標(biāo)題: Titlebook: Anoikis; How the Extracellula Steven M. Frisch Book 2021 Springer Nature Switzerland AG 2021 Anoikis.Apoptosis.Cancer.Epithelial-Mesenchyma [打印本頁] 作者: 分類 時間: 2025-3-21 19:27
書目名稱Anoikis影響因子(影響力)
書目名稱Anoikis影響因子(影響力)學(xué)科排名
書目名稱Anoikis網(wǎng)絡(luò)公開度
書目名稱Anoikis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Anoikis被引頻次
書目名稱Anoikis被引頻次學(xué)科排名
書目名稱Anoikis年度引用
書目名稱Anoikis年度引用學(xué)科排名
書目名稱Anoikis讀者反饋
書目名稱Anoikis讀者反饋學(xué)科排名
作者: justify 時間: 2025-3-21 23:26 作者: Infirm 時間: 2025-3-22 02:35
https://doi.org/10.1007/978-3-8349-8069-4f neuroectodermal origin and not epithelial in nature. As such, melanoma cells show key differences in specific parts of pathways leading to anoikis resistance. This chapter will focus on the specific mechanisms involved in anoikis resistance in cutaneous melanoma, a poorly studied tumor in this area.作者: 鍍金 時間: 2025-3-22 07:31 作者: certain 時間: 2025-3-22 08:47 作者: Override 時間: 2025-3-22 16:21
Anoikis Resistance in Melanoma,f neuroectodermal origin and not epithelial in nature. As such, melanoma cells show key differences in specific parts of pathways leading to anoikis resistance. This chapter will focus on the specific mechanisms involved in anoikis resistance in cutaneous melanoma, a poorly studied tumor in this area.作者: EXULT 時間: 2025-3-22 20:57 作者: 放肆的我 時間: 2025-3-22 23:17
kis, in a convenient format.Serves as a unique resource for .Anoikis is defined broadly as apoptosis that is inhibited by appropriate cell-matrix interactions.? Normal and tumor cells vary widely in their sensitivity to anoikis, but, in general, metastatic tumor cells are inevitably anoikis-resistan作者: BOOST 時間: 2025-3-23 02:46
Book 2021ulation by integrins, non-integrin matrix receptors, and the signaling complexes associated with them; b. regulation by metabolic changes occurring in response to attachment/detachment;? c. regulation by oncogenes and tumor suppressor genes d. regulation by tumor microenvironment;? e. regulation by EMT..作者: 信任 時間: 2025-3-23 08:08
d with them; b. regulation by metabolic changes occurring in response to attachment/detachment;? c. regulation by oncogenes and tumor suppressor genes d. regulation by tumor microenvironment;? e. regulation by EMT..978-3-030-73856-3作者: Hla461 時間: 2025-3-23 11:23
Metabolic Contributions to Anoikis-Resistance in Metastatic Dissemination,c and apoptogenic pathways converge on the mitochondria. Thus, accumulating evidence now links cellular metabolism after matrix detachment to both anoikis induction and anoikis resistance. Accordingly, the metabolic regulation of anoikis has become a topic of interest in cancer metastasis, since met作者: ALLEY 時間: 2025-3-23 14:31 作者: 喚起 時間: 2025-3-23 18:26
The Intersection of Anoikis Resistance and Fatty Acid Metabolism in Cancer, cells enhance fatty acid oxidation activity to avoid anoikis and successfully colonize a secondary site. Targeting metabolic reprogramming and fatty acid oxidation is a feasible approach to inhibit cancer progression. Emphasizing ovarian cancer as a model system, this chapter will focus on the meta作者: 暫時休息 時間: 2025-3-24 00:01 作者: Orgasm 時間: 2025-3-24 03:26
Anoikis and the Human Gut Epithelium in Health and Disease,indings concerning the regulation of anoikis in hIECs, as well as those pertaining to anoikis deregulation in CRC and IBD—with a partiality geared towards integrins, E-cadherins and the signaling they enact to this effect.作者: 空中 時間: 2025-3-24 09:25
Anoikis Mediated by Stress-Activated MAPK Signaling Pathways,ndantly to increase BIM and BMF apoptotic activity. The mechanism of cell death is mediated by the BAX/BAK-dependent intrinsic cell death pathway. Blocking either the JNK or the p38 MAPK pathway suppresses epithelial cell anoikis in vitro and prevents luminal clearance of mammary epithelial ducts in作者: 松緊帶 時間: 2025-3-24 12:25
,The TGFβ and Androgen Receptor Signaling Pathways Converge to Support Anoikis Resistance in Triple tly studied in TNBC. Gene expression profiling combined with reverse phase protein array of TNBC cells in anchorage independent culture conditions revealed increased components of the TGFβ signaling pathway at the level of ligands, receptors and phosphorylated, nuclear Smad 3 localization and activi作者: Spangle 時間: 2025-3-24 17:42 作者: FOR 時間: 2025-3-24 19:29 作者: Hamper 時間: 2025-3-25 00:03 作者: 沉積物 時間: 2025-3-25 04:00
