標(biāo)題: Titlebook: BLyS Ligands and Receptors; Michael P. Cancro Book 2010 Humana Press 2010 Antigen.autoimmunity.diseases.pathogenesis.pathophysiology.physi [打印本頁] 作者: 板條箱 時間: 2025-3-21 16:35
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作者: 極小 時間: 2025-3-21 22:35
Systemic Immune-Based Rheumatic Diseases: Blissless States of BLySfulness,clinical trials with BLyS antagonists in SLE and RA have documented in vivo biological activities along with favorable safety profiles for these agents and have pointed to beneficial effects in subsets of patients. These features collectively point to BLyS as an attractive therapeutic target in human SIRDs.作者: engrave 時間: 2025-3-22 02:34 作者: 討好女人 時間: 2025-3-22 06:56 作者: Physiatrist 時間: 2025-3-22 11:06
Book 2010l, activation, and homeostasis. Previously, the B-cell antigen receptor (BCR) was viewed as the sole mediator of these parameters, in which BCR signals were not only dominant but were also linearly related to consequent outcomes. However, appreciating that BLyS signaling is an equal partner in estab作者: Lament 時間: 2025-3-22 16:10
Role of BAFF and APRIL in Antibody Production and Diversification,ss switching, somatic hypermutation, plasmacytoid differentitation and tolerance. The understanding of their roles in these processes will offer novel insights in the therapeutic intervention of antibody-related disorders such as immunodeficiency, autoimmunity, infection and inflammation.作者: 衰老 時間: 2025-3-22 20:29 作者: LUDE 時間: 2025-3-22 21:12 作者: INERT 時間: 2025-3-23 04:24
Mitsuharu Yoshiyama,Hidehiro Kakizakiacterised by perturbed B-cell function and behaviour, including autoimmunity, malignancy, and immunodeficiency. This chapter will discuss the role of BAFF and APRIL in normal B-cell physiology as well as the emerging evidence of their involvement in the pathogenesis of these human immunopathologies.作者: 性行為放縱者 時間: 2025-3-23 07:09
nd B cell tumors.Includes supplementary material: Discovery of the BLyS (also known as BAFF) family of ligands and receptors has yielded a paradigm shift in our view of B-lymphocyte selection, survival, activation, and homeostasis. Previously, the B-cell antigen receptor (BCR) was viewed as the sole作者: 貪婪地吃 時間: 2025-3-23 11:15 作者: gentle 時間: 2025-3-23 17:35 作者: Banquet 時間: 2025-3-23 21:27 作者: 調(diào)情 時間: 2025-3-23 23:31
BAFF Receptor Regulation of Peripheral B-Lymphocyte Survival and Development, that BR3 and BCR are functionally linked and that Bruton’s cytoplasmic tyrosine kinase (Btk)/NF-κB signaling plays an essential role in this process. Therefore, the primary objective of this article is to discuss BR3-signaling pathways, and the cooperation with BCR signals, that regulate B-cell sur作者: 凹槽 時間: 2025-3-24 04:27 作者: Salivary-Gland 時間: 2025-3-24 07:25 作者: 高歌 時間: 2025-3-24 12:23 作者: gangrene 時間: 2025-3-24 15:29
Tipping the Scales of Survival: The Role of BLyS in B-Cell Malignancies,n the growth and survival of B cells from patients with non-Hodgkin lymphoma, chronic lymphocytic leukemia, multiple myeloma, and Hodgkin lymphoma. Additionally, we will discuss the clinical relevance and regulatory control of BLyS expression in B-cell malignancies.作者: 外科醫(yī)生 時間: 2025-3-24 21:53 作者: LUT 時間: 2025-3-25 01:37
Book 2010s revealing novel translational strategies for autoimmunity, neoplasia, and transplant tolerance. This book includes 12 chapters that together yield an overview of these advances and ideas. The initial excitement generated by associations with humoral autoimmunity, coupled with profound B lineage ph作者: BURSA 時間: 2025-3-25 05:56
BLyS/BR3 Receptor Signaling in the Biology and Pathophysiology of Aggressive B-Cell Lymphomas,作者: conservative 時間: 2025-3-25 08:49 作者: prodrome 時間: 2025-3-25 13:04
Gregg D. Cappon,Donald G. Stump that BR3 and BCR are functionally linked and that Bruton’s cytoplasmic tyrosine kinase (Btk)/NF-κB signaling plays an essential role in this process. Therefore, the primary objective of this article is to discuss BR3-signaling pathways, and the cooperation with BCR signals, that regulate B-cell sur作者: 意外 時間: 2025-3-25 17:21
https://doi.org/10.1007/978-981-99-1659-7 basis for the unique dependence of B cells on survival signals delivered by BAFF. In the absence of BAFF, B-cell survival is constitutively suppressed through the cooperative actions of the TRAF2 and TRAF3 signal adapters. BAFF circumvents this activity by triggering the recruitment of TRAF3 to BAF作者: 確定無疑 時間: 2025-3-25 20:08
https://doi.org/10.1007/978-981-99-1659-7ity self-reactive B cells. Interestingly, the expression of the toll-like receptor (TLR) signalling element MyD88 is required for disease, and a reciprocal stimulatory loop exists between TACI and TLR signalling. As TACI is an essential receptor driving T-cell-independent B-cell responses, it raises作者: 注射器 時間: 2025-3-26 03:43 作者: 青石板 時間: 2025-3-26 07:57
Handbook of New Institutional Economicsn the growth and survival of B cells from patients with non-Hodgkin lymphoma, chronic lymphocytic leukemia, multiple myeloma, and Hodgkin lymphoma. Additionally, we will discuss the clinical relevance and regulatory control of BLyS expression in B-cell malignancies.作者: Herd-Immunity 時間: 2025-3-26 09:13 作者: 變化 時間: 2025-3-26 16:24
