標題: Titlebook: BCL?2 Protein Family; Essential Regulators Claudio Hetz Book 2010 The Editor(s) (if applicable) and The Author(s), under exclusive license [打印本頁] 作者: 作業(yè) 時間: 2025-3-21 19:36
書目名稱BCL?2 Protein Family影響因子(影響力)
書目名稱BCL?2 Protein Family影響因子(影響力)學科排名
書目名稱BCL?2 Protein Family網(wǎng)絡公開度
書目名稱BCL?2 Protein Family網(wǎng)絡公開度學科排名
書目名稱BCL?2 Protein Family被引頻次
書目名稱BCL?2 Protein Family被引頻次學科排名
書目名稱BCL?2 Protein Family年度引用
書目名稱BCL?2 Protein Family年度引用學科排名
書目名稱BCL?2 Protein Family讀者反饋
書目名稱BCL?2 Protein Family讀者反饋學科排名
作者: 柱廊 時間: 2025-3-21 22:27 作者: HPA533 時間: 2025-3-22 01:59 作者: AVOID 時間: 2025-3-22 07:34
http://image.papertrans.cn/b/image/180078.jpg作者: 挑剔為人 時間: 2025-3-22 09:40
,Later Nyāya Logic: Computational Aspects,ells have evolved a highly integrated network of control mechanisms, including positive and negative feedback loops, to safeguard homeostasis in face of a wide range of stimuli. Such control mechanisms ultimately orchestrate cell death, division and repair in a manner concordant with cellular energy作者: 極端的正確性 時間: 2025-3-22 16:42
Logical Proofs in the , of Somānandaeins is a group of evolutionarily conserved regulators of cell death that comprises both anti- and pro-apoptotic members, that operate at the mitochondrial membrane to control caspase activation. Different BCL-2-related proteins are also located in the endoplasmic reticulum (ER), where important rol作者: cleaver 時間: 2025-3-22 20:20 作者: 獨裁政府 時間: 2025-3-23 00:23
General Introduction to Buddhist Logic Stringent regulation of redox and calcium homeostasis is paramount, failure of which leads accumulation of unfolded and aggregating proteins resulting in a condition known as ER stress. Eukaryotic cells deal with ER stress by eliciting the unfolded protein response (UPR). This pathway splits into t作者: demote 時間: 2025-3-23 04:09 作者: 羅盤 時間: 2025-3-23 09:10
Grace A. Mucci,Lilibeth R. Tornoulminates in the release of cytochrome c from the organelle in the cytoplasm, where it is required for the activation of effector caspases. The complete release of cytochrome c is the result of the combined action of proapoptotic BCL-2 family members and of changes in the complex morphology and ultr作者: consolidate 時間: 2025-3-23 09:47 作者: Vertebra 時間: 2025-3-23 17:13
https://doi.org/10.1007/978-1-4899-7584-3ll. Mitochondrial structure in the cell is predominantly regulated by cycles of fusion and fission. These two processes are tightly regulated and under physiological conditions, mitochondrial fusion is evenly counterbalanced by fission. During apoptosis, mitochondria undergo extensive fragmentation,作者: Sarcoma 時間: 2025-3-23 22:01
Book 2010 apoptosis, physiology and disease. An important focus of this volume is considering the potential TH ERA PEUT IC benefits of targeting apoptosis pathways in the context of human disease. Readers interested in understanding how a cell handles stress and the consequences of dysregulation of this proc作者: 內(nèi)閣 時間: 2025-3-24 01:55 作者: 易改變 時間: 2025-3-24 02:52 作者: 內(nèi)閣 時間: 2025-3-24 09:59
BH3-Only Proteins and Their Effects on Cancer,ssary for cell killing. Failure to properly sense death cues and relay the death signal can have a major impact on cancer. This chapter will discuss our current models of how BH3-only proteins function as well as their impact on carcinogenesis and cancer treatment.作者: 支架 時間: 2025-3-24 11:14 作者: reaching 時間: 2025-3-24 15:33
The Interplay between BCL-2 Family Proteins and Mitochondrial Morphology in the Regulation of Apoptastructure of the organelle, controlled by the balance between fusion and fission processes. Here we discuss recent findings pointing to a role for changes in mitochondrial morphology during apoptosis and how these might be regulated by members of the BCL-2 family.作者: 輪流 時間: 2025-3-24 20:06 作者: 禁令 時間: 2025-3-25 00:02
