標(biāo)題: Titlebook: Brain Edema X; Proceedings of the T Hector E. James,Lawrence F. Marshall,Zbigniew Czer Conference proceedings 1997 Springer-Verlag/Wien 199 [打印本頁] 作者: papertrans 時(shí)間: 2025-3-21 16:45
書目名稱Brain Edema X影響因子(影響力)
書目名稱Brain Edema X影響因子(影響力)學(xué)科排名
書目名稱Brain Edema X網(wǎng)絡(luò)公開度
書目名稱Brain Edema X網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Brain Edema X被引頻次
書目名稱Brain Edema X被引頻次學(xué)科排名
書目名稱Brain Edema X年度引用
書目名稱Brain Edema X年度引用學(xué)科排名
書目名稱Brain Edema X讀者反饋
書目名稱Brain Edema X讀者反饋學(xué)科排名
作者: 監(jiān)禁 時(shí)間: 2025-3-21 21:31
https://doi.org/10.1007/3-540-32382-1hown to cause disruption of blood-brain barrier (BBB) by destruction of capillary endothelial cell membrane. However, the exact mechanism of BBB breakdown by cerebral ischemia/reperfusion remains undetermined..The aim of the present study is to clarify the mechanism of intracellular calcium ion ([Ca作者: overrule 時(shí)間: 2025-3-22 03:07 作者: BANAL 時(shí)間: 2025-3-22 04:50 作者: 環(huán)形 時(shí)間: 2025-3-22 12:33 作者: PANEL 時(shí)間: 2025-3-22 16:03 作者: 走調(diào) 時(shí)間: 2025-3-22 20:09 作者: SUGAR 時(shí)間: 2025-3-22 21:40
Effects on Measurement Accuracy,leading to neuronal injury. The effects of hyperglycemia on another possible primary target, the cerebral microvasculature, is examined in this study. Focal cerebral ischemia was achieved by thread occlusion of the middle cerebral artery (MCA). Preischemic hyperglycemia was induced by intraperitonea作者: 合乎習(xí)俗 時(shí)間: 2025-3-23 04:55 作者: Overthrow 時(shí)間: 2025-3-23 06:05
https://doi.org/10.1007/978-1-4419-6020-7brain edema formation. Preischemic hyperglycemia was induced by intraperitoneal administration of D-glucose solution. Laser-Doppler flowmetry, indicated that after the initial decline in blood flow with carotid occlusion (36 ± 4% of preischemic), hyperglycemic but not control rats showed a further p作者: 鐵砧 時(shí)間: 2025-3-23 13:32
MEMS Lorentz Force Magnetometersed from rat cerebral cortices by a two step enzymatic treatment. The monolayers of BCECs were subjected to anoxia for 20 minutes followed by reoxygenation for 3 hours. Cell damage was assessed by measuring the leakage of intracellular lactic dehydrogenase (LDH). The control group was anoxia/reoxygen作者: 槍支 時(shí)間: 2025-3-23 16:39
MEMS Lorentz Force Magnetometersate the effects of mannitol in comatose patients on PtiO., PtiCO. and brain tissue pH using Clark-type electrodes implanted into cerebral white matter. In the neurosurgical intensive care unit PtiO., PtiCO., brain tissue pH, arterial blood pressure, intracranial pressure (ICP), cerebral perfusion pr作者: 允許 時(shí)間: 2025-3-23 21:19
Ronald G. Polcawich,Jeffrey S. Pulskampof recent stroke studies. Two magnetic resonance imaging techniques; diffusion-weighted (DW) and magnetization transfer contrast (MTC) imaging were introduced in experimental studies and were shown to have sensitivity for different stages of brain infarct...We used a reproducible middle cerebral art作者: BRAVE 時(shí)間: 2025-3-24 00:55
Shawn J. Cunningham,Mario Kupnikma cells have revealed that viable astrocytes swell, spatially buffering various metabolites which are increased by the metabolic turmoil following ischemic insults. In the present study, we have studied the temporal profile of ultrastructural changes of astrocytes in the cerebral cortex associated 作者: Abjure 時(shí)間: 2025-3-24 05:49
What Is the Product? Requirements Analysiscal ischemia. Two hours of tandem occlusion of the middle cerebral artery (MCA) and the conunon carotid artery produced a scattered reduction of SDH activity and corresponding brain edema in the cortex. Recirculation ameliorated the SDH reduction and the scattered pattern disappeared, although the b作者: 彩色 時(shí)間: 2025-3-24 08:10
Design for Back-end-of-Line Processes cerebral ischemia (GCI) by intravital fluorescence microscopy. Four days after 15 min of bilateral common carotid artery occlusion the ischemic tissue damage was histologically analysed in selectively vulnerable areas of the brain. The frequency of Rhodamine 6G labeled leukocytes rolling along (“ro作者: GULF 時(shí)間: 2025-3-24 12:00 作者: Dedication 時(shí)間: 2025-3-24 17:27
What Is the Product? Requirements Analysiscerebrovascular reserve capacity after the brain ischemia. The experiments were carried out in rabbits. Administration of acetazolamide (Diamox) of 20 mg/kg was performed in all experimental animals and the blood flow velocity in the middle cerebral artery (MCA) and the basilar artery (BA) was measu作者: 相信 時(shí)間: 2025-3-24 21:11 作者: 攤位 時(shí)間: 2025-3-25 02:58 作者: Glucocorticoids 時(shí)間: 2025-3-25 05:38 作者: Ceramic 時(shí)間: 2025-3-25 10:42 作者: 閃光你我 時(shí)間: 2025-3-25 14:58 作者: coagulation 時(shí)間: 2025-3-25 17:58
