標(biāo)題: Titlebook: Apoptosis; Enrico Mihich,Robert T. Schimke Book 1994 Springer Science+Business Media New York 1994 Antigen.DNA.apoptosis.cancer.cell.cell [打印本頁] 作者: INFER 時(shí)間: 2025-3-21 19:43
書目名稱Apoptosis影響因子(影響力)
書目名稱Apoptosis影響因子(影響力)學(xué)科排名
書目名稱Apoptosis網(wǎng)絡(luò)公開度
書目名稱Apoptosis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Apoptosis被引頻次
書目名稱Apoptosis被引頻次學(xué)科排名
書目名稱Apoptosis年度引用
書目名稱Apoptosis年度引用學(xué)科排名
書目名稱Apoptosis讀者反饋
書目名稱Apoptosis讀者反饋學(xué)科排名
作者: Merited 時(shí)間: 2025-3-21 23:41 作者: dowagers-hump 時(shí)間: 2025-3-22 01:13 作者: 上漲 時(shí)間: 2025-3-22 08:39
Elemente der Achten Nebengruppedependent 32Dcl3 murine myeloid cell line. 32D cells are absolutely dependent upon IL-3 for proliferation and survival,. and have no propensity for developing factor-independent growth. In the absence of IL-3, 32D cells rapidly arrest in the Gl phase of the cell cycle and eventually undergo apoptosis..作者: Legion 時(shí)間: 2025-3-22 10:08 作者: 內(nèi)行 時(shí)間: 2025-3-22 13:14 作者: 碎片 時(shí)間: 2025-3-22 17:16 作者: 狂熱語言 時(shí)間: 2025-3-23 00:40
Regulation of Apoptosis in Interleukin-3-Dependent Myeloid Progenitor Cells,dependent 32Dcl3 murine myeloid cell line. 32D cells are absolutely dependent upon IL-3 for proliferation and survival,. and have no propensity for developing factor-independent growth. In the absence of IL-3, 32D cells rapidly arrest in the Gl phase of the cell cycle and eventually undergo apoptosis..作者: 討厭 時(shí)間: 2025-3-23 04:58 作者: 加入 時(shí)間: 2025-3-23 08:15
Wild Type p53 Activity Contributes to Dependence on Hematopoietic Survival Factors,s found in cancer cells are typically devoid of such activities. Hence, the principal outcome of such mutations is probably the inactivation of the tumor suppressor function of wt p53. In addition, at least certain mutations may also confer a distinct gain of function., thereby possibly contributing a novel oncogenic activity to the affected cell.作者: Endometrium 時(shí)間: 2025-3-23 11:03 作者: 思想 時(shí)間: 2025-3-23 17:35
Book 1994 to Programmed Cell Death (PCD) or Apoptosis, and the mechanisms involved. With presentations at the cutting edge of progress and stimulating discussions, this Symposium addressed the genetics and molecular mechanisms determining PCD and the role of this suicidal process in cancer and the immune sys作者: amorphous 時(shí)間: 2025-3-23 19:25
https://doi.org/10.1007/978-3-662-30606-2 replicate its DNA prior to repairing lesions in the DNA may be more likely to result in daughter cells which have altered genetic information relative to the parental cell. It would be predicted that such altered daughter cells would be more likely to develop the genetic changes which contribute to a transformed phenotype (Figure 1).作者: 純樸 時(shí)間: 2025-3-24 00:39
Molecular Controls of Cell Cycle Progression Following DNA Damage: Roles of P53 and Ataxia-Telangie replicate its DNA prior to repairing lesions in the DNA may be more likely to result in daughter cells which have altered genetic information relative to the parental cell. It would be predicted that such altered daughter cells would be more likely to develop the genetic changes which contribute to a transformed phenotype (Figure 1).作者: Genteel 時(shí)間: 2025-3-24 06:03 作者: Overdose 時(shí)間: 2025-3-24 07:26 作者: rods366 時(shí)間: 2025-3-24 11:41 作者: 空洞 時(shí)間: 2025-3-24 15:42
Elemente der Achten Nebengruppe, however, extremely rare as demonstrated by the fact that cancer arises in only one in three individuals during the entire course of their lives. Part of the answer to this paradox appears to reside in the multifunctional nature of the components that mediate cell proliferation. One of these, c-Myc, is the subject of this paper.作者: Needlework 時(shí)間: 2025-3-24 20:44
Elemente der Achten Nebengruppe1992), whereas viruses (Rao et al., 1992) and bacterial toxins (Fiorentini et al, 1993) can directly elicit an apoptotic response in target cells. Finally, exposure to ionizing radiation (Zhivotovsky et al., 1981 and 1993b) or chemical toxicants (McConkey et al., 1988; Aw et al., 1990) can accelerate apoptosis in immature T cells.作者: 吸氣 時(shí)間: 2025-3-25 02:11 作者: thalamus 時(shí)間: 2025-3-25 05:51
