標(biāo)題: Titlebook: Advances in Research on Neurodegeneration; Volume 7 Y. Mizuno,D. B. Calne,M. B. H. Youdim Conference proceedings 2000 Springer-Verlag Wien [打印本頁(yè)] 作者: 類(lèi)屬 時(shí)間: 2025-3-21 17:17
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration影響因子(影響力)
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration被引頻次
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration被引頻次學(xué)科排名
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration年度引用
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration年度引用學(xué)科排名
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration讀者反饋
書(shū)目名稱(chēng)Advances in Research on Neurodegeneration讀者反饋學(xué)科排名
作者: 無(wú)效 時(shí)間: 2025-3-21 21:09 作者: interior 時(shí)間: 2025-3-22 03:06 作者: compose 時(shí)間: 2025-3-22 06:23
Computational Methods for Physicistshat continuous sustained stimulation of dopamine receptor may be responsible for its unusual pharmacological activity. Apomorphine has a far more broad neuroprotective activity in the various models as compared with 1-selegiline and may therefore be an ideal drug to study neuroprotection in parkinso作者: 黃瓜 時(shí)間: 2025-3-22 09:10 作者: avarice 時(shí)間: 2025-3-22 15:17 作者: MARS 時(shí)間: 2025-3-22 20:56 作者: NUDGE 時(shí)間: 2025-3-22 22:17 作者: 擔(dān)心 時(shí)間: 2025-3-23 01:30
https://doi.org/10.1007/978-0-387-68825-1of therapeutic delivery systems, which would reach the site of disease activity within the CNS, will permit the administration of inhibitors either of the cell death pathway or of effector target inter action and opens new avenues to neuroprotection approach.作者: 深陷 時(shí)間: 2025-3-23 06:41 作者: 泥土謙卑 時(shí)間: 2025-3-23 13:08 作者: AND 時(shí)間: 2025-3-23 15:59
Huntington disease: new insights on the role of huntingtin cleavage,ragments into the nucleus of striatal neurons. Increasing nuclear localization of huntingtin N-terminal fragments within medium spiny neurons of the striatum occurs concomitantly with the onset of selective neurodegeneration. Because huntingtin is a caspase substrate and truncated huntingtin fragmen作者: Collected 時(shí)間: 2025-3-23 18:00
,Autosomal-dominantly inherited forms of Parkinson’s disease,ce and typical Lewy-body pathology (PARK 1), and mutations have been identified in the gene for α-synuclein in these kindreds. Two other loci in families with dominant inheritance have been mapped, to chromosome 2p13 (PARK 3) and to chromosome 4p, respectively. A gene causing autosomal recessive par作者: hematuria 時(shí)間: 2025-3-23 23:43
,Subcellular localization of α-synuclein in primary neuronal cultures: effect of missense mutations,n appears to aggregate into distinct punctate inclusions in the cytoplasm and proximal neurites. Unlike Lewy bodies, these structures are not ubiquitin positive. These regions of α-synuclein accumulation are observed following transfections with wild-type, Ala30Pro or Ala53Thr α-synuclein; neither m作者: 戲法 時(shí)間: 2025-3-24 04:42
Iron chelating, antioxidant and cytoprotective properties of dopamine receptor agonist; apomorphinehat continuous sustained stimulation of dopamine receptor may be responsible for its unusual pharmacological activity. Apomorphine has a far more broad neuroprotective activity in the various models as compared with 1-selegiline and may therefore be an ideal drug to study neuroprotection in parkinso作者: 命令變成大炮 時(shí)間: 2025-3-24 08:08 作者: white-matter 時(shí)間: 2025-3-24 13:59 作者: indubitable 時(shí)間: 2025-3-24 18:43 作者: invulnerable 時(shí)間: 2025-3-24 20:46 作者: Influx 時(shí)間: 2025-3-25 00:16
Mechanisms of tissue injury in multiple sclerosis: opportunities for neuroprotective therapy,of therapeutic delivery systems, which would reach the site of disease activity within the CNS, will permit the administration of inhibitors either of the cell death pathway or of effector target inter action and opens new avenues to neuroprotection approach.作者: certitude 時(shí)間: 2025-3-25 04:39
https://doi.org/10.1007/978-0-387-74778-1 some members are implicated in cancer and nuronal diseases..Here, I overview the mechanism of how Bcl-2 family proteins regulate cell death, and how they are implicated in human diseases, particularly focusing on role of Bcl-2 in spinal muscular atropy.作者: Aerophagia 時(shí)間: 2025-3-25 09:13 作者: Evolve 時(shí)間: 2025-3-25 13:47
