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標(biāo)題: Titlebook: Blood Pressure and Arterial Wall Mechanics in Cardiovascular Diseases; Michel E. Safar,Michael F. O‘Rourke,Edward D. Froh Book 2014 Spring [打印本頁(yè)]

作者: 斷巖    時(shí)間: 2025-3-21 17:31
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書目名稱Blood Pressure and Arterial Wall Mechanics in Cardiovascular Diseases讀者反饋




書目名稱Blood Pressure and Arterial Wall Mechanics in Cardiovascular Diseases讀者反饋學(xué)科排名





作者: 聰明    時(shí)間: 2025-3-21 21:40
https://doi.org/10.1007/978-3-030-56787-3rease NO and EET release under physiological conditions, and the reduced pulse pressure-dependent strain in stiff conduits may suppress this adaptive response, thus amplifying the stiffening process. This article details fundamental experiments and clinical studies supporting these mechanisms.
作者: arthrodesis    時(shí)間: 2025-3-22 00:50
Mechanical Stress and the Arterial Wallascular pathology is pulse pressure, the difference between systolic and diastolic blood pressures. Both large and small arteries are exposed to changes in the mechanical environment and are remodeled in response to it.
作者: ADOPT    時(shí)間: 2025-3-22 07:13

作者: 減弱不好    時(shí)間: 2025-3-22 12:46

作者: neutralize    時(shí)間: 2025-3-22 13:56
e.Defines the clinical issues in relation to the study of thIn cardiovascular prevention, there is classically a small number of cardiovascular risk factors to treat, such as hypertension, diabetes, hyperlipidemia and smoking excess, which are widely detected and treated. Recently, it has been widel
作者: 放牧    時(shí)間: 2025-3-22 20:14

作者: 慢跑鞋    時(shí)間: 2025-3-22 23:37
https://doi.org/10.1007/3-540-37672-0in the pathogenesis of hypertension, atherosclerosis, and vascular stiffening. Here recent findings regarding angiotensin II biology and their potential relevance for vascular biology and disease are discussed
作者: brassy    時(shí)間: 2025-3-23 02:24
Determination of Systemic and Regional Arterial Structure and Functiones gives also direct, hypothesis-free measurement of arterial stiffness. Other techniques are derived from models of circulation and can approximate arterial stiffness. Details about techniques, parameter definition, are given here to help researchers and practitioners to make the best choice of technique for their applications.
作者: HEW    時(shí)間: 2025-3-23 05:56
Emerging Aspects of Angiotensin Biology and Their Potential Role in the Vasculaturein the pathogenesis of hypertension, atherosclerosis, and vascular stiffening. Here recent findings regarding angiotensin II biology and their potential relevance for vascular biology and disease are discussed
作者: 不幸的人    時(shí)間: 2025-3-23 13:12
https://doi.org/10.1007/978-94-007-6220-6long-term control mechanism of blood pressure throughout the vascular tree. Hypertension is characterized by vascular structural changes that cause increased arterial stiffness and resistance to flow. This chapter discusses the major structural changes in the vascular tree in hypertension.
作者: Entrancing    時(shí)間: 2025-3-23 13:52
https://doi.org/10.1007/3-540-37672-0angiotensin II (Ang II) and its downstream signaling cascade. The aforementioned structural changes result in a loss of the dampening function of central arteries, widening of pulse pressure, and subsequent adverse effects on the heart and end-organ systems, i.e. the brain and kidneys.
作者: heterodox    時(shí)間: 2025-3-23 19:31
Large Arteries, Microcirculation, and Mechanisms of Hypertensionlong-term control mechanism of blood pressure throughout the vascular tree. Hypertension is characterized by vascular structural changes that cause increased arterial stiffness and resistance to flow. This chapter discusses the major structural changes in the vascular tree in hypertension.
作者: nonsensical    時(shí)間: 2025-3-24 01:26
The Reality of Aging Viewed from the Arterial Wallangiotensin II (Ang II) and its downstream signaling cascade. The aforementioned structural changes result in a loss of the dampening function of central arteries, widening of pulse pressure, and subsequent adverse effects on the heart and end-organ systems, i.e. the brain and kidneys.
作者: 尊嚴(yán)    時(shí)間: 2025-3-24 05:37
Victor G. Farafonov,Vladimir B. Il’in to HFPEF in many patients. Pressure-volume loop analysis identifies three distinct hemodynamic phenotypes for HFPEF, a finding which may have significant implications for clinical trial enrollment and individual patient treatment.
作者: lesion    時(shí)間: 2025-3-24 07:29

