標(biāo)題: Titlebook: Biological Markers of Alzheimer’s Disease; Fran?ois Boller,Robert Katzman,Yves Christen Conference proceedings 1989 Springer-Verlag Berlin [打印本頁(yè)] 作者: misperceive 時(shí)間: 2025-3-21 17:13
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease影響因子(影響力)
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease被引頻次
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease被引頻次學(xué)科排名
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease年度引用
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease年度引用學(xué)科排名
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease讀者反饋
書(shū)目名稱(chēng)Biological Markers of Alzheimer’s Disease讀者反饋學(xué)科排名
作者: 虛情假意 時(shí)間: 2025-3-21 22:41 作者: podiatrist 時(shí)間: 2025-3-22 03:48 作者: inflate 時(shí)間: 2025-3-22 04:38 作者: 虛假 時(shí)間: 2025-3-22 09:27 作者: 迷住 時(shí)間: 2025-3-22 16:40 作者: countenance 時(shí)間: 2025-3-22 20:43
,Potential Diagnostic Markers for Alzheimer’s Disease, are claims being made that such tests will be available in the very near future. This chapter examines the potential sensitivity and specificity of some possible diagnostic markers for Alzheimer’s disease.作者: 身體萌芽 時(shí)間: 2025-3-23 00:48 作者: 使成核 時(shí)間: 2025-3-23 04:05
,The Amyloid Gene of Alzheimer’s Disease and Neuronal Dysfunction, Down’s syndrome revealed the β A4 protein to have a length of 42 to 43 residues (Kang et al. 1987). The βΑ4 amyloid arises from a much larger precursor protein, Pre A4. Elucidation of the mechanism leading to βA4 deposition is crucial in understanding AD.作者: 原來(lái) 時(shí)間: 2025-3-23 07:13
Wirkmechanismen des Mentalen Trainings are claims being made that such tests will be available in the very near future. This chapter examines the potential sensitivity and specificity of some possible diagnostic markers for Alzheimer’s disease.作者: Champion 時(shí)間: 2025-3-23 12:34
Mentales Training erlernen und anwendense who have only occasional changes of Alzheimer type. Occasional macrophages immunostained for paired helical filaments can also be observed in the cerebrospinal fluid. Therefore it is conceivable that laboratory tests for Alzheimer dementia based on cerebrospinal fluid cytology could become useful.作者: AROMA 時(shí)間: 2025-3-23 17:40
Wirksamkeit des Mentalen Trainings Down’s syndrome revealed the β A4 protein to have a length of 42 to 43 residues (Kang et al. 1987). The βΑ4 amyloid arises from a much larger precursor protein, Pre A4. Elucidation of the mechanism leading to βA4 deposition is crucial in understanding AD.作者: arthroplasty 時(shí)間: 2025-3-23 18:10
Mentales Training erlernen und anwendenr evaluation of the concentrations of both PHF antigens and beta-protein in CSF of AD patients and their correlation with the degree of dementia are needed to determine their usefulness in the diagnosis of AD.作者: 平項(xiàng)山 時(shí)間: 2025-3-24 00:13
https://doi.org/10.1007/978-3-540-76352-9he etiology of Alzheimer’s disease with respect to their interactions with APP. Approaching the study of Alzheimer’s disease from both ends should accelerate the understanding of the molecular changes underyling this disorder and, consequently, provide clues for new therapies aimed at ameliorating the effects of both sporadic and familial AD.作者: Acupressure 時(shí)間: 2025-3-24 03:56 作者: transplantation 時(shí)間: 2025-3-24 08:14 作者: ECG769 時(shí)間: 2025-3-24 13:02
,Genetic Studies of the Alzheimer’s Disease-Associated Amyloid β-Protein Precursor Gene and Familialhe etiology of Alzheimer’s disease with respect to their interactions with APP. Approaching the study of Alzheimer’s disease from both ends should accelerate the understanding of the molecular changes underyling this disorder and, consequently, provide clues for new therapies aimed at ameliorating the effects of both sporadic and familial AD.作者: 古董 時(shí)間: 2025-3-24 15:00
