標(biāo)題: Titlebook: Advances in Multiple Sclerosis and Experimental Demyelinating Diseases; Moses Rodriguez Book 2008 The Editor(s) (if applicable) and The Au [打印本頁] 作者: 不幸的你 時(shí)間: 2025-3-21 16:19
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases影響因子(影響力)
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases影響因子(影響力)學(xué)科排名
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書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases被引頻次
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases被引頻次學(xué)科排名
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases年度引用
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases年度引用學(xué)科排名
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases讀者反饋
書目名稱Advances in Multiple Sclerosis and Experimental Demyelinating Diseases讀者反饋學(xué)科排名
作者: Lineage 時(shí)間: 2025-3-21 23:09 作者: 菊花 時(shí)間: 2025-3-22 03:53 作者: FOIL 時(shí)間: 2025-3-22 07:31
Advances in Multiple Sclerosis and Experimental Demyelinating Diseases978-3-540-73677-6Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: Dislocation 時(shí)間: 2025-3-22 12:33
Jerry R. Miller,Suzanne M. Orbock Millerclerosis. Based on the Olmsted County population prevalence cohort study performed at the Mayo Clinic, MS patients with EDSS scores of 2 or lower followed for a period of 5–10 years have a very small risk of developing disability over the next 10–20 years. Based on these findings, this chapter revie作者: 喚起 時(shí)間: 2025-3-22 13:57 作者: Charade 時(shí)間: 2025-3-22 19:03
The Handbook of Environmental Chemistryility and multifaceted gene-environment interactions. The major histocompatibility complex (MHC) is the only genomic region consistently associated with MS, and susceptible MHC haplotypes have been identified. Although the MHC does not account for all genetic contribution to MS, the other genetic co作者: Ondines-curse 時(shí)間: 2025-3-22 22:08
W. James Catallo,Jay L. Comeaux,Thomas Junkthe disease process. While it is possible to establish the presence of remyelination with microscopy of tissue samples, it is important to have noninvasive or minimally invasive methods to measure remyelination in living animals and humans. Such tools are critical to establishing the efficacy of age作者: 軟弱 時(shí)間: 2025-3-23 02:09 作者: resuscitation 時(shí)間: 2025-3-23 07:22
Lecture Notes in Earth Sciencesn patterns in MS lesions, sera, and/or cerebrospinal fluid (CSF). Growing evidence exists regarding their mechanistic roles in inflammatory and neurodegenerative aspects of this disease. Proteolytic events participate in demyelination, axon injury, apoptosis, and development of the inflammatory resp作者: BORE 時(shí)間: 2025-3-23 11:50 作者: 雇傭兵 時(shí)間: 2025-3-23 17:10
https://doi.org/10.1007/BFb0011119y and maintenance of axonal health. It occurs as a spontaneous regenerative response following demyelination in a range of pathologies including traumatic injury as well as primary demyelinating disease such as multiple sclerosis (MS). Experimental models of demyelination based on the use of toxins,作者: NATAL 時(shí)間: 2025-3-23 21:19 作者: rectocele 時(shí)間: 2025-3-23 22:26 作者: 小樣他閑聊 時(shí)間: 2025-3-24 02:33
Lecture Notes in Earth Sciencesfluence inflammation and neuron and glial function is being slowly unraveled. There is increasing evidence that estrogen, progesterone, and testosterone contain immune responses and influence damage repair in the nervous system. Hormones such as prolactin and vitamin D are being explored as immunomo作者: Judicious 時(shí)間: 2025-3-24 10:27 作者: abject 時(shí)間: 2025-3-24 11:44 作者: 興奮過度 時(shí)間: 2025-3-24 17:25
