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標(biāo)題: Titlebook: Bacteria and Complement; Michael Loos Book 1985 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verla [打印本頁(yè)]

作者: 中間時(shí)期    時(shí)間: 2025-3-21 17:55
書(shū)目名稱Bacteria and Complement影響因子(影響力)




書(shū)目名稱Bacteria and Complement影響因子(影響力)學(xué)科排名




書(shū)目名稱Bacteria and Complement網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱Bacteria and Complement網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Bacteria and Complement被引頻次




書(shū)目名稱Bacteria and Complement被引頻次學(xué)科排名




書(shū)目名稱Bacteria and Complement年度引用




書(shū)目名稱Bacteria and Complement年度引用學(xué)科排名




書(shū)目名稱Bacteria and Complement讀者反饋




書(shū)目名稱Bacteria and Complement讀者反饋學(xué)科排名





作者: exigent    時(shí)間: 2025-3-21 21:42

作者: Prognosis    時(shí)間: 2025-3-22 01:16

作者: 長(zhǎng)處    時(shí)間: 2025-3-22 07:25

作者: chalice    時(shí)間: 2025-3-22 09:38

作者: Pcos971    時(shí)間: 2025-3-22 16:12
Mathematics and Its Applicationsclassical” pathway:. C1q, C1r, C1s, C4, C2, C3, C5, C6, C7, C8, and C9. There are three proteins of the “alternative” pathway (IUIS-WHO Nomenclature Committee 1981) B, D, and P. Finally, there are four control proteins: C1 inhibitor (Clˉ INH) and C4b binding protein (C4b-bp) for the classical pathwa
作者: vibrant    時(shí)間: 2025-3-22 19:39
Gary G. Schwartz,Leonard A. Rosenblumffect is abolished when serum has been incubated for 30 min at 56° C. Gram-positive bacteria are less sensitive than gram-negative bacteria to direct killing, although gram-positive cocci are opsonized by the action of serum mediated by antibodies and complement (. et al. 1968; . et al. 1969). Norma
作者: antenna    時(shí)間: 2025-3-22 21:38

作者: acetylcholine    時(shí)間: 2025-3-23 01:33
Development of Stability Indicating Methodst defence against invading microorganisms and in cooperation with cellular and humoral immune mechanisms. Pathogenic bacteria can be divided into two groups with regard to their fate within phagocytes: (a) extracellular bacteria which are promptly killed after phagocytosis and (b) facultative intrac
作者: Defraud    時(shí)間: 2025-3-23 08:22
https://doi.org/10.1007/978-0-387-85627-8 that fresh blood could kill certain types of bacteria (. 1985) has evolved detailed understanding of (a) the gram-negative cell wall constituents that activate complement, (b) the role of antibody in initiating or facilitating the process of complement activation, and (c) the biochemical localizati
作者: 搜尋    時(shí)間: 2025-3-23 12:58

作者: 鉆孔    時(shí)間: 2025-3-23 17:25

作者: 單純    時(shí)間: 2025-3-23 20:11

作者: 環(huán)形    時(shí)間: 2025-3-24 02:07
The Role of the Classical Pathway for the Bactericidal Effect of Normal Sera Against Gram-Negative (. and . 1976). Furthermore, it was found that most of the smooth strains of gram-negative bacteria are serum resistant; whereas, the corresponding rough forms are extremely serum sensitive (. and . 1970; . 1968; . and . 1977). These investigations provided evidence that the composition of the bacte
作者: cruise    時(shí)間: 2025-3-24 03:22

