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標題: Titlebook: Arterial Disorders; Definition, Clinical Adel Berbari,Giuseppe Mancia Book 2015 Springer International Publishing Switzerland 2015 Arterial [打印本頁]

作者: Ensign    時間: 2025-3-21 17:24
書目名稱Arterial Disorders影響因子(影響力)




書目名稱Arterial Disorders影響因子(影響力)學科排名




書目名稱Arterial Disorders網(wǎng)絡公開度




書目名稱Arterial Disorders網(wǎng)絡公開度學科排名




書目名稱Arterial Disorders被引頻次




書目名稱Arterial Disorders被引頻次學科排名




書目名稱Arterial Disorders年度引用




書目名稱Arterial Disorders年度引用學科排名




書目名稱Arterial Disorders讀者反饋




書目名稱Arterial Disorders讀者反饋學科排名





作者: 失誤    時間: 2025-3-21 23:08

作者: Maximizer    時間: 2025-3-22 00:30
Atherosclerosisesponse resulting in sub-endothelial lipoprotein retention and endothelial activation with migration of immune and smooth muscle cells to the inflamed intima. This leads to formation of atheromas with chronic vascular stenosis and tissue ischemia which is often complicated by acute atherothrombotic
作者: output    時間: 2025-3-22 08:23

作者: 多山    時間: 2025-3-22 10:40
Vascular Changes in the Microcirculation: Arterial Remodeling and Capillary Rarefactionhe renin-angiotensin-aldosterone system, endothelin, catecholamines, substances produced in perivascular fat, and inflammatory and immune mediators such as lymphocytes and macrophages and their products. The vascular phenotype of hypertension varies according to the age of subjects. In younger indiv
作者: LUMEN    時間: 2025-3-22 15:28
Perinatal Programming of Cardiovascular Diseaseoriginally proposed the hypothesis that several chronic adult diseases can be programmed in early life. These authors examined the mortality records of 5,654 men who were born during 1911–1930 in Hertfordshire (UK). In this report was found that the men with the lowest weights at birth subsequently
作者: 顯赫的人    時間: 2025-3-22 20:21
Hemodynamic and Mechanical Factors Acting on Arteriesss. Tensile stress corresponds to changes in dimension according to changes in forces applied on the vessel. Shear stress is of a different nature; it corresponds to the friction of viscous fluid (here the blood) on the inner surface of the vessel (here the endothelium). It is to be noted that direc
作者: 豪華    時間: 2025-3-22 21:46
Role of Metabolic Factors: Lipids, Glucose/Insulin Intoleranced the cardiorenal syndrome (CRS). Obesity, insulin resistance, and diabetes mellitus are associated with a substantially increased prevalence of arterial disease (AD), which is involved in the risk of coronary, cerebral, and peripheral atherosclerosis and the clinical consequences of myocardial infa
作者: Crohns-disease    時間: 2025-3-23 04:23

作者: 聲音刺耳    時間: 2025-3-23 08:25
Neurohormonal Interactionsogically interact with each other and participate in the regulation of cardiac as well as vascular function. Both endothelial and sympathetic functions seem to be the main important regulatory mechanisms involved in maintaining the homeostatic balance and participating in the cardiovascular response
作者: 和藹    時間: 2025-3-23 12:17
Arterial Interactions with Mineral and Bone Disordersomplications in hypertension, diabetes, chronic kidney disease, and chronic inflammatory diseases [1–3]. In many circumstances, these changes are in many aspects similar to those occurring with aging, with this age-related process accelerated and intensified in diabetes and chronic kidney disease (C
作者: 忘川河    時間: 2025-3-23 17:32

作者: callous    時間: 2025-3-23 18:41

作者: Barter    時間: 2025-3-23 23:03
Renal Arterial Agingrial aging represents a clinicopathologic entity characterized by progressive loss of renal mass and nephrons, decrease in renal blood flow and glomerular filtration rate, and a wide spectrum of renal vascular lesions involving both the large arteries and microcirculation. The changes in the renal m
作者: 事情    時間: 2025-3-24 04:09
https://doi.org/10.1007/978-3-319-57054-9adaptive metabolic responses develop a complex network and contribute to the progression of AD. In this review, we will focus on the interaction of metabolic risk factors, adaptive metabolic response, and AD, highlighting pathophysiology and molecular mechanisms, as well as the contemporary understanding of potential therapeutic strategies.
作者: osculate    時間: 2025-3-24 08:11
https://doi.org/10.1007/978-3-319-57054-9ilitating the pathophysiology of atherosclerosis. These important endothelial characteristics will be described in detail in this chapter, whilst we also discuss prominent cardiovascular disease risk factors affecting endothelial function.
作者: 同義聯(lián)想法    時間: 2025-3-24 11:50
Role of Metabolic Factors: Lipids, Glucose/Insulin Intoleranceadaptive metabolic responses develop a complex network and contribute to the progression of AD. In this review, we will focus on the interaction of metabolic risk factors, adaptive metabolic response, and AD, highlighting pathophysiology and molecular mechanisms, as well as the contemporary understanding of potential therapeutic strategies.
作者: 符合國情    時間: 2025-3-24 16:23
Endothelial Function in Health and Diseaseilitating the pathophysiology of atherosclerosis. These important endothelial characteristics will be described in detail in this chapter, whilst we also discuss prominent cardiovascular disease risk factors affecting endothelial function.
作者: intellect    時間: 2025-3-24 20:18
in systemic disorders.Explores the role of macrocirculationAs our knowledge?about arterial disease is greatly expanding, the aim of this book is to explore all aspects of arterial pathology, including classification, clinical manifestations, pathogenesis, and therapeutic options. The discussion of
作者: 羞辱    時間: 2025-3-25 02:45

