標題: Titlebook: Biochemistry of Hypertrophy and Heart Failure; Lorrie A. Kirshenbaum,Ian M. C. Dixon,Pawan K. Sin Book 2003 Springer Science+Business Medi [打印本頁] 作者: Holter-monitor 時間: 2025-3-21 19:30
書目名稱Biochemistry of Hypertrophy and Heart Failure影響因子(影響力)
書目名稱Biochemistry of Hypertrophy and Heart Failure影響因子(影響力)學科排名
書目名稱Biochemistry of Hypertrophy and Heart Failure網(wǎng)絡公開度
書目名稱Biochemistry of Hypertrophy and Heart Failure網(wǎng)絡公開度學科排名
書目名稱Biochemistry of Hypertrophy and Heart Failure被引頻次
書目名稱Biochemistry of Hypertrophy and Heart Failure被引頻次學科排名
書目名稱Biochemistry of Hypertrophy and Heart Failure年度引用
書目名稱Biochemistry of Hypertrophy and Heart Failure年度引用學科排名
書目名稱Biochemistry of Hypertrophy and Heart Failure讀者反饋
書目名稱Biochemistry of Hypertrophy and Heart Failure讀者反饋學科排名
作者: 泰然自若 時間: 2025-3-21 23:18
Lysophospholipids do not directly modulate NA+-H+ exchange,phosphatidylinositol (LPI) and lysoplasmenylcholine (LP.C) also did not alter Na.-H. exchange at all concentrations and reaction times examined. We conclude that any stimulatory effects of lysolipids on Na.-H. exchange do not occur through a direct action on the exchanger or its membrane lipid envir作者: Serenity 時間: 2025-3-22 03:56
Antioxidant enzyme gene expression in congestive heart failure following myocardial infarction, remained unchanged at all time points. The mRNA levels for catalase remained unchanged at 1 and 4 weeks PMI and were significantly reduced by about 44% at 16 weeks PMI as compared to the controls. The protein levels for MnSOD, CuZnSOD, GSHPx at 1 and 16 weeks remained unchanged in treated and untre作者: 講個故事逗他 時間: 2025-3-22 06:51 作者: 銼屑 時間: 2025-3-22 08:43 作者: GOUGE 時間: 2025-3-22 16:21
Analysis of postextrasystolic relaxation response in the human heart,uding patients and controls, correlation analysis between (EDVI, ESVI, and EF) and PRR yielded the following: (a) EDVI vs. PRR: R = 0.273, p = 0.036; (b) ESVI vs. PRR: R = 0.446, p < 0.001; and (c) EF vs. PRR: R = -0.520, p < 0.001. Thus, normal or non-failing human hearts showed a decline of -dP/dt作者: COWER 時間: 2025-3-22 19:00
Cardiac adaptation to endurance exercise in rats,myocardial stiffness (22 ± 1.1 to 19.1 ± 0.3) and reduced purine efflux during pacing-induced workload increases. .P-NMR spectroscopy in isolated hearts from trained rats showed decreased PCr and PCr/ATP ratios with increased creatine, AMP and ADP concentrations. Thus, this endurance exercise protoc作者: DOTE 時間: 2025-3-23 00:08 作者: 社團 時間: 2025-3-23 04:58 作者: 公理 時間: 2025-3-23 06:01
Calpain-1-dependent degradation of troponin I mutants found in familial hypertrophic cardiomyopathyh the use of Western immunoblotting and densitometry. The results indicated that both the wild-type and the mutant cTnI molecules were susceptible to Calpain-1. However, the degradation of the cTnI molecules in the troponin complex was less intense than that of the non-complexed forms. Moreover, pho作者: AGOG 時間: 2025-3-23 10:58 作者: 獨裁政府 時間: 2025-3-23 14:11 作者: 地殼 時間: 2025-3-23 22:03
Losartan inhibits myosin isoform shift after myocardial infarction in rats,ease in the relative contribution of V. (p < 0.05). Untreated cMI showed 39.7% more V. and 38.2% less V. when compared with untreated uMI (p < 0.05). Losartan treatment after myocardial infarction reduced the incidence of cMI from 30.4 to 4.5% and scar size from 1.52 ± 0.07 to 0.94 ± 0.11 cm. respec作者: Meander 時間: 2025-3-23 22:25
