標(biāo)題: Titlebook: Basis and Treatment of Cardiac Arrhythmias; Robert E. Kass,Colleen E. Clancy Book 2006 Springer-Verlag Berlin Heidelberg 2006 cardiac arrh [打印本頁] 作者: 民俗學(xué) 時間: 2025-3-21 19:53
書目名稱Basis and Treatment of Cardiac Arrhythmias影響因子(影響力)
書目名稱Basis and Treatment of Cardiac Arrhythmias影響因子(影響力)學(xué)科排名
書目名稱Basis and Treatment of Cardiac Arrhythmias網(wǎng)絡(luò)公開度
書目名稱Basis and Treatment of Cardiac Arrhythmias網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Basis and Treatment of Cardiac Arrhythmias被引頻次
書目名稱Basis and Treatment of Cardiac Arrhythmias被引頻次學(xué)科排名
書目名稱Basis and Treatment of Cardiac Arrhythmias年度引用
書目名稱Basis and Treatment of Cardiac Arrhythmias年度引用學(xué)科排名
書目名稱Basis and Treatment of Cardiac Arrhythmias讀者反饋
書目名稱Basis and Treatment of Cardiac Arrhythmias讀者反饋學(xué)科排名
作者: 相符 時間: 2025-3-21 21:20 作者: 可能性 時間: 2025-3-22 03:36 作者: Mettle 時間: 2025-3-22 06:51
Cardiac Na+ Channels as Therapeutic Targets for Antiarrhythmic Agents,ty. Different drugs have variable affinities to VGSC isoforms, and drug efficacy is affected by implicit tissue properties such as resting potential, action potential morphology, and action potential frequency. The presence of polymorphisms and mutations in the drug target can also influence drug ou作者: 燕麥 時間: 2025-3-22 09:33 作者: Hot-Flash 時間: 2025-3-22 14:02 作者: infelicitous 時間: 2025-3-22 19:09 作者: Jingoism 時間: 2025-3-23 01:08
AKAPs as Antiarrhythmic Targets?,substrate recognition sequences. Therefore, the targeting of kinases and phosphatases to specific sites enhances the regulation of diverse signaling events. Ion channel macromolecular complexes can be formed by the association of A-kinase anchoring proteins(AKAPs) or other adaptor proteins directly 作者: Picks-Disease 時間: 2025-3-23 02:41 作者: STEER 時間: 2025-3-23 06:46
Experimental Therapy of Genetic Arrhythmias: Disease-Specific Pharmacology,anagement of patients. In the last decade, the study of genetic bases of human diseases has achieved several milestones, and it is now possible to apply the knowledge that stems from the identification of the genetic substrate of diseases to clinical practice. The first step along the process of lin作者: 新陳代謝 時間: 2025-3-23 11:50 作者: 標(biāo)準(zhǔn) 時間: 2025-3-23 15:19 作者: harmony 時間: 2025-3-23 20:14 作者: 固執(zhí)點好 時間: 2025-3-23 22:34 作者: 潰爛 時間: 2025-3-24 06:24
Higher Education Marketing in Africar the diagnosis and treatment of genetic arrhythmias that arise from defects in Na+ channels and can underlie idiopathic clinical syndromes. These defective channels suggest themselves as prime targets of disease and perhaps even mutation specific pharmacological interventions.作者: Generalize 時間: 2025-3-24 08:55
Convergence in Higher Education Governance?,ity profoundly impacts function in the brain and heart. Recent findings that CaMKII expression in the heart changes during hypertrophy, heart failure,myocardial ischemia, and infarction suggest that CaMKII may be a viable therapeutic target for patients suffering from common forms of heart disease.作者: 頭盔 時間: 2025-3-24 13:19 作者: 記憶 時間: 2025-3-24 16:21
A Role for Calcium/Calmodulin-Dependent Protein Kinase II in Cardiac Disease and Arrhythmia,ity profoundly impacts function in the brain and heart. Recent findings that CaMKII expression in the heart changes during hypertrophy, heart failure,myocardial ischemia, and infarction suggest that CaMKII may be a viable therapeutic target for patients suffering from common forms of heart disease.作者: ARC 時間: 2025-3-24 20:36 作者: 食草 時間: 2025-3-25 00:05
International and Development Educationhe numbers of hERG channels at the membrane. Some trafficking-defective mutants can be rescued by pharmacological agents or temperature. Here we review evidence for rescue of mutant hERG subunits expressed in heterologous systems and discuss the potential for therapeutic approaches to correcting . . defects associated with LQTS.作者: agitate 時間: 2025-3-25 07:06
Pacemaker Current and Automatic Rhythms: Toward a Molecular Understanding,ily, best fits these criteria. The biophysical and molecular characteristics of this current are reviewed, and progress to date in developing selective pharmacologic agents targeting . . and in using gene and cell-based therapies to modulate the current are reviewed.作者: GLARE 時間: 2025-3-25 11:12
hERG Trafficking and Pharmacological Rescue of LQTS-2 Mutant Channels,he numbers of hERG channels at the membrane. Some trafficking-defective mutants can be rescued by pharmacological agents or temperature. Here we review evidence for rescue of mutant hERG subunits expressed in heterologous systems and discuss the potential for therapeutic approaches to correcting . . defects associated with LQTS.作者: nitric-oxide 時間: 2025-3-25 13:36
