標(biāo)題: Titlebook: Advances in Research on Neurodegeneration; Volume 6 W. Poewe,G. Ransmayr Conference proceedings 1999 Springer-Verlag Wien 1999 Huntington‘s [打印本頁] 作者: 交叉路口 時(shí)間: 2025-3-21 16:28
書目名稱Advances in Research on Neurodegeneration影響因子(影響力)
書目名稱Advances in Research on Neurodegeneration影響因子(影響力)學(xué)科排名
書目名稱Advances in Research on Neurodegeneration網(wǎng)絡(luò)公開度
書目名稱Advances in Research on Neurodegeneration網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Advances in Research on Neurodegeneration被引頻次
書目名稱Advances in Research on Neurodegeneration被引頻次學(xué)科排名
書目名稱Advances in Research on Neurodegeneration年度引用
書目名稱Advances in Research on Neurodegeneration年度引用學(xué)科排名
書目名稱Advances in Research on Neurodegeneration讀者反饋
書目名稱Advances in Research on Neurodegeneration讀者反饋學(xué)科排名
作者: GULP 時(shí)間: 2025-3-22 00:02 作者: 艱苦地移動 時(shí)間: 2025-3-22 04:02
,Molecular mimicry and multiple sclerosis — a possible role for degenerate T cell recognition in theort the hypothesis that the pathogenesis is mediated by autoreactive T lymphocytes. Molecular mimicry has been proposed as a possible mechanism for the development of an autoimmune response to myelin antigens. According to this model, an immune reaction to self antigens could be initiated by T cells作者: 水獺 時(shí)間: 2025-3-22 07:02 作者: 安撫 時(shí)間: 2025-3-22 10:41 作者: 自愛 時(shí)間: 2025-3-22 14:13 作者: 多嘴 時(shí)間: 2025-3-22 19:29 作者: enumaerate 時(shí)間: 2025-3-23 00:31
,Antiglutamate therapy of ALS — which is the next step?, in men indicates that antiglutamatergic strategies are the first to have an influence on its pathogenesis and slow down the disease process. Since the effect of the drugs is still small, this progress cannot only be seen as a success of the present but most also be acknowledged as a starting point 作者: ACTIN 時(shí)間: 2025-3-23 05:10
,Antiglutamate therapies in Huntington’s disease,netic defect leads to neuronal injury and death is unknown, but is thought to include glutamate-mediated excitotoxicity and abnormalities of mitochondrial energy production. Both of these mechanisms may lead to a final common pathway of increased production of free radical species. Prior clinical tr作者: 顯示 時(shí)間: 2025-3-23 08:36
Neural transplantation in animal models of multiple system atrophy: a review,Due to combined degeneration of substantia nigra pars compacta (SNC) and of striatum, antiparkinsonian therapy based on levodopa substitution eventually fails in more than 90% of patients. Animal models of MSA-SND are urgently required as test-bed for the evaluation of novel therapeutic intervention作者: 砍伐 時(shí)間: 2025-3-23 12:15
Striatal reconstruction by striatal grafts,, integrate into the lesioned host brain and alleviate the behavioural deficits in both motor and cognitive spheres. How? Since normal striatal function is dependent upon it’s integration within a connected corticalsubcortical neuronal circuitry, and the deficits following striatal damage appear to 作者: BAIL 時(shí)間: 2025-3-23 16:13 作者: pus840 時(shí)間: 2025-3-23 21:09 作者: 妨礙 時(shí)間: 2025-3-24 00:05
Derived Tubularity: a Computational Approach an overview on the development of different strategies in gene therapy in PD animal models and point out new and possibly more successful directions, including the transplantation of neural precursor cells and pig tissue.作者: Tailor 時(shí)間: 2025-3-24 05:13
Myelin dysfunction/degradation in the central nervous system: why are myelin sheaths susceptible to became apparent from analyses of animals with altered amounts of otherwise normal myelin components: Due to limited redundance in function between different myelin proteins, dysfunction or loss of one protein may cause loss of function and instability of the entire myelin sheath.作者: 值得 時(shí)間: 2025-3-24 08:09
