標(biāo)題: Titlebook: Animal Models of Neurological Disease, II; Metabolic Encephalop Alan A. Boulton,Glen B. Baker,Roger F. Butterworth Book 1992 Springer Scien [打印本頁] 作者: chondrocyte 時(shí)間: 2025-3-21 17:36
書目名稱Animal Models of Neurological Disease, II影響因子(影響力)
書目名稱Animal Models of Neurological Disease, II影響因子(影響力)學(xué)科排名
書目名稱Animal Models of Neurological Disease, II網(wǎng)絡(luò)公開度
書目名稱Animal Models of Neurological Disease, II網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Animal Models of Neurological Disease, II被引頻次
書目名稱Animal Models of Neurological Disease, II被引頻次學(xué)科排名
書目名稱Animal Models of Neurological Disease, II年度引用
書目名稱Animal Models of Neurological Disease, II年度引用學(xué)科排名
書目名稱Animal Models of Neurological Disease, II讀者反饋
書目名稱Animal Models of Neurological Disease, II讀者反饋學(xué)科排名
作者: Obverse 時(shí)間: 2025-3-21 23:54
Animal Models of Brain Hypoxia, brain function has implications for understanding other metabolic encephalopathies as well as aging and age-related disorders, such as Alzheimer’s disease. Utilizing a variety of models of hypoxia is necessary to determine the effects of hypoxia on brain function and to test hypotheses about the un作者: 褲子 時(shí)間: 2025-3-22 02:04
Animal Models of the Wernicke-Korsakoff Syndrome,nicke-Korsakoff syndrome. Disturbances of thiamine metabolism may also play a role in other human diseases, such as infantile subacute necrotizing encephalomyelopathy (Leigh Disease), as well as in some forms of inherited ataxias. One of the characteristics of a number of these thiamine-deficiency d作者: 發(fā)生 時(shí)間: 2025-3-22 05:02
Chemically Induced Models of Seizures,tive review of all chemical models, but rather a “how to” description of some of the more popular models that have yielded important data concerning mechanisms of seizure propagation. This chapter will examine seizure models that are directly related to chemical or drug administration, and not, for 作者: Demulcent 時(shí)間: 2025-3-22 11:58 作者: Decrepit 時(shí)間: 2025-3-22 14:30 作者: blithe 時(shí)間: 2025-3-22 18:15 作者: 新手 時(shí)間: 2025-3-22 22:28
,Animal Models for Reye’s Syndrome, entity, but one that at that time was associated with an 80% mortality rate (.). Although the apparent incidence and outcome of the disease have improved, it continues to be one of the major causes of noninflammatory neurologic death after a viral illness in children (.; .).作者: 大暴雨 時(shí)間: 2025-3-23 05:11
Animal Models of Niacin-Nicotinamide Deficiency, function of niacin was discovered. The recognition of pellagra as an endemic disease in the US dates from Searcy’s report in 1907 (.) describing 88 cases of dementia in the Mount Vernon, Alabama Insane Asylum. Extensive knowledge about the course of pellagra has since been obtained and this disease作者: Anecdote 時(shí)間: 2025-3-23 09:36
Pyridoxine Deficiency,al, and pyridoxamine, with similar physiological actions. They are referred to as vitamin B. vitamers. Pyridoxine is used synonymously with vitamin B.. Pyridoxal phosphate (PLP) is the major coenzymatic form of pyridoxine. There are over 100 known pyridoxal 5′-phosphate-dependent reactions, most of 作者: Aspirin 時(shí)間: 2025-3-23 11:25
Animal Models of Hereditary Hyperammonemias, of the affected individuals, causing an abnormal accumulation of ammonia in the blood. These altered gene products primarily include the enzymes and transport proteins that are part of the Krebs-Hensleit ornithine-urea cycle (.), the principal metabolic pathway for the disposal of ammonia in the ma作者: profligate 時(shí)間: 2025-3-23 17:48 作者: 不可侵犯 時(shí)間: 2025-3-23 20:41
