標題: Titlebook: Brain Injury; Robert S. B. Clark,Patrick Kochanek Book 2001 Springer Science+Business Media New York 2001 Alzheimer.Neurointensiv.Trauma.a [打印本頁] 作者: Helmet 時間: 2025-3-21 19:19
書目名稱Brain Injury影響因子(影響力)
書目名稱Brain Injury影響因子(影響力)學科排名
書目名稱Brain Injury網絡公開度
書目名稱Brain Injury網絡公開度學科排名
書目名稱Brain Injury被引頻次
書目名稱Brain Injury被引頻次學科排名
書目名稱Brain Injury年度引用
書目名稱Brain Injury年度引用學科排名
書目名稱Brain Injury讀者反饋
書目名稱Brain Injury讀者反饋學科排名
作者: 天賦 時間: 2025-3-21 20:14
openSAP – der Enterprise-MOOC-Pioniersent novel targets for therapies aimed at limiting damage after acute or chronic CNS injury. This chapter will provide a general review of death receptors and their signaling mechanisms, focusing on the two best characterized death receptors, Fas and TNF-α.作者: jocular 時間: 2025-3-22 01:57
Machen elektromagnetische Felder krank?l disintegration, nuclear condensation, and digestion of DNA. Finally, the engulfment of the dead cell or cell fragments by phagocytosis peacefully terminates the death program. Incomplete execution of programmed cell death may redirect the cell to necrosis, which is an unfavorable event for the org作者: 杠桿支點 時間: 2025-3-22 05:35
Machiavelli in Contemporary Media neurocytoskeletal elements sustain damage as a result of trauma, potentially contributing to neuronal dysfunction. Some of the major neurocytoskeletal proteins include neurofilaments (NFs), tubulin, microtubule-associated proteins (MAPs) such as MAP2 and tau, actin, and spectrin. Because cytoskelet作者: 焦慮 時間: 2025-3-22 09:35 作者: 偶像 時間: 2025-3-22 13:51
The Multifaceted Role of Adenosine in Experimental and Clinical Traumatic Brain Injury,thermia—putative pharmacological agents that might simultaneously or sequentially confer multiple beneficial effects in the injured brain. One endogenous mediator that fits this category is adenosine—which has the potential to favorably influence excitotoxicity, energy failure, hypoperfusion, calciu作者: instructive 時間: 2025-3-22 18:10
Death Receptors in Acute Brain Injury,sent novel targets for therapies aimed at limiting damage after acute or chronic CNS injury. This chapter will provide a general review of death receptors and their signaling mechanisms, focusing on the two best characterized death receptors, Fas and TNF-α.作者: PANIC 時間: 2025-3-22 22:51 作者: 遺傳學 時間: 2025-3-23 03:50
Neurocytoskeletal Changes Following Traumatic Brain Injury, neurocytoskeletal elements sustain damage as a result of trauma, potentially contributing to neuronal dysfunction. Some of the major neurocytoskeletal proteins include neurofilaments (NFs), tubulin, microtubule-associated proteins (MAPs) such as MAP2 and tau, actin, and spectrin. Because cytoskelet作者: 注意力集中 時間: 2025-3-23 07:10
Reproductive Hormones as Neuroprotectants in Brain Injury,d molecular mechanisms by which estrogen salvages brain are detailed. We focus on estrogen’s mechanisms since there is a large, and rapidly developing, understanding of this hormone’s neuroprotective potential. Limited data are available at present for progesterone or testosterone (reviewed in .; .)作者: 倔強不能 時間: 2025-3-23 10:07
Book 2001vered that are highly clinically relevant includethe effect of genetic background and gender differences in outcomeafter brain injury, preconditioning, and the effects of currently usedanesthetics and sedative agents in patients with brain injury.作者: 復習 時間: 2025-3-23 16:43 作者: faculty 時間: 2025-3-23 21:11 作者: 裂隙 時間: 2025-3-24 00:52 作者: 強化 時間: 2025-3-24 03:31
Preconditioning,may serve to mitigate or exacerbate the sequelae of the acute event and one process that may attenuate the damage of an acute injury is preconditioning. Preconditioning is the phenomenon whereby a non-lethal stimulus sets in motion a cascade of biochemical events that renders cells, tissues or the w作者: Gratulate 時間: 2025-3-24 08:44
