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標題: Titlebook: Bacterial Virulence Factors and Rho GTPases; Patrice Boquet (Professor Dr.),Emmanuel Lemichez ( Book 2005 The Editor(s) (if applicable) an [打印本頁]

作者: Impacted    時間: 2025-3-21 17:57
書目名稱Bacterial Virulence Factors and Rho GTPases影響因子(影響力)




書目名稱Bacterial Virulence Factors and Rho GTPases影響因子(影響力)學科排名




書目名稱Bacterial Virulence Factors and Rho GTPases網(wǎng)絡公開度




書目名稱Bacterial Virulence Factors and Rho GTPases網(wǎng)絡公開度學科排名




書目名稱Bacterial Virulence Factors and Rho GTPases被引頻次




書目名稱Bacterial Virulence Factors and Rho GTPases被引頻次學科排名




書目名稱Bacterial Virulence Factors and Rho GTPases年度引用




書目名稱Bacterial Virulence Factors and Rho GTPases年度引用學科排名




書目名稱Bacterial Virulence Factors and Rho GTPases讀者反饋




書目名稱Bacterial Virulence Factors and Rho GTPases讀者反饋學科排名





作者: institute    時間: 2025-3-21 22:36
Current Topics in Microbiology and Immunologyhttp://image.papertrans.cn/b/image/180330.jpg
作者: 淘氣    時間: 2025-3-22 01:24
Handbuch Erwachsenenbildung/Weiterbildungence mechanisms. Small GTPases, including Rho GTPases, make particularly attractive targets for pathogens because of their central roles in modulating cellular functions such as cytoskeletal control. Such modifications of these GTPases can include direct chemical modification of the GTPase or interf
作者: habitat    時間: 2025-3-22 05:58
Bibliotheken und Erwachsenenbildung, attachment on the surface of human cells; a dialogue takes place between bacteria and infected cells. These pathogens modulate cellular functions to their advantage, leading to survival and proliferation at the cell surface. Furthermore, there is increasing evidence that a variety of extracellular
作者: LEVY    時間: 2025-3-22 10:33
,Grundbegriffe der Erz?hlanalyse,lls. Here, we focus on the functional mechanism of three of these toxins: SopE, SopE2, and SptP. All three effector proteins change the GTP/GDP loading state of RhoGTPases by transient interactions. SopE and SopE2 mimic eukaryotic G-nucleotide exchange factors and thereby activate RhoGTPase signalin
作者: DEMN    時間: 2025-3-22 15:41

作者: 察覺    時間: 2025-3-22 19:12
Essst?rungen und Leistungssportnow recognized as pivotal regulators of antigen-specific T cell activation by APCs and immunological synapse formation. This review summarizes recent advances in our understanding of how Rho GTPase-dependent pathways control T lymphocyte motility, polarization and activation.
作者: 手銬    時間: 2025-3-22 22:49
Norbert Quadflieg,Manfred Fichteren species (ROS) that are used to kill bacteria within the phagosome. This chapter describes this multicomponent NADPH oxidase system, one of the first cellular systems shown to be directly regulated by Rac GTPases. We present current models of NADPH oxidase regulation by Rac2 and describe how Rac2
作者: ANTIC    時間: 2025-3-23 02:14

作者: graphy    時間: 2025-3-23 06:37

作者: ACE-inhibitor    時間: 2025-3-23 13:26
Handbuch Essst?rungen und Adipositash a type III secretion (TTS) apparatus. One of the six effector Yops, YopT, inactivates the small GTPase RhoA by removing the geranylgeranylated C-terminal cysteine. This cleavage results in release of RhoA from the cell membrane and subsequently in blockage of stress fiber formation. Thus YopT impa
作者: famine    時間: 2025-3-23 14:05

作者: Mediocre    時間: 2025-3-23 22:04
The Immunological Synapse and Rho GTPases,now recognized as pivotal regulators of antigen-specific T cell activation by APCs and immunological synapse formation. This review summarizes recent advances in our understanding of how Rho GTPase-dependent pathways control T lymphocyte motility, polarization and activation.
作者: wangle    時間: 2025-3-24 01:47
0070-217X to ensure their replication and persistence into their hosts. The authors in this volume show a synthesis on how the various host cellular Rho GTPases activities are manipulated by bacteria to fulfil their virulence..978-3-642-06280-3978-3-540-27511-4Series ISSN 0070-217X Series E-ISSN 2196-9965
作者: 顧客    時間: 2025-3-24 02:58

作者: EVADE    時間: 2025-3-24 08:30

作者: Glucocorticoids    時間: 2025-3-24 12:39

作者: Bravura    時間: 2025-3-24 15:01
https://doi.org/10.1007/3-540-27511-8Cell-cell interactions; Escherichia coli; Pseudomonas aeruginosa; Salmonella; bacteria; bacterial pathoge
作者: 先驅(qū)    時間: 2025-3-24 20:41
978-3-642-06280-3The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
作者: Anticoagulants    時間: 2025-3-25 01:13

