標(biāo)題: Titlebook: Angiotensin and the Heart; H. Grobecker,Gerd Heusch,B. E. Strauer Book 1993 Dr. Dietrich Steinkopff Verlag GmbH & Co. KG, Darmstadt 1993 A [打印本頁] 作者: 娛樂某人 時(shí)間: 2025-3-21 19:30
書目名稱Angiotensin and the Heart影響因子(影響力)
書目名稱Angiotensin and the Heart影響因子(影響力)學(xué)科排名
書目名稱Angiotensin and the Heart網(wǎng)絡(luò)公開度
書目名稱Angiotensin and the Heart網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Angiotensin and the Heart被引頻次
書目名稱Angiotensin and the Heart被引頻次學(xué)科排名
書目名稱Angiotensin and the Heart年度引用
書目名稱Angiotensin and the Heart年度引用學(xué)科排名
書目名稱Angiotensin and the Heart讀者反饋
書目名稱Angiotensin and the Heart讀者反饋學(xué)科排名
作者: LAVE 時(shí)間: 2025-3-21 22:48
European Union Protection Systemality of life appear modest. During prolonged administration to patients with ischemic left-ventricular dysfunction, ACE-I also significantly reduce the incidence of new ischemic events (myocardial infarction, unstable angina).作者: 冷淡一切 時(shí)間: 2025-3-22 03:04
https://doi.org/10.1007/978-3-642-02242-5hnology have resulted in a high primary success rate (over 90%) and a low compilation rate (death, non-fatal myocardial infarction; 4–5%) (9). However, the late restenosis rate (17% to 40%) still limits the long-term benefit of the procedure (21, 23, 47, 52).作者: CRUMB 時(shí)間: 2025-3-22 05:13 作者: 不吉祥的女人 時(shí)間: 2025-3-22 12:34
Angiotensin-converting enzyme inhibitor in a human model of restenosishnology have resulted in a high primary success rate (over 90%) and a low compilation rate (death, non-fatal myocardial infarction; 4–5%) (9). However, the late restenosis rate (17% to 40%) still limits the long-term benefit of the procedure (21, 23, 47, 52).作者: 偏離 時(shí)間: 2025-3-22 15:24 作者: 充足 時(shí)間: 2025-3-22 19:07
https://doi.org/10.1007/978-3-642-72497-8Angiotensin II; Renin-Angiotensin-System; cardiovascular; circulation; coronary artery disease; heart; hyp作者: 驚呼 時(shí)間: 2025-3-22 21:25 作者: palliate 時(shí)間: 2025-3-23 04:18
Overview: 978-3-7985-0936-8978-3-642-72497-8作者: 征兵 時(shí)間: 2025-3-23 05:40 作者: 同步左右 時(shí)間: 2025-3-23 13:01
https://doi.org/10.1007/978-3-030-69583-5oint to a role in long-term cardiovascular regulation for endogenous RAS in target tissues. This article focuses on the multiple effects of tissue angiotensin enzyme (ACE) and angiotensin II (Ang II), its active peptide product. Ang II has been shown to be a potent growth factor in vascular smooth m作者: 難管 時(shí)間: 2025-3-23 15:36 作者: bizarre 時(shí)間: 2025-3-23 19:44 作者: heartburn 時(shí)間: 2025-3-23 23:39
https://doi.org/10.1007/978-3-030-54001-2tors have also been used in the treatment of patients with coronary artery disease (CAD), since from experimental data an antiischemic action of these agents is suggested. Antiischemic effects of ACE-inhibitors may be exerted through a reduction of myocardial oxygen demand, by a reduction of angiote作者: 細(xì)胞學(xué) 時(shí)間: 2025-3-24 03:37 作者: Ligneous 時(shí)間: 2025-3-24 06:41 作者: Phenothiazines 時(shí)間: 2025-3-24 12:14
EU Islands and the Clean Energy Transition patients with arterial hypertension often have symptoms of angina pectoris and a positive exercise tolerance test. The angina pectoris-symptoms in patients with arterial hypertension are due to functional and structural alterations of the coronary microcirculation. Consequently, an antihypertensive作者: 胎兒 時(shí)間: 2025-3-24 15:27 作者: 引起痛苦 時(shí)間: 2025-3-24 21:08
The Policy Paradigm from 1997 to 2007,ocardium have been summarized under the term “l(fā)eft ventricular remodeling” post myocardial infarction. An increase in left ventricular volume may be a consequence, and associated with an adverse prognosis. Although left ventricular dilatation may increase stroke volume and, thus, be compensatory at 作者: muffler 時(shí)間: 2025-3-25 00:20
