標(biāo)題: Titlebook: Antiarrhythmic Drugs; Mechanisms of Antiar Günter Breithardt,Martin Borggrefe,Gerhard Hindric Book 1995 Springer-Verlag Berlin Heidelberg 1 [打印本頁] 作者: Jefferson 時間: 2025-3-21 16:06
書目名稱Antiarrhythmic Drugs影響因子(影響力)
書目名稱Antiarrhythmic Drugs影響因子(影響力)學(xué)科排名
書目名稱Antiarrhythmic Drugs網(wǎng)絡(luò)公開度
書目名稱Antiarrhythmic Drugs網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Antiarrhythmic Drugs被引頻次
書目名稱Antiarrhythmic Drugs被引頻次學(xué)科排名
書目名稱Antiarrhythmic Drugs年度引用
書目名稱Antiarrhythmic Drugs年度引用學(xué)科排名
書目名稱Antiarrhythmic Drugs讀者反饋
書目名稱Antiarrhythmic Drugs讀者反饋學(xué)科排名
作者: Deduct 時間: 2025-3-21 22:39
Die Physiologie der peripheren Nerven I type [2]. The fundamental question that should be answered is whether better results may be expected from the new approach of using drugs prolonging the action potential duration. Is this new hope justified?作者: 飛來飛去真休 時間: 2025-3-22 02:13
being about pharmacological treatment to a situation in which there is now marked uncertainty and general apprehension about the role of antiarrhythmic drugs. Until relatively recently the prevailing concept in antiarrhythmic therapy was that arrhythmias could be controlled by drugs which slowed con作者: forecast 時間: 2025-3-22 07:33 作者: 使閉塞 時間: 2025-3-22 10:21
Jan Niklas Collet,Jan-Hendrik Herbstles at those site(s), may be an important tool to understand variability in response to antiarrhythmic drug therapy. Such understanding, in turn, may lead to an improved ability to predict patients at risk for serious adverse events, as well as to adjust doses to maximize efficacy.作者: 精致 時間: 2025-3-22 14:54
Interaction of Transient Ischemia with Antiarrhythmic Drugsic reflexes, as well as direct damage to the cell membrane, its constituent channels, pumps and gap junctions, and the supporting collagen matrix. Preexisting scarring due to myocardial infarction, cardiac hypertrophy, congestive heart failure, pulmonary disease and the like may contribute to the arrhythmogenic potential of transient ischemia.作者: 邪惡的你 時間: 2025-3-22 18:50
The Study of Pharmacokinetics and Pharmacodynamics as a Tool for Understanding Mechanisms of Antiarrles at those site(s), may be an important tool to understand variability in response to antiarrhythmic drug therapy. Such understanding, in turn, may lead to an improved ability to predict patients at risk for serious adverse events, as well as to adjust doses to maximize efficacy.作者: 愚蠢人 時間: 2025-3-23 00:37
Pharmacology of T and L Type Ca2+ Channels in Cardiac Tissueture describing the pharmacology of these two cardiac Ca channels is limited, particularly in reference to the pharmacology of the T type channel. Therefore, the emphasis here is to discuss the known pharmacology of both channels with particular attention to the T type Ca channel.作者: 拍翅 時間: 2025-3-23 03:38
A Shift from Class I to Class III Drugs in the Medical Treatment of Arrhythmias: Wishful Thinking? I type [2]. The fundamental question that should be answered is whether better results may be expected from the new approach of using drugs prolonging the action potential duration. Is this new hope justified?作者: fertilizer 時間: 2025-3-23 07:33 作者: Osteoarthritis 時間: 2025-3-23 11:09 作者: Cocker 時間: 2025-3-23 14:41
Use Dependence and Reverse Use Dependence of Antiarrhythmic Agents: Pro- and Antiarrhythmic Actionsod stops, while in tachycardia the heart contracts so frequently that it contracts before it has properly filled. As a result cardiac output is very low. Because of the seriousness of these two arrhythmias, I concentrate on the electropharmacology of agents that interfere with tachycardias and fibrillation.作者: 加強(qiáng)防衛(wèi) 時間: 2025-3-23 18:51 作者: 污穢 時間: 2025-3-24 01:57 作者: 案發(fā)地點 時間: 2025-3-24 02:25 作者: Expediency 時間: 2025-3-24 06:52 作者: Ventilator 時間: 2025-3-24 14:29
