標(biāo)題: Titlebook: Advances in TNF Family Research; Proceedings of the 1 David Wallach,Andrew Kovalenko,Marc Feldmann Conference proceedings 2011 The Editor(s [打印本頁] 作者: Eisenhower 時間: 2025-3-21 19:36
書目名稱Advances in TNF Family Research影響因子(影響力)
書目名稱Advances in TNF Family Research影響因子(影響力)學(xué)科排名
書目名稱Advances in TNF Family Research網(wǎng)絡(luò)公開度
書目名稱Advances in TNF Family Research網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Advances in TNF Family Research被引頻次
書目名稱Advances in TNF Family Research被引頻次學(xué)科排名
書目名稱Advances in TNF Family Research年度引用
書目名稱Advances in TNF Family Research年度引用學(xué)科排名
書目名稱Advances in TNF Family Research讀者反饋
書目名稱Advances in TNF Family Research讀者反饋學(xué)科排名
作者: 憲法沒有 時間: 2025-3-22 00:17
David Wallach,Andrew Kovalenko,Marc FeldmannMost up-to-date book on current research of the TNF family.TNF meetings follow the enormous progress in this field.The conference focused on the physiological, pathophysiological, and medical signific作者: ALTER 時間: 2025-3-22 03:02
Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/a/image/149982.jpg作者: photophobia 時間: 2025-3-22 06:42
Structural Studies of NEMO and TRAF6: Implications in NF-κB Activationological processes including innate and adaptive immunity, oncogenesis, and development. They are activated in response to ligation of many receptors including T-cell receptors, B-cell receptors, members of the tumor necrosis factor (TNF) receptor superfamily, and the Toll-like receptor/interleukin-1 receptor (TLR/IL-1R) superfamily.作者: 施魔法 時間: 2025-3-22 11:34
Advances in TNF Family Research978-1-4419-6612-4Series ISSN 0065-2598 Series E-ISSN 2214-8019 作者: HPA533 時間: 2025-3-22 15:10
Lecture Notes in Computer Scienceological processes including innate and adaptive immunity, oncogenesis, and development. They are activated in response to ligation of many receptors including T-cell receptors, B-cell receptors, members of the tumor necrosis factor (TNF) receptor superfamily, and the Toll-like receptor/interleukin-1 receptor (TLR/IL-1R) superfamily.作者: fatty-acids 時間: 2025-3-22 17:26
Lucas Bechberger,Kai-Uwe Kühnbergerat LT-deficient mice lacked lymph nodes and the discovery of multiple additional members of the family with distinctly non-inflammatory roles brought about the realization that many members of the family were crucial for normal development. Furthermore, TNF’s role in tumor defense has come under scr作者: 貧困 時間: 2025-3-23 00:58
Mauri Kaipainen,Antti Hautam?kiealth care. Over the last decade, a major focus of research was the identification of factors controlling bone metabolism and osteoclastogenesis, respectively. In particular, three tumor necrosis factor (TNF) family molecules, the receptor activator of NF-κB (RANK), its ligand RANKL, and the decoy r作者: Daily-Value 時間: 2025-3-23 04:16 作者: 苦惱 時間: 2025-3-23 08:36 作者: 蚊帳 時間: 2025-3-23 10:55
Implications in Triadic Formal Contexts cell death. In a large-scale ectopic-expression screen in . for identifying genes regulating cell death, the fly homolog of tumor necrosis factor (TNF) encoded by . (.....) was identified [7]. . was also isolated independently through a molecular approach [15]. . encodes a protein of 409 amino acid作者: 抵消 時間: 2025-3-23 15:41 作者: Adenoma 時間: 2025-3-23 20:55
Lecture Notes in Computer Sciencetor 2 (TRAF2) were described as putative mediators of TNFR2 signaling. The following years witnessed an immense number of studies confirming the seminal role of both cellular IAPs and of TRAF2 in transducing signaling initiated by TNF family ligands. As such, c-IAP1 and c-IAP2 suppress TNF-stimulate作者: Vo2-Max 時間: 2025-3-24 00:31 作者: Infusion 時間: 2025-3-24 05:35 作者: Generator 時間: 2025-3-24 06:59 作者: infantile 時間: 2025-3-24 11:12 作者: 比目魚 時間: 2025-3-24 15:07 作者: 碎石頭 時間: 2025-3-24 20:17 作者: GIDDY 時間: 2025-3-25 01:53 作者: CANDY 時間: 2025-3-25 03:28