Molecular and Cellular Mechanisms of Traumatic Cerebral Vascular Injury,uctivity annually in the United States alone (.). The physiologic deficit most likely responsible for this higher mortality is inadequate cerebral blood flow (CBF). Cerebral ischemia is caused by complex interactions between reduced blood pressure, impaired cerebral vasodilatory responsiveness, cere作者: 緩解 時間: 2025-3-24 12:12 作者: prosthesis 時間: 2025-3-24 18:42
Free Radicals and Acute Brain Injury: Mechanisms of Oxidative Stress and Therapeutic Potentials,CI) and cerebral ischemia, as well as chronic neurodegenerative diseases. Free radicals are also produced during normal oxidative metabolism. Free radicals generated during energy production are involved in enzymatic reactions, mitochondrial electron transport, signal transduction, activation of nuc作者: FICE 時間: 2025-3-24 20:04 作者: 智力高 時間: 2025-3-24 23:35
Cell Signaling: Serine/Threonine Protein Kinases and Traumatic Brain Injury, to the magnitude and type of TBI and the resulting systemic as well as neural complications. In this brief review we will focus on several features of neurotransmitter and trophic factor proximal signal transduction that normally function in the transfer of neuronal information within and between n作者: 混沌 時間: 2025-3-25 05:59
Death Receptors in Acute Brain Injury,tion of caspases (.; .; .; .; .; .; .). Although the cellular events downstream of activated caspases have been the subject of intense study, the exact mechanisms that initiate activation of cell death programs after acute CNS injury are less clear. Death receptors are transmembrane, multi-domain pr作者: Demulcent 時間: 2025-3-25 08:42 作者: Resistance 時間: 2025-3-25 13:27 作者: myriad 時間: 2025-3-25 17:23 作者: 大猩猩 時間: 2025-3-25 21:48 作者: 露天歷史劇 時間: 2025-3-26 01:04 作者: 植物學 時間: 2025-3-26 07:32
,Role of Genetic Background: Influence of Apolipoprotein E Genotype in Alzheimer’s Disease and Aftervent secondary brain injury. Despite intense interest in furthering our understanding of the pathogenesis of the events that occur after injury, results from multiple clinical trials based on current pathophysiological principles have failed to uncover therapies that improve outcomes in this populat作者: DUST 時間: 2025-3-26 09:37
Anesthetics and the Injured Brain,ensate to noxious stimuli. Because of these effects, major interactions between anesthetics and injured neural tissue can be expected. Effects of anesthetics on cerebral pathophysiology were reported more than 120 years ago. Since then, considerable scientific inquiry has been devoted to defining bo作者: Matrimony 時間: 2025-3-26 13:06 作者: vanquish 時間: 2025-3-26 17:34 作者: 閃光你我 時間: 2025-3-26 22:33 作者: 不適 時間: 2025-3-27 04:07 作者: 不能仁慈 時間: 2025-3-27 05:25
Excitotoxic Damage in Traumatic Brain Injury,ting disabilities in 25% of cases. This represents an enormous social and economical cost estimated to be around 38 billion dollars per year in the USA (.). However, at the present there is no available treatment to reduce the extent of cerebral damage following brain injury, other than supportive intensive care.作者: oxidant 時間: 2025-3-27 13:13 作者: Multiple 時間: 2025-3-27 14:27 作者: VEST 時間: 2025-3-27 21:39
Machen elektromagnetische Felder krank?echanisms, since its neurochemistry over the first 28-postnatal days has similarities with changes in humans over the first few years of life (.; .; .). This review will therefore focus on the experimental work about ischemia-induced ionic failure and its interrelation with excitatory amino acid- and oxidant-related injury in the developing brain.作者: Peristalsis 時間: 2025-3-28 01:30