作者: Rheumatologist    時間: 2025-3-25 05:57
Atherosclerosis intima. This leads to formation of atheromas with chronic vascular stenosis and tissue ischemia which is often complicated by acute atherothrombotic events such as myocardial infarction or stroke. Even today, atherosclerosis and its consequences remain the leading causes of mortality and morbidity in the industrialised nations.
作者: 安定    時間: 2025-3-25 09:27
Definition and Epidemiology of Arterial Diseaseed to rise to around 23 million by 2030 [5], thus highlighting the need for better understanding of these disorders and exploration of new treatment and prevention strategies both at individual and population level. Developing countries will suffer a similar fate if steps are not taken urgently.
作者: adipose-tissue    時間: 2025-3-25 15:39
Hemodynamic and Mechanical Factors Acting on Arteriest measurement of stress is difficult in vivo and that stress is most of the time deduced from stretch (elongation) and force (derived from pressure). Stress can also be derived from mechanical modelling.
作者: 有惡臭    時間: 2025-3-25 16:16
Biomarkers of Vascular Inflammation and Cardiovascular Diseasepredictors of vascular damage and could provide insights into mechanisms, risk and targeted treatment. The present chapter provides a brief overview of vascular damage in cardiovascular disease and discusses recently identified novel biomarkers of vascular inflammation and oxidative stress. The potential clinical relevance is also highlighted.
作者: 可憎    時間: 2025-3-25 20:28

作者: BLINK    時間: 2025-3-26 03:48
Book 2015cation, clinical manifestations, pathogenesis, and therapeutic options. The discussion of pathophysiologic mechanisms of arterial disease is wide ranging, encompassing hemodynamic, metabolic, humoral, inflammatory, genetic and environmental factors. Particular emphasis is placed on recent concepts,
作者: flammable    時間: 2025-3-26 04:39

作者: insincerity    時間: 2025-3-26 11:03
https://doi.org/10.1007/978-3-319-57054-9t measurement of stress is difficult in vivo and that stress is most of the time deduced from stretch (elongation) and force (derived from pressure). Stress can also be derived from mechanical modelling.
作者: Hemodialysis    時間: 2025-3-26 16:28

作者: progestin    時間: 2025-3-26 20:35

作者: ACME    時間: 2025-3-26 21:45
Adel Berbari,Giuseppe ManciaDiscusses in detail the pathophysiologic mechanisms of arterial disease.Explains a range of important novel concepts.Covers arterial disease in systemic disorders.Explores the role of macrocirculation
作者: Breach    時間: 2025-3-27 02:00
http://image.papertrans.cn/b/image/161886.jpg
作者: kyphoplasty    時間: 2025-3-27 05:22
https://doi.org/10.1007/978-3-319-14556-3Arterial ageing; Calcium phosphorus and bone metabolism; Macro-/microcirculation cross-talk; Renal resi
作者: affinity    時間: 2025-3-27 10:21
978-3-319-35348-7Springer International Publishing Switzerland 2015
作者: resistant    時間: 2025-3-27 17:25
Pennee Narot,Narong Kiettikunwongstimated 17.3 million people died from these conditions which is 48 % of noncommunicable diseases; of these, 6.2 million were a consequence of stroke and 7.3 million due to coronary artery disease [3]. In the UK alone, 160,000 people died of CVD in 2001 [4]. The number of deaths from CVDs is predict
作者: Constant    時間: 2025-3-27 20:33
Pennee Narot,Narong Kiettikunwongial tree to the arterioles and capillaries (microcirculation) to meet the metabolic requirements of surrounding tissues. The walls of arteries are composed of living cells, including smooth muscles cells (SMC), stiff collagen fibers, and compliant elastic fibers. The proximal aorta has large amounts
作者: 聯(lián)合    時間: 2025-3-28 01:16
https://doi.org/10.1007/978-981-16-3326-3esponse resulting in sub-endothelial lipoprotein retention and endothelial activation with migration of immune and smooth muscle cells to the inflamed intima. This leads to formation of atheromas with chronic vascular stenosis and tissue ischemia which is often complicated by acute atherothrombotic
作者: 愚蠢人    時間: 2025-3-28 02:23
Pennee Narot,Narong Kiettikunwongover 100 years ago. At that time, atherosclerosis was called nodular arteriosclerosis, and arteriosclerosis, as we now know it, was called senile arteriosclerosis. Arteriosclerosis is caused by fatigue and fracture of elastin laminae in the proximal aorta. The proximal thoracic aorta is principally
作者: 馬具    時間: 2025-3-28 09:20
Equity in Health Services for Ageinghe renin-angiotensin-aldosterone system, endothelin, catecholamines, substances produced in perivascular fat, and inflammatory and immune mediators such as lymphocytes and macrophages and their products. The vascular phenotype of hypertension varies according to the age of subjects. In younger indiv
作者: Hectic    時間: 2025-3-28 14:16