Mechanism of cell death of rat cardiac fibroblasts induced by serum depletion,he mRNA expression of Bax and Bcl-2 was investigated by quantitative real-time PCR. Serum withdrawal led to the death of 26% of cultured isolated cardiac fibroblasts during the first 5 h. The activity of the p42/p44. as well as of Akt kinase was partially reduced. For p46/p54. and p38., elevated pho作者: innate 時間: 2025-3-24 03:47 作者: observatory 時間: 2025-3-24 07:39
Revisiting the surgical creation of volume load by aorto-caval shunt in rats,puncture site and glue seeping through the aortic hole resulting in shunt occlusion. We have modified the procedure using aortic adventitial suture instead of glue and 18G angiocatheter instead of 16G needle, which eliminated the technical difficulties from the former method. The ACS was visually co作者: llibretto 時間: 2025-3-24 14:41 作者: Muscularis 時間: 2025-3-24 15:17
Beeinflussung der Lebenslaufkosten, remained unchanged at all time points. The mRNA levels for catalase remained unchanged at 1 and 4 weeks PMI and were significantly reduced by about 44% at 16 weeks PMI as compared to the controls. The protein levels for MnSOD, CuZnSOD, GSHPx at 1 and 16 weeks remained unchanged in treated and untre作者: MELON 時間: 2025-3-24 23:01 作者: canvass 時間: 2025-3-25 02:31 作者: 供過于求 時間: 2025-3-25 07:16
Kostenverantwortung der Produktentwickler,uding patients and controls, correlation analysis between (EDVI, ESVI, and EF) and PRR yielded the following: (a) EDVI vs. PRR: R = 0.273, p = 0.036; (b) ESVI vs. PRR: R = 0.446, p < 0.001; and (c) EF vs. PRR: R = -0.520, p < 0.001. Thus, normal or non-failing human hearts showed a decline of -dP/dt作者: 合法 時間: 2025-3-25 09:19 作者: xanthelasma 時間: 2025-3-25 15:33
Beeinflussung der Selbstkosten,so caused a loss of 72 kDa MMP-2 activity in the ventricles and the perfusate. Although MMP-9 activity was not detected in the ventricles, LPS resulted in an increase in perfusate 92 kDa MMP-9 activity. The MMP inhibitors significantly improved cardiac function of LPS-treated rats (Ro 31?9790: 38 ± 作者: 隱士 時間: 2025-3-25 19:46 作者: 嘲弄 時間: 2025-3-25 19:59 作者: galley 時間: 2025-3-26 01:24 作者: 基因組 時間: 2025-3-26 06:33 作者: 官僚統(tǒng)治 時間: 2025-3-26 11:07
Kostengünstig Entwickeln und Konstruierenease in the relative contribution of V. (p < 0.05). Untreated cMI showed 39.7% more V. and 38.2% less V. when compared with untreated uMI (p < 0.05). Losartan treatment after myocardial infarction reduced the incidence of cMI from 30.4 to 4.5% and scar size from 1.52 ± 0.07 to 0.94 ± 0.11 cm. respec作者: 激怒 時間: 2025-3-26 13:09
Kostengünstig Entwickeln und Konstruierenhe mRNA expression of Bax and Bcl-2 was investigated by quantitative real-time PCR. Serum withdrawal led to the death of 26% of cultured isolated cardiac fibroblasts during the first 5 h. The activity of the p42/p44. as well as of Akt kinase was partially reduced. For p46/p54. and p38., elevated pho作者: 蚊帳 時間: 2025-3-26 18:28 作者: 返老還童 時間: 2025-3-26 23:51 作者: BUMP 時間: 2025-3-27 04:53
Book 2003ovided in this special issue can be utilized to design new treatment modalities that will reduce the incidence of cardiac failure which will improve quality of life in patients with chronic heart disease.作者: 無禮回復 時間: 2025-3-27 07:32
,Functional and structural characterization of anti-β1-adrenoceptor autoantibodies of spontaneously . The antibodies recognized an epitope of the second extracellular loop of the β.-adrenoceptor identical to that epitope identified in Chagas’ disease. In conclusion, our assumption is supported that old SHR-rat are an useful animal model for investigating the role of anti-β.-adrenoceptor anti-bodies in the induction of human cardiomyopathy.作者: 含糊 時間: 2025-3-27 12:23 作者: 喧鬧 時間: 2025-3-27 14:00 作者: Alienated 時間: 2025-3-27 20:38
