標題: Titlebook: Brain Aging and Therapeutic Interventions; Mahendra K. Thakur,Suresh I.S. Rattan Book 2012 Springer Science+Business Media Dordrecht 2012 [打印本頁] 作者: 爆裂 時間: 2025-3-21 17:56
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書目名稱Brain Aging and Therapeutic Interventions網(wǎng)絡公開度學科排名
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書目名稱Brain Aging and Therapeutic Interventions讀者反饋
書目名稱Brain Aging and Therapeutic Interventions讀者反饋學科排名
作者: 充氣女 時間: 2025-3-21 21:39
Base Excision DNA Repair: The House Keeping Guardian for Genomic Stability in the Brain, keep driving metabolic changes that would lead to more viable progenies for survival and propagation. Thus natural evolution uses two opposing forces to improve the genetic makeup of the species. With the advent of organisms turning aerobic, the advantage of gains in energy production also paved th作者: arrogant 時間: 2025-3-22 00:24
Role of Brain-Derived Neurotrophic Factor as a Modulator of Synaptic Plasticity,ifferentiation, but also functions in activity-dependent plasticity processes such as long-term potentiation (LTP), long-term depression (LTD), learning and memory. BDNF is synthesized as a precursor protein (proBDNF) that is proteolytically cleaved to form the mature protein. BDNF mediates its effe作者: 清楚 時間: 2025-3-22 05:12 作者: hegemony 時間: 2025-3-22 08:45
,Brain Aging: Influence of Early–Life Events on Late-Life Brain Disorders,ellectual abilities. AD usually appears late in adult life, but when the disease initiates and how long the disease processes take to develop are presently unknown. To address this issue, the “Latent Early–life Associated Regulation” (LEARn) model has been proposed. This model explains the etiology 作者: 輕浮女 時間: 2025-3-22 16:19
The Importance of the Environment in Brain Aging: Be Happy, Live Longer!,his demands development of effective therapeutic/preventive interventions aimed to slow down the negative effects of aging and extend health-span. Here, we overview the mechanisms underlying brain aging in the context of the oxi-inflamm-aging theory, and discuss cutting edge promising findings openi作者: 表被動 時間: 2025-3-22 19:09 作者: 密切關系 時間: 2025-3-22 23:39 作者: Immunoglobulin 時間: 2025-3-23 04:02
Neurolipofuscin in Aging and Aluminum-Induced Aging: Possible Therapeutic Interventions,igment but it distorts the protein building machinery of the cell and interferes with the important autophagic process. Numerous forms of cellular stresses activate macroautophagy. Increased autophagic vacuoles (AVs) are associated with not only aging and ceroid disorders but also Alzheimer’s diseas作者: 馬具 時間: 2025-3-23 06:57
Dietary Restriction as a Potential Intervention to Retard Age-associated Impairment of Brain Functi various environmental factors that are known to facilitate healthy aging process, dietary restriction (DR) seems to be the most promising regimen as it is known to prolong the lifespan, attenuate aging and prevent and/or delay the onset of numerous age associated diseases in model systems. In our r作者: 反饋 時間: 2025-3-23 11:30 作者: 使糾纏 時間: 2025-3-23 14:07
Neurodegeneration in Hypoxia: Implications in Aging, Similar decrease in partial pressure of oxygen occurs on ascent to high altitude, a situation referred to as hypobaric hypoxia that limits oxygen availability to the brain. Our studies on human subjects reveal decrease in vigilance and response time along with decreased cerebral oxygenation which i作者: 讓你明白 時間: 2025-3-23 19:29 作者: Jargon 時間: 2025-3-23 22:22 作者: Alopecia-Areata 時間: 2025-3-24 05:39 作者: palpitate 時間: 2025-3-24 08:51 作者: 異端 時間: 2025-3-24 11:08 作者: harangue 時間: 2025-3-24 16:27 作者: Rustproof 時間: 2025-3-24 22:52 作者: slipped-disk 時間: 2025-3-25 00:17
ntions.Identifying cognitive impairment and therapeutic inte.This book brings together most up-to-date information on different aspects of brain aging and on the strategies for intervention and therapy of age-related brain disorders. It includes 18 chapters by leading researchers, and each chapter i作者: ARM 時間: 2025-3-25 04:04
Book 2012elopments in the field of neurodegenerative disorders and their therapeutic interventions. This book will be of much interest to pharmaceutical, nutrition and healthcare industry for an easy access to accurate and reliable information in the field of aging research and intervention..作者: 爭議的蘋果 時間: 2025-3-25 07:45 作者: Amenable 時間: 2025-3-25 13:45
https://doi.org/10.1007/978-3-8348-8117-5remature senescence and progeria like phenotype. These studies have demonstrated that mortalin-mediated mitochondrial functions are not only the key factors in maintaining the continued proliferation of cancer cells but also the normal neuronal physiology. Lack of a functional mortalin leads to cancer cell death and neurodegenerative phenotype.作者: Enthralling 時間: 2025-3-25 19:11
https://doi.org/10.1007/978-3-8348-9007-8 on oxidative stress and cognitive decline with age on animal models and human subjects showing the importance of non-polyphenolic and polyphenolic-rich plant products in alleviating age-related cognitive loss.作者: Synchronism 時間: 2025-3-25 22:50 作者: constitute 時間: 2025-3-26 02:46
Consequences of Altered Mortalin Expression in Control of Cell Proliferation and Brain Function,remature senescence and progeria like phenotype. These studies have demonstrated that mortalin-mediated mitochondrial functions are not only the key factors in maintaining the continued proliferation of cancer cells but also the normal neuronal physiology. Lack of a functional mortalin leads to cancer cell death and neurodegenerative phenotype.作者: Femish 時間: 2025-3-26 06:00 作者: cluster 時間: 2025-3-26 11:52