Sodium Channel Blockade as an Antiarrhythmic Mechanismly lead to safer, more effective treatment. This paper reviews the normal function of the sodium channel, the mechanism(s) of its blockade by drugs and the implication of the blocking mechanisms to the clinical use of these drugs.作者: 狼群 時間: 2025-3-24 17:11 作者: receptors 時間: 2025-3-24 21:13
Experimental Models for the Assessment of Antiarrhythmic and Proarrhythmic Drug Action: Traditional Obviously, there is a need for an effective form of therapy capable of safely treating patients at risk of sudden cardiac death and therefore, a priori, a need for appropriate animal models for preclinical drug testing.作者: Indecisive 時間: 2025-3-25 01:21
Book 1995t pharmacological treatment to a situation in which there is now marked uncertainty and general apprehension about the role of antiarrhythmic drugs. Until relatively recently the prevailing concept in antiarrhythmic therapy was that arrhythmias could be controlled by drugs which slowed conduction or作者: 裂隙 時間: 2025-3-25 03:24
Sodium Channel Blockade as an Antiarrhythmic Mechanismsthetic-class drugs are the most widely prescribed antiarrhythmic agents. They exert their antiarrhythmic effect by blockade of the inward sodium current [1]. The adverse impact of drug treatment on survival of patients in Cardiac Arrhythmia Suppression Trial and in patients with atrial fibrillation作者: conception 時間: 2025-3-25 09:38 作者: 摻假 時間: 2025-3-25 15:20
Molecular Biology and Ion Channel Biophysics: Their Role in Rational Pharmacological Antiarrhythmic in the sophistication of the basic research tools available to study the heart and its electrical activity. First, the introduction of the patch clamp technique (Hamill et al. 1981) made it relatively easy to record membrane currents in single cardiac myocytes from a variety of species, without conc作者: 幸福愉悅感 時間: 2025-3-25 17:19 作者: Bone-Scan 時間: 2025-3-25 21:45 作者: Diaphragm 時間: 2025-3-26 02:45 作者: Lacerate 時間: 2025-3-26 04:47 作者: FECT 時間: 2025-3-26 11:31
Management of Patients with Sustained Ventricular Tachyarrhythmias: Different Clinical Studies, Difftime of sudden death, approximately 80 % of deaths are due to ventricular tachyarrhythmias [1–3]. In addition, approximately 3–6% of postmyocardial infarction patients develop monomorphic ventricular tachycardia that does not lead to cardiac arrest. Thus, overall, approximately 400 000 patients each作者: GEON 時間: 2025-3-26 14:02 作者: BARB 時間: 2025-3-26 16:56 作者: 最低點 時間: 2025-3-26 22:13 作者: HERTZ 時間: 2025-3-27 01:32
Experimental Models for the Assessment of Antiarrhythmic and Proarrhythmic Drug Action: Traditional may be defined as death which is nonviolent, unexpected, witnessed and instantaneous or occurs within a few minutes of an abrupt change in a previous clinical state [2]. Sudden cardiac death refers to such death with a cardiac cause, primarily ventricular fibrillation, and is a major health care pro作者: Dictation 時間: 2025-3-27 07:35
Influence of the Autonomic Nervous System on the Action of Antiarrhythmic Drugsients, have eventually turned the interest toward arrhythmogenic interactions that often have not been foreseen or, at least, have not been thoroughly analyzed. As an example, when the Task Force of the Working Group on Arrhythmias of the European Society Cardiology called attention to the fact that作者: 公社 時間: 2025-3-27 11:03 作者: incubus 時間: 2025-3-27 17:30 作者: SIT 時間: 2025-3-27 21:30 作者: 音樂會 時間: 2025-3-28 00:53 作者: GENUS 時間: 2025-3-28 04:11
Antiarrhythmic Drug Action: Modulation by Disease and Injury been disappointing. While some drugs can achieve high-grade suppression of extrasystoles, their effects on other components of vulnerability to sustained ventricular tachyarrhythmias may even result in a net increase in risk as demonstrated by the Cardiac Arrhythmia Suppression Trial (CAST) [9] and作者: 劇毒 時間: 2025-3-28 08:55 作者: Truculent 時間: 2025-3-28 13:50