https://doi.org/10.1007/978-3-322-85163-5hat this recognition prompted the appearance of models of immune tolerance, and served as the impetus for the development of the clonal selection theory (2,3). The application of this theory to explain tolerance through the deletion of potentially autoreactive cells as proposed by Burnet (3,4) persist in almost unchanged form today.作者: seroma 時(shí)間: 2025-3-25 09:22 作者: AGATE 時(shí)間: 2025-3-25 13:57 作者: agnostic 時(shí)間: 2025-3-25 19:38
,: Oncogene and Tumour Suppressor Gene,, however, extremely rare as demonstrated by the fact that cancer arises in only one in three individuals during the entire course of their lives. Part of the answer to this paradox appears to reside in the multifunctional nature of the components that mediate cell proliferation. One of these, c-Myc, is the subject of this paper.作者: Introvert 時(shí)間: 2025-3-25 21:50
Ion Signalling in Apoptosis,1992), whereas viruses (Rao et al., 1992) and bacterial toxins (Fiorentini et al, 1993) can directly elicit an apoptotic response in target cells. Finally, exposure to ionizing radiation (Zhivotovsky et al., 1981 and 1993b) or chemical toxicants (McConkey et al., 1988; Aw et al., 1990) can accelerate apoptosis in immature T cells.作者: Corral 時(shí)間: 2025-3-26 04:01 作者: 織物 時(shí)間: 2025-3-26 07:17
MYC, FAS, Apoptosis, and Immune Tolerance,hat this recognition prompted the appearance of models of immune tolerance, and served as the impetus for the development of the clonal selection theory (2,3). The application of this theory to explain tolerance through the deletion of potentially autoreactive cells as proposed by Burnet (3,4) persist in almost unchanged form today.作者: 無瑕疵 時(shí)間: 2025-3-26 10:03 作者: Optometrist 時(shí)間: 2025-3-26 14:30 作者: Heterodoxy 時(shí)間: 2025-3-26 20:32
978-1-4757-9219-5Springer Science+Business Media New York 1994作者: Hiatus 時(shí)間: 2025-3-27 01:01
Pezcoller Foundation Symposiahttp://image.papertrans.cn/a/image/158994.jpg作者: HAIL 時(shí)間: 2025-3-27 03:24
https://doi.org/10.1007/978-3-662-30588-1occurs during the development of the mammalian fetus, particularly in the fetal brain; in areas of the developing vertebrate nervous system as many as 85% of the developing neurons die. Similarly, about 95% of developing thymocytes die without ever leaving the thymus. Why such cell deaths occur and 作者: Leisureliness 時(shí)間: 2025-3-27 05:17
Elemente der Achten Hauptgruppehe causes of photoreceptor death. Mutations of many different proteins interfere with normal development of photoreceptors or initiate a process that leads to premature photoreceptor death for obscure reasons. These mutations include the rhodopsin gene at many different sites (reviewed by Berson.), 作者: 恭維 時(shí)間: 2025-3-27 10:42 作者: 枕墊 時(shí)間: 2025-3-27 16:18 作者: investigate 時(shí)間: 2025-3-27 21:28
Elemente der Achten Nebengruppes underscores a major problem facing multicellular organisms: namely, how to allow the rapid proliferation of component cells and at the same time never allow those component cells to engage in competition with each other. In principle, any cell that acquires a growth advantage through mutation shou作者: 無目標(biāo) 時(shí)間: 2025-3-27 23:27 作者: 碎石 時(shí)間: 2025-3-28 03:57 作者: Outmoded 時(shí)間: 2025-3-28 07:27 作者: hypnotic 時(shí)間: 2025-3-28 14:30
Elemente der Achten Nebengruppe IIs growth. Prostatic cancer is thus often highly responsive to androgen ablation therapy. Based upon this, patients with non-organ confined disease eventually require systemic androgen ablation therapy. Nearly all men with metastatic prostatic cancer treated with surgically or chemically induced cast作者: 手勢 時(shí)間: 2025-3-28 17:13
https://doi.org/10.1007/978-3-662-25642-8llicular lymphomas are indolent tumors, difficult to eradicate but slow to progress, and are believed to originate from centrocytic B cells in germinal centers.. 85% or more of follicular lymphomas carry a t(14;18) (q32;q21) leading to the expectation that a new cellular oncogene would be activated 作者: glans-penis 時(shí)間: 2025-3-28 21:49
Elemente der Achten Nebengruppe IIinding chains and immunoglobulin are generated randomly from the rearrangement of independent gene segments. While this process ensures the diversity of the immune cell receptor repertoire, it also results in the generation of T cells that express receptors that recognize self antigens in the contex作者: 虛弱的神經(jīng) 時(shí)間: 2025-3-29 02:47
