標(biāo)題: Titlebook: Apoptosis, Cell Signaling, and Human Diseases; Molecular Mechanisms Rakesh Srivastava Book 2007 Humana Press 2007 DNA.HIV.gene expression.g [打印本頁] 作者: ossicles 時(shí)間: 2025-3-21 17:58
書目名稱Apoptosis, Cell Signaling, and Human Diseases影響因子(影響力)
書目名稱Apoptosis, Cell Signaling, and Human Diseases影響因子(影響力)學(xué)科排名
書目名稱Apoptosis, Cell Signaling, and Human Diseases網(wǎng)絡(luò)公開度
書目名稱Apoptosis, Cell Signaling, and Human Diseases網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Apoptosis, Cell Signaling, and Human Diseases被引頻次
書目名稱Apoptosis, Cell Signaling, and Human Diseases被引頻次學(xué)科排名
書目名稱Apoptosis, Cell Signaling, and Human Diseases年度引用
書目名稱Apoptosis, Cell Signaling, and Human Diseases年度引用學(xué)科排名
書目名稱Apoptosis, Cell Signaling, and Human Diseases讀者反饋
書目名稱Apoptosis, Cell Signaling, and Human Diseases讀者反饋學(xué)科排名
作者: 控訴 時(shí)間: 2025-3-21 20:16
Protein Kinase C and Apoptosisproliferation and survival and their expression or activity is altered in some human diseases, particularly cancer. The development and utilization of PKC isoform specific tools, including dominant inhibitory kinases, mouse models in which specific PKC isoforms have been disrupted, and PKC isoform s作者: 改革運(yùn)動(dòng) 時(shí)間: 2025-3-22 03:54 作者: 頌揚(yáng)本人 時(shí)間: 2025-3-22 08:18 作者: ACTIN 時(shí)間: 2025-3-22 10:10 作者: Alveoli 時(shí)間: 2025-3-22 16:09
MAPK Signaling in Human Diseasesng cell proliferation, differentiation, motility, and survival. Three subfamilies of MAPK have been extensively studied: extracellular signal-regulated kinases (ERK), p38-MAPK, and c-Jun N-terminal kinase (JNK). The ERKs play roles in cell proliferation, survival, and motility, and inhibitors of thi作者: panorama 時(shí)間: 2025-3-22 19:28
Serine/Threonine Protein Phosphatases in Apoptosis, myosin light chain, lamin B, some caspases and their inhibitors, as well as transcriptions factors like CREB and NF-kB/I-kB. Most is known about the protein phosphatases (PPs) PP1, PP2A, PP2B/calcineurin, PP4/PPX, and PP5. With the possible exception of PP5, which so far appears to mainly promote 作者: 歸功于 時(shí)間: 2025-3-22 22:17
Urokinase/Urokinase Receptor-Mediated Signaling in Cancerits receptor (uPAR), and its major inhibitor plasminogen activator inhibtor-1 (PAI-1) or -2 (PAI-2) are activated by common signaling mechanisms. In tumor cells of mesenchymal or epithelial origin uPA, uPAR, and PAIs or metallo-proteinases (MMPs) are overexpressed and these molecules are implicated 作者: 階層 時(shí)間: 2025-3-23 03:40
Signaling Pathways That Protect the Heart Against Apoptosis Induced by Ischemia and Reperfusionly regulated. Two mechanisms of apoptosis involve the extrinsic death receptor pathway and the intrinsic mitochondrial pathway. Both pathways lead to the activation of effector caspases, resulting in cell death. The mitochondrial pathway plays a key role in initiating apoptosis after ischemia and re作者: 吞下 時(shí)間: 2025-3-23 06:36 作者: antedate 時(shí)間: 2025-3-23 11:22
Death Receptorshanism to remove infected, damaged or mutated cells. Although a number of stimuli trigger apoptosis, it is mainly mediated through at least three major pathways that are regulated by (1) the death receptors, (2) the mitochondria, and (3) the ER (endoplasmic reticulum). Under certain conditions, thes作者: Urgency 時(shí)間: 2025-3-23 14:24
DNA Damage-Dependent Apoptosisanisms are activated to allow more time for effective repair. However, if these mechanisms fail or the damage is irreparable, then apoptosis is induced to eliminate cells carrying DNA lesions and preserve genomic integrity. This chapter discusses how DNA damage-dependent apoptosis is induced and exe作者: 微枝末節(jié) 時(shí)間: 2025-3-23 20:25 作者: Euphonious 時(shí)間: 2025-3-23 23:36
Apoptosis Induction in T Lymphocytes by HIV immune response and the onset of opportunistic infections and neoplasms. The mechanism responsible of CD4 T-cell depletion is mainly the induction of apoptosis, which can be activated by HIV through various pathways. In this chapter direct and indirect mechanisms of HIV-induced apoptosis in infecte作者: 任意 時(shí)間: 2025-3-24 04:18 作者: 顯微鏡 時(shí)間: 2025-3-24 10:11
