標(biāo)題: Titlebook: Autoimmunity; Andrzej Górski,Hubert Krotkiewski,Micha? Zimecki Book 2001 Springer Science+Business Media New York 2001 Arthritis.Diabetes. [打印本頁] 作者: VIRAL 時(shí)間: 2025-3-21 18:14
書目名稱Autoimmunity影響因子(影響力)
書目名稱Autoimmunity影響因子(影響力)學(xué)科排名
書目名稱Autoimmunity網(wǎng)絡(luò)公開度
書目名稱Autoimmunity網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Autoimmunity被引頻次
書目名稱Autoimmunity被引頻次學(xué)科排名
書目名稱Autoimmunity年度引用
書目名稱Autoimmunity年度引用學(xué)科排名
書目名稱Autoimmunity讀者反饋
書目名稱Autoimmunity讀者反饋學(xué)科排名
作者: 畫布 時(shí)間: 2025-3-21 21:42 作者: 愚笨 時(shí)間: 2025-3-22 04:08
Complement Regulatory Proteins and Autoimmunity,indings suggest that complement regulatory proteins have dual roles at inflammatory sites: enhancement of cellular resistance to complement attack and acceleration of theclearance of cells injurious to the organism due to complement-mediated mechanisms. To assist the former function, a therapeutic a作者: 圓桶 時(shí)間: 2025-3-22 08:05 作者: 大門在匯總 時(shí)間: 2025-3-22 09:41
Evidence that Fas and Fasl Contribute to the Pathogenesis of Experimental Autoimmune Encephalomyelirse can be acute or chronic relapsing, characterized by a rapid onset of hind limb weakness that commonly progresses to paralysis, followed by spontaneous remission starting 7–10 days after the initial appearance of symptoms. EAE can also be induced passively by the adoptive transfer of . activated 作者: Annotate 時(shí)間: 2025-3-22 16:04 作者: Exposition 時(shí)間: 2025-3-22 20:09
Is Lack of Peripheral Tolerance Induction a Cause for Diabetes in the Non-Obese Diabetic Mouse?,ing naive T cells, the dendritic cells are also involved in generating central and peripheral tolerance to self molecules. In this process DC2 cells appear to be more important for the development of peripheral tolerance than DC1 cells. Besides abnormalities in the development of bone marrow-derived作者: Connotation 時(shí)間: 2025-3-22 23:26 作者: 反對(duì) 時(shí)間: 2025-3-23 05:15
The Role of T Cells in Rheumatoid Arthritis,ed CD4.CD28. T cells is fundamentally changed and is shifted towards tissue-injurious capabilities. CD4.CD28. T cells are particularly important in patients with extra-articular manifestations of RA, where they may have a direct role in vascular injury. Understanding the mechanisms underlying the lo作者: Armory 時(shí)間: 2025-3-23 06:00 作者: Meditate 時(shí)間: 2025-3-23 13:36
https://doi.org/10.1007/978-3-658-07889-8s increases the susceptibility of the affected cells to apoptosis induced by tumor necrosis factor α (TNF-α). The proteasome dysfunction is both tissue and developmental stagespecific and likely contributes to disease pathogenesis and tissue targeting.作者: Osteons 時(shí)間: 2025-3-23 16:11
,Factor de éxito Estructura de Conducción,indings suggest that complement regulatory proteins have dual roles at inflammatory sites: enhancement of cellular resistance to complement attack and acceleration of theclearance of cells injurious to the organism due to complement-mediated mechanisms. To assist the former function, a therapeutic a作者: 是比賽 時(shí)間: 2025-3-23 21:36 作者: excursion 時(shí)間: 2025-3-24 01:14 作者: fulmination 時(shí)間: 2025-3-24 04:16 作者: 現(xiàn)實(shí) 時(shí)間: 2025-3-24 08:28
,Teilstudie I: Machen Kinder unglücklich?,ing naive T cells, the dendritic cells are also involved in generating central and peripheral tolerance to self molecules. In this process DC2 cells appear to be more important for the development of peripheral tolerance than DC1 cells. Besides abnormalities in the development of bone marrow-derived作者: 形狀 時(shí)間: 2025-3-24 13:24 作者: 暗諷 時(shí)間: 2025-3-24 17:46
Conclusions and Future Directions,ed CD4.CD28. T cells is fundamentally changed and is shifted towards tissue-injurious capabilities. CD4.CD28. T cells are particularly important in patients with extra-articular manifestations of RA, where they may have a direct role in vascular injury. Understanding the mechanisms underlying the lo作者: aesthetician 時(shí)間: 2025-3-24 21:54 作者: 后退 時(shí)間: 2025-3-24 23:18
The Paradigms of Causality and Treatment for Autoimmune Disease, hope to learn the most efficient way to manipulate the disease process. How do we identify a cause when we see one? Quite simply, a single cause is that which is both necessary and sufficient to produce the effect. Here, I explore the general paradigm of autoimmune causality, using multiple sclerosis as a specific example.作者: Salivary-Gland 時(shí)間: 2025-3-25 05:58
Gestión de la Cooperación en la Prácticanown about T cell activation and function .. In the last decade, a number of models have been developed which allow the tracking of Ag-activated T cells . and these studies have been instrumental in advancing the field of T cell biology. In particular, a new and emerging paradigm of T cell immunity is evolving.作者: adj憂郁的 時(shí)間: 2025-3-25 09:56 作者: nominal 時(shí)間: 2025-3-25 14:14 作者: fulcrum 時(shí)間: 2025-3-25 19:42 作者: 血友病 時(shí)間: 2025-3-25 23:08
