it diffuses to the postsynaptic terminals where it binds to glutamate receptors, allowing Na. influx that depolarizes the membrane. This opens Ca. channels allowing Ca. to enter the postsynaptic cell. Under ischemic conditions, this process becomes excessive—the “glutamate-calcium cascade”.作者: Condense 時(shí)間: 2025-3-29 01:47 作者: 復(fù)習(xí) 時(shí)間: 2025-3-29 04:38 作者: sigmoid-colon 時(shí)間: 2025-3-29 07:40 作者: 不可救藥 時(shí)間: 2025-3-29 14:19
Fuwei Li,Lifeng Lai,Shuguang CuiWhen effective blood flow is recovered before irreversible necrotic damage forms, a stereotype reparative process will have been started in the brain tissue.作者: 怕失去錢(qián) 時(shí)間: 2025-3-29 16:04
Studies in Computational IntelligenceModern scientific and technological advances give reason to revise the previous pessimistic opinion about the therapeutic possibilities for acute focal brain ischemia, i.e. ischemic stroke.作者: lobster 時(shí)間: 2025-3-29 22:49 作者: Preamble 時(shí)間: 2025-3-30 01:07 作者: Kidnap 時(shí)間: 2025-3-30 05:05
Cellular Reactions in Response to Acute Focal Brain IschemiaAcute focal brain ischemia triggers pathological biochemical reactions that take place in all neural tissue cell pools and cause neuronal dysfunction, astrocytosis, microglial activation, and accompanying changes in neutrophils, macrophages, and endothelial cells.作者: 低位的人或事 時(shí)間: 2025-3-30 09:37 作者: amputation 時(shí)間: 2025-3-30 13:56
Delayed Neuronal Death Following Acute Focal Brain IschemiaThe major part of a brain infarction is formed within the first 3–6 h of the first clinical signs of strake (Fig. 6.1). However, the temporal evolution of infarction and its “up-formation” are connected with secondary brain cell damage which takes place in hours to days after the initial vascular event.作者: Negotiate 時(shí)間: 2025-3-30 16:47 作者: avarice 時(shí)間: 2025-3-30 22:37
Programmed Cell Death. Apoptosis in Focal Brain IschemiaUntil the 1980s it was believed that neuronal death in acute brain ischemia occurred only by the mechanism of necrosis. However, later studies elucidated the role of apoptosis, another pathway to infarction, which is a variant of programmed cell death.作者: 有花 時(shí)間: 2025-3-31 04:28
Molecular Mechanisms of Post-Ischemic Reparation EventsWhen effective blood flow is recovered before irreversible necrotic damage forms, a stereotype reparative process will have been started in the brain tissue.作者: Kidnap 時(shí)間: 2025-3-31 08:14
Modern Therapeutic Approaches to Acute Focal Brain Ischemia. Basic Strategies for NeuroprotectionModern scientific and technological advances give reason to revise the previous pessimistic opinion about the therapeutic possibilities for acute focal brain ischemia, i.e. ischemic stroke.作者: 公式 時(shí)間: 2025-3-31 10:12 作者: Meander 時(shí)間: 2025-3-31 16:10
Lisa French. “Phototherapy” 100 years ago. The first American treatise on the uses of blue light. thought of light as a mode of medical treatment and ignored its use in the management of jaundice in the newborn infant, a condition which, if unchecked, can be crippling to a severe degree.作者: Conjuction 時(shí)間: 2025-3-31 20:41 作者: mutineer 時(shí)間: 2025-3-31 22:28 作者: famine 時(shí)間: 2025-4-1 01:51 作者: 革新 時(shí)間: 2025-4-1 09:14
Dietmar Berger,Monika Engelhardt,Roland MertelsmanFundiertes und therapieorientiertes Nachschlagewerk.Detailinformationen zur Chemotherapie.CD-ROM mit der M?glichkeit individualisierte Patientenb?gen und Bestellformulare an die Apotheke auszudrucken.