作者: 捐助    時(shí)間: 2025-3-24 12:43
Heart Failure with Preserved Ejection Fraction to HFPEF in many patients. Pressure-volume loop analysis identifies three distinct hemodynamic phenotypes for HFPEF, a finding which may have significant implications for clinical trial enrollment and individual patient treatment.
作者: mosque    時(shí)間: 2025-3-24 17:17

作者: investigate    時(shí)間: 2025-3-24 19:55

作者: negligence    時(shí)間: 2025-3-25 01:52
pecialty. Much has been learned regarding the management of these patients in recent years and this book presents extensive data on the techniques needed to maximize outcomes.?978-1-4471-7216-1978-1-4471-5198-2
作者: PACT    時(shí)間: 2025-3-25 05:28

作者: 同音    時(shí)間: 2025-3-25 09:33

作者: 語(yǔ)源學(xué)    時(shí)間: 2025-3-25 14:39
Arterial Stiffness, Wave Reflection, Wave Amplification: Basic Concepts, Principles of Measurement af pulsatile strain on non-living elastic fibres in the highly pulsatile aorta lead to their fracture and to progressive passive aortic dilation, with transfer of stress to more rigid collagen fibres in the media (Nichols et al., McDonald’s blood flow in arteries, 6th edn. Arnold Hodder, London, 2011
作者: 顧客    時(shí)間: 2025-3-25 16:44
Direct Measurement of Local Arterial Stiffness and Pulse Pressure(DC) and cross-sectional compliance (CC) can be calculated. Distensibility is a measure of wall elasticity and the inverse of stiffness. Compliance reflects the buffering capacity of the artery. Local stiffness assessment is the only method that can assess both wall properties. In addition, if the p
作者: 骯臟    時(shí)間: 2025-3-25 23:24
Animal Models for Studies of Arterial Stiffnessed by attempting to try and interfere directly with these different factors..Hypertensive rodents were the first experimental models used and employed to test remodelling and vascular function in an environment mimicking human physiology. Then, investigations were completed with other cardiovascular
作者: 真實(shí)的你    時(shí)間: 2025-3-26 03:28

作者: 真繁榮    時(shí)間: 2025-3-26 07:09

作者: Minatory    時(shí)間: 2025-3-26 11:43
Hypoxia, Arterial Blood Pressure, and Microcirculationon to a significant reduction in capillary density and a complete blunting of the endothelial function..Taken together, these experimental and clinical results suggest that angiogenesis and antiangiogenesis could have rapid and marked effects on the peripheral resistance. Moreover, the density of th
作者: 來(lái)自于    時(shí)間: 2025-3-26 15:42

作者: Preamble    時(shí)間: 2025-3-26 17:08
Limiting values for light pollution,f pulsatile strain on non-living elastic fibres in the highly pulsatile aorta lead to their fracture and to progressive passive aortic dilation, with transfer of stress to more rigid collagen fibres in the media (Nichols et al., McDonald’s blood flow in arteries, 6th edn. Arnold Hodder, London, 2011
作者: Armada    時(shí)間: 2025-3-27 01:01
https://doi.org/10.1007/978-94-007-6220-6(DC) and cross-sectional compliance (CC) can be calculated. Distensibility is a measure of wall elasticity and the inverse of stiffness. Compliance reflects the buffering capacity of the artery. Local stiffness assessment is the only method that can assess both wall properties. In addition, if the p
作者: 1FAWN    時(shí)間: 2025-3-27 04:35

作者: 妨礙議事    時(shí)間: 2025-3-27 08:55
https://doi.org/10.1007/978-3-658-29723-7unctional elements such as the scleroprotein elastin, an essential component of elastic fibres. This deterioration is the basis of many important pathological conditions in man such as stiffening of the arterial wall..In this article we will examine how ageing can modify elastin and the role of calc
作者: Instinctive    時(shí)間: 2025-3-27 12:02
https://doi.org/10.1007/978-3-658-29723-7ffening. Available data on genome-wide association (GWA) have been initiated on PWV and have identified common genetic variation in specific loci or single-nucleotide polymorphisms (SNP) significantly associated with PWV. Telomere length at birth is strongly determined genetically and is the main de
作者: 廚師    時(shí)間: 2025-3-27 16:34

