標(biāo)題: Titlebook: Arteriosclerosis; New Insights into Pa H. Just,W. Hort,A. M. Zeiher Conference proceedings 1994 Dr. Dietrich Steinkopff Verlag GmbH & Co. K [打印本頁(yè)] 作者: Maculate 時(shí)間: 2025-3-21 17:15
書目名稱Arteriosclerosis影響因子(影響力)
作者: 圖畫文字 時(shí)間: 2025-3-21 21:15
Changes in components and structure of atherosclerotic lesions developing from childhood to middle Adaptive thickening occurs in defined locations from birth and represents a self-limited response of the intima to hemodynamic forces present within specific locations. Adaptive thickening does not indicate or presage an atherosclerotic lesion. However, some of the identical intima locations (progre作者: seduce 時(shí)間: 2025-3-22 03:14
Immunological control mechanisms in plaque formation,atherosclerotic plaques and obtained evidence for T lymphocyte activation and cytokine secretion. A molecular genetical characterization of T cell clones obtained from atherosclerotic lesions revealed that the cells are heterogeneous with regard to antigen receptor gene organization. This indicates 作者: 甜瓜 時(shí)間: 2025-3-22 08:25
,Proliferation versus atrophy — the ambivalent role of smooth muscle cells in human atherosclerosis,ain cellular events: 1) activation of macrophages leading to lipoprotein phagocytosis by scavenger cells, and 2) proliferation of smooth muscle cells (SMC). SMC-like cells producing collagenous fibers and extracellular matrix are particularly involved in the formation of the so-called fibrous caps s作者: Jingoism 時(shí)間: 2025-3-22 09:52 作者: 少量 時(shí)間: 2025-3-22 16:25 作者: isotope 時(shí)間: 2025-3-22 20:28 作者: 失望未來(lái) 時(shí)間: 2025-3-22 23:37 作者: 變白 時(shí)間: 2025-3-23 03:49
Hyperlipidemic endothelial injury and angiogenesis,products in artery walls. Oxidized low-density lipoprotein (LDL) and its products have been incriminated in impairing various endothelial functions including G-protein-dependent transmembrane signaling, calcium regulation, phosphoinositide turnover, protein kinase C activation and others. Modificati作者: 水槽 時(shí)間: 2025-3-23 08:25
Assessment of endothelial modulation of coronary vasomotor tone: Insights into a fundamental functi the endothelium is both a target for and a mediator of vascular disease, functional alterations in coronary vascular reactivity due to endothelial dysfunction might play an important integral part in the clinical presentation of coronary artery disease. Recent advances in interventional techniques 作者: 俗艷 時(shí)間: 2025-3-23 11:13 作者: Allowance 時(shí)間: 2025-3-23 14:20
Therapeutic approaches to the control of coronary atherosclerosis, lipid, monocyte adhesion followed by penetration across the endothelium, the conversion of monocytes to macrophages and smooth muscle cell proliferation and migration are some of the events involved in the early stages of lesion formation. Late events include the formation of excess ground substanc作者: strdulate 時(shí)間: 2025-3-23 21:55 作者: colony 時(shí)間: 2025-3-23 23:59
Role of calcium antagonists in progression of arteriosclerosis. Evidence from animal experiments an arteries during the whole life span (0–90 years). However, this normal ratio increasingly changes as soon as arteriosclerotic alterations of the coronary walls set in. Accordingly, the mural calcium content steadily rises from fatty streaks over severe arteriosclerosis and, lastly, seems to reach a作者: 天真 時(shí)間: 2025-3-24 03:40
,Lipid-lowering therapy — Implications for the prevention of atherosclerosis,dies, epidemiologic surveys, and intervention studies in man. There have been recommendations by national and international advisory boards regarding the aims and means of such interventional strategies. These have not been unanimously accepted, however. The present article will review the evidence 作者: JOT 時(shí)間: 2025-3-24 09:20
http://image.papertrans.cn/b/image/161907.jpg作者: 放逐 時(shí)間: 2025-3-24 14:42 作者: 浮夸 時(shí)間: 2025-3-24 17:04
https://doi.org/10.1007/978-3-030-95833-6Adaptive thickening occurs in defined locations from birth and represents a self-limited response of the intima to hemodynamic forces present within specific locations. Adaptive thickening does not indicate or presage an atherosclerotic lesion. However, some of the identical intima locations (progre作者: frenzy 時(shí)間: 2025-3-24 20:14 作者: 甜得發(fā)膩 時(shí)間: 2025-3-25 02:17