Book 2010ess for human disease will find this book very valuable. It attempts to describe a fascinating area of research where physiology and biomedicine converge at different levels, revealing a trip from the molecular regulation of apoptosis to the impact of this process to the physiology of a whole organism.作者: 代理人 時間: 2025-3-25 05:52
Grace A. Mucci,Lilibeth R. Tornoastructure of the organelle, controlled by the balance between fusion and fission processes. Here we discuss recent findings pointing to a role for changes in mitochondrial morphology during apoptosis and how these might be regulated by members of the BCL-2 family.作者: badinage 時間: 2025-3-25 08:09
https://doi.org/10.1007/978-1-4899-7584-3 which precedes caspase activation, whereas inhibition of the mitochondrial fission machinery blocks or delays cell death. Aberrant mitochondrial fusion and fission have also emerged as important mechanisms in the development of disease.作者: 有節(jié)制 時間: 2025-3-25 12:55 作者: Nomadic 時間: 2025-3-25 16:54 作者: 擴大 時間: 2025-3-25 21:48
Alternative Functions of the BCL-2 Protein Family at the Endoplasmic Reticulum,in the control of diverse cellular processes including calcium homeostasis, autophagy and ER morphogenesis. Here we describe the emerging concept that BCL-2 family members are important regulators of essential cellular processes beyond apoptosis.作者: 樸素 時間: 2025-3-26 03:26 作者: AMEND 時間: 2025-3-26 06:06
Noncanonical Functions of BCL-2 Proteins in the Nervous System,l of antagonism between BCL-2 family proteins. Emphasis is logically placed on those phenotypes that support the model. However, this working model of BCL-2 family interactions has become so engrained that alternative, potentially valid interpretations are sometimes dismissed. Therefore, it is usefu作者: 使成波狀 時間: 2025-3-26 10:21 作者: 粗魯性質(zhì) 時間: 2025-3-26 16:20
,Later Nyāya Logic: Computational Aspects,ghly selective network of functional interactions that ultimately governs the permeabilization of the mitochondrial outer membrane and subsequent release of apoptogenic factors such as cytochrome ... The advent of loss- and gain-of-function genetic models for the various BCL-2 family proteins has no作者: Amnesty 時間: 2025-3-26 20:52 作者: GOAT 時間: 2025-3-26 21:36 作者: 淺灘 時間: 2025-3-27 02:46
Gastrointestinal and Hepatic Systemsl of antagonism between BCL-2 family proteins. Emphasis is logically placed on those phenotypes that support the model. However, this working model of BCL-2 family interactions has become so engrained that alternative, potentially valid interpretations are sometimes dismissed. Therefore, it is usefu作者: 怎樣才咆哮 時間: 2025-3-27 08:47 作者: Factual 時間: 2025-3-27 10:00 作者: blight 時間: 2025-3-27 15:52 作者: 古老 時間: 2025-3-27 20:34 作者: CONE 時間: 2025-3-27 22:56
Targeting Survival Pathways in Lymphoma,ath have been distinguished, including: apoptosis, autophagy, and necrosis. Increasing evidence has begun to support a role of Bcl-2-family members in the cellular pathways involved in each of these processes. The induction of apoptosis in different types of tissue and in response to various stresso作者: 移動 時間: 2025-3-28 05:42 作者: SMART 時間: 2025-3-28 07:53 作者: adipose-tissue 時間: 2025-3-28 13:06
Mitochondria on Guard: Role of Mitochondrial Fusion and Fission in the Regulation of Apoptosis,ll. Mitochondrial structure in the cell is predominantly regulated by cycles of fusion and fission. These two processes are tightly regulated and under physiological conditions, mitochondrial fusion is evenly counterbalanced by fission. During apoptosis, mitochondria undergo extensive fragmentation,作者: 彩色 時間: 2025-3-28 18:21
10樓作者: labyrinth 時間: 2025-3-28 22:32
10樓作者: 吝嗇性 時間: 2025-3-29 01:02
10樓作者: Indelible 時間: 2025-3-29 05:40
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