The Policy Paradigm from 1997 to 2007,nd positive inotropic agent. This has raised the possibility that angiotensin converting enzyme (ACE) inhibitors might be “cardioprotective” (that is, might attenuate myocardial injury, dysfunction and necrosis) in the setting of acute ischemia and infarction. Captopril, enalapril and ramipril have,作者: 強(qiáng)所 時(shí)間: 2025-3-25 05:08
Lessons from Migration after EU Enlargementf angiotensin II ii) decreased metabolism of bradykinin, iii) antioxidant activity, and iv) possibly other unknown mechanisms. Reduced formation of angiotensin II should be beneficial because this peptide exerts several actions that are potentially detrimental to the ischemic/reperfused myocardium, 作者: humectant 時(shí)間: 2025-3-25 09:44
https://doi.org/10.1007/978-3-642-02242-5ass grafting (CABG) to restore coronary blood flow in patients with obstructive coronary artery disease (15). Increased experience and advances in technology have resulted in a high primary success rate (over 90%) and a low compilation rate (death, non-fatal myocardial infarction; 4–5%) (9). However作者: 溝通 時(shí)間: 2025-3-25 13:50 作者: 小樣他閑聊 時(shí)間: 2025-3-25 15:56 作者: Hyaluronic-Acid 時(shí)間: 2025-3-25 22:06 作者: Ambulatory 時(shí)間: 2025-3-26 00:18
Local expression and pathophysiological role of renin-angiotensin in the blood vessels and heartoint to a role in long-term cardiovascular regulation for endogenous RAS in target tissues. This article focuses on the multiple effects of tissue angiotensin enzyme (ACE) and angiotensin II (Ang II), its active peptide product. Ang II has been shown to be a potent growth factor in vascular smooth m作者: 禁止 時(shí)間: 2025-3-26 06:16 作者: Water-Brash 時(shí)間: 2025-3-26 10:18
Local and neurohumoral control of coronary blood flowr, despite substantial experimental efforts the responsible mediators are still largely unknown. Adenosine, a purported mediator of local metabolic control of coronary blood flow, is probably only involved in transient flow adaptations, but not in steady-state coronary autoregulation. Even below the作者: Stress 時(shí)間: 2025-3-26 14:50 作者: 處理 時(shí)間: 2025-3-26 20:04 作者: 笨拙的你 時(shí)間: 2025-3-26 21:13
Myocardial perfusion dependent and independent mechanisms of regional myocardial dysfunction in hypeicular (LV) function, it could be of greater importance during periods of stress such as occurs during exercise where increased metabolic demands induced by the stress may not be fully met by increases in coronary blood flow. The impaired subendocardial coronary reserve is caused not only by the hyp作者: SIT 時(shí)間: 2025-3-27 01:21
ACE-inhibitors and coronary microcirculation patients with arterial hypertension often have symptoms of angina pectoris and a positive exercise tolerance test. The angina pectoris-symptoms in patients with arterial hypertension are due to functional and structural alterations of the coronary microcirculation. Consequently, an antihypertensive作者: GEST 時(shí)間: 2025-3-27 08:10 作者: GRUEL 時(shí)間: 2025-3-27 10:42 作者: 斗爭(zhēng) 時(shí)間: 2025-3-27 16:16
“Cardioprotection” by ACE-inhibitors in acute myocardial ischemia and infarction?nd positive inotropic agent. This has raised the possibility that angiotensin converting enzyme (ACE) inhibitors might be “cardioprotective” (that is, might attenuate myocardial injury, dysfunction and necrosis) in the setting of acute ischemia and infarction. Captopril, enalapril and ramipril have,作者: Magnitude 時(shí)間: 2025-3-27 21:46 作者: neolith 時(shí)間: 2025-3-28 00:47
Angiotensin-converting enzyme inhibitor in a human model of restenosisass grafting (CABG) to restore coronary blood flow in patients with obstructive coronary artery disease (15). Increased experience and advances in technology have resulted in a high primary success rate (over 90%) and a low compilation rate (death, non-fatal myocardial infarction; 4–5%) (9). However作者: overture 時(shí)間: 2025-3-28 05:22 作者: critic 時(shí)間: 2025-3-28 06:21
