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標(biāo)題: Titlebook: Aging of Cells in and Outside the Body; Sunil C. Kaul,Renu Wadhwa Book 2003 Springer Science+Business Media Dordrecht 2003 Chaperone.aging [打印本頁]

作者: Interpolate    時間: 2025-3-21 18:24
書目名稱Aging of Cells in and Outside the Body影響因子(影響力)




書目名稱Aging of Cells in and Outside the Body影響因子(影響力)學(xué)科排名




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書目名稱Aging of Cells in and Outside the Body讀者反饋學(xué)科排名





作者: Conflict    時間: 2025-3-21 23:29
Climate Change and Energy Transitionan important source of vasoactive substances, such as endothelin-1 (ET-1) and nitric oxide (NO), vascular ECs play a key role in the control of vascular tone. Because EC functions change during aging, EC senescence is an important pathogenetic factor in age-related vascular dysfunction [9].
作者: SAGE    時間: 2025-3-22 04:29
Aging of Endothelial Cells,an important source of vasoactive substances, such as endothelin-1 (ET-1) and nitric oxide (NO), vascular ECs play a key role in the control of vascular tone. Because EC functions change during aging, EC senescence is an important pathogenetic factor in age-related vascular dysfunction [9].
作者: 越自我    時間: 2025-3-22 07:00
Book 2003l 40 years ago. Contributions of genetic and environmental factors, failure of genetic and cellular repair mechanisms, and the epigenetic modifications determine the final lifespan of cells. This book also provides an understanding on how aging mechanisms in mice, a most frequently used model, diffe
作者: 營養(yǎng)    時間: 2025-3-22 11:03

作者: 挑剔小責(zé)    時間: 2025-3-22 14:09

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作者: Preamble    時間: 2025-3-23 05:14

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作者: 合唱隊    時間: 2025-3-23 13:39
Molecular Chaperones and Cellular Aging,ts. Their decrease in expression during aging could account for numerous features of cellular senescence. As many proteins have been shown to have chaperone activity, we focus here on heat shock proteins (Hsp), a major class of chaperones, and on their involvement in the aging process.
作者: ARIA    時間: 2025-3-23 16:03

作者: jabber    時間: 2025-3-23 19:14
Climate Governance in the Digital Erafects on the skeleton. The aim of this chapter is to give a review on the current understanding of the contribution of aging of the osteoblasts (the bone forming cells) to the phenomenon of age-related boneloss.
作者: 昆蟲    時間: 2025-3-24 00:55
Stellina Jolly,Nafees Ahmad,Matthew Scottts. Their decrease in expression during aging could account for numerous features of cellular senescence. As many proteins have been shown to have chaperone activity, we focus here on heat shock proteins (Hsp), a major class of chaperones, and on their involvement in the aging process.
作者: 大包裹    時間: 2025-3-24 05:35

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作者: Resection    時間: 2025-3-24 12:21
?zge Yaren Türkseven,Orhan Gündüz,Alper Babarom the body did not undergo aging and were potentially immortal. The dramatic shift in paradigm began to occur with the publication of a series of papers by Leonard Hayflick and colleagues [1, 2], followed by hundreds of other publications which helped to establish the new paradigm that normal, dip
作者: 抵押貸款    時間: 2025-3-24 16:18
Cultural Heritage and Resilience,e signals could lead to loss of cell proliferative response, as observed in the aging immune system, or alternatively to uncontrolled cell division, one step in the progression toward neoplasia. There is good evidence that the likelihood of both these changes increases with age. Therefore, changes i
作者: 藝術(shù)    時間: 2025-3-24 22:24
https://doi.org/10.1007/978-3-031-61242-8ological and social issue. In the effort to unravel the basic mechanisms that regulate the aging process fibroblasts was and still is the most “popular” cellular model of experimentation. This article will try to focus on some of the features of aging human fibroblasts, the mechanisms underlying the
作者: 樂意    時間: 2025-3-25 01:31
Climate Change and Energy Transitione functional integrity of the endothelium monolayer, which is composed of endothelial cells (ECs), is essential to the prevention of vascular leakage and the formation of atherosclerotic lesions [1, 2], which are often found in the human vasculature of the elderly [3, 4]. Aging is an independent ris
作者: Electrolysis    時間: 2025-3-25 04:11

作者: Expurgate    時間: 2025-3-25 08:45
https://doi.org/10.1007/978-3-031-56423-9wever, always has to be validated by reference to the situation . and the particular cell and organ system being examined. As I shall limit my remarks in this chapter mostly to the human system we also need to distinguish between the use of the term “.” to mean what happens inside the organism and “
作者: Indecisive    時間: 2025-3-25 13:01

作者: 包裹    時間: 2025-3-25 18:45
U. C. Mohanty,Palash Sinha,K. K. Singhself, or defects in its regulation, are probably the most common genetic markers of all human cancers [1]. Despite the fact that tumor etiologies differ widely, this evidence shows that there are common pathways in tumor development. A key step in tumor development is the conversion of a cell from o
作者: 暫時過來    時間: 2025-3-25 21:51