,Somatic gene therapy in animal models of Parkinson’s disease, an overview on the development of different strategies in gene therapy in PD animal models and point out new and possibly more successful directions, including the transplantation of neural precursor cells and pig tissue.作者: 衰老 時(shí)間: 2025-3-24 14:15 作者: Flawless 時(shí)間: 2025-3-24 17:33 作者: 教育學(xué) 時(shí)間: 2025-3-24 23:02 作者: 物種起源 時(shí)間: 2025-3-25 01:03 作者: bromide 時(shí)間: 2025-3-25 04:35
Multidimensional Feller Processes implies that we are likely to use combination therapies and have to try to treat patients early. The latter will be necessarily connected with the demand for a novel clinical attitude to the diagnosis of the disease.作者: Venules 時(shí)間: 2025-3-25 08:24 作者: itinerary 時(shí)間: 2025-3-25 13:30
Neuroprotective effect of chronic inactivation of the subthalamic nucleus in a rat model of Parkinsra dopaminergic somata when carried out one week prior to 6-hydroxydopamine injection in the striatum. Nevertheless neurochemical results showed that this lesion did not antagonize the striatal 6-hydroxydopamine-induced dopamine depletion in the striatum 15 days after 6-hydroxydopamine injection.作者: 我們的面粉 時(shí)間: 2025-3-25 19:01
,Antiglutamate therapy of ALS — which is the next step?, implies that we are likely to use combination therapies and have to try to treat patients early. The latter will be necessarily connected with the demand for a novel clinical attitude to the diagnosis of the disease.作者: Electrolysis 時(shí)間: 2025-3-25 21:56
,Antiglutamate therapies in Huntington’s disease,ydrochloride and Coenzyme Q10 in 340 patients with Huntington’s disease is described. This is the largest and longest multi-center trial in Huntington’s disease to address the glutamate- and mitochondrialmediated hypotheses of neurodegeneration.作者: 水槽 時(shí)間: 2025-3-26 03:09
Striatal reconstruction by striatal grafts,ence to corroborate or refute this hypothesis, in comparison with a lessspecific mechanism (or mechanisms) of recovery, is considered, including anatomical, electrophysiological and neurochemical demonstrations of functional circuit reconstruction in the host brain by striatal tissue transplants.作者: Intend 時(shí)間: 2025-3-26 04:46 作者: VICT 時(shí)間: 2025-3-26 08:33
,Dopamine agonists: what is the place of the newer compounds in the treatment of Parkinson’s diseasehan bromocriptine in this indication, at the cost of very similar adverse effects..In de novo levodopa naive patients, pramipexole and ropinirole did significantly better than placebo in short-term (few months) follow-up trials, at the cost again of classical dopaminergic adverse effects. Ropinirole作者: Reservation 時(shí)間: 2025-3-26 13:02
Apomorphine has a potent antiproliferative effect on Chinese hamster ovary cells, during incubation; CHO-K1 cells exposed to apomorphine for a period as short as 1 h and then allowed to grow for three days were significantly reduced in number with respect to untreated control cells. After four hours of exposition to apomorphine (10 μM) the antiproliferative effect was similar to作者: 澄清 時(shí)間: 2025-3-26 17:25
Potent neuroprotective and antioxidant activity of apomorphine in MPTP and 6-hydroxydopamine inducetyrosine hydroxylase activity. It is suggested that the neuroprotective effect of (R)-apomorphine against MPTP neurotoxicity derives from its radical scavenging and MAO inhibitory actions and not from its agonistic activity, since the mechanism of MPTP dopaminergic neurotoxicity involves the generat作者: Pudendal-Nerve 時(shí)間: 2025-3-26 23:31
Neural transplantation in animal models of multiple system atrophy: a review,opamine (6OHDA) and quinolinic acid (QA) into medial forebrain bundle and ipsilateral striatum, respectively (“double toxin — double lesion” approach). Preliminary evidence in rodents subjected to such lesions indicates that embryonic transplantation may partially reverse behavioural abnormalities. 作者: jabber 時(shí)間: 2025-3-27 04:08 作者: Original 時(shí)間: 2025-3-27 06:44
https://doi.org/10.1007/978-3-642-35401-4han bromocriptine in this indication, at the cost of very similar adverse effects..In de novo levodopa naive patients, pramipexole and ropinirole did significantly better than placebo in short-term (few months) follow-up trials, at the cost again of classical dopaminergic adverse effects. Ropinirole作者: Mosaic 時(shí)間: 2025-3-27 12:51