Dick Hoogendoorn,Wim H. Rulkens Ph.D., and results in early permanent disability for most affected patients. A new autoantibody marker called neuromyelitis optica immunoglobulin G (NMO-IgG), which targets the water channel protein aquaporin-4, is highly specific for NMO. The marker has demonstrated that the NMO spectrum of disorders is作者: 絆住 時(shí)間: 2025-3-24 22:25 作者: Budget 時(shí)間: 2025-3-24 23:45 作者: 上釉彩 時(shí)間: 2025-3-25 04:56
https://doi.org/10.1007/BFb0011119 injury and promotes maximal remyelination within 5 weeks after a single low dose (25 μg/kg). The IgM isoform of this reparative antibody is required for in vivo function. We hypothesize that the IgM clusters membrane domains and associated signaling molecules on the surface of target cells. Current作者: pulmonary 時(shí)間: 2025-3-25 10:25
https://doi.org/10.1007/BFb0011119 axonal damage, and remyelination, and studies utilizing in vivo T1 and T2 relaxometry, provide insight into the pathology of demyelinating diseases at previously unprecedented details. The technical challenges of small voxel in vivo MR spectroscopy and the biologically relevant information obtained作者: motor-unit 時(shí)間: 2025-3-25 13:24 作者: 廚房里面 時(shí)間: 2025-3-25 18:29
0070-217X etic tools for identifying patients who are most likely to experience spontaneous remyelination. Epidemiology studies offer additional avenues of treatment. Exa978-3-642-43783-0978-3-540-73677-6Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: NEG 時(shí)間: 2025-3-25 22:07 作者: 清醒 時(shí)間: 2025-3-26 02:43 作者: Obscure 時(shí)間: 2025-3-26 06:22 作者: ASSAY 時(shí)間: 2025-3-26 10:55 作者: CLAY 時(shí)間: 2025-3-26 15:14
Advances in Multiple Sclerosis and Experimental Demyelinating Diseases作者: 委派 時(shí)間: 2025-3-26 17:53
Benign Multiple Sclerosis: A Distinct Clinical Entity with Therapeutic Implications,clerosis. Based on the Olmsted County population prevalence cohort study performed at the Mayo Clinic, MS patients with EDSS scores of 2 or lower followed for a period of 5–10 years have a very small risk of developing disability over the next 10–20 years. Based on these findings, this chapter revie作者: predict 時(shí)間: 2025-3-26 23:24
Pathological Heterogeneity of Idiopathic Central Nervous System Inflammatory Demyelinating Disorderchemical tools to examine the MS lesion microscopically, as well as technological advances in neuroimaging, which permit a dynamic assessment of lesion formation and disease progression. The heterogeneous pathology of MS in relation to stage of lesion activity, phase of disease, and clinical course 作者: CBC471 時(shí)間: 2025-3-27 02:46 作者: cacophony 時(shí)間: 2025-3-27 05:21 作者: 確定方向 時(shí)間: 2025-3-27 10:15 作者: 激怒某人 時(shí)間: 2025-3-27 15:50
The Multiple Sclerosis Degradome: Enzymatic Cascades in Development and Progression of Central Nervn patterns in MS lesions, sera, and/or cerebrospinal fluid (CSF). Growing evidence exists regarding their mechanistic roles in inflammatory and neurodegenerative aspects of this disease. Proteolytic events participate in demyelination, axon injury, apoptosis, and development of the inflammatory resp作者: Osteoarthritis 時(shí)間: 2025-3-27 20:17
Genetic Analysis of CNS Remyelination,l role in determining the level of accompanying neurologic disability. While remyelination can be quite robust, in multiple sclerosis it often fails. Understanding and stimulating the remyelination process are therefore important goals in MS research. Remyelination is a complex cellular process that作者: 公理 時(shí)間: 2025-3-27 23:25
Remyelination in Experimental Models of Toxin-Induced Demyelination,y and maintenance of axonal health. It occurs as a spontaneous regenerative response following demyelination in a range of pathologies including traumatic injury as well as primary demyelinating disease such as multiple sclerosis (MS). Experimental models of demyelination based on the use of toxins,作者: Obvious 時(shí)間: 2025-3-28 05:31 作者: 不連貫 時(shí)間: 2025-3-28 08:42