On Causality and Control in Economic Systemsconsiderable study during the past 20 years, and the number of publications related to kindling has increased dramatically (.). Despite efforts to understand kindling and the many parameters that have been explored, the mechanism underlying the phenomenon remains elusive.作者: 浪費(fèi)物質(zhì) 時(shí)間: 2025-3-23 22:41 作者: flamboyant 時(shí)間: 2025-3-24 02:48
Computing Optimal Tolls in a Money Economyephalomyelopathy (Leigh Disease), as well as in some forms of inherited ataxias. One of the characteristics of a number of these thiamine-deficiency disorders is the predilection to neuropathologic damage of specific regions of the central nervous system (CNS).作者: 蔑視 時(shí)間: 2025-3-24 08:43
Arnold H. Q. M. Merkies,Marjan W. Hofkesscussed in their respective chapters. In the present chapter, we will discuss several genetic models of epilepsy. These models have unique characteristics that relate to the onset and course of seizure activity.作者: 藐視 時(shí)間: 2025-3-24 13:41
Animal Models of the Wernicke-Korsakoff Syndrome,ephalomyelopathy (Leigh Disease), as well as in some forms of inherited ataxias. One of the characteristics of a number of these thiamine-deficiency disorders is the predilection to neuropathologic damage of specific regions of the central nervous system (CNS).作者: Endearing 時(shí)間: 2025-3-24 18:16 作者: enlist 時(shí)間: 2025-3-24 22:45 作者: Conflict 時(shí)間: 2025-3-25 02:59
Animal Models of Hereditary Hyperammonemias,other mutations that hinder the availability of energy-producing substrates necessary for ammonia detoxification at the mitochondrial level have also been included among the list of secondary hyperammonias.作者: 耕種 時(shí)間: 2025-3-25 04:06
Book 1992orders. Because of increasing public concern over the ethical treatment of animals in research, we felt it incumbent upon us to include this general preface to these volumes in order to indicate why we think further - search using animals is necessary and why animal models of psychiatric disorders, 作者: 箴言 時(shí)間: 2025-3-25 08:02
Chemically Induced Models of Seizures,example, those that may result from a chemical imbalance, such as produced by hypoxia or hypoglycemia. Finally, in vivo seizure models will be described. Important information can be derived from in vitro or slice studies, but these models are beyond the scope of this chapter.作者: 個(gè)人長篇演說 時(shí)間: 2025-3-25 15:30 作者: 點(diǎn)燃 時(shí)間: 2025-3-25 18:06
Alan A. Boulton,Glen B. Baker,Roger F. Butterworth作者: glacial 時(shí)間: 2025-3-25 20:56
Book 1992 the animals outweighs the anticipated gains for human welfare, then the research is not ethical and should not be carried out. It is imperative that each individual researcher examine his/her own research from a critical moral standpoint - fore engaging in it, taking into consideration the animals’作者: 不要不誠實(shí) 時(shí)間: 2025-3-26 01:58 作者: 拋射物 時(shí)間: 2025-3-26 07:02
Growth and Income Distribution in Iranne release impairs mental function, whereas the excessive release of dopamine and glutamate damages cells postsynaptically. Fundamental alterations in calcium homeostasis, particularly the ability of mitochondria to buffer calcium, appear to underlie these deficits. Furthermore, these changes in cal作者: 組成 時(shí)間: 2025-3-26 10:23
https://doi.org/10.1007/978-3-642-51675-7s channels or simply reverses the direction of flow in the existing ones. These shunts permit toxins of intestinal origin to bypass the liver into the systemic circulation; hence the term “portal-systemic encephalopathy.”作者: 纖細(xì) 時(shí)間: 2025-3-26 14:57 作者: 性行為放縱者 時(shí)間: 2025-3-26 20:19
https://doi.org/10.1007/978-3-642-57615-7n view of this, during progressive pyridoxine deficiency, the PLP-dependent enzymes are decreased to various extents, from no decrease to almost a complete depletion. The decreases in the activities of glutamic acid and 5-hydroxytryptophan decarboxylases during pyridoxine deficiency result in the re作者: idiopathic 時(shí)間: 2025-3-26 23:30 作者: 適宜 時(shí)間: 2025-3-27 02:51 作者: KEGEL 時(shí)間: 2025-3-27 08:38
