標(biāo)題: Titlebook: Autoantibodies in Neurological Diseases; Angela Vincent,Gianvito Martino Book 2002 Springer-Verlag Italia 2002 Antigen.Nervous System.Neur [打印本頁] 作者: onychomycosis 時(shí)間: 2025-3-21 18:38
書目名稱Autoantibodies in Neurological Diseases影響因子(影響力)
書目名稱Autoantibodies in Neurological Diseases影響因子(影響力)學(xué)科排名
書目名稱Autoantibodies in Neurological Diseases網(wǎng)絡(luò)公開度
書目名稱Autoantibodies in Neurological Diseases網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Autoantibodies in Neurological Diseases被引頻次
書目名稱Autoantibodies in Neurological Diseases被引頻次學(xué)科排名
書目名稱Autoantibodies in Neurological Diseases年度引用
書目名稱Autoantibodies in Neurological Diseases年度引用學(xué)科排名
書目名稱Autoantibodies in Neurological Diseases讀者反饋
書目名稱Autoantibodies in Neurological Diseases讀者反饋學(xué)科排名
作者: 捐助 時(shí)間: 2025-3-21 22:51
Clinical Presentation and Mechanisms of Immune-Mediated Cerebellar Ataxia,e also been described in patients with non-paraneoplastic cerebellar ataxia (N-PCA), suggesting that the immune system may be involved in certain cases of sporadic cerebellar ataxia. In this review, the clinical presentation of the different subtypes of potentially immune-mediated PCA and N-PCA will作者: 吞沒 時(shí)間: 2025-3-22 03:30 作者: SPASM 時(shí)間: 2025-3-22 05:52 作者: larder 時(shí)間: 2025-3-22 09:56
Overview: This book provides an updated review of immune-mediated diseases from a clinical and neuroimmunological point of view.It will be of great help to neuroimmunologists and to clinicians, who will gain a 978-88-470-2163-1978-88-470-2097-9作者: FLING 時(shí)間: 2025-3-22 13:52 作者: BARGE 時(shí)間: 2025-3-22 19:37
Topics in Neurosciencehttp://image.papertrans.cn/b/image/166100.jpg作者: 褻瀆 時(shí)間: 2025-3-23 00:47
Der Weg zum Semantischen ObjektmodellPregnancy is an immunological balancing act in which the mother’s immune system has to remain tolerant of paternal major histocompatibility (MHC) antigens while maintaining normal immune competence for defence against microorganisms.作者: defile 時(shí)間: 2025-3-23 05:27 作者: Acupressure 時(shí)間: 2025-3-23 09:33 作者: 厭惡 時(shí)間: 2025-3-23 12:33 作者: 征稅 時(shí)間: 2025-3-23 17:28 作者: 冷漠 時(shí)間: 2025-3-23 18:08
Immunological Mechanisms in the Lambert-Eaton Myasthenic Syndrome,The Lambert-Eaton myasthenic syndrome (LEMS) is an antibody-mediated disorder of neuromuscular and autonomic synaptic transmission. It is paraneoplastic in about 50% of cases, generally being associated with small-cell lung carcinoma (SCLC). In non-cancer cases, it can be associated with other autoimmune diseases [.].作者: Between 時(shí)間: 2025-3-24 00:44 作者: 變白 時(shí)間: 2025-3-24 03:15
Paraneoplastic Neuropathies: a Neuropathological Overview,Neuropathies in patients with cancer are due to the direct effect of nerve compression or infiltration by tumour, or may occur as an indirect consequence of metabolic and nutritional disorders, infections, vascular disorders, or as a result of neurotoxic chemotherapy such as with vincristine, cisplatin and taxols [., .].作者: 移動(dòng) 時(shí)間: 2025-3-24 08:48 作者: Muffle 時(shí)間: 2025-3-24 11:34 作者: 高深莫測 時(shí)間: 2025-3-24 18:27 作者: 易達(dá)到 時(shí)間: 2025-3-24 22:05 作者: 勤勞 時(shí)間: 2025-3-25 01:45 作者: conscribe 時(shí)間: 2025-3-25 03:53 作者: 沖擊力 時(shí)間: 2025-3-25 11:21 作者: 倔強(qiáng)不能 時(shí)間: 2025-3-25 11:43
https://doi.org/10.1007/978-3-642-55921-1with neuroblastoma [.] and other neural-crest-derived tumours has been described in some children (paraneoplastic OMS, OMS-NB.), whilst several other potential causal relationships have also been found (Table 1), but little progress has been made in understanding the etiology and treatment of this s作者: Headstrong 時(shí)間: 2025-3-25 19:08
Wissensmanagement im Projektgesch?ftune response. This weak response was explained by: (1) the presence of a blood-brain barrier (BBB); (2) constitutive lack or low expression of proteins of the immune system, such as major histocompatibility complex (MHC) class I and II, co-stimulatory and accessory molecules, on cells of the CNS (gU作者: sparse 時(shí)間: 2025-3-25 23:17
https://doi.org/10.1007/978-3-662-07470-1a, sustained inflammation sometimes compromises vessel integrity. The central nervous system (CNS) is the target organ of isolated, or primary, vasculitis (primary angiitis of the CNS, or PACNS). Far more frequently, the involvement of the CNS is part of a more complex scenario involving other organ作者: Outwit 時(shí)間: 2025-3-26 01:44
https://doi.org/10.1007/978-88-470-2097-9Antigen; Nervous System; Neuromuscular disorders; antibodies; autoimmunity; inflammation; paraneoplastic s作者: 鉆孔 時(shí)間: 2025-3-26 06:02
978-88-470-2163-1Springer-Verlag Italia 2002作者: Acetaldehyde 時(shí)間: 2025-3-26 08:52
Introduction - Approaches to Understanding Immune-Mediated Neurological Disorders: Measuring and Ev junction disorders, immune-mediated demyelinating disorders, and also some form of epilepsy, stroke, Alzheimer’s and Parkinsons’ diseases, and prion diseases. It is, therefore, particularly important to distinguish those conditions which are caused by dysfunction of the immune system, from those in作者: 運(yùn)動(dòng)吧 時(shí)間: 2025-3-26 14:26 作者: Enteropathic 時(shí)間: 2025-3-26 19:54 作者: Diatribe 時(shí)間: 2025-3-26 22:53
