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標(biāo)題: Titlebook: Alzheimer’s and Parkinson’s Diseases; Recent Developments Israel Hanin,Mitsuo Yoshida,Abraham Fisher Book 1995 Springer Science+Business Me [打印本頁(yè)]

作者: BID    時(shí)間: 2025-3-21 16:37
書目名稱Alzheimer’s and Parkinson’s Diseases影響因子(影響力)




書目名稱Alzheimer’s and Parkinson’s Diseases影響因子(影響力)學(xué)科排名




書目名稱Alzheimer’s and Parkinson’s Diseases網(wǎng)絡(luò)公開度




書目名稱Alzheimer’s and Parkinson’s Diseases網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Alzheimer’s and Parkinson’s Diseases被引頻次




書目名稱Alzheimer’s and Parkinson’s Diseases被引頻次學(xué)科排名




書目名稱Alzheimer’s and Parkinson’s Diseases年度引用




書目名稱Alzheimer’s and Parkinson’s Diseases年度引用學(xué)科排名




書目名稱Alzheimer’s and Parkinson’s Diseases讀者反饋




書目名稱Alzheimer’s and Parkinson’s Diseases讀者反饋學(xué)科排名





作者: 牢騷    時(shí)間: 2025-3-21 23:45

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作者: 哥哥噴涌而出    時(shí)間: 2025-3-22 14:27
Correlation between Severity of Dementia and Decreased Cerebral Blood Flow in Alzheimer’s Disease anto the age of onset (i.e.; before or after age 65), this view remains controversial and has not been confirmed by all investigators. The neurological basis for this difference also remains undetermined.. The purpose of this study was to investigate the relationships between the perfusion defects and
作者: 送秋波    時(shí)間: 2025-3-22 20:13

作者: 絕種    時(shí)間: 2025-3-22 21:36

作者: 小爭(zhēng)吵    時(shí)間: 2025-3-23 04:43

作者: 舊式步槍    時(shí)間: 2025-3-23 08:04
Cellular Acetylcholine Receptor Expression in the Brain of Patients with Alzheimer’s and Parkinson’sices.. A detailed account of these changes requires a study of neurochemical phenotype of individual neurons as basic elements of networks constituting the substrate of cortical functions.. Examples will be given for cell-type specific AChR localization in normal and diseased human cerebral cortex.
作者: JADED    時(shí)間: 2025-3-23 10:02

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作者: GROWL    時(shí)間: 2025-3-23 19:53
Creutzfeldt-Jakob Disease (CJD) is Not an Infectious DiseaseRecent advances in molecular biology have identified the molecular pathology of the disease, consisting of accumulation of “prion protein”. Epidemiologic studies demonstrate that CJD is in most cases a sporadic disease. Cases with a familial background have been shown to result, in many cases, from
作者: Mystic    時(shí)間: 2025-3-24 01:13

作者: 神經(jīng)    時(shí)間: 2025-3-24 06:11
Modulation of APP Processing by Neurotransmissioner amounts in brains of neuropsychologically normal, aged human subjects. AD-type brain amyloid consists of aggregated A. peptides which are 39–43 amino acid residues in length. A. is derived, by proteolytic processing, from a larger amyloid .-protein precursor (APP), which is a transmembrane glycop
作者: accrete    時(shí)間: 2025-3-24 08:07
Amyloid Precursor Protein (APP) Gene Expression is Controlled by a NFkB/Rel Related Proteince cleavage occurs in the endosomal-lysosomal compartment to generate the entire .A4 peptide sequence; the second which is “non-amyloidogenic” and involves the secretory cleavage of APP at a site within the (3A4 sequence (for a review see Selkoe, D.J., 1993). Since APP metabolism may become disrupte
作者: 割公牛膨脹    時(shí)間: 2025-3-24 13:13

作者: MORPH    時(shí)間: 2025-3-24 18:27

作者: 減弱不好    時(shí)間: 2025-3-24 19:18

作者: adduction    時(shí)間: 2025-3-25 02:57
Stereopsis in Alzheimer’s Disease: Measuring Binocular Eye Movementvisuospatial cognition and skills (Mishkin et al., 1991). Saccadic and smooth pursuit eye movement in AD were studied and the response time was found to be prolonged, while the gain of smooth pursuit eye movement following a sinusoidal moving target was reduced, but the motor control of eye movement
作者: instill    時(shí)間: 2025-3-25 04:36

作者: 投票    時(shí)間: 2025-3-25 09:45

作者: 熔巖    時(shí)間: 2025-3-25 13:10

作者: instill    時(shí)間: 2025-3-25 17:01
Modulation of APP Processing by Neurotransmissionlevels in brain and, at lower levels, in many peripheral tissues. The biological function of APP is unclear but accumulating evidence suggests roles in cell adhesion (Schubert et al., 1989), in neurite outgrowth (Milward et al., 1992), as well as excitoprotective functions via the regulation of intr
作者: 背帶    時(shí)間: 2025-3-25 22:27