Angiotensin-converting enzyme inhibitors in the treatment of clinical heart failuree, there is convincing evidence that they improve survival and that, in this respect, they are superior to ordinary vasodilators. ACE-I administration also improves New York Heart Association functional class and the left-ventricular function. But their long-term effects on exercise tolerance and qu作者: Entirety 時(shí)間: 2025-3-28 13:02
Basic aspects the cardiac renin-angiotensin system has been made. It is now accepted that the renin-angiotensin system originally detected as a classical humoral system, which secretes active molecules from a gland reaching their receptors of a target organ via the blood, may also reside in many tissues, especia作者: 怒目而視 時(shí)間: 2025-3-28 14:34 作者: 嚴(yán)厲批評(píng) 時(shí)間: 2025-3-28 21:44
Angiotensin, ACE-inhibitors and endothelial control of vasomotor tonederived vasodilator system. Angiotensin II not only can activate vascular smooth muscle cells (where it causes contraction and proliferation), but also endothelial cells. In certain blood vessels, angiotensin II can stimulate prostacyclin production; in addition, angiotensin II activates endothelin 作者: 令人發(fā)膩 時(shí)間: 2025-3-29 00:22 作者: CHASM 時(shí)間: 2025-3-29 04:32
ACE-inhibitors in coronary artery disease?schemia at rest and during exercise, a significant fraction of about 30% of normotensive patients with CAD does not benefit or even deteriorates. Lowering of coronary perfusion pressure and alteration of transmural blood flow distribution may be responsible for this. In patients with left ventricula作者: LEERY 時(shí)間: 2025-3-29 09:51
Molecular mechanisms of cardiac gene expressionexpression but has also significant practical applications for gene therapy. It is now possible to analyze the role of different regulatory gene sequences and identify their corresponding trans-active factors. In addition, direct gene injection makes it possible to study gene expression in a natural作者: 玩笑 時(shí)間: 2025-3-29 12:50 作者: 令人悲傷 時(shí)間: 2025-3-29 16:27 作者: heartburn 時(shí)間: 2025-3-29 23:33 作者: expunge 時(shí)間: 2025-3-30 01:04 作者: 壕溝 時(shí)間: 2025-3-30 07:38 作者: 竊喜 時(shí)間: 2025-3-30 09:14 作者: 胡言亂語 時(shí)間: 2025-3-30 14:01 作者: 慢慢沖刷 時(shí)間: 2025-3-30 19:30
https://doi.org/10.1007/978-3-030-69583-5ddition, cardiac RAS may contribute to the pathophysiology of heart failure. Experimental and clinical studies with ACE inhibitors point to a role for tissue ACE activity in the development of atherosclerosis, as well as cardiac hypertrophy and remodeling.作者: dragon 時(shí)間: 2025-3-30 20:59
https://doi.org/10.1007/978-3-030-54001-2derived vasodilator system. Angiotensin II not only can activate vascular smooth muscle cells (where it causes contraction and proliferation), but also endothelial cells. In certain blood vessels, angiotensin II can stimulate prostacyclin production; in addition, angiotensin II activates endothelin 作者: Loathe 時(shí)間: 2025-3-31 02:21
https://doi.org/10.1007/978-3-030-54001-2ncreases to match the increased oxygen consumption, α-adrenergic coronary constriction remains operative in ischemic myocardium, thus precipitating or contributing to acute myocardial ischemia during sympathetic activation and exercise in experimental animals as well as in patients with stable angin作者: GRIPE 時(shí)間: 2025-3-31 07:52
https://doi.org/10.1007/978-3-030-54001-2schemia at rest and during exercise, a significant fraction of about 30% of normotensive patients with CAD does not benefit or even deteriorates. Lowering of coronary perfusion pressure and alteration of transmural blood flow distribution may be responsible for this. In patients with left ventricula
