標題: Titlebook: Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics; Konrad Maurer,Peter Riederer,Helmut Beckmann Book 1990 Spr [打印本頁] 作者: Ferret 時間: 2025-3-21 17:54
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics影響因子(影響力)
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics影響因子(影響力)學科排名
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics網(wǎng)絡公開度
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics網(wǎng)絡公開度學科排名
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics被引頻次
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics被引頻次學科排名
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics年度引用
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics年度引用學科排名
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics讀者反饋
書目名稱Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics讀者反饋學科排名
作者: arthrodesis 時間: 2025-3-21 21:46 作者: Maximizer 時間: 2025-3-22 00:35 作者: Rodent 時間: 2025-3-22 05:17 作者: anaerobic 時間: 2025-3-22 10:50
Die Behandlung von Gelenkknorpeldefektene sole change. In dementia of Alzheimer’s type white matter disease in the nature of a selective incomplete white matter infarction was found in around 60% of cases. It was regarded as being most likely due to regional hypoperfusion of the white matter and might therefore be prevented, halted or even partly reversed by therapy.作者: CHANT 時間: 2025-3-22 16:16
https://doi.org/10.1007/978-3-322-98830-0re affected by the neurofibrillary degeneration (NFD). We suggest here that these likely early markers of the NFD might define an in vitro model for the study of the NFD. A such model might be useful to verify the different etiopathogenic hypothesis which have been proposed up to here.作者: 不愛防注射 時間: 2025-3-22 17:03
https://doi.org/10.1007/978-3-322-98830-0tudies have shown that an extra tau species is present in Alzheimer brain and tangle-reactive neurofilament antibodies were found to be more selective towards phosphorylated neurofilaments than those that do not recognise tangles. A fragment of MAP2 was found to be present in tangles.作者: aptitude 時間: 2025-3-22 21:45
https://doi.org/10.1007/978-3-7091-3396-5Alzheimer; Parkinson; SPECT; alzheimer‘s disease; brain; computed tomography (CT); dementia; depression; dia作者: SEVER 時間: 2025-3-23 04:23 作者: FLAGR 時間: 2025-3-23 05:44 作者: Herpetologist 時間: 2025-3-23 12:17
Some philosophical aspects of Alzheimer’s discovery: an American perspectivery of the disease that bears his name. At present that disease remains poorly understood and even less well treated. However, the speed with which new insights into its characteristic clinical and pathological findings now arise gives us optimism that afflicted patients will, someday soon, be better served than they can be at present.作者: implore 時間: 2025-3-23 17:17
A proposed classification of familial Alzheimer’s disease based on analysis of 32 multigeneration pedementia. The classification can briefly be stated as follows I. Early onset FAD with mean age of onset before 50 years; II. Late onset FAD with mean age of onset after 60 years; III. Intermediate/variable onset FAD; IV. Familial dementia with atypical Alzheimer changes; V. FAD without multigeneration involvement.作者: handle 時間: 2025-3-23 20:34
Morphology of white matter, subcortical, dementia in Alzheimer’s diseasee sole change. In dementia of Alzheimer’s type white matter disease in the nature of a selective incomplete white matter infarction was found in around 60% of cases. It was regarded as being most likely due to regional hypoperfusion of the white matter and might therefore be prevented, halted or even partly reversed by therapy.作者: 流動性 時間: 2025-3-23 22:44
An in vitro model for the study of the neurofibrillary degeneration of the Alzheimer typere affected by the neurofibrillary degeneration (NFD). We suggest here that these likely early markers of the NFD might define an in vitro model for the study of the NFD. A such model might be useful to verify the different etiopathogenic hypothesis which have been proposed up to here.作者: Credence 時間: 2025-3-24 05:10 作者: 無能的人 時間: 2025-3-24 09:53
https://doi.org/10.1007/978-3-642-91642-7ry of the disease that bears his name. At present that disease remains poorly understood and even less well treated. However, the speed with which new insights into its characteristic clinical and pathological findings now arise gives us optimism that afflicted patients will, someday soon, be better作者: fixed-joint 時間: 2025-3-24 11:21 作者: 細胞膜 時間: 2025-3-24 16:07 作者: 建筑師 時間: 2025-3-24 19:36 作者: 大雨 時間: 2025-3-24 23:37 作者: compel 時間: 2025-3-25 05:57