Influence of the Autonomic Nervous System on the Action of Antiarrhythmic Drugs experimental observation that flecainide has the potential to actually favor the onset of life-threatening arrhythmias when acute myocardial ischemia is accompanied by sympathetic activation [2] (Fig. 1).作者: 寒冷 時間: 2025-3-28 17:48 作者: cauda-equina 時間: 2025-3-28 19:27
Peripheres vegetatives Nervensystem in action potential waveform (Antzelevich et al. 1991) and their modification by disease (Furukawa et al. 1991; Beuckelmann et al. 1993). Finally, the capacity to resolve current flow at the single channel level provided an opportunity to obtain direct information about the channel gating process a作者: Ophthalmoscope 時間: 2025-3-29 02:07 作者: OFF 時間: 2025-3-29 06:06 作者: Stress 時間: 2025-3-29 07:23 作者: 自制 時間: 2025-3-29 14:25
Einführung in die Physiologische Embryologie experimental observation that flecainide has the potential to actually favor the onset of life-threatening arrhythmias when acute myocardial ischemia is accompanied by sympathetic activation [2] (Fig. 1).作者: 盡責(zé) 時間: 2025-3-29 17:03
Einführung in die Physiologische Embryologie with a familial pattern in children and young adults. The sporadic occurrence of the syndrome without a familial pattern has also been described [197]. In contrast, the acquired form usually occurs in older patients after administration of drugs or other inverventions and circumstances capable of p作者: Meditative 時間: 2025-3-29 21:13 作者: Mechanics 時間: 2025-3-30 00:18
inical prac- tice, but also on the whole approach of those involved in the research, development and regulation of antiarrhythmic drugs. The results of the CAST trial, designed to redress the shortcomings of earlier trials which had failed to demonstrate the anticipated improvement in mortality post-myocardia978-3-642-85626-6978-3-642-85624-2作者: atopic 時間: 2025-3-30 07:46 作者: muffler 時間: 2025-3-30 11:16 作者: pericardium 時間: 2025-3-30 13:17
Die Physiologie der peripheren Nerventhe L type Ca channel (i.) and the T type Ca channel (i.), is vast. It is not the intent of this chapter to review this exhaustive literature; however, initially we describe some of the physiological and biophysical facts essential for the study of channel pharmacology. On the other hand, the litera作者: Acetaminophen 時間: 2025-3-30 19:53 作者: Jacket 時間: 2025-3-30 23:24
Die Physiologie der ?inneren Sekretion“rdium. Evidence has been advanced in support of the existence of at least four electrophysiologically distinct cell types in the canine ventricle including: epicardial, M, endocardial and Purkinje cells. A number of studies have described distinctions between ventricular epicardium and endocardium [作者: 克制 時間: 2025-3-31 01:05
Die Physiologie der peripheren Nervenrugs prolonging the action potential duration. The immediate reason was the Cardiac Arrhythmia Suppression Trial (CAST) study (1989), which showed an increased mortality in patients treated for premature ventricular extrasystoles following myocardial infarction with drugs representative of the class作者: ACME 時間: 2025-3-31 08:32
https://doi.org/10.1007/978-3-662-36786-5on is slow. With some delay it enters the His-Purkinje system which quickly activates the ventricular myocardium. When activation of the heart deviates from this normal activation sequence an arrhythmia results. The most serious arrhythmias are those where the normal pumping function of the heart is作者: 天賦 時間: 2025-3-31 10:19
Peripheres vegetatives Nervensysteme accumulation of toxic metabolites, shifts in intracellular and extracellular ionic activities, and modulation of neurohumoral influences and autonomic reflexes, as well as direct damage to the cell membrane, its constituent channels, pumps and gap junctions, and the supporting collagen matrix. Pre