Strukturerhaltende Abbildungen,s of cytokines is the interferons (IFNs), which represent a heterogeneous family of multifunctional cytokines that typically manifest antiviral activity. In fact, viral infections commonly trigger induction of the type-I IFN (IFN-αs and IFN-β) genes in a variety of cells, and these newly produced IF作者: MUTED 時(shí)間: 2025-3-29 06:07
https://doi.org/10.1007/978-3-322-85163-5ield of immunology. Arguably, Ehrlich’s description of the phenomenon and the ensuing belief that it is a fundamental attribute of the immune system delayed the recognition of the existence of autoimmune disease until the 1950’s (1). However, once it was realized that the immune system . respond to 作者: 造反,叛亂 時(shí)間: 2025-3-29 09:11
Mengen, Relationen, Abbildungen,complex and highly varied.. It is clear that the actions of cytotoxic agents in mammalian cells frequently feed into the apoptosis pathway, and an extremely wide variety of agents/treatments have been shown to induce the events of apoptosis in cultured mammalian cells.. It is important to note, howe作者: 含水層 時(shí)間: 2025-3-29 13:47
Elemente der Arithmetik und Algebran control of growth of malignant lymphocytes we raised monoclonal antibodies against the human B lymphoblast cell line SKW6.4. One antibody, anti-APO-1, reacted with a 48-kilodalton antigen (APO-1) on a set of activated human lymphocytes, on malignant human lymphocyte lines, and on a fraction of leu作者: FLIP 時(shí)間: 2025-3-29 17:00
https://doi.org/10.1007/978-3-642-92119-3the nuclear membrane. This occurs well in advance of loss of membrane integrity. Concurrent with chromatin condensation, the nuclear DNA is usually digested into oligonucleosomal fragments by an endogenous endonuclease. This chromatin condensation together with DNA digestion has been called one of t作者: 他很靈活 時(shí)間: 2025-3-29 22:57
Genetic Control of Programmed Cell Death in the Nematode ,,occurs during the development of the mammalian fetus, particularly in the fetal brain; in areas of the developing vertebrate nervous system as many as 85% of the developing neurons die. Similarly, about 95% of developing thymocytes die without ever leaving the thymus. Why such cell deaths occur and 作者: Scintillations 時(shí)間: 2025-3-30 02:02
Apoptosis in Inherited Retinal Degenerations,he causes of photoreceptor death. Mutations of many different proteins interfere with normal development of photoreceptors or initiate a process that leads to premature photoreceptor death for obscure reasons. These mutations include the rhodopsin gene at many different sites (reviewed by Berson.), 作者: 出來 時(shí)間: 2025-3-30 05:25 作者: fluffy 時(shí)間: 2025-3-30 09:05
Regulation of Apoptosis by the Transforming Gene Products of Adenovirus,nslation, and DNA replication machinery to reproduce viral proteins and DNA. In rodent cells which are semipermissive for adenovirus infection, cell growth is deregulated but virus replication is ineffective. As the viral infection does not progress to completion, the deregulation of cell growth con作者: COST 時(shí)間: 2025-3-30 15:07
,: Oncogene and Tumour Suppressor Gene,s underscores a major problem facing multicellular organisms: namely, how to allow the rapid proliferation of component cells and at the same time never allow those component cells to engage in competition with each other. In principle, any cell that acquires a growth advantage through mutation shou作者: SENT 時(shí)間: 2025-3-30 17:46
Regulation of Apoptosis in Interleukin-3-Dependent Myeloid Progenitor Cells,in hemopoietins.. To examine signalling pathways which may influence apoptosis in hematopoietic progenitors we have utilized the Interleukin-3 (IL-3) dependent 32Dcl3 murine myeloid cell line. 32D cells are absolutely dependent upon IL-3 for proliferation and survival,. and have no propensity for de作者: 欲望 時(shí)間: 2025-3-31 00:43 作者: anniversary 時(shí)間: 2025-3-31 01:05
Molecular Controls of Cell Cycle Progression Following DNA Damage: Roles of P53 and Ataxia-Telangiere, an understanding of the molecular events involved in the cellular responses to such exposures should provide insights into mechanisms of human carcinogenesis. Much effort over the past 25 years has been focused on how altered nucleotide bases are removed and how the linear integrity of the DNA s
