標(biāo)題: Titlebook: Artemisinin and Nitric Oxide; Mechanisms and Impli Qing-Ping Zeng Book 2015 The Author(s) 2015 Aging-Related Disease.Artemisinin-Mediated A [打印本頁(yè)] 作者: Aggrief 時(shí)間: 2025-3-21 17:00
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide影響因子(影響力)
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide被引頻次
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide被引頻次學(xué)科排名
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書(shū)目名稱(chēng)Artemisinin and Nitric Oxide讀者反饋
書(shū)目名稱(chēng)Artemisinin and Nitric Oxide讀者反饋學(xué)科排名
作者: 招待 時(shí)間: 2025-3-21 21:33 作者: 不容置疑 時(shí)間: 2025-3-22 00:43 作者: Ejaculate 時(shí)間: 2025-3-22 06:03 作者: 知道 時(shí)間: 2025-3-22 11:13
ART for Anti-inflammation,eeding. While a donor of NO replicates the modeling of synovitis, an inhibitor of NOS blocks the progression of synovitis, suggesting inflammation-triggered NO burst represents an etiological cause of synovitis. ART, via inhibiting iNOS, can ameliorate the synovial inflammation by mitigating NO-driv作者: 評(píng)論者 時(shí)間: 2025-3-22 15:21 作者: irradicable 時(shí)間: 2025-3-22 20:14 作者: 平息 時(shí)間: 2025-3-22 23:52
Book 2015ains why we should maintain a steady-state NO level that is derived from neuronal or epithelial NO synthase, and avoid the extremely high NO level resulting from inducible NO synthase. The book offers a valuable resource for medical chemists, clinicians, biologists and all those interested in health and disease..作者: Blanch 時(shí)間: 2025-3-23 01:48
2191-5407 tric oxide is harmful for health.Guides people to understand.This book discusses both the beneficial and harmful aspects of NO in biology and medicine, and also introduces the emerging discovery of artemisinin in antitumor, antibacterial infection, anti-inflammation, and antiaging contexts. In 1992 作者: hematuria 時(shí)間: 2025-3-23 07:09 作者: legacy 時(shí)間: 2025-3-23 12:11 作者: microscopic 時(shí)間: 2025-3-23 17:55 作者: 笨重 時(shí)間: 2025-3-23 19:34 作者: Intellectual 時(shí)間: 2025-3-24 01:25 作者: forbid 時(shí)間: 2025-3-24 03:44 作者: 減至最低 時(shí)間: 2025-3-24 08:14
Artemisinin and Nitric Oxide978-3-662-47688-8Series ISSN 2191-5407 Series E-ISSN 2191-5415 作者: Mendicant 時(shí)間: 2025-3-24 11:18 作者: chastise 時(shí)間: 2025-3-24 15:29
Dermatochalasis and Blepharoplasty,RT benefits tumor propagation due to induce cytoprotective NO production. Pro-oxidants that antagonize antioxidants can potentiate ART’s antitumor capacity. The combination of ART with pro-oxidants should provide an effective solution to the?chemotherapy of multidrug resistant?tumors.作者: 細(xì)微的差異 時(shí)間: 2025-3-24 19:07
Histopathology of Eyelid Tumors,n antibiotic sensitizing role through inhibiting bNOS and CAT. Either in vivo or in vitro, ART can increase the bacteriocidal activity of antibiotics by abrogating the beneficial NO and enhancing the harmful H.O., demonstrating a potential role in combating multidrug?resistant bacteria.作者: 一瞥 時(shí)間: 2025-3-25 00:43
https://doi.org/10.1007/978-3-030-36606-3eeding. While a donor of NO replicates the modeling of synovitis, an inhibitor of NOS blocks the progression of synovitis, suggesting inflammation-triggered NO burst represents an etiological cause of synovitis. ART, via inhibiting iNOS, can ameliorate the synovial inflammation by mitigating NO-driven hypoxia, angiogenesis, and hyperplasia.作者: 外形 時(shí)間: 2025-3-25 03:55
https://doi.org/10.1007/978-3-030-36606-3ttern from biosynthesis to degradation. ART also mimics CR to compromise mouse?telomere shortening by upregulating antioxidative enzymes for effective ROS scavenging, which is followed by the alleviation of DNA damage and downregulation of tumor suppressors. This is the first time for?having found ART exerting an antiaging role.作者: 杠桿 時(shí)間: 2025-3-25 09:15 作者: Expostulate 時(shí)間: 2025-3-25 15:12 作者: 租約 時(shí)間: 2025-3-25 16:32 作者: Optimum 時(shí)間: 2025-3-25 22:10