Die Behandlung von Gelenkknorpeldefekten current criteria that are critically discussed. The morphologic diagnosis of AD is based on the finding of more than certain minimum age-related numbers of neuritic plaques (NP) and neurofibrillary tangles (NFT) that may occur independently from each other and show specific, often bilaterally symme作者: 減去 時間: 2025-3-25 09:13
Die Behandlung von Gelenkknorpeldefektene sole change. In dementia of Alzheimer’s type white matter disease in the nature of a selective incomplete white matter infarction was found in around 60% of cases. It was regarded as being most likely due to regional hypoperfusion of the white matter and might therefore be prevented, halted or eve作者: Gourmet 時間: 2025-3-25 12:32
Die ?tiologie von Gelenkknorpelsch?den is different from that of neurofibrillary changes. Plaque-like deposits of amyloid should carefully be distinguished from the well known neuritic plaques. The brunt of the cortical pathology is borne by the entorhinal region, the hippocampal formation, and the isocortical association areas. The ear作者: Proclaim 時間: 2025-3-25 19:17
Die Behandlung von Gelenkknorpeldefektenoca (area CH.) and of spine density of fascia dentata dendrites was performed in DAT and control cases. The data revealed that compensatory axonal sprouting of surviving axons seems to replace lost cholinergic afference of fascia dentata cells after degeneration of the basal forebrain nuclei. Change作者: 縮影 時間: 2025-3-25 20:40
https://doi.org/10.1007/978-3-7985-1938-1ons. Their presence is associated with a disappearance of microtubules and neurofilaments, and an accumulation of dense bodies, altered mitochondria and smooth endoplasmic reticulum. Their ultrastructure differs from normal cytoskeletal fibers but one of their main components are the tau proteins. S作者: 容易生皺紋 時間: 2025-3-26 02:35
https://doi.org/10.1007/978-3-322-98830-0re affected by the neurofibrillary degeneration (NFD). We suggest here that these likely early markers of the NFD might define an in vitro model for the study of the NFD. A such model might be useful to verify the different etiopathogenic hypothesis which have been proposed up to here.作者: CHART 時間: 2025-3-26 05:47
https://doi.org/10.1007/978-3-322-98830-0tudies have shown that an extra tau species is present in Alzheimer brain and tangle-reactive neurofilament antibodies were found to be more selective towards phosphorylated neurofilaments than those that do not recognise tangles. A fragment of MAP2 was found to be present in tangles.作者: 不可比擬 時間: 2025-3-26 08:31 作者: fatuity 時間: 2025-3-26 16:08
Wissenschaftliche Taschenbücherned on tissue sections immunocytochemically stained with two monoclonal antibodies to two distinct epitopes of the beta-protein. Amyloid deposits were found in all seven brains examined. Amyloid occurred in three morphological forms: 1. focal amyloid deposits (plaques), 2. large diffuse amyloid depo作者: Generalize 時間: 2025-3-26 18:19
Die Behandlung von GelenkknorpeldefektenIn a quantitative study on thick frozen sections stained by the Gallyas’ silver impregnation method neurofibrillary tangles (NFT), neuritic plaques (NP) and neuropil threads (NT) in the entorhinal as well as transentorhinal area (Braak and Braak, 1985) have been evaluated. The amount of NT exceeded by far that of NFT and NP.作者: insular 時間: 2025-3-26 22:32
Quantitative investigations of presenile and senile changes of the human entorhinal regionIn a quantitative study on thick frozen sections stained by the Gallyas’ silver impregnation method neurofibrillary tangles (NFT), neuritic plaques (NP) and neuropil threads (NT) in the entorhinal as well as transentorhinal area (Braak and Braak, 1985) have been evaluated. The amount of NT exceeded by far that of NFT and NP.作者: Minikin 時間: 2025-3-27 04:40 作者: ensemble 時間: 2025-3-27 06:49 作者: interrogate 時間: 2025-3-27 11:10
Descriptive and analytic epidemiology of Alzheimer’s disease for Alzheimer’s disease are concerned, our knowledges principally base on findings derived from case-control studies. A family history of dementia and a previous head trauma were found to be associated with Alzheimer’s disease in case-control studies. Indications of the role of genetic factors in t作者: 小臼 時間: 2025-3-27 15:56
Morphology of Alzheimer’s disease and related disordersrtical atrophy with 40 to 60% loss of large neurons and 45 to 55% decline of synaptic density in frontal, temporal, parietal cortex and hippocampus are probable correlates of progressive dementia. Many mediator specific brainstem nuclei show progressive neuronal loss and presence of NFT with severe 作者: 命令變成大炮 時間: 2025-3-27 18:34 作者: encomiast 時間: 2025-3-28 01:17 作者: AMITY 時間: 2025-3-28 03:33
