標題: Titlebook: Biological Basis of Alcohol-Induced Cancer; Vasilis Vasiliou,Samir Zakhari,Jan B. Hoek Conference proceedings 2015 Springer International [打印本頁] 作者: 掩飾 時間: 2025-3-21 20:00
書目名稱Biological Basis of Alcohol-Induced Cancer影響因子(影響力)
書目名稱Biological Basis of Alcohol-Induced Cancer影響因子(影響力)學科排名
書目名稱Biological Basis of Alcohol-Induced Cancer網(wǎng)絡公開度
書目名稱Biological Basis of Alcohol-Induced Cancer網(wǎng)絡公開度學科排名
書目名稱Biological Basis of Alcohol-Induced Cancer被引頻次
書目名稱Biological Basis of Alcohol-Induced Cancer被引頻次學科排名
書目名稱Biological Basis of Alcohol-Induced Cancer年度引用
書目名稱Biological Basis of Alcohol-Induced Cancer年度引用學科排名
書目名稱Biological Basis of Alcohol-Induced Cancer讀者反饋
書目名稱Biological Basis of Alcohol-Induced Cancer讀者反饋學科排名
作者: 鴕鳥 時間: 2025-3-21 23:05 作者: prediabetes 時間: 2025-3-22 04:04
Alcohol and Cancer: An Overview with Special Emphasis on the Role of Acetaldehyde and Cytochrome P4g DNA repair and DNA methylation, ROS results in lipid peroxidation with the generation of lipid peroxidation products such as 4-hydoxynonenal which binds to DNA-forming highly carcinogenic exocyclic DNA adducts. ROS production correlates significantly with CYP2E1 in the liver but also in the esopha作者: Gastric 時間: 2025-3-22 06:59 作者: 厭倦嗎你 時間: 2025-3-22 08:48
The Role of Iron in Alcohol-Mediated Hepatocarcinogenesis,lso strongly affected by alcohol. The few available studies suggest various molecular mechanisms that involve iron regulatory protein (IRP1), transferrin receptor 1 (TfR1), and the systemic iron master switch hepcidin, but not classical mutations of the HFE gene. Notably, reactive oxygen species (RO作者: 絕種 時間: 2025-3-22 13:49
,Synergistic Toxic Interactions Between CYP2E1, LPS/TNFα, and JNK/p38 MAP Kinase and Their Implicatisms involved including activation of the mitogen-activated kinase kinase kinase ASK-1 as a result of CYP2E1-derived reactive oxygen intermediates promoting dissociation of the inhibitory thioredoxin from ASK-1. This activation of ASK-1 is followed by activation of the mitogen-activated kinase kinase作者: 影響帶來 時間: 2025-3-22 20:23 作者: Slit-Lamp 時間: 2025-3-22 23:55
,Alcohol Consumption, Wnt/β-Catenin Signaling, and Hepatocarcinogenesis,PF?+?DEN and chow?+?DEN groups, .?
作者: 發(fā)微光 時間: 2025-3-23 03:21
Application of Mass Spectrometry-Based Metabolomics in Identification of Early Noninvasive Biomarkecould be distinguished from control animals. It also showed that a combined endogenous biomarker panel helps to identify subjects with ALD as well as those at risk of developing ALD even without any information on alcohol intake or genetics. Quantitative analysis showed that increased excretion of i作者: 諂媚于性 時間: 2025-3-23 07:59
Alcohol Metabolism by Oral Streptococci and Interaction with Human Papillomavirus Leads to Malignanspectively. . V2016 did not show a detectable aldehyde dehydrogenase. AdhE is the major alcohol dehydrogenase in .. Acetaldehyde and malondialdehyde production from permissible . species significantly increased the bacterial attachment to keratinocytes, which was associated with an enhanced expressi作者: 朝圣者 時間: 2025-3-23 09:44 作者: 重疊 時間: 2025-3-23 14:44 作者: 北京人起源 時間: 2025-3-23 20:03 作者: jungle 時間: 2025-3-23 23:20
Alcohol Consumption and Antitumor Immunity: Dynamic Changes from Activation to Accelerated Deteriorlected in the following: (1) inhibition in the expansion of memory CD8. T cells, (2) accelerated decay of Th1 cytokine-producing cells, (3) increased myeloid-derived suppressor cells, (4) compromised circulation of B cells and T cells, and (5) increased NKT cells that exhibit an IL-4 dominant cytoki作者: agenda 時間: 2025-3-24 05:42 作者: critique 時間: 2025-3-24 08:46
Kasseler Edition Soziale Arbeitssociation and variation in results across studies leave the causal role of alcohol in question.” Furthermore, breast cancer develops over decades; thus, correlations between alcohol consumption and breast cancer cannot be determined in epidemiological studies with windows of alcohol exposure that c作者: aneurysm 時間: 2025-3-24 11:12 作者: –吃 時間: 2025-3-24 16:56
Der Begriff ?Gewalt gegen Kinder“g DNA repair and DNA methylation, ROS results in lipid peroxidation with the generation of lipid peroxidation products such as 4-hydoxynonenal which binds to DNA-forming highly carcinogenic exocyclic DNA adducts. ROS production correlates significantly with CYP2E1 in the liver but also in the esopha作者: DNR215 時間: 2025-3-24 19:23 作者: 織物 時間: 2025-3-25 00:06
https://doi.org/10.1007/978-3-658-41198-5lso strongly affected by alcohol. The few available studies suggest various molecular mechanisms that involve iron regulatory protein (IRP1), transferrin receptor 1 (TfR1), and the systemic iron master switch hepcidin, but not classical mutations of the HFE gene. Notably, reactive oxygen species (RO作者: Alveolar-Bone 時間: 2025-3-25 05:38
Kasseler Edition Soziale Arbeitsms involved including activation of the mitogen-activated kinase kinase kinase ASK-1 as a result of CYP2E1-derived reactive oxygen intermediates promoting dissociation of the inhibitory thioredoxin from ASK-1. This activation of ASK-1 is followed by activation of the mitogen-activated kinase kinase作者: 火光在搖曳 時間: 2025-3-25 11:16
Kasseler Edition Soziale Arbeitverview of the current knowledge on molecular mechanisms of dysregulation of PTEN expression/activity in the liver and their relationship to development of ethanol-induced hepatocellular damage and cancer.作者: 圣人 時間: 2025-3-25 15:41
Kinderschutz und ?ffentlichkeitPF?+?DEN and chow?+?DEN groups, .?