Workshop Summary: ,at LT-deficient mice lacked lymph nodes and the discovery of multiple additional members of the family with distinctly non-inflammatory roles brought about the realization that many members of the family were crucial for normal development. Furthermore, TNF’s role in tumor defense has come under scr作者: TAP 時間: 2025-3-25 07:33 作者: 和平 時間: 2025-3-25 11:49 作者: narcissism 時間: 2025-3-25 17:14 作者: ESPY 時間: 2025-3-25 22:18 作者: labile 時間: 2025-3-26 00:42
Workshop Summary: ,refully balanced between a pro-survival and pro-apoptotic response and, as might be expected for proteins containing these E3 ligase domains, much of the data and discussion revolved around the regulation of these alternative pathways by ubiquitylation. The session was also notable for talks that ex作者: COWER 時間: 2025-3-26 05:43
TRAF2 and Cellular IAPs: A Critical Link in TNFR Family Signalingtor 2 (TRAF2) were described as putative mediators of TNFR2 signaling. The following years witnessed an immense number of studies confirming the seminal role of both cellular IAPs and of TRAF2 in transducing signaling initiated by TNF family ligands. As such, c-IAP1 and c-IAP2 suppress TNF-stimulate作者: enfeeble 時間: 2025-3-26 10:14
New Perspectives in TNF-R1-Induced NF-,B Signalingion of the kinase RIPK1 is required for recruitment of the TAB2/TAB3/TAK1 and IKK1/IKK2/NEMO complexes that are in turn required to phosphorylate and degrade IκBα allowing freed NF-κB dimers to nuclear translocation. The exact involvement of the E3 ligases, TRAF2, TRAF5, cIAP1, and cIAP2 in this pro作者: 傲慢物 時間: 2025-3-26 14:38 作者: Fresco 時間: 2025-3-26 16:57 作者: 包裹 時間: 2025-3-26 23:02
Selective Binding of Linear Ubiquitin Chains to NEMO in NF-kappaB Activationan acute stimulation of cells with variety of cytokines leads to Lys63-linked ubiquitin chain conjugation on the receptor-associated complexes to activate the TAK1 kinase. In addition, function of the linear ubiquitin chain assembly (LUBAC) ligase is required for the activation of IkappaB kinase com作者: 拱形大橋 時間: 2025-3-27 01:21 作者: 慢慢啃 時間: 2025-3-27 09:01
Temporal Control of TNFα Signaling by Miz1apoptosis, and tumorigenesis mainly through its cytoplasm membrane receptor TNF-R1 complex 1 [6,7], which activates downstream effectors such as NF-κB, caspases and JNK. However, the control of TNF-R1 complex 1 activity is incompletely understood. We report here that the transcription factor zinc fi作者: aspect 時間: 2025-3-27 12:48 作者: Integrate 時間: 2025-3-27 16:11
The Edar Subfamily in Hair and Exocrine Gland Developmentnown about the physiological role of Troy and Xedar. Research on the Edar pathway has focused on hair follicle and tooth biology, but recent progress has been made in uncovering its relevance in morphogenesis of glandular appendages as well.作者: Pelago 時間: 2025-3-27 20:56
High Endothelial Venule Reporter Mice to Probe Regulation of Lymph Node Vasculatureice express the reporter gene in HEVs in concurrence with the endogenous gene and PNAd. Additional mice transgenic for lymphatic vessel reporter constructs are in development. These will provide material for in vivo imaging and allow us to evaluate the regulation and interaction of HEVs and LVs.作者: 付出 時間: 2025-3-27 23:23
Eiger and Wengen: The , Orthologs of TNF/TNFRs with a C-terminal TNF homology domain and a hydrophobic transmembrane domain. The sequence of the Eiger’s TNF domain shows highest homology with human EDA-A2 (28%) and also shows significant homology with all known mammalian TNF superfamily members.作者: 貪心 時間: 2025-3-28 02:47 作者: HARP 時間: 2025-3-28 07:39