Energy Technologies and Economicsain cellular events: 1) activation of macrophages leading to lipoprotein phagocytosis by scavenger cells, and 2) proliferation of smooth muscle cells (SMC). SMC-like cells producing collagenous fibers and extracellular matrix are particularly involved in the formation of the so-called fibrous caps s作者: 單獨(dú) 時(shí)間: 2025-3-25 05:21 作者: 值得贊賞 時(shí)間: 2025-3-25 09:27 作者: 愛(ài)好 時(shí)間: 2025-3-25 13:48
https://doi.org/10.1007/978-4-431-68325-4red in a variety of disease states, and alterations of vasomotion may be responsible for the development of hypertension, altered tissue perfusion, and an enhanced propensity for vasoconstriction in several common disorders. In hypercholesterolemia and atherosclerosis, this alteration of vasomotor c作者: Suggestions 時(shí)間: 2025-3-25 17:40
Environmental Biotechnology Vol. 1ies. One determinant of this is the LDL concentration in the blood (4). We are investigating other factors which may influence the uptake of LDL and of fibrinogen, another atherogenic plasma protein, by artery walls, as well as the mechanism(s) of this uptake. In order to model human atherogenesis a作者: 勛章 時(shí)間: 2025-3-25 20:08
Md Kaisar Ali,Sajjad Husain Mirproducts in artery walls. Oxidized low-density lipoprotein (LDL) and its products have been incriminated in impairing various endothelial functions including G-protein-dependent transmembrane signaling, calcium regulation, phosphoinositide turnover, protein kinase C activation and others. Modificati作者: 軟弱 時(shí)間: 2025-3-26 01:39
https://doi.org/10.1007/978-981-10-7284-0 the endothelium is both a target for and a mediator of vascular disease, functional alterations in coronary vascular reactivity due to endothelial dysfunction might play an important integral part in the clinical presentation of coronary artery disease. Recent advances in interventional techniques 作者: Intruder 時(shí)間: 2025-3-26 08:09
https://doi.org/10.1007/978-981-10-7284-0s with a mechanical ultrasound device (CVIS) comparing the results of ultrasound with the corresponding histological specimens. Ultrasound was performed with and without echogenic contrast dye (Laevovist, Schering AG, FRG)..After application of contrast dye, correlation of luminal area between histo作者: cartilage 時(shí)間: 2025-3-26 09:58 作者: 極大痛苦 時(shí)間: 2025-3-26 13:28
https://doi.org/10.1007/978-981-10-7284-0oad up to excessive degrees, and the severity of plaque formation, whereas only small amounts of cholesterol were found, even in complicated lesions. The pathogenetic role of Ca was tested in three types of experimental arteriosclerosis and atheromatosis, using Ca antagonists (verapamil, nitrendipin作者: Negotiate 時(shí)間: 2025-3-26 17:05 作者: 獨(dú)白 時(shí)間: 2025-3-26 22:36 作者: 特征 時(shí)間: 2025-3-27 01:44 作者: harbinger 時(shí)間: 2025-3-27 08:04 作者: 一起平行 時(shí)間: 2025-3-27 11:25
Lipid and cellular constituents of unstable human aortic plaques,作者: Palpate 時(shí)間: 2025-3-27 14:56
https://doi.org/10.1007/978-3-319-49875-1ual cases, however, show significant differences in the distribution and extent of lesions..Today, arteriosclerotic research is focused on arteries being most important in clinical investigations. Nevertheless, there are also other arteries with severe arteriosclerotic lesions; for example, the degr作者: 天真 時(shí)間: 2025-3-27 21:02 作者: KEGEL 時(shí)間: 2025-3-28 00:47 作者: Capitulate 時(shí)間: 2025-3-28 02:24 作者: HARP 時(shí)間: 2025-3-28 10:01
Basic Physical Processes and Economics,ncentration-dependent manner, Ro 31–4724 also inhibited outgrowth. Rabbit aortic smooth muscle cells were further shown to release MMPs, namely a 95 and a 72 kDa gelatinases that were inhibited by Ro 31–4724 and Ro 31–7467. The evidence suggests that degradation of basement membrane by gelatinase is作者: oncologist 時(shí)間: 2025-3-28 14:05
https://doi.org/10.1007/978-4-431-68325-4ion of elastic arteries upregulates the local renin-angiotensin system in these arteries and thereby downregulates nitric oxide releasability. Enhanced local synthesis of the trophic factor angiotensin-II and reduced releasability of the antitrophic factor nitric oxide appear to cooperate in the tro作者: Epithelium 時(shí)間: 2025-3-28 15:15 作者: 流動(dòng)性 時(shí)間: 2025-3-28 22:28 作者: 向下五度才偏 時(shí)間: 2025-3-28 22:53