Intracellular Pathways Involved in DNA Damage and Repair to Neuronal Apoptosislangiectasia. However, other syndromes associated with defective DNA damage response also include neurological symptoms as a primary feature of their phenotypes. This argues that defects in the repair of, or response to, DNA damage impact significantly on brain function. This chapter summarizes some作者: 假裝是你 時(shí)間: 2025-3-24 11:05
The Biology of Caspasesses, to affect the controlled demise of cells. When the regulatory pathways controlling cell death are perturbed through mutation, disease states, including cancers and neurodegenerative disorders, can occur. Therefore, caspases and the molecules regulating their function are potential candidates fo作者: aviator 時(shí)間: 2025-3-24 18:52
DNA Damage-Dependent Apoptosisanisms are activated to allow more time for effective repair. However, if these mechanisms fail or the damage is irreparable, then apoptosis is induced to eliminate cells carrying DNA lesions and preserve genomic integrity. This chapter discusses how DNA damage-dependent apoptosis is induced and executed.作者: incontinence 時(shí)間: 2025-3-24 19:20 作者: 橫截,橫斷 時(shí)間: 2025-3-25 02:02 作者: Mast-Cell 時(shí)間: 2025-3-25 03:34 作者: 挫敗 時(shí)間: 2025-3-25 09:09
https://doi.org/10.1007/978-88-470-2309-3final step is completed by phosphorylation of a number of substrate proteins mediating the diverse functional responses of the cell. The study of cyclic adenosine monophosphate (cAMP) has indicated a dual effect for this nucleotide on proliferation and differentiation processes. Elevation of intrace作者: Keshan-disease 時(shí)間: 2025-3-25 13:28 作者: Conflict 時(shí)間: 2025-3-25 16:47
Le sequenze RM: tecniche fondamentali,cles. This success, however also depends on the evasion of a multitude of antiviral signaling mechanisms. Many viruses are capable of averting antiviral signals through modulation of host cell signaling pathways. Apoptotic inhibition, for example, is a universal intracellular antiviral response, whi作者: 猛烈責(zé)罵 時(shí)間: 2025-3-25 20:55 作者: 輕浮女 時(shí)間: 2025-3-26 02:28 作者: STERN 時(shí)間: 2025-3-26 05:46 作者: 祝賀 時(shí)間: 2025-3-26 10:10
https://doi.org/10.1007/978-3-030-85889-6, myosin light chain, lamin B, some caspases and their inhibitors, as well as transcriptions factors like CREB and NF-kB/I-kB. Most is known about the protein phosphatases (PPs) PP1, PP2A, PP2B/calcineurin, PP4/PPX, and PP5. With the possible exception of PP5, which so far appears to mainly promote 作者: 值得贊賞 時(shí)間: 2025-3-26 15:22 作者: 切掉 時(shí)間: 2025-3-26 17:49 作者: 燒烤 時(shí)間: 2025-3-27 00:33 作者: 兒童 時(shí)間: 2025-3-27 04:57
An exact and scientific metaphysicshanism to remove infected, damaged or mutated cells. Although a number of stimuli trigger apoptosis, it is mainly mediated through at least three major pathways that are regulated by (1) the death receptors, (2) the mitochondria, and (3) the ER (endoplasmic reticulum). Under certain conditions, thes作者: Assemble 時(shí)間: 2025-3-27 08:35 作者: 辯論 時(shí)間: 2025-3-27 12:56
https://doi.org/10.1007/978-3-540-31107-2ls. It was previously considered a humble garbage collector, performing housekeeping duties to remove misfolded or spent proteins. Recent findings also substantiate a pivotal role of the proteasome in the regulation of apoptosis. Polyubiquitin conjugation of key pro-and antiapoptotic molecules targe作者: filicide 時(shí)間: 2025-3-27 14:27
https://doi.org/10.1007/978-3-540-31107-2 immune response and the onset of opportunistic infections and neoplasms. The mechanism responsible of CD4 T-cell depletion is mainly the induction of apoptosis, which can be activated by HIV through various pathways. In this chapter direct and indirect mechanisms of HIV-induced apoptosis in infecte作者: intercede 時(shí)間: 2025-3-27 19:55 作者: 狂怒 時(shí)間: 2025-3-27 22:32 作者: 假 時(shí)間: 2025-3-28 03:29
Quantities and Measurable Qualities,ses, to affect the controlled demise of cells. When the regulatory pathways controlling cell death are perturbed through mutation, disease states, including cancers and neurodegenerative disorders, can occur. Therefore, caspases and the molecules regulating their function are potential candidates fo作者: EVEN 時(shí)間: 2025-3-28 10:15