Book 2012elated brain disorders. It includes 18 chapters by leading researchers, and each chapter is a comprehensive and critical review of the topic in question, discusses the current scenario and focuses on future perspectives. The target readership is the undergraduate and graduate students in the univers作者: expire 時間: 2025-3-26 13:46 作者: collagenase 時間: 2025-3-26 17:38
https://doi.org/10.1007/978-3-658-39516-2in maintaining the structural integrity of DNA that is quite often threatened from events taking place within the cell itself. Implicit in this crucial role is also the fact that any perturbation in this repair pathway could lead to deleterious consequences—particularly in a post-mitotic tissue like作者: 小步走路 時間: 2025-3-26 22:15 作者: corpuscle 時間: 2025-3-27 01:54
Manfred Bornhofen,Martin C. Bornhofenotein and amyloid beta peptides in mitochondria in AD brain has been the subject of intense and interesting research and the review has described this aspect in the context of mitochondrial dysfunction. The cross-talk of mitochondrial dysfunction with oxidative stress and inflammatory response in AD作者: Definitive 時間: 2025-3-27 07:14 作者: Inoperable 時間: 2025-3-27 11:07
https://doi.org/10.1007/978-3-658-13846-2al effects on sleep as well as age related diseases. This review aims to summarize the current knowledge on the changes of the circadian system in advanced age and the therapeutic effects of antioxidant melatonin on such changes.作者: 樂器演奏者 時間: 2025-3-27 15:14 作者: Confess 時間: 2025-3-27 21:34 作者: Fortuitous 時間: 2025-3-28 01:08 作者: Glucose 時間: 2025-3-28 06:07 作者: podiatrist 時間: 2025-3-28 09:12
https://doi.org/10.1007/978-3-8348-9007-8 including our ICER mutant mice, is discussed. Finally, based on the developments emerging from research using genetically modified mice, potential molecular targets are proposed for ameliorating memory impairments and dementia.作者: DOSE 時間: 2025-3-28 13:24 作者: 舊式步槍 時間: 2025-3-28 17:19
https://doi.org/10.1007/978-3-8348-9007-8ly controlled and standardized. The challenge is even greater when targeting human cognitive function, a multi faceted process which is demanding to measure and hard to improve. However, good progress is being made in this field; standardized cognitive tests are available and gold-standard experimen作者: 減少 時間: 2025-3-28 19:39 作者: 中古 時間: 2025-3-29 01:17
Brain Aging: A Critical Reappraisal,ticity, perturbed calcium homeostasis, neuronal survival and neuroinflammation, eventually affecting brain functions with increased incidence of neurological disorders. Factors like stress, depression, hypertension as well as obesity accelerate the aging of brain contributing to neurodegeneration an作者: 群島 時間: 2025-3-29 03:11
Base Excision DNA Repair: The House Keeping Guardian for Genomic Stability in the Brain,in maintaining the structural integrity of DNA that is quite often threatened from events taking place within the cell itself. Implicit in this crucial role is also the fact that any perturbation in this repair pathway could lead to deleterious consequences—particularly in a post-mitotic tissue like作者: 孵卵器 時間: 2025-3-29 09:50 作者: 違反 時間: 2025-3-29 15:13 作者: 違抗 時間: 2025-3-29 16:41
,Brain Aging: Influence of Early–Life Events on Late-Life Brain Disorders,ly later in life, if an additional perturbation were to occur. The LEARn model postulates latent expression of specific genes triggered at an early stage of life. The LEARn model operates via the regulatory region (promoter) of the gene, specifically through changes in methylation and oxidation stat作者: 延期 時間: 2025-3-29 21:02 作者: Repetitions 時間: 2025-3-30 00:37 作者: CRUDE 時間: 2025-3-30 05:47 作者: outer-ear 時間: 2025-3-30 09:31 作者: 統(tǒng)治人類 時間: 2025-3-30 14:24 作者: 止痛藥 時間: 2025-3-30 17:35
Potential Therapeutic Targets for Improving Memory Impairments and Dementia: Clues Obtained from Me including our ICER mutant mice, is discussed. Finally, based on the developments emerging from research using genetically modified mice, potential molecular targets are proposed for ameliorating memory impairments and dementia.作者: 委托 時間: 2025-3-30 21:44 作者: 考得 時間: 2025-3-31 02:49 作者: 藐視 時間: 2025-3-31 06:39
Stress and Memory: from Mechanisms to Long-Lasting Consequences,作者: opportune 時間: 2025-3-31 11:01 作者: 異端 時間: 2025-3-31 14:41 作者: 半圓鑿 時間: 2025-3-31 18:39
https://doi.org/10.1007/978-3-658-39516-2ifferentiation, but also functions in activity-dependent plasticity processes such as long-term potentiation (LTP), long-term depression (LTD), learning and memory. BDNF is synthesized as a precursor protein (proBDNF) that is proteolytically cleaved to form the mature protein. BDNF mediates its effe作者: Deduct 時間: 2025-3-31 23:28 作者: critique 時間: 2025-4-1 03:01
,Teil 2: Zus?tzliche F?lle und L?sungen,ellectual abilities. AD usually appears late in adult life, but when the disease initiates and how long the disease processes take to develop are presently unknown. To address this issue, the “Latent Early–life Associated Regulation” (LEARn) model has been proposed. This model explains the etiology 作者: Critical 時間: 2025-4-1 06:05
https://doi.org/10.1007/978-3-658-26179-5his demands development of effective therapeutic/preventive interventions aimed to slow down the negative effects of aging and extend health-span. Here, we overview the mechanisms underlying brain aging in the context of the oxi-inflamm-aging theory, and discuss cutting edge promising findings openi