Multidimensional Diffusion Models during incubation; CHO-K1 cells exposed to apomorphine for a period as short as 1 h and then allowed to grow for three days were significantly reduced in number with respect to untreated control cells. After four hours of exposition to apomorphine (10 μM) the antiproliferative effect was similar to作者: FILTH 時(shí)間: 2025-3-27 16:19
Multidimensional Diffusion Modelstyrosine hydroxylase activity. It is suggested that the neuroprotective effect of (R)-apomorphine against MPTP neurotoxicity derives from its radical scavenging and MAO inhibitory actions and not from its agonistic activity, since the mechanism of MPTP dopaminergic neurotoxicity involves the generat作者: Axillary 時(shí)間: 2025-3-27 20:04
P. Dr?xler,C. M. Ringel,G. O. Michleropamine (6OHDA) and quinolinic acid (QA) into medial forebrain bundle and ipsilateral striatum, respectively (“double toxin — double lesion” approach). Preliminary evidence in rodents subjected to such lesions indicates that embryonic transplantation may partially reverse behavioural abnormalities. 作者: 收養(yǎng) 時(shí)間: 2025-3-28 00:17 作者: pellagra 時(shí)間: 2025-3-28 05:48
0303-6995 Overview: 978-3-211-83261-5978-3-7091-6369-6Series ISSN 0303-6995 作者: Trochlea 時(shí)間: 2025-3-28 08:48
Journal of Neural Transmission. Supplementahttp://image.papertrans.cn/a/image/149582.jpg作者: Ejaculate 時(shí)間: 2025-3-28 13:17 作者: Petechiae 時(shí)間: 2025-3-28 16:03 作者: 使無效 時(shí)間: 2025-3-28 19:38 作者: restrain 時(shí)間: 2025-3-28 23:24
Local Structure Prediction of Proteins,highly complex structures, which are often destructed in neurological disorders. One possible reason for the vulnerability of myelin sheaths to damage became apparent from analyses of animals with altered amounts of otherwise normal myelin components: Due to limited redundance in function between di作者: 廣口瓶 時(shí)間: 2025-3-29 06:13
https://doi.org/10.1007/978-3-642-35401-4ort the hypothesis that the pathogenesis is mediated by autoreactive T lymphocytes. Molecular mimicry has been proposed as a possible mechanism for the development of an autoimmune response to myelin antigens. According to this model, an immune reaction to self antigens could be initiated by T cells作者: 進(jìn)步 時(shí)間: 2025-3-29 08:43
https://doi.org/10.1007/978-3-642-35401-4son’s disease. Like any marketed dopamine agonists, the new compounds bind to the D2-like receptors. Pramipexole and ropinirole appear to be quite close drugs. Both are selective non ergot D2 (and preferentially D3) agonists, with an elimination half-life of 5 to 10 hours. Conversely, cabergoline is作者: CRANK 時(shí)間: 2025-3-29 15:13 作者: 不可比擬 時(shí)間: 2025-3-29 15:39 作者: Enteropathic 時(shí)間: 2025-3-29 19:52
https://doi.org/10.1007/978-3-642-35401-4th NMDA antagonists have been shown to induce a neuroprotective effect in animal models of this disease. As subthalamic nucleus neurons send direct glutamatergic projections to the substantia nigra, we studied the effects of kainic acid lesion of this nucleus on the degeneration of dopaminergic neur作者: DECRY 時(shí)間: 2025-3-30 00:55
Multidimensional Feller Processes in men indicates that antiglutamatergic strategies are the first to have an influence on its pathogenesis and slow down the disease process. Since the effect of the drugs is still small, this progress cannot only be seen as a success of the present but most also be acknowledged as a starting point 作者: 充滿人 時(shí)間: 2025-3-30 05:17
Multidimensional Feller Processesnetic defect leads to neuronal injury and death is unknown, but is thought to include glutamate-mediated excitotoxicity and abnormalities of mitochondrial energy production. Both of these mechanisms may lead to a final common pathway of increased production of free radical species. Prior clinical tr作者: 音樂戲劇 時(shí)間: 2025-3-30 11:59