Ontogeny of Skeletal Muscle Cells,e early stages.Skeletal myogenesis begins shortly after gastrulation but persists, at least in mammals, until the end of postnatal growth, and the potential for myogenesis continues for the entire life span of the animal [.]. Local signalling commits mesodermal cells to a myogenic fate, and shortly 作者: 財(cái)主 時(shí)間: 2025-3-27 01:39 作者: hysterectomy 時(shí)間: 2025-3-27 09:11 作者: Diluge 時(shí)間: 2025-3-27 11:58
Stiff-Man Syndrome: Pathogenetic, Nosological and Therapeutic Considerations, muscle spasms. It was first described by Moersch and Woltmann in 1956 [.]. A set of diagnostic criteria was proposed by Gordon et al. [.] and Lorish et al. [.]. Diagnostic criteria included: 1) a prodrome of stiffness and rigidity in axial muscles; 2) a slow progression of stiffness involving proxi作者: Alpha-Cells 時(shí)間: 2025-3-27 17:10
Opsoclonus-Myoclonus Syndrome in Childhood,with neuroblastoma [.] and other neural-crest-derived tumours has been described in some children (paraneoplastic OMS, OMS-NB.), whilst several other potential causal relationships have also been found (Table 1), but little progress has been made in understanding the etiology and treatment of this s作者: paragon 時(shí)間: 2025-3-27 21:33
Epilepsy and Autoantibodies,une response. This weak response was explained by: (1) the presence of a blood-brain barrier (BBB); (2) constitutive lack or low expression of proteins of the immune system, such as major histocompatibility complex (MHC) class I and II, co-stimulatory and accessory molecules, on cells of the CNS (gU作者: 轎車 時(shí)間: 2025-3-28 02:01 作者: A簡潔的 時(shí)間: 2025-3-28 03:34
Introduction - Approaches to Understanding Immune-Mediated Neurological Disorders: Measuring and Ev which immune responses play a purely secondary role. In this chapter, we first provide an abbreviated introduction to neuroimmunology, and then discuss ways of measuring immune responses to neuronal antigens, and of approaches that evaluate their pathogenic roles.作者: Adrenal-Glands 時(shí)間: 2025-3-28 06:53 作者: 膝蓋 時(shí)間: 2025-3-28 13:23
Idiopathic Inflammatory Myopathies: Immunological Aspects,DM), polymyositis (PM) and inclusion body myositis (IBM). The latter includes sporadic (s-IBM) and hereditary inclusion body myopathy (h-IBM), which is an a hereditary progressive muscle disease with muscle pathology similar to the s-IBM,but lacking lymphocytic inflammation [.].作者: ABIDE 時(shí)間: 2025-3-28 15:00 作者: 不愿 時(shí)間: 2025-3-28 22:32 作者: Abjure 時(shí)間: 2025-3-28 23:08
Thomas Benker,Carsten Jürck,Matthias Wolfplification of the post-synaptic membrane [., .]. The reduced number of functional AChR accounts for the impaired neuromuscular transmission, leading to the muscle weakness and fatigability typical of MG patients. For a review of the clinical features of MG, see [.].作者: Ballad 時(shí)間: 2025-3-29 03:44
Immunotherapy of Myasthenia Gravis,plification of the post-synaptic membrane [., .]. The reduced number of functional AChR accounts for the impaired neuromuscular transmission, leading to the muscle weakness and fatigability typical of MG patients. For a review of the clinical features of MG, see [.].作者: ARIA 時(shí)間: 2025-3-29 09:07 作者: athlete’s-foot 時(shí)間: 2025-3-29 14:43
Wissensmanagement im Projektgesch?ftdies against brain antigens exist and might lead to brain impairment and, in some disorders, are associated with seizures [.]. In this chapter, we will focus on the CNS disorders characterized by epilepsy and the presence of autoantibodies against neuronal antigens (Table 1)...作者: 持續(xù) 時(shí)間: 2025-3-29 15:37 作者: CHOP 時(shí)間: 2025-3-29 21:33
Epilepsy and Autoantibodies,dies against brain antigens exist and might lead to brain impairment and, in some disorders, are associated with seizures [.]. In this chapter, we will focus on the CNS disorders characterized by epilepsy and the presence of autoantibodies against neuronal antigens (Table 1)...作者: onlooker 時(shí)間: 2025-3-30 02:58
,Zusammenfassung und Schlu?folgerungen, which immune responses play a purely secondary role. In this chapter, we first provide an abbreviated introduction to neuroimmunology, and then discuss ways of measuring immune responses to neuronal antigens, and of approaches that evaluate their pathogenic roles.作者: 眉毛 時(shí)間: 2025-3-30 05:39 作者: implore 時(shí)間: 2025-3-30 11:59
Thomas Benker,Carsten Jürck,Matthias WolfDM), polymyositis (PM) and inclusion body myositis (IBM). The latter includes sporadic (s-IBM) and hereditary inclusion body myopathy (h-IBM), which is an a hereditary progressive muscle disease with muscle pathology similar to the s-IBM,but lacking lymphocytic inflammation [.].作者: Intervention 時(shí)間: 2025-3-30 13:59 作者: affluent 時(shí)間: 2025-3-30 16:39