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作者: Binge-Drinking    時(shí)間: 2025-3-27 00:49
Die Berechnung der Scheibenkolben,hat the processing of .APP into A. can actually cause Alzheimer’s disease has come from the identification by several laboratories of missense mutations in the .APP gene within and flanking the A. region in affected members of certain families having Alzheimer’s disease or hereditary cerebral hemorr
作者: 油膏    時(shí)間: 2025-3-27 01:10

作者: Hyaluronic-Acid    時(shí)間: 2025-3-27 07:20
https://doi.org/10.1007/978-3-662-30329-0tial contribution of APP gene overexpression to Alzheimer’s disease neuropathology: 1) in trisomy 21 (Down Syndrome) there seems to be a deregulated expression of APP since the difference in APP mRNA levels between patients and controls is higher than the expected 3:2 ratio (Neve et al., 1988; Tanzi
作者: 清楚說話    時(shí)間: 2025-3-27 12:15
Advances in Behavioral Biologyhttp://image.papertrans.cn/a/image/154276.jpg
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作者: 調(diào)整校對(duì)    時(shí)間: 2025-3-28 13:19
https://doi.org/10.1007/978-3-642-50888-2to the age of onset (i.e.; before or after age 65), this view remains controversial and has not been confirmed by all investigators. The neurological basis for this difference also remains undetermined.. The purpose of this study was to investigate the relationships between the perfusion defects and
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作者: 哥哥噴涌而出    時(shí)間: 2025-3-29 09:06
Rotierende Luft- und Kondensatpumpened by Brown and Marsden, 1988; Sagar and Sullivan, 1988). The cognitive deficits occur particularly on “executive” tasks, which are known to be sensitive to frontal lobe dysfunction raising the possibility that a frontal lobe disorder is the basis of the majority of the cognitive impairment in the d
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作者: GREEN    時(shí)間: 2025-3-29 16:31
https://doi.org/10.1007/978-3-322-98722-8Recent advances in molecular biology have identified the molecular pathology of the disease, consisting of accumulation of “prion protein”. Epidemiologic studies demonstrate that CJD is in most cases a sporadic disease. Cases with a familial background have been shown to result, in many cases, from
作者: AMOR    時(shí)間: 2025-3-29 21:53

作者: 舊石器時(shí)代    時(shí)間: 2025-3-30 03:21
https://doi.org/10.1007/978-3-662-02209-2er amounts in brains of neuropsychologically normal, aged human subjects. AD-type brain amyloid consists of aggregated A. peptides which are 39–43 amino acid residues in length. A. is derived, by proteolytic processing, from a larger amyloid .-protein precursor (APP), which is a transmembrane glycop
作者: certain    時(shí)間: 2025-3-30 07:52
https://doi.org/10.1007/978-3-662-30329-0ce cleavage occurs in the endosomal-lysosomal compartment to generate the entire .A4 peptide sequence; the second which is “non-amyloidogenic” and involves the secretory cleavage of APP at a site within the (3A4 sequence (for a review see Selkoe, D.J., 1993). Since APP metabolism may become disrupte
作者: 反話    時(shí)間: 2025-3-30 09:39

作者: Liability    時(shí)間: 2025-3-30 12:55

作者: Hamper    時(shí)間: 2025-3-30 18:27
Articulatory Deficits in Parkinson’s and Huntington’s Disease: An Acoustic Analysision between movements, in Huntington’s (HD) and Parkinson’s disease (PD).. Since both striatal disorders may give rise to speech motor disturbances, the question emerges whether HD and PD dysarthria are characterized by similar articulatory deteriorations as well.
作者: 創(chuàng)作    時(shí)間: 2025-3-30 22:07
Cellular Acetylcholine Receptor Expression in the Brain of Patients with Alzheimer’s and Parkinson’sices.. A detailed account of these changes requires a study of neurochemical phenotype of individual neurons as basic elements of networks constituting the substrate of cortical functions.. Examples will be given for cell-type specific AChR localization in normal and diseased human cerebral cortex.
作者: 痛得哭了    時(shí)間: 2025-3-31 01:06

作者: epicondylitis    時(shí)間: 2025-3-31 07:39

作者: Mingle    時(shí)間: 2025-3-31 13:15
https://doi.org/10.1007/978-1-4757-9145-7Alzheimer; Parkinson; dementia; epidemiology; genetics
作者: 一個(gè)攪動(dòng)不安    時(shí)間: 2025-3-31 14:59
978-1-4757-9147-1Springer Science+Business Media New York 1995
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作者: 懶鬼才會(huì)衰弱    時(shí)間: 2025-4-1 10:35





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