作者: Munificent 時間: 2025-3-25 19:07
https://doi.org/10.1007/978-3-658-09862-9could be distinguished from control animals. It also showed that a combined endogenous biomarker panel helps to identify subjects with ALD as well as those at risk of developing ALD even without any information on alcohol intake or genetics. Quantitative analysis showed that increased excretion of i作者: phlegm 時間: 2025-3-25 19:57
Kasseler Edition Soziale Arbeitspectively. . V2016 did not show a detectable aldehyde dehydrogenase. AdhE is the major alcohol dehydrogenase in .. Acetaldehyde and malondialdehyde production from permissible . species significantly increased the bacterial attachment to keratinocytes, which was associated with an enhanced expressi作者: 用手捏 時間: 2025-3-26 03:39 作者: 多產(chǎn)子 時間: 2025-3-26 05:22 作者: 性上癮 時間: 2025-3-26 11:42
Sirikit Krone,Sybille St?be-Blosseyupffer cells (KCs) and secondarily, by hepatocytes via the asialoglycoprotein receptor (ASGPR). Since both KCs and hepatocytes are known to be significantly impacted by alcohol, it is hypothesized that alcohol-related effects to these liver cells will lead to altered CEA processing, including impair作者: insidious 時間: 2025-3-26 15:09 作者: jaundiced 時間: 2025-3-26 18:49 作者: Adjourn 時間: 2025-3-27 00:55
Biological Basis of Alcohol-Induced Cancer978-3-319-09614-8Series ISSN 0065-2598 Series E-ISSN 2214-8019 作者: 信徒 時間: 2025-3-27 02:27
https://doi.org/10.1007/978-3-658-10742-0 but also significant secondary effects on families and friends and a tertiary effect on society through the increased cost of health care and losses in productivity. The consumption of alcoholic beverages has long been an integral part of celebrations throughout the world. Some have even suggested 作者: hardheaded 時間: 2025-3-27 08:37 作者: 間諜活動 時間: 2025-3-27 10:44
https://doi.org/10.1007/978-3-658-10742-0iver, colorectum, and female breast, as assessed by the International Agency for Research on Cancer (IARC). Alcohol and other beverage constituents, their metabolic effects, and alcohol-related unhealthy lifestyles have been suggested as etiological factors. The aim of the present survey is to evalu作者: 可以任性 時間: 2025-3-27 14:13 作者: ALERT 時間: 2025-3-27 21:18 作者: 完整 時間: 2025-3-28 01:52 作者: Exclaim 時間: 2025-3-28 04:30 作者: stress-test 時間: 2025-3-28 07:51 作者: countenance 時間: 2025-3-28 12:21
Kasseler Edition Soziale Arbeitress. Considerable attention has been given to alcohol-elevated production of lipopolysaccharide (LPS) and TNFα and to alcohol induction of CYP2E1. These two pathways are not exclusive of each other; however, associations and interactions between them, especially in vivo, have not been extensively e作者: Indebted 時間: 2025-3-28 15:30
Kasseler Edition Soziale Arbeitactivation via PtdIns 3 kinase. Changes in PTEN expression and/or activity have been identified in a variety of chronic hepatocellular disorders including obesity, NAFLD, NASH, and alcoholism. In cancer biology, PTEN is frequently mutated or deleted in a wide variety of tumors. Mutations, decreased 作者: 序曲 時間: 2025-3-28 20:43 作者: 指耕作 時間: 2025-3-28 23:13 作者: 修飾語 時間: 2025-3-29 03:04
Kasseler Edition Soziale Arbeitfully known. This study examines alcohol metabolism in . and its interaction with HPV-16 in the malignant transformation of oral keratinocytes. The acetaldehyde-producing strain . V2016 was analyzed for . genes and activities of alcohol and aldehyde dehydrogenases. . attachment to immortalized HPV-1作者: DEFT 時間: 2025-3-29 10:28
Kinderschutzbezogene Zusammenarbeitcopic screening of 6,014 Japanese alcoholics yielded high detection rates of esophageal squamous cell carcinoma (SCC; 4.1 %) and head and neck SCC (1.0 %). The risks of upper aerodigestive tract SCC/dysplasia, especially of multiple SCC/dysplasia, were increased in a multiplicative fashion by the pr作者: Grasping 時間: 2025-3-29 15:08