Ischemia-Induced Ionic Mechanisms of Injury in the Developing Brain,echanisms, since its neurochemistry over the first 28-postnatal days has similarities with changes in humans over the first few years of life (.; .; .). This review will therefore focus on the experimental work about ischemia-induced ionic failure and its interrelation with excitatory amino acid- and oxidant-related injury in the developing brain.作者: Mediocre 時間: 2025-3-28 03:23 作者: 我悲傷 時間: 2025-3-28 07:27 作者: photopsia 時間: 2025-3-28 13:46 作者: ELUDE 時間: 2025-3-28 15:39
Lucio Biasiori,Giuseppe Marcoccith beneficial and detrimental effects of these drugs. Evidence from both animal and human investigations now allows reasonable scientific basis for use of these compounds in humans suffering acute brain injury.作者: Projection 時間: 2025-3-28 22:45 作者: Apoptosis 時間: 2025-3-29 02:15
Management of Emergency Situations,l perfusion pressure is critical for the effective treatment of head-injured patients, an important additional therapeutic strategy would be to restore the vasodilatory and vasoconstrictory properties of the cerebral vasculature that are intended to protect the brain by maintaining as constant and ideal an environment as possible.作者: 打包 時間: 2025-3-29 05:19 作者: 反感 時間: 2025-3-29 08:12
Petra H?rn,Frank Günther,Simon Dückert however, the complexity of these interactions suggests we still have much to learn. This review will focus on recent insights into how interactions between trophic and neurotransmitter signals may participate in neuronal survival after TBI.作者: 啞劇 時間: 2025-3-29 15:12 作者: 拋物線 時間: 2025-3-29 15:44
Molecular and Cellular Mechanisms of Traumatic Cerebral Vascular Injury,l perfusion pressure is critical for the effective treatment of head-injured patients, an important additional therapeutic strategy would be to restore the vasodilatory and vasoconstrictory properties of the cerebral vasculature that are intended to protect the brain by maintaining as constant and ideal an environment as possible.作者: engender 時間: 2025-3-29 20:54 作者: 吼叫 時間: 2025-3-30 03:21
Cell Signaling: Serine/Threonine Protein Kinases and Traumatic Brain Injury, however, the complexity of these interactions suggests we still have much to learn. This review will focus on recent insights into how interactions between trophic and neurotransmitter signals may participate in neuronal survival after TBI.作者: engagement 時間: 2025-3-30 06:46
Neurotransmitter and Growth Factor Alterations in Functional Deficits and Recovery Following Traumargic systems as prototypic neurotransmitter systems for memory and frontal lobe function. Neuronal survival and/or regeneration may be influenced by changes in growth factors following TBI. Therefore, this chapter will also review studies of growth factor responses to TBI and the therapeutic efficacy of exogeneously administered growth factors.作者: Narcissist 時間: 2025-3-30 09:17
Christoph Zimmermann,Albrecht W. Kraastted each year as a result of TBI in the USA alone (.). This disease affects mainly young adults in their productive stage of life, producing long lasting disabilities in 25% of cases. This represents an enormous social and economical cost estimated to be around 38 billion dollars per year in the US作者: conjunctivitis 時間: 2025-3-30 14:58 作者: abstemious 時間: 2025-3-30 19:46 作者: arthrodesis 時間: 2025-3-30 21:00 作者: 友好關系 時間: 2025-3-31 04:11 作者: 障礙 時間: 2025-3-31 08:37 作者: gait-cycle 時間: 2025-3-31 12:36 作者: Lice692 時間: 2025-3-31 15:10 作者: FOR 時間: 2025-3-31 18:35