ART for Antibacterial Infection,n antibiotic sensitizing role through inhibiting bNOS and CAT. Either in vivo or in vitro, ART can increase the bacteriocidal activity of antibiotics by abrogating the beneficial NO and enhancing the harmful H.O., demonstrating a potential role in combating multidrug?resistant bacteria.作者: 不吉祥的女人 時(shí)間: 2025-3-26 00:28 作者: 富饒 時(shí)間: 2025-3-26 08:02
ART for Antiaging,ttern from biosynthesis to degradation. ART also mimics CR to compromise mouse?telomere shortening by upregulating antioxidative enzymes for effective ROS scavenging, which is followed by the alleviation of DNA damage and downregulation of tumor suppressors. This is the first time for?having found ART exerting an antiaging role.作者: 被告 時(shí)間: 2025-3-26 09:28 作者: macular-edema 時(shí)間: 2025-3-26 12:52 作者: SHRIK 時(shí)間: 2025-3-26 17:35
Eyeblink Classical Conditioning in Amnesia,se of ART can kill cancer cells and bacteria through compromising protective NO production. A low dose of ART can mimic CR to extend lifespan and reduce weight by triggering mitochondrial biogenesis. Therefore, ART can exert versatile beneficial effects on human health in addition to antimalaria.作者: Plaque 時(shí)間: 2025-3-26 22:57 作者: MERIT 時(shí)間: 2025-3-27 04:53 作者: Cocker 時(shí)間: 2025-3-27 08:56
Histopathology of Eyelid Tumors,n antibiotic sensitizing role through inhibiting bNOS and CAT. Either in vivo or in vitro, ART can increase the bacteriocidal activity of antibiotics by abrogating the beneficial NO and enhancing the harmful H.O., demonstrating a potential role in combating multidrug?resistant bacteria.作者: obstinate 時(shí)間: 2025-3-27 11:58
https://doi.org/10.1007/978-3-030-36606-3eeding. While a donor of NO replicates the modeling of synovitis, an inhibitor of NOS blocks the progression of synovitis, suggesting inflammation-triggered NO burst represents an etiological cause of synovitis. ART, via inhibiting iNOS, can ameliorate the synovial inflammation by mitigating NO-driv作者: ORBIT 時(shí)間: 2025-3-27 15:14
https://doi.org/10.1007/978-3-030-36606-3ttern from biosynthesis to degradation. ART also mimics CR to compromise mouse?telomere shortening by upregulating antioxidative enzymes for effective ROS scavenging, which is followed by the alleviation of DNA damage and downregulation of tumor suppressors. This is the first time for?having found A作者: harmony 時(shí)間: 2025-3-27 18:02
https://doi.org/10.1007/978-3-030-36606-3otein misfolding and structural/functional abnormality have been supposed to be the plausible pathogenic initiators of neurodegenerative diseases. Obesity might be an outcome of the activation of iNOS and inactivation of eNOS, leading to mitochondrial loss and adipose whitening without adipose burni作者: ETCH 時(shí)間: 2025-3-27 22:41
The Female Reproductive System in Health and Disease,on. The ovum is then swept into the fallopian tube and starts its journey towards the uterus. After the ovum has been expelled, the remaining follicle is transformed into yellowish body called corpus luteum. This structure continues to secret the hormone estrogen, and starts secreting the other fema作者: Blemish 時(shí)間: 2025-3-28 04:04
individual writings is Kipling‘s concern with the heroic life, but of equal importance is the analysis and evaluation of them as works of art. Avoiding the tangled and special language of some recent literary theory, this will appeal to a wide audience of those interested in Kipling‘s mind and art.978-1-4039-7868-4作者: 膽小懦夫 時(shí)間: 2025-3-28 10:05 作者: paltry 時(shí)間: 2025-3-28 13:15