The Physiological Role of Regulatory T Cells in the Prevention of Autoimmunity: Generation, Specifiy or suppressor T cells. This article examines data from one model of rodent autoimmunity, where autoimmune pathology develops following thymectomy and irradiation of normal laboratory rats, which characterize the development and function of these so-called regulatory T cells.作者: Inculcate 時(shí)間: 2025-3-26 00:24
,Factor de éxito Aprendizaje e innovación,y or suppressor T cells. This article examines data from one model of rodent autoimmunity, where autoimmune pathology develops following thymectomy and irradiation of normal laboratory rats, which characterize the development and function of these so-called regulatory T cells.作者: Connotation 時(shí)間: 2025-3-26 07:06
Overview: 978-94-010-3877-5978-94-010-0981-2作者: 哪有黃油 時(shí)間: 2025-3-26 12:22 作者: 護(hù)航艦 時(shí)間: 2025-3-26 14:40
978-94-010-3877-5Springer Science+Business Media New York 2001作者: 膽小鬼 時(shí)間: 2025-3-26 20:29 作者: Fortify 時(shí)間: 2025-3-26 23:30 作者: Colonnade 時(shí)間: 2025-3-27 01:32
Gestión de la Cooperación en la Prácticaments of activation. During this time, a compendium of information has been collected and has proven to be invaluable. Paradoxically, very little is known about T cell activation and function .. In the last decade, a number of models have been developed which allow the tracking of Ag-activated T cel作者: 為敵 時(shí)間: 2025-3-27 07:51 作者: 舞蹈編排 時(shí)間: 2025-3-27 12:18 作者: 極為憤怒 時(shí)間: 2025-3-27 15:43 作者: 衰弱的心 時(shí)間: 2025-3-27 19:49 作者: 果仁 時(shí)間: 2025-3-28 01:27
,Factor de éxito Estructura de Conducción,ssociated with viral infection. The majority of experimental models rely heavily on the autoimmune components since similar diseases can be induced following immunization with various myelin antigens. A very attractive alternative model is the Theiler’s murine encephalomyelitis virus-induced demyeli作者: pancreas 時(shí)間: 2025-3-28 02:32 作者: amphibian 時(shí)間: 2025-3-28 07:36 作者: 初學(xué)者 時(shí)間: 2025-3-28 13:02
https://doi.org/10.1007/978-3-658-41224-1mptoms of dry mouth and eyes due to insufficient secretion. Although it has been assumed that a combination of immunologic, genetic and environmental factors may play a key role in the development of autoimmune lesions in the salivary and lacrimal glands, little is known about the disease pathogenes作者: Cardiac-Output 時(shí)間: 2025-3-28 17:33 作者: exacerbate 時(shí)間: 2025-3-28 21:52
https://doi.org/10.1007/978-3-319-62212-5. In patients with RA, proinflammatory cytokines secreted by synovial cells provoke local inflammation in the joints and, simultaneously, initiate a systemic acute phase response. Thus, profound changes of the neuro-endocrine-immune axis could take place in the patients. Defects in the hypothalamus-作者: 有花 時(shí)間: 2025-3-29 00:27 作者: panorama 時(shí)間: 2025-3-29 05:44
The Paradigms of Causality and Treatment for Autoimmune Disease,ll not cure the patient. This does not mean that effective treatments cannot emerge from faulty diagnoses. In truth, much of our therapeutic ensemble is composed of drugs developed as a result of chance observation, random screening, intuition or pre-scientific tradition. Nevertheless, the way to ef作者: 寄生蟲 時(shí)間: 2025-3-29 11:01 作者: 藕床生厭倦 時(shí)間: 2025-3-29 15:18 作者: 修飾語 時(shí)間: 2025-3-29 16:41 作者: Magnificent 時(shí)間: 2025-3-29 20:17
,A Role for NF-κB and the Proteasome in Autoimmunity,th a pre-diabetic state characterized by the production of autoantibodies specific for proteins expressed by pancreatic β cells. The non-obese diabetic (NOD) mouse is a spontaneous model of type 1 diabetes with a strong genetic component that maps to the major histocompatibility complex (MHC) region作者: 有罪 時(shí)間: 2025-3-30 03:10 作者: 拖網(wǎng) 時(shí)間: 2025-3-30 07:21
,Pathogenic Immunity in Theiler’s Virus-Induced Demyelinating Disease: A Viral Model for Multiple Scssociated with viral infection. The majority of experimental models rely heavily on the autoimmune components since similar diseases can be induced following immunization with various myelin antigens. A very attractive alternative model is the Theiler’s murine encephalomyelitis virus-induced demyeli作者: 提名的名單 時(shí)間: 2025-3-30 08:59 作者: gospel 時(shí)間: 2025-3-30 12:44
The Role of Cytokines in Experimental Autoimmune Encephalomyelitis,ole in defining the Th1 or Th2 nature of the autoantigen directed immune response, and in propagating and regulating inflammation within the central nervous system. In this review we summarize some of the recent developments in the field of cytokine research that relate to this model of human diseas