https://doi.org/10.1007/978-3-8349-8069-4ike lineages. In this chapter, we review the Shc proteins as a specific example of how GTPases coordinate cytoskeletal dynamics, adhesion, and cell fate, and explore the ramifications of Shc dysregulation for malignant transformation.作者: Moderate 時間: 2025-3-25 08:15
https://doi.org/10.1007/978-3-8349-8069-4indings concerning the regulation of anoikis in hIECs, as well as those pertaining to anoikis deregulation in CRC and IBD—with a partiality geared towards integrins, E-cadherins and the signaling they enact to this effect.作者: Duodenitis 時間: 2025-3-25 12:48 作者: 新字 時間: 2025-3-25 19:00
https://doi.org/10.1007/978-3-8349-8069-4tly studied in TNBC. Gene expression profiling combined with reverse phase protein array of TNBC cells in anchorage independent culture conditions revealed increased components of the TGFβ signaling pathway at the level of ligands, receptors and phosphorylated, nuclear Smad 3 localization and activi作者: 極深 時間: 2025-3-25 20:22
Steven M. FrischIs the first to summarize the past 26 years of research in this field.Provides both mechanistic and translational information regarding anoikis, in a convenient format.Serves as a unique resource for 作者: BLINK 時間: 2025-3-26 00:14 作者: 賞錢 時間: 2025-3-26 04:55
https://doi.org/10.1007/978-3-030-73856-3Anoikis; Apoptosis; Cancer; Epithelial-Mesenchymal Transition; cell-matrix signalling作者: Concrete 時間: 2025-3-26 09:33 作者: 脫毛 時間: 2025-3-26 13:52 作者: 強(qiáng)制性 時間: 2025-3-26 20:27
Diskussion der Methode und der Ergebnisse,M) attachment [., .]. Cancer cells, on the other hand, can circumvent this process which enables them to survive matrix-deprivation, disseminate through blood or lymphatics, and attach to distant organs with different matrix compositions [.]. Part of their ability to do so is linked to the metabolic作者: 真實(shí)的你 時間: 2025-3-26 22:20
https://doi.org/10.1007/978-3-8349-8069-4lular matrix induces cellular stressors, including nutrient-deprivation and oxidative stress, and this elevated cellular stress leads to apoptosis. In contrast, cancer cells often have an increased capacity to adapt to cellular stressors, and these adaptations include metabolic reprogramming to acco作者: 職業(yè)拳擊手 時間: 2025-3-27 03:58 作者: Processes 時間: 2025-3-27 09:00 作者: 不能根除 時間: 2025-3-27 11:42 作者: Ballerina 時間: 2025-3-27 17:34 作者: Biguanides 時間: 2025-3-27 20:09 作者: Nonflammable 時間: 2025-3-28 00:46 作者: indenture 時間: 2025-3-28 04:11
https://doi.org/10.1007/978-3-8349-8069-4ween proliferation and the newly characterized biologic process called cell extrusion. Cell extrusion ejects live cells from overcrowded epithelia, where these cells may or may not die by anoikis, depending on their genome and microenvironment. Their failure to undergo anoikis may promote tumor meta作者: 觀點(diǎn) 時間: 2025-3-28 08:05 作者: 神圣不可 時間: 2025-3-28 11:38
Metabolic Reprogramming and the Control of Anoikis Resistance in Cancer,M) attachment [., .]. Cancer cells, on the other hand, can circumvent this process which enables them to survive matrix-deprivation, disseminate through blood or lymphatics, and attach to distant organs with different matrix compositions [.]. Part of their ability to do so is linked to the metabolic作者: 最低點(diǎn) 時間: 2025-3-28 15:42
The Intersection of Anoikis Resistance and Fatty Acid Metabolism in Cancer,lular matrix induces cellular stressors, including nutrient-deprivation and oxidative stress, and this elevated cellular stress leads to apoptosis. In contrast, cancer cells often have an increased capacity to adapt to cellular stressors, and these adaptations include metabolic reprogramming to acco作者: separate 時間: 2025-3-28 21:28 作者: 四目在模仿 時間: 2025-3-29 01:05 作者: extrovert 時間: 2025-3-29 06:06
The Roles of Anoikis in Cervical Cancer,ity in patients with cervical cancer. Resistance to anoikis is an important component of the multi-step cancer metastasis process. During this process, cancer cells utilize mechanisms to trigger survival signals and suppress death pathways, and take advantage of miRNAs or specific HPV oncogene varia作者: Corroborate 時間: 2025-3-29 07:22