Neuroimaging of Demyelination and Remyelination Models,ommonly used autoimmune, viral, and toxic models. Because most MS models are induced in rodents with brains and spinal cords of a smaller magnitude than humans, small-animal MRI must accomplish much higher resolution acquisition in order to generate useful data. In this review, we discuss key aspect作者: Offbeat 時(shí)間: 2025-3-28 10:46
Hormonal Influences in Multiple Sclerosis,fluence inflammation and neuron and glial function is being slowly unraveled. There is increasing evidence that estrogen, progesterone, and testosterone contain immune responses and influence damage repair in the nervous system. Hormones such as prolactin and vitamin D are being explored as immunomo作者: 殺子女者 時(shí)間: 2025-3-28 15:45
Statins and Demyelination,n order to reduce atherogenesis and cardiovascular morbidity. Moreover, statins have been shown to exert pleiotropic immunomodulatory effects that might be of therapeutic benefit in autoimmune disorders. Statins appear to alter immune function largely independent of lipid lowering and rather through作者: 娘娘腔 時(shí)間: 2025-3-28 22:46 作者: 宣誓書 時(shí)間: 2025-3-29 00:55 作者: 發(fā)酵 時(shí)間: 2025-3-29 05:45
W. James Catallo,Jay L. Comeaux,Thomas Junknts purported to promote or enhance remyelination. This chapter reviews the technology of imaging of the brain, its application to MS, and the current state of imaging techniques for measuring remyelination and the health of the associated neurons in the setting of MS.作者: 制造 時(shí)間: 2025-3-29 11:16 作者: Gum-Disease 時(shí)間: 2025-3-29 15:05
Lecture Notes in Earth Sciencesdulators and may influence diseases such as multiple sclerosis (MS) or may be used therapeutically to modulate the immune response. More recently identified hormones, such as leptin and gherlin, may also influence the course of disease. This chapter reviews some of the evidence that supports a role for hormones in MS.作者: HAUNT 時(shí)間: 2025-3-29 18:19
Lecture Notes in Earth Sciencesderate success. Several recent studies have reported lower levels of uric acid (UA), a major scavenger of reactive nitrogen species, in MS patients, while other studies found no such correlation. Here, we discuss these studies as well as current efforts to manipulate serum UA levels in MS patients.作者: 招募 時(shí)間: 2025-3-29 22:57
Book 2008tory response is the primary contributor to demyelination, Dr. Rodriguez and colleagues take a fresh, bold look at the causes and possible treatments of MS....Assuming oligodendrocyte injury as a prerequisite to MS, the authors explore viruses, toxins and genetic defects as possible culprits. They p作者: Emg827 時(shí)間: 2025-3-30 02:14
The Handbook of Environmental ChemistryS from the expression consequences of MS. The use of new methodologies and technologies to refine the phenotype, such as brain spectroscopy, PET and functional magnetic resonance imaging combined with novel computational tools and a better understanding of the human genome architecture, may help resolve the genetic causes of MS.作者: Wernickes-area 時(shí)間: 2025-3-30 08:08
https://doi.org/10.1007/BFb0011119w they can be used to study different aspects of remyelination. We also describe the optimal use of these models, highlighting potential pitfalls in interpretation, and how remyelination can be unequivocally recognized. Finally, we discuss the role of toxin models alongside viral and immune-mediated models of demyelination.作者: 梯田 時(shí)間: 2025-3-30 11:59 作者: 過份 時(shí)間: 2025-3-30 16:27
Multiple Sclerosis Genetics,S from the expression consequences of MS. The use of new methodologies and technologies to refine the phenotype, such as brain spectroscopy, PET and functional magnetic resonance imaging combined with novel computational tools and a better understanding of the human genome architecture, may help resolve the genetic causes of MS.