Mutation-Specific Pharmacology of the Long QT Syndrome,eview, we summarize some of the key clinical and genetic characteristics of this disease and highlight novel findings about ion channel structure, function, and the causal relationship between channel dysfunction and human disease, that have come from investigations of this disorder.作者: Commodious 時間: 2025-3-25 16:12 作者: landfill 時間: 2025-3-25 22:58 作者: tendinitis 時間: 2025-3-26 00:43 作者: concentrate 時間: 2025-3-26 04:55
https://doi.org/10.1007/3-540-29715-4cardiac arrhythmias; ion channels; mechanisms of arrhythmia; molecular targets; tissue; treatment of arrh作者: Cirrhosis 時間: 2025-3-26 08:55 作者: Prostaglandins 時間: 2025-3-26 12:49 作者: Grating 時間: 2025-3-26 20:35
Handbook of Experimental Pharmacologyhttp://image.papertrans.cn/b/image/181290.jpg作者: 現(xiàn)代 時間: 2025-3-26 23:45
https://doi.org/10.1007/978-3-030-39379-3g of arrhythmias, general mechanisms of arrhythmias, mechanisms of some specific arrhythmias and nonpharmacological forms of therapy. The unravelling of arrhythmia mechanisms depends, of course, on the ability to record the electrical activity of the heart. It is therefore no surprise that following作者: Missile 時間: 2025-3-27 04:51 作者: farewell 時間: 2025-3-27 08:47
Yirakpoa Ikaba,Charles Enyindahmes, is not new. Abnormal cardiac rhythms due to digitalis or quinidine have been recognized for decades. This phenomenon, termed “proarrhythmia,” was generally viewed as a clinical curiosity, since it was thought to be rare and unpredictable. However, the past 20 years have seen the recognition tha作者: condescend 時間: 2025-3-27 11:43 作者: 結(jié)束 時間: 2025-3-27 15:28 作者: BARB 時間: 2025-3-27 21:00 作者: construct 時間: 2025-3-27 23:26 作者: Foreknowledge 時間: 2025-3-28 05:14
Convergence in Higher Education Governance?,substrate recognition sequences. Therefore, the targeting of kinases and phosphatases to specific sites enhances the regulation of diverse signaling events. Ion channel macromolecular complexes can be formed by the association of A-kinase anchoring proteins(AKAPs) or other adaptor proteins directly 作者: 招募 時間: 2025-3-28 08:41 作者: LIEN 時間: 2025-3-28 11:30 作者: 牲畜欄 時間: 2025-3-28 18:38 作者: 果核 時間: 2025-3-28 22:19
Analysis of Causal and Conditioning Factors,cterized by a distinct coved-type ST segment elevation in the right precordial leads, the syndrome is associated with a high risk for sudden cardiac death in young and otherwise healthy adults, and less frequently in infants and children. The ECG manifestations of the Brugada syndrome are often dyna作者: 隱藏 時間: 2025-3-29 00:27
https://doi.org/10.1007/978-3-319-08054-3ker implantation. Isolated cardiac conduction disease (ICCD) can affect various regions within the heart, and therefore the clinical features also vary from case to case. Typically, it is characterized by progressive alteration of cardiac conduction through the atrioventricular node, His-Purkinje sy作者: 裂口 時間: 2025-3-29 03:41
International and Development Educationt.Mutations in . cause long QT syndrome (LQTS) by disrupting . ., increasing cardiac excitability and, in some cases, triggering catastrophic torsades de pointes arrhythmias and sudden death. More than 200 putative disease-causing mutations in . have been identified in affected families to date, but作者: 厚顏無恥 時間: 2025-3-29 09:30 作者: 者變 時間: 2025-3-29 12:15 作者: Benzodiazepines 時間: 2025-3-29 19:05 作者: 王得到 時間: 2025-3-29 22:54 作者: saturated-fat 時間: 2025-3-30 00:23 作者: MEN 時間: 2025-3-30 04:22 作者: confide 時間: 2025-3-30 10:36
Experimental Therapy of Genetic Arrhythmias: Disease-Specific Pharmacology,gorithms. Finally, the most ambitious step in the study of genetic disease is to discover a novel pharmacological therapy targeted at correcting the inborn defect (locus-specific therapy) or even to “cure” the DNA abnormality by replacing the defective gene with gene therapy. At present, this curati作者: 破布 時間: 2025-3-30 15:11 作者: Extricate 時間: 2025-3-30 16:48 作者: Anterior 時間: 2025-3-31 00:32
https://doi.org/10.1007/978-3-030-39379-3r nodal re-entry and atrioventricular re-entrant tachycardia in hearts with an accessory atrioventricular connection. The components of the electrocardiogram, and of extracellular electrograms directly recorded from the heart, could only be well understood by comparing such registrations with record作者: subacute 時間: 2025-3-31 04:46
Yirakpoa Ikaba,Charles Enyindahficacy, but very frequently to minimize the likelihood of proarrhythmia. . avoiding proarrhythmia has become a key element of contemporary new antiarrhythmic drug development. Further, recognition of the magnitude of the problem has led to important advances in understanding basic mechanisms.While t作者: cumulative 時間: 2025-3-31 07:08 作者: 改良 時間: 2025-3-31 10:14
Robert Ebo Hinson,Emmanuel Mogajidrugs. Very selective agents have great potential as tools for further study of the role the Na/Ca exchanger plays in arrhythmogenesis. For therapy, they may have their specific indications, but they carry the risk of increasing Ca. load of the cell. Agentswith a broader action that includes Ca. cha