Conference proceedings 1997re not accepted by the Editorial Board. Therefore, the excellent quality of those that are in the book are a reflection of the authors‘ dedication and work and that of those of the Editorial Board in their review process. For the reader‘s convenience, the papers are structured according to the vario作者: REIGN 時(shí)間: 2025-3-25 22:16
The Role of Calcium Ion in Anoxia/Reoxygenation Damage of Cultured Brain Capillary Endothelial Cellsy decreased after reoxygenation..Indomethacin and SOD suppressed the elevation of [Ca.]. during anoxia. N.-nitro-L-arginine methyl ester and catalase moderately supressed the elevation, however nifedipine did not supress it at all. In this model, rapid [Ca.]. change was not observed during the reoxy作者: ARENA 時(shí)間: 2025-3-26 02:55 作者: Phonophobia 時(shí)間: 2025-3-26 04:32
Increase in Surface Expression of ICAM-1, VCAM-1 and E-Selectin in Human Cerebromicrovascular Endothh the cytokine- and ischemia-induced up-regulation of adhesion molecules were completely abolished by the transcriptional inhibitor, actinomycin D (10 μg/ml), and inhibited by the cycloxygenase (COX) inhibitor, indomethacin (300 μM). These findings implicate HCEC in the processes of leukocyte adhesi作者: 遺產(chǎn) 時(shí)間: 2025-3-26 10:20
Assessment of the Relationship Between Ischemic Damage and Brain Swelling in Frozen Brain Slicesently observed, whereas sham-operated rats demonstrated no brain infarction or significant differences between two hemispheres. There were good correlationships not only between the volumes of brain edema and infarction (p < 0.05) but between the volume of brain edema and the difference in weight (p作者: Amenable 時(shí)間: 2025-3-26 16:42
Brain Edema Associated with Progressive Selective Neuronal Death or Impending Infarction in the Cere ischemia. Evans blue extravasated only in face A, corresponding to focal infarction at 24 and 48 hr after the 2nd 10-min ischemia. The specific gravities of the ischemic cortex of both faces decreased significantly from control at 15 min (P < 0.05) and had recovered by 5 hr after ischemia. By 12 hr作者: Abbreviate 時(shí)間: 2025-3-26 16:47 作者: Abbreviate 時(shí)間: 2025-3-26 23:26
Hyperglycemia and the Vascular Effects of Cerebral Ischemiaerglycemia led to a further progressive reduction during ischemia. On reperfusion, hyperglycemia resulted in poor restoration of CBF, increased occurrence of hemorrhagic infarction (12 of 12) and a large infarct volume. Hyperglycemia induces progressive cerebrovascular changes during ischemia and af作者: 英寸 時(shí)間: 2025-3-27 04:38 作者: grandiose 時(shí)間: 2025-3-27 05:35
Effects of Hyperglycemia on Cerebral Blood Flow and Edema Formation After Carotid Artery Occlusion i appears to primarily reflect the effects of hyperglycemia on CBF. Cerebral plasma volume (CPV) 2 hours after occlusion was also reduced in hyper-compared to normoglycemic rats (3.9 ± 0.9 and 7.2 ± 0.1 μl/g; p < 0.01). Thus, hyperglycemia in a model of global ischemia induces a reduction in CPV and 作者: Subdue 時(shí)間: 2025-3-27 10:26 作者: galley 時(shí)間: 2025-3-27 16:03
Mannitol Decreases ICP but Does Not Improve Brain-Tissue pO2 in Severely Head-Injured Patients with ve 20 mmHg a significant effect was seen on ICP (decrease from 23 ± 1 to 16 ± 2 mmHg at 60 min) and CPP (increase from 68 ± 2 to 80 ± 3 mmHg at 120 min). These effects were not reflected in PtiO. or SjvO., which were 29 ± 4 mmHg and 61 ± 3%, respectively, at the beginning of mannitol injection and r作者: BET 時(shí)間: 2025-3-27 18:42
A Chronological Evaluation of Experimental Brain Infarct by Diffusion-Mapping and Magnetization Tran high signal intensity, while T. imaging does not raise significant contrast of the lesion at this early stage. Three sub regions of the lesion having different ADCs, are discernible on diffusion-mapping images from 1 day to 7 days. The decrease of MTC effect was measured within the infarct lesion f作者: 輕快帶來危險(xiǎn) 時(shí)間: 2025-3-28 01:27