作者: doxazosin    時(shí)間: 2025-3-27 21:51
https://doi.org/10.1007/3-540-37672-0duction in distensibility of muscular arteries shown under particular conditions of stress, such as exposure to high altitude and, in general, to hypoxia. Changes in sympathetic activity may be influenced by baroreflex regulation of cardiovascular homeostasis. Reflex changes of arterial tone and mod
作者: Colonoscopy    時(shí)間: 2025-3-27 23:31

作者: 嫌惡    時(shí)間: 2025-3-28 05:01
Large Arteries, Microcirculation, and Mechanisms of Hypertensionl function of the cardiovascular system. Extrinsic neurohormonal control systems contribute to this match. In addition, blood pressure transfers energy, mechanical signals, and metabolic factors independently of these neurohormonal control mechanisms. The plasticity of vascular structure provides a
作者: 吝嗇性    時(shí)間: 2025-3-28 09:36

作者: CLAIM    時(shí)間: 2025-3-28 12:44
Ventricular-Arterial Coupling and Mechanism of Wave Reflectionsventricular and arterial elastance (analysis in pressure-volume plane) and (ii) a novel approach based on assessment of time-varying myocardial stress. The latter analysis, in the time domain, allows to directly link left ventricular myocardial stress with systemic arterial properties and with the m
作者: GRACE    時(shí)間: 2025-3-28 16:01

作者: integrated    時(shí)間: 2025-3-28 22:20
Animal Models for Studies of Arterial Stiffnesstuents and their organization to the physiology of normal and early vascular ageing..The most widely used parameter to investigate arterial stiffness in rodents is pulse wave velocity (PWV). The relation between strain and stress is also established to characterize the intrinsic behaviour of the art
作者: 值得贊賞    時(shí)間: 2025-3-29 01:48
Elastin, Calcium and Age-Related Stiffening of the Arterial Walland high (for renal and other functions) pressure components. The latter required a change from a low-pressure peristaltic to a high-pressure pulsatile, cardiac function. In this high-pressure system arteries near the heart damp the pulse pressure wave. Damping, which depends upon the visco-elastic
作者: 量被毀壞    時(shí)間: 2025-3-29 03:18
Genetic and Cellular Aspects of Arterial Stiffnessof cardiovascular complications. Cellular and molecular determinants of arterial stiffness have not been completely elucidated. Clinically, the carotid-femoral pulse wave velocity (PWV) is the gold standard parameter of arterial stiffness. A recent genome-wide scan of the Framingham Heart Study popu
作者: Meander    時(shí)間: 2025-3-29 10:29

作者: 吹牛者    時(shí)間: 2025-3-29 11:47
Pulsatile Stress, Arterial Stiffness, and Endothelial Function effect results from the shear stress-dependent release of vasoactive factors, as nitric oxide (NO) and endothelium-derived hyperpolarizing factors as epoxyeicosatrienoic acids (EETs) to modify arterial tone, geometry, and trophicity and to optimize vascular functions and cardiovascular coupling. En
作者: Defiance    時(shí)間: 2025-3-29 18:43

作者: Malleable    時(shí)間: 2025-3-29 23:44

作者: Postulate    時(shí)間: 2025-3-30 02:34

作者: 新字    時(shí)間: 2025-3-30 07:22

作者: helper-T-cells    時(shí)間: 2025-3-30 08:20
Heart Failure with Preserved Ejection Fraction). HFPEF has high rates of morbidity and mortality, yet therapy remains empirical due to incomplete understanding of disease pathophysiology. Systemic hypertension is the most prominent risk factor for HFPEF. Patients often have multiple other comorbid conditions, which may contribute to symptom bur
作者: RAGE    時(shí)間: 2025-3-30 14:44

作者: exorbitant    時(shí)間: 2025-3-30 18:49

作者: emission    時(shí)間: 2025-3-30 23:37

作者: ITCH    時(shí)間: 2025-3-31 01:49

作者: 性滿足    時(shí)間: 2025-3-31 08:25

作者: Pathogen    時(shí)間: 2025-3-31 09:43

作者: 委托    時(shí)間: 2025-3-31 14:58
Biological Definition of Light Pollutionventricular and arterial elastance (analysis in pressure-volume plane) and (ii) a novel approach based on assessment of time-varying myocardial stress. The latter analysis, in the time domain, allows to directly link left ventricular myocardial stress with systemic arterial properties and with the magnitude and timing of arterial wave reflections.
作者: 單調(diào)性    時(shí)間: 2025-3-31 19:01

作者: Hyperalgesia    時(shí)間: 2025-3-31 22:07
978-1-4471-7216-1Springer-Verlag London 2014




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