作者: 幼稚    時(shí)間: 2025-3-24 09:32

作者: PLIC    時(shí)間: 2025-3-24 13:28

作者: anatomical    時(shí)間: 2025-3-24 15:50

作者: cancer    時(shí)間: 2025-3-24 22:20
Development of Stability Indicating Methodsembranes (. and .-. 1978). There is a large body of evidence indicating that insertion of complexes into the bacterial envelope is directly responsible for initiating the sequence of events that results in the death of the target bacterial cell (. et al. 1968 b; . et al. 1979; . et al. 1982 a, b; .
作者: 善于    時(shí)間: 2025-3-24 23:59
Development of Stability Indicating Methodsk by the lytic components of complement. In general, therefore, gram-positive organisms are not directly killed by complement and are defined as serum resistant. The mechanism of serum resistance, however, differs for gram-positive and gram-negative bacteria, and the serum resistance of the gram-pos
作者: Lipoprotein    時(shí)間: 2025-3-25 06:12
,Bacteria and Complement — A Historical Review,se infectious diseases, extensive studies were untertaken to elucidate the mechanisms of protection from and resistance to infections. This early history of the investigation of immunity is characterized by the dispute between two opposing schools, the protagonists of the “cellular” and the “humoral
作者: Oration    時(shí)間: 2025-3-25 11:28
The Complement System: Activation and Control,classical” pathway:. C1q, C1r, C1s, C4, C2, C3, C5, C6, C7, C8, and C9. There are three proteins of the “alternative” pathway (IUIS-WHO Nomenclature Committee 1981) B, D, and P. Finally, there are four control proteins: C1 inhibitor (Clˉ INH) and C4b binding protein (C4b-bp) for the classical pathwa
作者: 努力趕上    時(shí)間: 2025-3-25 13:12
The Role of the Classical Pathway for the Bactericidal Effect of Normal Sera Against Gram-Negative ffect is abolished when serum has been incubated for 30 min at 56° C. Gram-positive bacteria are less sensitive than gram-negative bacteria to direct killing, although gram-positive cocci are opsonized by the action of serum mediated by antibodies and complement (. et al. 1968; . et al. 1969). Norma
作者: 都相信我的話    時(shí)間: 2025-3-25 17:57
Evidence for Direct Binding of the First Component of Complement, C1, to Outer Membrane Proteins frin . and . that LPS molecules are exclusively localized in the outer layer of the outer membrane (. and . 1975; . et al. 1975; . and . 1980). Localization of proteins in the outer membrane is also indicated by the fact that various major outer membrane proteins in association with LPS, serve as rece
作者: 完成才會(huì)征服    時(shí)間: 2025-3-25 20:19
Differences in Attachment and Phagocytosis of , Strains (S Form, Re Mutant) by Mouse Peritoneal Mact defence against invading microorganisms and in cooperation with cellular and humoral immune mechanisms. Pathogenic bacteria can be divided into two groups with regard to their fate within phagocytes: (a) extracellular bacteria which are promptly killed after phagocytosis and (b) facultative intrac
作者: Nucleate    時(shí)間: 2025-3-26 02:49
Studies on the Mechanism of Bacterial Resistance to Complement-Mediated Killing and on the Mechanis that fresh blood could kill certain types of bacteria (. 1985) has evolved detailed understanding of (a) the gram-negative cell wall constituents that activate complement, (b) the role of antibody in initiating or facilitating the process of complement activation, and (c) the biochemical localizati
作者: Brain-Imaging    時(shí)間: 2025-3-26 07:36
Effect of Lethal Doses of Complement on the Functional Integrity of Target Enterobacteria,nd the generation of macromolecular C5b-9 protein complexes. When formed on the surface of susceptible . (. et al. 1966) or other gramnegative bacteria (. et al. 1967; . and . 1969; . et al. 1982), C5b-9 complexes form remarkably stable lesions embedded in the bacterial envelope where they behave es
作者: BABY    時(shí)間: 2025-3-26 08:44
Interaction of Gram-Positive Microorganisms with Complement,e. The complex outer membrane of the gram-negative organisms, which is the focus of complement attack, does not exist for those that are gram positive. Rather, the peptidoglycan layer, which underlies the outer membrane in gramnegative organisms, is very often the outermost structure of the gram-pos
作者: LAVE    時(shí)間: 2025-3-26 15:19
0070-217X -to-date knowledge in this field allows identification of many unspecific as well as highly specific defense reactions involved in the struggle against infectious diseases. On the other hand, protective structures on the microbial cell surfaces have been adapted and improve the counterpart‘s chances
作者: 浪費(fèi)物質(zhì)    時(shí)間: 2025-3-26 17:03

作者: myelography    時(shí)間: 2025-3-27 00:40
Evidence for Direct Binding of the First Component of Complement, C1, to Outer Membrane Proteins frtion of proteins in the outer membrane is also indicated by the fact that various major outer membrane proteins in association with LPS, serve as receptors for phages (Datta et al. 1977; Mu-TOH et al. 1978; . and . 1979; . and . 1982) and colicins (. et al. 1979; . 1979).
作者: finite    時(shí)間: 2025-3-27 04:40
Mathematics and Its Applicationsy, I (C3b inactivator or C3b INA) and H (β1 or C3b INA accelerator) for the alternative pathway, and anaphylatoxin inactivator. Due to the dramatic advances in protein chemistry, these 19 distinct serum proteins have been highly purified and characterized physiochemically (Table 1).
作者: 蠟燭    時(shí)間: 2025-3-27 07:30
Development of Stability Indicating Methodsellular bacteria which are resistant to intracellular killing unless macrophages are activated. Humoral immune mechanisms (antibody, complement) deal mainly with extracellular bacteria, while cellular immune mechanisms (T cells, macrophages) deal with facultative intracellular bacteria (. 1983).
作者: anachronistic    時(shí)間: 2025-3-27 10:43
https://doi.org/10.1007/978-0-387-85627-8on and biological consequences of complement deposition and complement activation by pathogenic gram-negative bacteria. The purpose of this chapter is to provide a brief review of existing data in these areas and to outline in detail the work done in our laboratory over the last 4 years on complement activation by gramnegative bacteria.
作者: 不怕任性    時(shí)間: 2025-3-27 17:20

作者: liaison    時(shí)間: 2025-3-27 21:50
Differences in Attachment and Phagocytosis of , Strains (S Form, Re Mutant) by Mouse Peritoneal Macellular bacteria which are resistant to intracellular killing unless macrophages are activated. Humoral immune mechanisms (antibody, complement) deal mainly with extracellular bacteria, while cellular immune mechanisms (T cells, macrophages) deal with facultative intracellular bacteria (. 1983).
作者: Dysarthria    時(shí)間: 2025-3-27 22:47
Studies on the Mechanism of Bacterial Resistance to Complement-Mediated Killing and on the Mechanison and biological consequences of complement deposition and complement activation by pathogenic gram-negative bacteria. The purpose of this chapter is to provide a brief review of existing data in these areas and to outline in detail the work done in our laboratory over the last 4 years on complement activation by gramnegative bacteria.




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