New Perspectives in TNF-R1-Induced NF-,B Signalingcess has been unclear. We summarize a recent study from our lab defining the roles of TRAF2 and cIAP1?s in activation of NF-κB downstream of TNF-R1 and report the surprising finding that RIPK1 is not essential for TNF-R1-induced NF-κB. Alternative mechanisms of how NF-κB may be activated by TNF-R1 in the light of these findings are discussed.作者: FECT 時間: 2025-3-28 12:20 作者: arrogant 時間: 2025-3-28 17:17
Contexts, canons and coreferent types,the family of human deubiquitinating enzymes (DUBs), we summarize requirements for setting up a screen and the follow-up of candidate genes. In conclusion we give an overview about current therapeutic options targeting TNFα signaling and discuss the relevance of target discovery by RNAi for progress in cancer therapy.作者: 有限 時間: 2025-3-28 22:25 作者: 黃油沒有 時間: 2025-3-29 00:42
Selma Strahringer,Rudolf Wille,Uta Willeing requires multiple monoubiquitinations, as progressive elimination of lysines in p105 is accompanied by gradual inhibition of p50 generation. Last, the proteasome recognizes the multiply monoubiquitinated p105. These findings suggest that a proteolytic signal can be comprised of a cluster of single ubiquitins, and not necessarily of a chain.作者: 鳴叫 時間: 2025-3-29 06:33
Modification by Single Ubiquitin Moieties Rather Than Polyubiquitination Is Sufficient for Proteasoming requires multiple monoubiquitinations, as progressive elimination of lysines in p105 is accompanied by gradual inhibition of p50 generation. Last, the proteasome recognizes the multiply monoubiquitinated p105. These findings suggest that a proteolytic signal can be comprised of a cluster of single ubiquitins, and not necessarily of a chain.作者: 認(rèn)識 時間: 2025-3-29 10:07 作者: adduction 時間: 2025-3-29 14:12 作者: Neolithic 時間: 2025-3-29 16:50
https://doi.org/10.1007/978-3-030-12800-5ice express the reporter gene in HEVs in concurrence with the endogenous gene and PNAd. Additional mice transgenic for lymphatic vessel reporter constructs are in development. These will provide material for in vivo imaging and allow us to evaluate the regulation and interaction of HEVs and LVs.作者: 偏見 時間: 2025-3-29 19:57
Implications in Triadic Formal Contextss with a C-terminal TNF homology domain and a hydrophobic transmembrane domain. The sequence of the Eiger’s TNF domain shows highest homology with human EDA-A2 (28%) and also shows significant homology with all known mammalian TNF superfamily members.作者: Grievance 時間: 2025-3-30 00:24 作者: 露天歷史劇 時間: 2025-3-30 05:14
Rajeev Sangal,Soma Paul,P. Kiran Mayeecess has been unclear. We summarize a recent study from our lab defining the roles of TRAF2 and cIAP1?s in activation of NF-κB downstream of TNF-R1 and report the surprising finding that RIPK1 is not essential for TNF-R1-induced NF-κB. Alternative mechanisms of how NF-κB may be activated by TNF-R1 in the light of these findings are discussed.作者: Harpoon 時間: 2025-3-30 11:29 作者: negotiable 時間: 2025-3-30 16:08 作者: AGATE 時間: 2025-3-30 17:25 作者: deadlock 時間: 2025-3-31 00:42 作者: Vldl379 時間: 2025-3-31 02:33 作者: Orgasm 時間: 2025-3-31 08:42
0065-2598 essions at the meeting specifically address their involvement in immunity, development, apoptosis, autoimmunity, cancer, and infection, the normal function and pathology of the neuronal system, as well as major unresolved questions about their mechanisms of action.978-1-4614-2783-4978-1-4419-6612-4Series ISSN 0065-2598 Series E-ISSN 2214-8019 作者: 揉雜 時間: 2025-3-31 12:39
Mauri Kaipainen,Antti Hautam?kiT cells and other immune cells via RANKL induce osteoclastogenesis and thereby can shift the intricate balance of bone deposition and resorption. Through knockout mice it became also clear that the RANKL–RANK–OPG system is involved in other processes, such as in controlling immune responses in the s