Animal Models of Hepatic Encephalopathies,s channels or simply reverses the direction of flow in the existing ones. These shunts permit toxins of intestinal origin to bypass the liver into the systemic circulation; hence the term “portal-systemic encephalopathy.”作者: 獨(dú)行者 時(shí)間: 2025-3-27 09:26 作者: 極端的正確性 時(shí)間: 2025-3-27 14:33
Pyridoxine Deficiency,n view of this, during progressive pyridoxine deficiency, the PLP-dependent enzymes are decreased to various extents, from no decrease to almost a complete depletion. The decreases in the activities of glutamic acid and 5-hydroxytryptophan decarboxylases during pyridoxine deficiency result in the re作者: craving 時(shí)間: 2025-3-27 18:12
0893-2336 tive that each individual researcher examine his/her own research from a critical moral standpoint - fore engaging in it, taking into consideration the animals’978-1-4899-4389-7978-1-59259-627-0Series ISSN 0893-2336 Series E-ISSN 1940-6045 作者: CHANT 時(shí)間: 2025-3-27 23:58
Econometric Contributions to Public Policyus rupture of an intracranial artery with hemorrhage into the brain parenchyma or subarachnoid space (.). Brain infarction, a localized lesion caused by the occlusion of a brain vessel (usually an artery), accounts for about 75% of the lesions produced by stroke, with brain hemorrhage (11%) and suba作者: 事先無準(zhǔn)備 時(shí)間: 2025-3-28 04:30
Growth and Income Distribution in Iran brain function has implications for understanding other metabolic encephalopathies as well as aging and age-related disorders, such as Alzheimer’s disease. Utilizing a variety of models of hypoxia is necessary to determine the effects of hypoxia on brain function and to test hypotheses about the un作者: 嚴(yán)厲譴責(zé) 時(shí)間: 2025-3-28 08:36 作者: 結(jié)果 時(shí)間: 2025-3-28 11:24 作者: A簡潔的 時(shí)間: 2025-3-28 14:38
On Causality and Control in Economic Systemsh small burning twigs ultimately produce a large fire, repeated subconvulsive stimulation of the brain by electrical current or pharmacological agents produces seizure behavior of gradually increasing intensity that culminates in a full, clonic, motor seizure. This phenomenon has been the object of 作者: upstart 時(shí)間: 2025-3-28 20:50
Arnold H. Q. M. Merkies,Marjan W. Hofkes drug and chemical means, electroshock, and kindling models. Each of these methods of seizure induction has advantages and disadvantages, which are discussed in their respective chapters. In the present chapter, we will discuss several genetic models of epilepsy. These models have unique characteris作者: irreparable 時(shí)間: 2025-3-29 02:17
https://doi.org/10.1007/978-3-642-51675-7major forms: (.) Associated with ., a clinical syndrome resulting from severe inflammatory and/or necrotic liver disease of rapid onset. The neurological disorder progresses from altered mental status to coma, generally within hours or days. Death frequently results from increased intracranial press作者: hidebound 時(shí)間: 2025-3-29 04:08
https://doi.org/10.1007/978-3-642-51675-7 entity, but one that at that time was associated with an 80% mortality rate (.). Although the apparent incidence and outcome of the disease have improved, it continues to be one of the major causes of noninflammatory neurologic death after a viral illness in children (.; .).作者: Femish 時(shí)間: 2025-3-29 07:15
A. H. Q. M. Merkies,Th. E. Nijman function of niacin was discovered. The recognition of pellagra as an endemic disease in the US dates from Searcy’s report in 1907 (.) describing 88 cases of dementia in the Mount Vernon, Alabama Insane Asylum. Extensive knowledge about the course of pellagra has since been obtained and this disease