Quantities and Measurable Qualities,anisms are activated to allow more time for effective repair. However, if these mechanisms fail or the damage is irreparable, then apoptosis is induced to eliminate cells carrying DNA lesions and preserve genomic integrity. This chapter discusses how DNA damage-dependent apoptosis is induced and executed.作者: fructose 時(shí)間: 2025-3-28 12:04 作者: irreparable 時(shí)間: 2025-3-28 18:40
https://doi.org/10.1007/978-1-59745-199-4DNA; HIV; gene expression; genes; infection; molecular biology; molecular mechanisms; signal transduction; v作者: 運(yùn)動(dòng)的我 時(shí)間: 2025-3-28 20:02 作者: 任意 時(shí)間: 2025-3-28 23:14
Elementi di risonanza magneticai-apoptotic isoforms suggests that PKC isoforms may function as molecular sensors, promoting cell survival under favorable conditions, and executing the death of abnormal or damaged cells when needed.This chapter discusses what is currently known about the contribution of specific isoforms to apopto作者: tenuous 時(shí)間: 2025-3-29 04:30
Le sequenze RM: tecniche fondamentali,rease their survival, in addition to other established mechanisms such as expression of viral onco-genes and direct inhibition of proapoptotic proteins. Recent research has revealed that this pathway is up-regulated by a number of viruses during both short-term acute infections and long-term latent 作者: Cupping 時(shí)間: 2025-3-29 09:33 作者: Leisureliness 時(shí)間: 2025-3-29 12:29
Joaquim Sanz,Oriol Tomasa,Nor Sidki-Riusological functions of the MAPKs with an emphasis on the regulation of cell survival and proliferation by these enzymes, and will also discuss the development of MAPK pathway inhibitors for the treatment of human diseases.作者: 小平面 時(shí)間: 2025-3-29 17:52 作者: Hypopnea 時(shí)間: 2025-3-29 20:10 作者: CODE 時(shí)間: 2025-3-30 01:18
Significance of Protein Kinase A in Cancerent differences of the protein kinases, both under the control of cAMP, indicate that they act at different stages of cell life. The relative ratio of PKA-I and PKA-II varies not only throughout the cell cycle in cells of the same type, but also among tissues and during development. The data reporte作者: 凹室 時(shí)間: 2025-3-30 04:04 作者: POLYP 時(shí)間: 2025-3-30 10:10
The Role of Phosphoinositide 3-Kinase-Akt Signaling in Virus Infectionrease their survival, in addition to other established mechanisms such as expression of viral onco-genes and direct inhibition of proapoptotic proteins. Recent research has revealed that this pathway is up-regulated by a number of viruses during both short-term acute infections and long-term latent 作者: Graves’-disease 時(shí)間: 2025-3-30 13:19 作者: 脾氣暴躁的人 時(shí)間: 2025-3-30 19:00
MAPK Signaling in Human Diseasesological functions of the MAPKs with an emphasis on the regulation of cell survival and proliferation by these enzymes, and will also discuss the development of MAPK pathway inhibitors for the treatment of human diseases.作者: engrossed 時(shí)間: 2025-3-31 00:07
Cyclooxygenase-2 Gene Expressione. Many cellular transcription factors bind these elements to regulate the COX-2 gene transcription. Among them, the key factors are NF-kB and b-catenin. Of these, b-catenin is especially interesting because it can egulate COX-2 both directly by binding to the promoter elements as a comples with eit作者: cipher 時(shí)間: 2025-3-31 02:01 作者: 信條 時(shí)間: 2025-3-31 07:50
Book 2007ws they present systemically examine the most exciting and innovative aspects of their particular areas of expertise. Researchers will find these volumes of major benefit as they search for novel and more effective treatments for human diseases..作者: adipose-tissue 時(shí)間: 2025-3-31 10:50 作者: NADIR 時(shí)間: 2025-3-31 16:43
An exact and scientific metaphysics transformed cells typically overexpress uPA and uPAR and show increased activation of the above signaling modules. These signaling intermediaries are involved in the expression of uPA and uPAR as well as mitogenesis, neoplastic growth and metaststic spread and tissue remodeling as occurs in the pathogenesis of neoplasia.作者: HALO 時(shí)間: 2025-3-31 17:30 作者: Misgiving 時(shí)間: 2025-4-1 01:43 作者: 修飾語 時(shí)間: 2025-4-1 05:54
Quantities and Measurable Qualities, sensing and repairing: the tumor suppressor p53 and the component of the DNA mismatch repair system MSH2. Both proteins participate in the cancer prevention machinery for the body as well as in the neurodegenerative process.作者: Mindfulness 時(shí)間: 2025-4-1 06:41