https://doi.org/10.1007/978-981-10-7284-0lopment of atherosclerosis, and impaired endothelial vasodilation is the predominant mechanism underlying inappropriate vasoconstriction in atherosclerosis. However, endothelial vasodilator dysfunction is not only confined to atherosclerotic epicardial vessels, but may also extend into the coronary 作者: etiquette 時(shí)間: 2025-3-29 04:32
https://doi.org/10.1007/978-981-10-7284-0 accuracy of ultrasound measurements in the near field. Thus, further improvement of intracoronary ultrasound devices is mandatory. However, with the use of the currently available systems, additional application of echogenic contrast dye can improve accuracy of luminal measurements, especially in s作者: emission 時(shí)間: 2025-3-29 08:05 作者: 期滿 時(shí)間: 2025-3-29 13:45 作者: aplomb 時(shí)間: 2025-3-29 19:16 作者: Instantaneous 時(shí)間: 2025-3-29 21:03
Arteriosclerosis: Its morphology in the past and today,ual cases, however, show significant differences in the distribution and extent of lesions..Today, arteriosclerotic research is focused on arteries being most important in clinical investigations. Nevertheless, there are also other arteries with severe arteriosclerotic lesions; for example, the degr作者: 針葉類的樹(shù) 時(shí)間: 2025-3-30 01:08 作者: Individual 時(shí)間: 2025-3-30 07:31
Immunological control mechanisms in plaque formation, and foam cell formation by down-regulating the scavenger receptor on macrophages. It also inhibits smooth muscle proliferation in culture and the formation of arterial restenosis after angioplasty in experimental animals. Together, these studies emphasize the importance of vascular-immune interacti作者: 變化無(wú)常 時(shí)間: 2025-3-30 09:41 作者: 河流 時(shí)間: 2025-3-30 14:40
Extracellular matrix degrading metalloproteinases in the pathogenesis of arteriosclerosis,ncentration-dependent manner, Ro 31–4724 also inhibited outgrowth. Rabbit aortic smooth muscle cells were further shown to release MMPs, namely a 95 and a 72 kDa gelatinases that were inhibited by Ro 31–4724 and Ro 31–7467. The evidence suggests that degradation of basement membrane by gelatinase is作者: 奇怪 時(shí)間: 2025-3-30 18:38
Vascular renin-angiotensin-system, endothelial function and atherosclerosis?,ion of elastic arteries upregulates the local renin-angiotensin system in these arteries and thereby downregulates nitric oxide releasability. Enhanced local synthesis of the trophic factor angiotensin-II and reduced releasability of the antitrophic factor nitric oxide appear to cooperate in the tro作者: Indent 時(shí)間: 2025-3-30 23:29
Endothelial dysfunction in atherosclerosis,review, evidence for these alterations will be considered, potential interventions for restoring endothelium-dependent relaxations examined, and the possible impact of endothelial dysfunction in atherosclerosis considered.作者: Hemoptysis 時(shí)間: 2025-3-31 02:44
New determinants of the uptake of atherogenic plasma proteins by arteries,ectly supports the evidence that LDL leaves the plasma by transcytosis through the endothelial cells..In anesthetized rabbits and in conscious rats the uptake of LDL, methylated to prevent its removal by high-affinity receptors (m-LDL), is significantly increased by noradrenaline and by adrenaline a作者: 演繹 時(shí)間: 2025-3-31 08:15
Assessment of endothelial modulation of coronary vasomotor tone: Insights into a fundamental functilopment of atherosclerosis, and impaired endothelial vasodilation is the predominant mechanism underlying inappropriate vasoconstriction in atherosclerosis. However, endothelial vasodilator dysfunction is not only confined to atherosclerotic epicardial vessels, but may also extend into the coronary 作者: 議程 時(shí)間: 2025-3-31 12:53 作者: 男生戴手銬 時(shí)間: 2025-3-31 15:38
Therapeutic approaches to the control of coronary atherosclerosis,h lower plasma cholesterol provide one approach. Another approach is to use calcium antagonists which not only lower blood pressure, but also directly interfere with some of the metabolic events involved in lesion formation.