,Alzheimer’s Disease and Chromosome 21,have disease onset below 62 years of age, with affected individuals present in at least two generations. The inability of other groups to find linkage of FAD to chromosome 21 (Schellenberg et al. 1988; Pericak-Vance et al. 1988) is discussed and their data shown to be consistent with our finding in early onset Alzheimer’s disease.作者: Nonflammable 時(shí)間: 2025-3-24 19:40 作者: 冒號(hào) 時(shí)間: 2025-3-25 00:49 作者: Torrid 時(shí)間: 2025-3-25 04:57 作者: 干涉 時(shí)間: 2025-3-25 10:33 作者: 暴發(fā)戶 時(shí)間: 2025-3-25 12:45 作者: 鬧劇 時(shí)間: 2025-3-25 18:59 作者: 打火石 時(shí)間: 2025-3-25 20:58 作者: 粗俗人 時(shí)間: 2025-3-26 00:48
Wirkmechanismen des Mentalen Trainingsimportant for growth factor-mediated cell maintenance is the battery of protein kinases, in particular, PKC. PKC mediates many functions of nerve growth factor, the best-characterized neurotrophic factor. Both the kinases, including PKC, and the phosphorylation levels are altered in AD brains, where作者: visceral-fat 時(shí)間: 2025-3-26 07:35
Mentales Training erlernen und anwendend ultrastructural studies revealed fascicles of 10 nm filaments, but no PHF in these cells. Although the nature of the immunochemically reactive materials requires further characterization, these data do suggest that cultured cells from skin are one of the useful systems for investigating the fundam作者: LIMIT 時(shí)間: 2025-3-26 09:03 作者: headlong 時(shí)間: 2025-3-26 13:16
,Cerebrospinal Fluid Neurochemical Markers in Alzheimer’s Disease,e related to reduced HVA concentrations in subgroups of AD patients with extrapyramidal signs or myoclonus; HVA concentrations in AD without movement disorders did not differ from control values. A dopaminergic deficit in patients with extrapyramidal signs or myoclonus was further indicated by the f作者: GROSS 時(shí)間: 2025-3-26 17:07
Probes for the Molecular Components of Plaques and Tangles Point to a Broadening View of Alzheimer in the same cell. A major component of the neuritic response is the microtubule-associated protein tau, which we have shown to undergo development ally regulated splicing. One of the previously reported sequences obtained directly from paired helical filament preparations corresponds to an immature 作者: single 時(shí)間: 2025-3-26 22:22
Tau 64 and Tau 69: Two Early Biochemical Markers of Neurofibrillary Degeneration,, called tau 64 and tau 69, were also found in a biopsy, suggesting that they may be early markers of neurofibrillary degeneration. Tau 64 and tau 69 have a high MW due to their abnormal phosphorylation, as shown by the alkaline phosphatase treatment which provoked a decrease in their MW..A parallel作者: 敲竹杠 時(shí)間: 2025-3-27 03:38
A cDNA Encodes Epitopes Shared Between Microtubule-Associated Protein MAP2 and Alzheimer Neurofibrin of MAP2 immunoreactivity in neurofibrillary tangles is not directly related to an overex-pression of this protein. This also strengthens the hypothesis implicating post-translational modifications of cytoskeletal proteins as mechanisms of neurofibrillary tangles formation.作者: 啜泣 時(shí)間: 2025-3-27 06:32
Relationship Between Tau, Paired Helical Filaments, Amyloid and the Intellectual Deterioration in t. The three methods revealed a systematically different number of changes. The pathological changes best linked to dementia were those in NFT revealed by tau antiserum. A higher proportion of amyloid-rich plaques was noted in the least affected cases, suggesting that tau and PHF epitopes appeared se作者: enlist 時(shí)間: 2025-3-27 11:41