Ultrastructure of Astrocytes Associated with Selective Neuronal Death of Cerebral Cortex After Repean, 5 and 12 hr after the 2nd 10 min ischemia. Ultrathin sections including the 3rd and 5th cortical layers were prepared from the cut surface at the level of infundibulum. Mild swelling of astrocytic processes and perivascular end-feet was observed in the 15 min group. Glycogen granules were not pro作者: HAIRY 時(shí)間: 2025-3-28 04:24 作者: 小母馬 時(shí)間: 2025-3-28 08:22
Selective Impairments of Mitochondrial Respiratory Chain Activity During Aging and Ischemic Brain Danificant impairment of complex I activity in the lesioned hemisphere of both age groups. The age related decline in complex I activity may be important in the enhanced susceptibility of the aging brain to ischemic neuronal damage.作者: harpsichord 時(shí)間: 2025-3-28 11:57
Regional Differences in the Cerebrovascular Reserve Following Acute Global Ischemia in Rabbitsse in the BA. In some animals there was a decrease in the blood flow velocity in MCA. This could be a consequence of stealing phenomenon, as an increase of cerebral blood flow in the posterior fossa compartment, at the cost of the supratentorial circulation. Identification of the areas damaged after作者: motor-unit 時(shí)間: 2025-3-28 15:25 作者: 表皮 時(shí)間: 2025-3-28 22:00
0065-1419 e excellent quality of those that are in the book are a reflection of the authors‘ dedication and work and that of those of the Editorial Board in their review process. For the reader‘s convenience, the papers are structured according to the vario978-3-7091-7418-0978-3-7091-6837-0Series ISSN 0065-1419 Series E-ISSN 2197-8395 作者: chance 時(shí)間: 2025-3-28 23:04 作者: 抵消 時(shí)間: 2025-3-29 04:29
Algorithmic Developments in TAUlease. The production of ROS was calcium-dependent and not affected by AA or its metabolites. The findings indicate that the RBEC can produce superoxide dismutase (SOD)-inhibita-ble ROS which are augmented by hypoxia/reoxygenation. It is suggested that . cerebromicrovascular endothelium may contribu作者: Increment 時(shí)間: 2025-3-29 11:15 作者: 賄賂 時(shí)間: 2025-3-29 13:16 作者: 極少 時(shí)間: 2025-3-29 17:49 作者: 減去 時(shí)間: 2025-3-29 23:14 作者: orthodox 時(shí)間: 2025-3-29 23:54
Effects on Measurement Accuracy,erglycemia led to a further progressive reduction during ischemia. On reperfusion, hyperglycemia resulted in poor restoration of CBF, increased occurrence of hemorrhagic infarction (12 of 12) and a large infarct volume. Hyperglycemia induces progressive cerebrovascular changes during ischemia and af作者: CESS 時(shí)間: 2025-3-30 05:09 作者: Harridan 時(shí)間: 2025-3-30 10:17
https://doi.org/10.1007/978-1-4419-6020-7 appears to primarily reflect the effects of hyperglycemia on CBF. Cerebral plasma volume (CPV) 2 hours after occlusion was also reduced in hyper-compared to normoglycemic rats (3.9 ± 0.9 and 7.2 ± 0.1 μl/g; p < 0.01). Thus, hyperglycemia in a model of global ischemia induces a reduction in CPV and 作者: Champion 時(shí)間: 2025-3-30 16:05 作者: 強(qiáng)制性 時(shí)間: 2025-3-30 16:39
MEMS Lorentz Force Magnetometersve 20 mmHg a significant effect was seen on ICP (decrease from 23 ± 1 to 16 ± 2 mmHg at 60 min) and CPP (increase from 68 ± 2 to 80 ± 3 mmHg at 120 min). These effects were not reflected in PtiO. or SjvO., which were 29 ± 4 mmHg and 61 ± 3%, respectively, at the beginning of mannitol injection and r作者: Munificent 時(shí)間: 2025-3-30 21:18 作者: myocardium 時(shí)間: 2025-3-31 01:08
Shawn J. Cunningham,Mario Kupnikn, 5 and 12 hr after the 2nd 10 min ischemia. Ultrathin sections including the 3rd and 5th cortical layers were prepared from the cut surface at the level of infundibulum. Mild swelling of astrocytic processes and perivascular end-feet was observed in the 15 min group. Glycogen granules were not pro作者: 羽毛長成 時(shí)間: 2025-3-31 06:35 作者: 修剪過的樹籬 時(shí)間: 2025-3-31 10:13 作者: Hyperplasia 時(shí)間: 2025-3-31 15:25
What Is the Product? Requirements Analysisse in the BA. In some animals there was a decrease in the blood flow velocity in MCA. This could be a consequence of stealing phenomenon, as an increase of cerebral blood flow in the posterior fossa compartment, at the cost of the supratentorial circulation. Identification of the areas damaged after作者: patella 時(shí)間: 2025-3-31 18:31
Glutamate Uptake and Na,K-ATPase Activity in Rat Astrocyte Cultures Exposed to Ischemiachemic insult .. To measure sodium pump activity and glutamate uptake, .H-glutamate and .Rb were simultaneously added to the cultures in the presence or absence of 2 mM ouabain. Na,K-ATPase activity was defined as ouabainsensitive .Rb uptake. Cell death was assessed by exclusion of the vital dye, ca