BAFF Receptor Regulation of Peripheral B-Lymphocyte Survival and Development, continually provided by either tonic or antigen-mediated BCR signals and other trophic factors. B-cell-activating factor (BAFF) has emerged as a key growth factor for B lymphocytes. Through its interaction with a TNF-R family member, BAFF-R or BR3, BAFF promotes survival of both immature and mature作者: fiscal 時間: 2025-3-26 16:48 作者: 嗎啡 時間: 2025-3-26 21:45
Role of BAFF and APRIL in Antibody Production and Diversification,innate immune cells and stromal cells and are upregulated in response to various danger signals. By bindings to their receptors and signaling for the activation of NF-κB, they regulate multiple aspects of T cell-dependent and -independent antibody responses including B-cell survival, activation, cla作者: hedonic 時間: 2025-3-27 02:51
Signal Transduction by Receptors for BAFF and APRIL,ascades responsible for B-cell homeostasis. Transmitting these stimuli to the intracellular milieu are the receptors B-cell maturation antigen (BCMA), transmembrane activator and CAML interactor (TACI), and BAFF receptor (BAFFR). While evidence for the activation of several nuclear-independent pathw作者: Pituitary-Gland 時間: 2025-3-27 08:57 作者: Conjuction 時間: 2025-3-27 10:35 作者: alliance 時間: 2025-3-27 15:45 作者: 確定無疑 時間: 2025-3-27 20:41
The Role of BAFF and APRIL in Regulating Human B-Cell Behaviour: Implications for Disease PathogeneAFF and APRIL are members of the TNF superfamily of cytokines and have been identified as critical regulators of B-cell development and differentiation. Defects in the production of BAFF and APRIL, and/or expression of their receptors, have been associated with a diverse array of human diseases char作者: Endearing 時間: 2025-3-27 23:41 作者: 植物茂盛 時間: 2025-3-28 04:12
Tipping the Scales of Survival: The Role of BLyS in B-Cell Malignancies,buting mechanistic details underlying transformation events are not yet known. It is clear, however, that dysregulation of the balance between cell proliferation and programmed cell death is a central feature. While many factors have been shown to regulate the normal balance of B-cell growth and apo作者: Enervate 時間: 2025-3-28 09:59 作者: 演講 時間: 2025-3-28 12:24 作者: 澄清 時間: 2025-3-28 17:41 作者: CHURL 時間: 2025-3-28 21:38
Gregg D. Cappon,Donald G. Stump continually provided by either tonic or antigen-mediated BCR signals and other trophic factors. B-cell-activating factor (BAFF) has emerged as a key growth factor for B lymphocytes. Through its interaction with a TNF-R family member, BAFF-R or BR3, BAFF promotes survival of both immature and mature作者: 委托 時間: 2025-3-28 23:37
https://doi.org/10.1007/978-981-99-1659-7tain self-tolerance, B cells that express antigen receptors with significant reactivity against self-antigens are prevented from receiving or responding to these survival signals. The great majority of B cells produced from bone marrow precursors fail to join the mature peripheral B-cell pool either作者: BOLT 時間: 2025-3-29 05:30
Mitsuharu Yoshiyama,Hidehiro Kakizakiinnate immune cells and stromal cells and are upregulated in response to various danger signals. By bindings to their receptors and signaling for the activation of NF-κB, they regulate multiple aspects of T cell-dependent and -independent antibody responses including B-cell survival, activation, cla作者: FICE 時間: 2025-3-29 08:34 作者: 樂意 時間: 2025-3-29 15:22
Reference work 2023Latest editionportant regulator of immunity in both mice and humans. Although much has been learned about its signaling and physiological functions, its complexity in terms of ligand binding, cell-type-specific effects, as well as potential roles in human disease dictates that much work remains to be done in this作者: 邊緣帶來墨水 時間: 2025-3-29 19:04
https://doi.org/10.1007/978-981-99-1659-7nce for understanding B-cell biology. Excessive BAFF production is associated with the development of autoimmune disorders such as lupus, a problem attributed to the inappropriate survival of self-reactive B cells. These early findings prompted the development of BAFF inhibitors, which are now in hu作者: BLINK 時間: 2025-3-29 23:21
Pharmacology of the Lower Urinary Tractcal features of systemic lupus erythematosus (SLE) and Sj?gren’s syndrome (SS). Treatment with BLyS antagonists of mice with established SLE ameliorates disease progression and enhances survival. Moreover, similar treatment of mice with inflammatory arthritis ameliorates the ongoing inflammation and作者: 討好美人 時間: 2025-3-30 01:51 作者: Injunction 時間: 2025-3-30 08:01 作者: murmur 時間: 2025-3-30 09:44
Handbook of New Institutional Economicsbuting mechanistic details underlying transformation events are not yet known. It is clear, however, that dysregulation of the balance between cell proliferation and programmed cell death is a central feature. While many factors have been shown to regulate the normal balance of B-cell growth and apo作者: CHIP 時間: 2025-3-30 12:32
https://doi.org/10.1007/978-1-60327-013-7Antigen; autoimmunity; diseases; pathogenesis; pathophysiology; physiology作者: neutral-posture 時間: 2025-3-30 18:29
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