,Molecular Insights into Alzheimer’s Disease, micellar phases. Similar transformations in vivo could induce the formation of vesicles (micellar) and the fusion of membranes (hexagonal II) with important biological consequences. Computer modeling studies demonstrate the PME to have striking conformational similarities with the neurotransmitters作者: 幼稚 時(shí)間: 2025-3-27 16:24
,Altered Protein Kinase and Amyloid ?-Protein Precursor in Alzheimer’s Disease: Which Comes First?,important for growth factor-mediated cell maintenance is the battery of protein kinases, in particular, PKC. PKC mediates many functions of nerve growth factor, the best-characterized neurotrophic factor. Both the kinases, including PKC, and the phosphorylation levels are altered in AD brains, where作者: 音樂(lè)學(xué)者 時(shí)間: 2025-3-27 20:51 作者: 對(duì)待 時(shí)間: 2025-3-27 22:50 作者: 一個(gè)姐姐 時(shí)間: 2025-3-28 03:53 作者: Progesterone 時(shí)間: 2025-3-28 07:44
,Cerebrospinal Fluid-Based Laboratory Test for Alzheimer’s Disease,ntigens in cerebrospinal fluid (CSF) of patients with Alzheimer’s disease (AD) and non-AD controls. Our results showed higher levels of PHF antigens in CSF of AD patients than in CSF of non-AD controls. Although the range of values overlaps with those of non-AD controls, the higher values of PHF ant作者: Clumsy 時(shí)間: 2025-3-28 12:40 作者: 疼死我了 時(shí)間: 2025-3-28 14:53 作者: Myocyte 時(shí)間: 2025-3-28 21:33 作者: Melanocytes 時(shí)間: 2025-3-28 23:08 作者: 充足 時(shí)間: 2025-3-29 03:22 作者: Arable 時(shí)間: 2025-3-29 10:45 作者: definition 時(shí)間: 2025-3-29 12:46
,Altered Protein Kinase and Amyloid ?-Protein Precursor in Alzheimer’s Disease: Which Comes First?,dystrophic neuntes, collectively referred to as NP. NP are specific to AD, whereas NFT are found in many other neurodegenerative diseases. The purpose of this report is to present a hypothesis regarding the biochemical basis for these pathologic features of AD — neurodegeneration and NFT and NP form作者: Dorsal-Kyphosis 時(shí)間: 2025-3-29 19:06
,The Amyloid Gene of Alzheimer’s Disease and Neuronal Dysfunction,t al. 1985). The major constituent of these depositions is a protein that is now commonly termed the β A4 protein, according to its proposed secondary structure of beta pleated sheets and its relative molecular weight of 4 kDa. Protein sequencing of amyloid from brains of patients with AD as well as作者: 放逐 時(shí)間: 2025-3-29 23:29 作者: meritorious 時(shí)間: 2025-3-30 03:36 作者: CHOIR 時(shí)間: 2025-3-30 07:05 作者: Bricklayer 時(shí)間: 2025-3-30 08:15
,Calcium and the Cytoskeleton in Alzheimer’s Disease, protein tau in the paired helical filaments has led to an increase of interest in this protein. The function of tau in vivo is still poorly understood, but knowledge of its primary structure has permitted us to better define its actions in vitro. AD appears to involve tissues other than the brain i作者: Anthropoid 時(shí)間: 2025-3-30 15:01 作者: 傳染 時(shí)間: 2025-3-30 17:16 作者: 猛擊 時(shí)間: 2025-3-30 21:12 作者: oxidant 時(shí)間: 2025-3-31 02:40 作者: 袖章 時(shí)間: 2025-3-31 06:11
Research and Perspectives in Alzheimer‘s Diseasehttp://image.papertrans.cn/b/image/187394.jpg作者: 多節(jié) 時(shí)間: 2025-3-31 09:32
,Cloning of Different Amyloid Peptide Precursors from Brains of Patients with Alzheimer’s Disease,uding senile plaques and neurofibrillary tangles (Roth et al. 1966; Terry et al. 1981; Whitehouse et al. 1982; Glenner 1983). Recent studies have provided an amino acid sequence for a major polypeptide of the cerebral amyloid found in AD, the amyloid β-protein (Glenner and Wong 1984) or A4 protein (Masters et al. 1985).