Kostengünstig Entwickeln und Konstruierenphic response at two points, promoting hypertrophy at the first and limiting it at the second. Calpain appears to be the protease involved at the first point, and there may be another cysteine protease acting at the second site.作者: 出處 時間: 2025-3-27 22:05 作者: forebear 時間: 2025-3-28 02:37
Modulation of cardiac remodeling by adenosine: , and , effects,tors and beneficial actions on the cardiovascular system are reviewed. In addition new, . and . data will be presented supporting the concept that adenosine exerts actions that may ameliorate adverse cardiac remodeling.作者: 發(fā)源 時間: 2025-3-28 07:32
Differential effects of calpain inhibitors on hypertrophy of cardiomyocytes,phic response at two points, promoting hypertrophy at the first and limiting it at the second. Calpain appears to be the protease involved at the first point, and there may be another cysteine protease acting at the second site.作者: 瑣碎 時間: 2025-3-28 10:42
Protein kinase C isoform-selective signals that lead to cardiac hypertrophy and the progression of the actions of the conventional PKC isoforms, and suggest that these proteins may play a particularly significant role in pathways leading to cardiac growth and/or cardioprotection also are considered.作者: BRUNT 時間: 2025-3-28 18:12 作者: esculent 時間: 2025-3-28 19:21 作者: 哀求 時間: 2025-3-29 02:31 作者: 慢跑 時間: 2025-3-29 03:03
Beeinflussung der Lebenslaufkosten,subsequent to myocardial infarction (MI). The objective of the present study was to characterize changes in the mRNA abundance and protein levels for the enzymatic antioxidants, superoxide dismutase (SOD), glutathione peroxidase (GSHPx) and catalase during the sequelae of congestive heart failure in作者: 臭名昭著 時間: 2025-3-29 08:05 作者: Alveoli 時間: 2025-3-29 13:21 作者: 新義 時間: 2025-3-29 17:37
https://doi.org/10.1007/3-540-28557-1 hypertrophy, myocyte disarray, fibrosis, and cardiac arrhythmias that may lead to premature sudden death. Five distinct point mutations within α-tropomyosin are associated with the development of familial hypertrophic cardiomyopathy. Two of these mutations are found within a troponin T binding site作者: 條約 時間: 2025-3-29 22:42
Kostenverantwortung der Produktentwickler,sponse of diastolic function after an extrasystole is unknown. We studied 58 patients with chronic heart failure (CHF) and two control subjects to evaluate the response of relaxation following extrasystole. At cardiac catheterization, from the derivative of the left ventricular (LV) pressure, the ra作者: 彩色 時間: 2025-3-30 03:38 作者: 皺痕 時間: 2025-3-30 05:33 作者: FILTH 時間: 2025-3-30 08:55 作者: constitute 時間: 2025-3-30 12:54
Beeinflussung der Selbstkosten,milarly as known in human dilated cardiomyopathy or Chagas’ disease. The agonist-like anti-β. bodies were able to activate the β.-adrenoceptor mediated signal transduction cascade in cultured rat cardiomyocytes and induced a long-lasting stimulatory effect resulting in a harmful adrenergic overdrive作者: Magnitude 時間: 2025-3-30 20:25 作者: 花費 時間: 2025-3-30 22:19 作者: fringe 時間: 2025-3-31 03:37 作者: Hippocampus 時間: 2025-3-31 06:01 作者: LIMN 時間: 2025-3-31 09:20
Beeinflussung der Selbstkosten,produce functional improvements in contractility . and . While . experiments are often performed in rat, . manipulation of myocytes has been confined to rabbit and human cells. In the present study we make quantitative comparisons between cultured adult rat and rabbit myocytes in their responses to 作者: 易受騙 時間: 2025-3-31 15:45 作者: nephritis 時間: 2025-3-31 18:21 作者: Sinus-Node 時間: 2025-3-31 21:41 作者: 抑制 時間: 2025-4-1 05:51