作者: 放肆的我    時間: 2025-3-28 16:49

作者: 彩色的蠟筆    時間: 2025-3-28 20:44

作者: 丑惡    時間: 2025-3-29 00:18
https://doi.org/10.1007/978-3-319-57054-9erapies are key priorities in cardiovascular research. Growing evidence indicates that vascular inflammation and oxidative stress may play an important role in the genesis and progression of cardiovascular disease. Accordingly identification of markers reflecting these processes may be useful early
作者: subordinate    時間: 2025-3-29 03:36

作者: 審問    時間: 2025-3-29 08:28
https://doi.org/10.1007/978-3-319-57054-9omplications in hypertension, diabetes, chronic kidney disease, and chronic inflammatory diseases [1–3]. In many circumstances, these changes are in many aspects similar to those occurring with aging, with this age-related process accelerated and intensified in diabetes and chronic kidney disease (C
作者: 繼而發(fā)生    時間: 2025-3-29 13:26

作者: nonplus    時間: 2025-3-29 16:26
https://doi.org/10.1007/978-3-319-57054-9ce of risk factors and adverse lifestyle as well as negative social factors. For many years research focusing on the development of . has tried to find mechanisms and treatment targets for early manifestations of the pathophysiological changes leading to cardiovascular events. However, even prior to
作者: Decrepit    時間: 2025-3-29 22:47
https://doi.org/10.1007/978-3-030-47020-3rial aging represents a clinicopathologic entity characterized by progressive loss of renal mass and nephrons, decrease in renal blood flow and glomerular filtration rate, and a wide spectrum of renal vascular lesions involving both the large arteries and microcirculation. The changes in the renal m
作者: Entreaty    時間: 2025-3-30 03:29
https://doi.org/10.1007/978-3-319-57054-9Given the prognostic role of arterial stiffness and central hemodynamics [1–4], a large body of research has focused on the recognition of lifestyle interventions that may either worsen arterial function, and thus, they should be prohibited or at least discouraged, or exert favorable effects, and, consequently, they should be encouraged.
作者: URN    時間: 2025-3-30 06:49
Pharmacologic and Environmental Factors: Coffee, Smoking, and SodiumGiven the prognostic role of arterial stiffness and central hemodynamics [1–4], a large body of research has focused on the recognition of lifestyle interventions that may either worsen arterial function, and thus, they should be prohibited or at least discouraged, or exert favorable effects, and, consequently, they should be encouraged.
作者: Carminative    時間: 2025-3-30 10:23

作者: 鬧劇    時間: 2025-3-30 12:28

作者: 聯(lián)合    時間: 2025-3-30 20:08
Equity in Health Services for Ageing more typically found. In subjects older than 50 years of age, vascular changes are increasingly found in aorta, which becomes stiffer as arteriosclerosis develops, resulting in increased pulse pressure. Enhanced pulsatility may injure small arteries and arterioles, leading to remodeling of these an
作者: 榨取    時間: 2025-3-30 21:42

作者: Ambulatory    時間: 2025-3-31 03:33

作者: Gobble    時間: 2025-3-31 08:23

作者: 首創(chuàng)精神    時間: 2025-3-31 10:07
https://doi.org/10.1007/978-3-030-47020-3zation of systemic and glomerular hypertension with antihypertensive agents; lipid-lowering, antiplatelet agents; and lifestyle measures. Revascularization of stenotic renal artery(ies) does not appear to provide any improvement in malfunction and/or cardiovascular outcome.
作者: 使殘廢    時間: 2025-3-31 17:12
Book 2015microcirculation in target organ involvement. Evaluation procedures are carefully explained, and the full range of currently available therapeutic options, including lifestyle modifications and pharmacologic approaches, are described and appraised. ? ?




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