Die spezifisch antirachitischen Verfahren, for Alzheimer’s disease are concerned, our knowledges principally base on findings derived from case-control studies. A family history of dementia and a previous head trauma were found to be associated with Alzheimer’s disease in case-control studies. Indications of the role of genetic factors in t作者: 新奇 時間: 2025-3-28 08:43
Die Behandlung von Gelenkknorpeldefektenrtical atrophy with 40 to 60% loss of large neurons and 45 to 55% decline of synaptic density in frontal, temporal, parietal cortex and hippocampus are probable correlates of progressive dementia. Many mediator specific brainstem nuclei show progressive neuronal loss and presence of NFT with severe 作者: Figate 時間: 2025-3-28 13:02
Wissenschaftliche Taschenbücherer number of amyloid deposits. With age, the number of plaques decreased in superficial layers of the cerebral cortex (II–III) and increased in the deeper layers (IV–VI). In the oldest animals, diffuse amyloid deposits in the deeper layers of the cortex predominated. Our studies suggest that the fre作者: 無節(jié)奏 時間: 2025-3-28 16:28 作者: Tremor 時間: 2025-3-28 20:02 作者: 連鎖,連串 時間: 2025-3-29 02:34
https://doi.org/10.1007/978-3-7985-1938-1nd smooth endoplasmic reticulum. Their ultrastructure differs from normal cytoskeletal fibers but one of their main components are the tau proteins. Senile plaques are composed of a core of amyloid fibers surrounded by abnormal neurites containing PHF and accumulations of organelles, reactive astrocytes and microglia.作者: Constitution 時間: 2025-3-29 04:58 作者: FELON 時間: 2025-3-29 10:35 作者: Malleable 時間: 2025-3-29 14:07 作者: 蔓藤圖飾 時間: 2025-3-29 17:16
Die spezifisch antirachitischen Verfahren, deficiencies. Owing to failure of regulatory mechanisms neurotransmitters may prove harmful to the cells producing them as well as to cells exposed to them by innervation. The former mechanism is illustrated by the toxic potential of catecholamine autoxidation, the latter by excitotoxins.作者: 擔憂 時間: 2025-3-29 23:17
https://doi.org/10.1007/978-3-322-85300-4es, amyloid deposition, and neurofibrillary tangles [NFT]) that occur in these animals. These investigations have relevance for understanding some of the behavioral, neuropathological, and neurochemical abnormalities that occur in older humans and in individuals with AD.作者: BAN 時間: 2025-3-30 01:33
The aging brain and its disorders deficiencies. Owing to failure of regulatory mechanisms neurotransmitters may prove harmful to the cells producing them as well as to cells exposed to them by innervation. The former mechanism is illustrated by the toxic potential of catecholamine autoxidation, the latter by excitotoxins.作者: FLUSH 時間: 2025-3-30 04:49 作者: Aboveboard 時間: 2025-3-30 10:35
Some philosophical aspects of Alzheimer’s discovery: an American perspectivery of the disease that bears his name. At present that disease remains poorly understood and even less well treated. However, the speed with which new insights into its characteristic clinical and pathological findings now arise gives us optimism that afflicted patients will, someday soon, be better作者: ALIAS 時間: 2025-3-30 13:50 作者: 雄辯 時間: 2025-3-30 18:37
Epidemiology of Alzheimer’s diseaseor moderate and severe dementia at the age of 65 and over, and of Alzheimer’s and vascular dementia in particular. In the European countries about 60% of all cases of late-life dementia seem to belong to Alzheimer’s, about 20% to multi-infarct dementia. In Japan and the USSR the share of vascular de作者: Devastate 時間: 2025-3-31 00:21
Descriptive and analytic epidemiology of Alzheimer’s disease range between 0.6 in China (Shen et al., 1987), and 5.8 in Scotland (Broe et al., 1976). This variability can be influenced by different definitions of the disease under study and different case ascertainment procedures adopted. All the studies consistently showed an exponential increase of the pre作者: 巧辦法 時間: 2025-3-31 02:48
A proposed classification of familial Alzheimer’s disease based on analysis of 32 multigeneration pedementia. The classification can briefly be stated as follows I. Early onset FAD with mean age of onset before 50 years; II. Late onset FAD with mean age of onset after 60 years; III. Intermediate/variable onset FAD; IV. Familial dementia with atypical Alzheimer changes; V. FAD without multigenerati