作者: 深淵    時間: 2025-3-26 00:48

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作者: 吹牛需要藝術(shù)    時間: 2025-3-26 09:02
Stellina Jolly,Nafees Ahmad,Matthew Scottal to daughter cells, it puts genetic material to inherent risk of mutations that might occur during the process of replication and failure of genetic fidelity. Such mutations may cause genomic instability, neoplastic transformation and development of cancer [1]. Perhaps to avoid this risky journey,
作者: heterodox    時間: 2025-3-26 12:55
Haplotype-Assisted Breeding in Rice,is featured most typically by specific morphological alterations, flat and enlarged cell shape, and induction of senescence markers such as fibronectin, collagenase I, and senescence-associated β-galactosidase [2]. It is also shown that in normally senesced human fibroblasts the cyclin-dependent pro
作者: AMEND    時間: 2025-3-26 18:46

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作者: Harass    時間: 2025-3-27 04:32

作者: 原諒    時間: 2025-3-27 07:30
Danang Aditya Nizar,Ly Quoc Dangiferation arrest. This arrest is accompanied by a variety of morphological and biochemical changes in the cells, and the arrest state is referred to as senescence. The . senescence process has been widely studied as a model for . cellular aging [1].
作者: anchor    時間: 2025-3-27 10:04

作者: 類人猿    時間: 2025-3-27 16:12

作者: 難管    時間: 2025-3-27 21:48
Aging of Fibroblasts,ological and social issue. In the effort to unravel the basic mechanisms that regulate the aging process fibroblasts was and still is the most “popular” cellular model of experimentation. This article will try to focus on some of the features of aging human fibroblasts, the mechanisms underlying the
作者: 神刊    時間: 2025-3-27 22:11
Aging of Endothelial Cells,e functional integrity of the endothelium monolayer, which is composed of endothelial cells (ECs), is essential to the prevention of vascular leakage and the formation of atherosclerotic lesions [1, 2], which are often found in the human vasculature of the elderly [3, 4]. Aging is an independent ris
作者: 吹牛需要藝術(shù)    時間: 2025-3-28 05:38

作者: 走路左晃右晃    時間: 2025-3-28 08:17
T Cell Immunosenescence,wever, always has to be validated by reference to the situation . and the particular cell and organ system being examined. As I shall limit my remarks in this chapter mostly to the human system we also need to distinguish between the use of the term “.” to mean what happens inside the organism and “
作者: 使更活躍    時間: 2025-3-28 11:59

作者: 強制令    時間: 2025-3-28 14:41
The p53 Pathway, Cancer and Aging,self, or defects in its regulation, are probably the most common genetic markers of all human cancers [1]. Despite the fact that tumor etiologies differ widely, this evidence shows that there are common pathways in tumor development. A key step in tumor development is the conversion of a cell from o
作者: Enzyme    時間: 2025-3-28 22:35
Regulation of Cellular Senescence by the Retinoblastoma Pathway,ly exponential kinetics and eventually reach a state of permanent growth arrest, referred to as replicative senescence [1]. Senescent cells remain viable and metabolically active, but cannot re-initiate DNA replication in response to physiological mitogenic stimuli [1–3]. Expression of .enescence-.s
作者: 讓空氣進入    時間: 2025-3-29 00:56

作者: innovation    時間: 2025-3-29 04:37

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作者: 藥物    時間: 2025-3-29 14:43
Therapeutic Induction of Cellular Senescence: Nuclear Matrix in Senescence,is featured most typically by specific morphological alterations, flat and enlarged cell shape, and induction of senescence markers such as fibronectin, collagenase I, and senescence-associated β-galactosidase [2]. It is also shown that in normally senesced human fibroblasts the cyclin-dependent pro
作者: 流浪    時間: 2025-3-29 19:08
Use of Hammerhead Ribozymes for Aging and Cancer,scades (replication, transcription and translation) by catalyzing essential steps such as cleavage and ligation [2]. Hammerhead ribozymes (for resemblance of their two-dimensional structure to a hammerhead) are the smallest ribozymes that are used as molecular scissors in molecular biology and biote
作者: filial    時間: 2025-3-29 23:37
?zge Yaren Türkseven,Orhan Gündüz,Alper Babaoblasts, epithelial cells, endothelial cells, keratinocytes, melanocytes, chondrocytes, muscle satellite cells, articular cartilage cells, glial cells, lymphocytes, trabecular osteoblasts and bone marrow cells, isolated from a variety of tissues and animals.
作者: 止痛藥    時間: 2025-3-30 02:43
Cultural Heritage and Resilience,s that a direct correlation exists between species life span and . life span [2], but that this correlation is inverse with donorage [3–7]. The frequency of reversal of the senescence phenotype (i.e., immortalization) varies greatly with different animal species. The probability of immortalization (
作者: inferno    時間: 2025-3-30 08:08

作者: 人類學(xué)家    時間: 2025-3-30 09:08

作者: TSH582    時間: 2025-3-30 13:44

作者: 騙子    時間: 2025-3-30 16:44
Stellina Jolly,Nafees Ahmad,Matthew Scottnt cells from different organisms show similar biochemical and morphological features collectively referred to as senescent/aging phenotype, most consistently characterized by functional decline. The senescent cells are irreversibly arrested in G1 stage of the cell cycle but remain viable and metabo
作者: interpose    時間: 2025-3-30 21:14
Haplotype-Assisted Breeding in Rice,or genes, a phenomenon called “premature senescence.” They include, for instance, hydrogen peroxide [5], the activated . gene [6], histone deacetylase inhibitors [7], DNA topoisomerases inhibitors [8], and others. In sharp contrast, they have different effects on immortal cells, namely, having no ef




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