作者: Harridan    時間: 2025-3-25 04:57

作者: 最高點    時間: 2025-3-25 08:31

作者: Calibrate    時間: 2025-3-25 14:26

作者: PALMY    時間: 2025-3-25 18:48

作者: Meager    時間: 2025-3-25 20:47
0070-217X to ensure their replication and persistence into their hosts. The authors in this volume show a synthesis on how the various host cellular Rho GTPases activities are manipulated by bacteria to fulfil their virulence..978-3-642-06280-3978-3-540-27511-4Series ISSN 0070-217X Series E-ISSN 2196-9965
作者: 注意力集中    時間: 2025-3-26 01:29

作者: goodwill    時間: 2025-3-26 04:51
Anorexia nervosa im Kindes- und Jugendalternactivate the GTPases. Here Rhoinactivating toxins and effectors are reviewed, including the families of large clostridial cytotoxins and C3-like transferases, which inactivate Rho GTPases by glucosylation and ADP-ribosylation, respectively.
作者: Tartar    時間: 2025-3-26 09:34

作者: Legion    時間: 2025-3-26 15:11
Extracellular Bacterial Pathogens and Small GTPases of the Rho Family: An Unexpected Combination,their advantage, leading to survival and proliferation at the cell surface. Furthermore, there is increasing evidence that a variety of extracellular pathogens activate small GTPases of the Rho family during adhesion, placing these regulators at the center of the interaction between these bacteria and their infected host.
作者: 矛盾心理    時間: 2025-3-26 17:42

作者: 強制性    時間: 2025-3-26 23:40
Modulation of Rho GTPases and the Actin Cytoskeleton by YopT of , ,minal cysteine. This cleavage results in release of RhoA from the cell membrane and subsequently in blockage of stress fiber formation. Thus YopT impairs cellular functions associated with cytoskeleton rearrangements.
作者: 破裂    時間: 2025-3-27 04:14
,Grundbegriffe der Erz?hlanalyse,g pathways in infected host cells. In contrast, a domain of SptP inactivates RhoGTPases by mimicking the activity of eukaryotic GTPase-activating proteins. The .-host cell interaction provides an excellent example for the use of molecular mimicry by bacterial pathogens.
作者: 碎石    時間: 2025-3-27 08:24

作者: FRAX-tool    時間: 2025-3-27 10:04

作者: 使饑餓    時間: 2025-3-27 15:29
Triggered Phagocytosis by ,: Bacterial Molecular Mimicry of RhoGTPase Activation/Deactivation,lls. Here, we focus on the functional mechanism of three of these toxins: SopE, SopE2, and SptP. All three effector proteins change the GTP/GDP loading state of RhoGTPases by transient interactions. SopE and SopE2 mimic eukaryotic G-nucleotide exchange factors and thereby activate RhoGTPase signalin
作者: 彈藥    時間: 2025-3-27 20:45
Regulation of Phagocytosis by Rho GTPases,de, and eventually present peptides derived from particulate antigens. The phagocytic process comprises several sequential and complex events initiated by the recognition of ligands on the surface of the particles by specific receptors on the surface of the phagocytic cells. Receptor clustering at t
作者: 得罪    時間: 2025-3-28 01:18
The Immunological Synapse and Rho GTPases,now recognized as pivotal regulators of antigen-specific T cell activation by APCs and immunological synapse formation. This review summarizes recent advances in our understanding of how Rho GTPase-dependent pathways control T lymphocyte motility, polarization and activation.
作者: nephritis    時間: 2025-3-28 03:55
Rho GTPases and the Control of the Oxidative Burst in Polymorphonuclear Leukocytes,en species (ROS) that are used to kill bacteria within the phagosome. This chapter describes this multicomponent NADPH oxidase system, one of the first cellular systems shown to be directly regulated by Rac GTPases. We present current models of NADPH oxidase regulation by Rac2 and describe how Rac2
作者: 小丑    時間: 2025-3-28 07:13

作者: 難理解    時間: 2025-3-28 12:41
The Type III Cytotoxins of , and , That Modulate the Actin Cytoskeleton,tudies have addressed the molecular interactions of these toxins with host cell signaling pathways and how toxins modulate cellular physiology. Although each individual toxin has a unique mode of action, general themes have started to emerge between bacterial pathogens. During the course of an infec
作者: epinephrine    時間: 2025-3-28 15:30

作者: 打谷工具    時間: 2025-3-28 18:43
Bacterial Toxins Activating Rho GTPases,and toxins modifying cellular regulators of the actin cytoskeleton, namely the Rho GTPases. CNF1 autonomously enters the host cell cytosol, where it catalyzes the constitutive activation of Rho GTPases by deamidation. This activation is, however, attenuated because of activated Rho protein ubiquitin
作者: 好開玩笑    時間: 2025-3-29 00:43

作者: 得意人    時間: 2025-3-29 04:18

作者: 矛盾    時間: 2025-3-29 08:44





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