作者: consent 時(shí)間: 2025-3-21 23:07 作者: 性別 時(shí)間: 2025-3-22 02:05 作者: 火車車輪 時(shí)間: 2025-3-22 06:59 作者: 退出可食用 時(shí)間: 2025-3-22 12:26 作者: 要控制 時(shí)間: 2025-3-22 15:41 作者: Spirometry 時(shí)間: 2025-3-22 18:49
Jinkan Sai M.D.,Joe Ariyama M.D.ies [4,11,21,40,45,67]. Using models of diffuse and focal injuries, brain cooling has been shown to both protect against neuronal damage and improve behavioral outcome. Recently, post-traumatic hypothermia has also been reported to protect against axonal damage [39,43]. In addition, the blood-brain 作者: Conjuction 時(shí)間: 2025-3-22 23:12
Jinkan Sai M.D.,Joe Ariyama M.D.nal cord injury, the rat spinal cord was exposed at the 11th vertebra and compressed using 20 g of weight for 20 min. The temperature of the animals was fixed for 1 h from the beginning of the compression period at 33°C and 37°C for the mild hypothermic treatment and the control, respectively. At 24作者: 平躺 時(shí)間: 2025-3-23 04:09 作者: Accolade 時(shí)間: 2025-3-23 06:05 作者: 代替 時(shí)間: 2025-3-23 10:15
Image Contrast and Pulse Sequences,ge because of its harmful complications and the lack of basic knowledge of its beneficial effects. In 1987, it was shown in animal experiments that lowering brain temperature only by a few degrees Celsius during ischemia could protect central neurons against ischemic damages. Since then, widespread 作者: Allergic 時(shí)間: 2025-3-23 17:26 作者: Rankle 時(shí)間: 2025-3-23 20:57 作者: Offstage 時(shí)間: 2025-3-24 00:08 作者: 煞費(fèi)苦心 時(shí)間: 2025-3-24 04:39
https://doi.org/10.1007/978-1-4471-1413-0was complete destruction of neural tissue at the crush site, the area was filled in by other cells including neutrophils and connective tissue elements in both the hypothermia-treated and the untreated groups. These was a striking reduction in the progressive necrosis and cavitation that is characte作者: ALERT 時(shí)間: 2025-3-24 06:56 作者: 死亡 時(shí)間: 2025-3-24 12:20 作者: MUTED 時(shí)間: 2025-3-24 18:04
Enhanced Neuronal Damage in Severely Brain-Injured Patients by Hypothalamus, Pituitary, and Adrenal s a certain time window. The initial stages of brain injury involve destruction of the brain tissue, localized brain ischemia, cytokine inflammation, and synaptic dysfunction with release of vascular agonists, catecholamines, dopamine, neurogenous agonists such as choline, excitatory amino acids, an作者: 修飾 時(shí)間: 2025-3-24 21:16 作者: Exposition 時(shí)間: 2025-3-25 02:21
Mild Hypothermia Amelioration of Damage During Rat Spinal Cord Injury: Inhibition of Pathological Miry. A marked proliferation of positive isolectin-staining microglia was observed in the control animals from 48 h after the injury. On the other hand, in both hypothermia-treated and sham animals, a small number of microglia were observed up to 72 h after the injury. These results suggest that mild 作者: ARY 時(shí)間: 2025-3-25 05:53
Local Cooling: Limits on Secondary Injury and Neuronal Death Following Spinal Cord Injurywas complete destruction of neural tissue at the crush site, the area was filled in by other cells including neutrophils and connective tissue elements in both the hypothermia-treated and the untreated groups. These was a striking reduction in the progressive necrosis and cavitation that is characte作者: 反應(yīng) 時(shí)間: 2025-3-25 08:22 作者: 最初 時(shí)間: 2025-3-25 14:43 作者: 發(fā)芽 時(shí)間: 2025-3-25 17:19
mia, Basic Research of Hypothermia Treatment, and Clinical Studies of Brain Hypothermia. The book is a valuable source for practitioners and researchers in neurosurgery and neurology and in critical care and emergency medicine.978-4-431-70277-1978-4-431-66882-4作者: chastise 時(shí)間: 2025-3-25 23:05
Enhanced Neuronal Damage in Severely Brain-Injured Patients by Hypothalamus, Pituitary, and Adrenal e these pathophysiological changes, such as hypotension, hypoxia, free radicals, blood-brain barrier dysfunction, excitatory amino acid, and increased intracellular Ca., have been considered targets of treatment. This concept of brain injury mechanism has long been supported by many animal studies. 作者: Conscientious 時(shí)間: 2025-3-26 02:20
Molecular System Controlling Mammalian Hibernation with Circannual Rhythm. Such adaptation has been suggested to be due to readjustment of cells and organs to a new physiological state. As hibernation occurs with circannual rhythm, this physiological readjustment has been suggested to be under the control of a circannual rhythm. Recently, a novel protein complex (HP), fo作者: assail 時(shí)間: 2025-3-26 06:22 作者: subordinate 時(shí)間: 2025-3-26 09:38
Mild Hypothermia Amelioration of Damage During Rat Spinal Cord Injury: Inhibition of Pathological Minal cord injury, the rat spinal cord was exposed at the 11th vertebra and compressed using 20 g of weight for 20 min. The temperature of the animals was fixed for 1 h from the beginning of the compression period at 33°C and 37°C for the mild hypothermic treatment and the control, respectively. At 24作者: cluster 時(shí)間: 2025-3-26 15:17 作者: Binge-Drinking 時(shí)間: 2025-3-26 17:36
Effect of Intraischemic Hypothermia on Expression of c-Fos, c-Jun, and HSP72 After Transient Focal Cto either a normothermic (NT) or a hypothermic (HT) group. In the NT group the animals were left at ambient temperature (21°C), and the brain temperature was elevated to 40°C during ischemia. In the HT group the animals were put into a cold room (1°C), and the brain temperature was decreased to 30°C作者: Subjugate 時(shí)間: 2025-3-26 21:03
Neuroprotective Effect of Mild Hypothermia in Experimental Brain Ischemiage because of its harmful complications and the lack of basic knowledge of its beneficial effects. In 1987, it was shown in animal experiments that lowering brain temperature only by a few degrees Celsius during ischemia could protect central neurons against ischemic damages. Since then, widespread 作者: JADED 時(shí)間: 2025-3-27 03:16 作者: CHAFE 時(shí)間: 2025-3-27 06:54
Conference proceedings 2000he therapy for critically brain-injured patients and breakthroughs in understanding the pathophysiology of brain damage. The latest concept of brain hypothermia therapy clarifies targets such as brain thermal pooling, masking brain hypoxia associated with catecholamine surge, the metabolic shift fro作者: LIMN 時(shí)間: 2025-3-27 11:01
Jinkan Sai M.D.,Joe Ariyama M.D.els and various morphological and functional endpoints emphasize how remarkably effective hypothermia can be in protecting the brain. Importantly, these experimental findings with therapeutic hypothermia have been supported by clinical data where hypothermic protection has been demonstrated in patients with severe brain injury [12,13,42].作者: 交響樂(lè) 時(shí)間: 2025-3-27 14:32 作者: 愛(ài)哭 時(shí)間: 2025-3-27 20:50 作者: 友好關(guān)系 時(shí)間: 2025-3-27 22:31
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