Sirikit Krone,Sybille St?be-Blosseyant risk factor for these cancers. Ethanol is oxidized primarily by alcohol dehydrogenases to acetaldehyde, an agent capable of initiating tumors by forming adducts with proteins and DNA. Acetaldehyde is metabolized by ALDH2, ALDH1B1, and ALDH1A1 to acetate. Retinoic acid (RA) is required for cellul作者: 放肆的你 時間: 2025-3-29 17:16
Sirikit Krone,Sybille St?be-Blosseyajor health issues are associated with alcohol abuse, including colorectal cancer (CRC) and its metastatic growth to the liver. Although a correlation exists between alcohol use and the development of diseases, the search continues for a better understanding of specific mechanisms. Concerning the ro作者: MUMP 時間: 2025-3-29 20:32
Ethnografie als Forschungsstrategie,ol consumption affects antitumor immunity and host survival have largely been unexplored. We studied the effects of chronic alcohol consumption on the immune system and antitumor immunity in mice inoculated with B16BL6 melanoma and found that alcohol consumption activates the immune system leading t作者: affect 時間: 2025-3-30 03:33 作者: 使痛苦 時間: 2025-3-30 04:27
https://doi.org/10.1007/978-3-319-09614-8acetaldehyde; cirrhosis; hepatocarcinogenesis; liver cancer作者: 真繁榮 時間: 2025-3-30 08:54
978-3-319-37855-8Springer International Publishing Switzerland 2015作者: 好色 時間: 2025-3-30 13:11
Vasilis Vasiliou,Samir Zakhari,Jan B. HoekExplores effects of alcohol on cancer stem cells, metabolomics, biomarkers, and more.Challenges and updates the epidemiology of alcohol and cancer.Discusses how alcohol affects tissue-specific cancers作者: Lipoma 時間: 2025-3-30 17:17
Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/b/image/187310.jpg作者: medieval 時間: 2025-3-31 00:43 作者: organism 時間: 2025-3-31 03:52 作者: surrogate 時間: 2025-3-31 07:25
,Genetic–Epidemiological Evidence for the Role of Acetaldehyde in Cancers Related to Alcohol Drinkiniver, colorectum, and female breast, as assessed by the International Agency for Research on Cancer (IARC). Alcohol and other beverage constituents, their metabolic effects, and alcohol-related unhealthy lifestyles have been suggested as etiological factors. The aim of the present survey is to evalu作者: 傷心 時間: 2025-3-31 12:55 作者: 媒介 時間: 2025-3-31 15:49 作者: 曲解 時間: 2025-3-31 19:11
The Role of Iron in Alcohol-Mediated Hepatocarcinogenesis,. This iron overload is an independent factor of disease progression, hepatocellular carcinoma and it determines survival. Since simple phlebotomy does not allow the efficient removal of excess iron in ALD, a better understanding of the underlying mechanisms is urgently needed to identify novel targ作者: 違抗 時間: 2025-3-31 22:57
Alcoholic Cirrhosis and Hepatocellular Carcinoma,ic liver disease (ALD) and cirrhosis, the latter being the premier premalignant factor for HCC. The present chapter addresses key issues including the epidemiology of alcohol-associated HCC, and its link to other coexisting non-alcoholic liver diseases, and additional host and environmental risk fac作者: 大吃大喝 時間: 2025-4-1 02:13 作者: 使更活躍 時間: 2025-4-1 07:46
,Synergistic Toxic Interactions Between CYP2E1, LPS/TNFα, and JNK/p38 MAP Kinase and Their Implicatiress. Considerable attention has been given to alcohol-elevated production of lipopolysaccharide (LPS) and TNFα and to alcohol induction of CYP2E1. These two pathways are not exclusive of each other; however, associations and interactions between them, especially in vivo, have not been extensively e作者: Brocas-Area 時間: 2025-4-1 11:44 作者: acclimate 時間: 2025-4-1 14:19
,Alcohol Consumption, Wnt/β-Catenin Signaling, and Hepatocarcinogenesis,sted that ethanol (EtOH) metabolism may enhance tumor progression by increasing hepatocyte proliferation. To test this hypothesis, ethanol (EtOH) feeding of male mice began 7 weeks post-injection of the chemical carcinogen diethylnitrosamine (DEN), and continued for 16 weeks, with a final EtOH conce