SpringerBriefs in Computer Sciencethat the aggregate formation and apoptosis are partially suppressed by trans glutaminase inhibitors, raising the possibility that trans glutaminase is involved in the aggregate body. The results may open new prospects for developing therapeutic measures for the polyglutamine diseases.作者: 寬大 時(shí)間: 2025-3-25 16:28
Structure Prediction of Membrane Proteins,ne therapy of PD is the protection of dopaminergic neurons in the substantia nigra using an AAV vector containing a glial cell line-derived neurotrophic factor (GDNF) gene. Combination of dopamine-supplement gene therapy and GDNF gene therapy would be a logical approach to the treatment of PD.作者: Hemiplegia 時(shí)間: 2025-3-25 21:15
Structure Prediction of Membrane Proteins, immune surveillance against HTLV-I and found that significant cytotoxic T lymphocyte activity, and suppressed natural killer activity and antibody-dependent cell-mediated cytotoxicity in the patients. These altered immune surveillance may be associated with the spread of HTLV-I infection and the pathogenesis of HAM/TSP.作者: Ejaculate 時(shí)間: 2025-3-26 04:00
Role of anti-apoptotic Bcl-2 protein in spinal muscular atrophy, some members are implicated in cancer and nuronal diseases..Here, I overview the mechanism of how Bcl-2 family proteins regulate cell death, and how they are implicated in human diseases, particularly focusing on role of Bcl-2 in spinal muscular atropy.作者: Perceive 時(shí)間: 2025-3-26 06:13 作者: entitle 時(shí)間: 2025-3-26 09:17
Dentatorubral-pallidoluysian atrophy (DRPLA),that the aggregate formation and apoptosis are partially suppressed by trans glutaminase inhibitors, raising the possibility that trans glutaminase is involved in the aggregate body. The results may open new prospects for developing therapeutic measures for the polyglutamine diseases.作者: SEED 時(shí)間: 2025-3-26 13:07
,Gene therapy of Parkinson’s disease using Adeno-Associated Virus (AAV) vectors,ne therapy of PD is the protection of dopaminergic neurons in the substantia nigra using an AAV vector containing a glial cell line-derived neurotrophic factor (GDNF) gene. Combination of dopamine-supplement gene therapy and GDNF gene therapy would be a logical approach to the treatment of PD.作者: Geyser 時(shí)間: 2025-3-26 19:34 作者: debacle 時(shí)間: 2025-3-26 21:32
The myelin oligodendrocyte glycoprotein (MOG): a model for antibody-mediated demyelination in expersingly, recent data suggest that this autoimmune response is in part regulated by polymorphisms in the MOG gene itself, an observation that may have important implications for the genetic and immunological stratification of patients with multiple sclerosis.作者: aspersion 時(shí)間: 2025-3-27 03:44 作者: Noctambulant 時(shí)間: 2025-3-27 06:17
David T. Melnekoff,Alessandro Laganàsingly, recent data suggest that this autoimmune response is in part regulated by polymorphisms in the MOG gene itself, an observation that may have important implications for the genetic and immunological stratification of patients with multiple sclerosis.作者: Emg827 時(shí)間: 2025-3-27 10:24
refecture in Japan on January 20 to 22, 1999. Karuizawa is a nice summer as well as winter resort close to the place for winter Olympic game in the year of 1998. Over 40 scientists gathered together and made hot discussion. Neurodegeneration and neuroinflammation are two major and important neurolog作者: Badger 時(shí)間: 2025-3-27 17:37 作者: Mitigate 時(shí)間: 2025-3-27 20:45
PARKIN as a pathogenic gene for autosomal recessive juvenile parkinsonism,ethnic origins, providing a definitive evidence for the Parkin to be a causative gene for AR-JP. The predicted structure of Parkin protein and its mutation provide important clues for studying the functional role of the Parkin protein in leading to selective degeneration of nigral neurons in the brains of AR-JP patients.作者: Euphonious 時(shí)間: 2025-3-27 22:52
https://doi.org/10.1007/978-3-7091-6284-2Alzheimer‘s Disease; Multiple Sclerosis; Neurodegeneration; Neuroinflammation; Parkinson; Parkinson‘s Dis作者: seruting 時(shí)間: 2025-3-28 03:41 作者: DAMP 時(shí)間: 2025-3-28 08:30
http://image.papertrans.cn/a/image/149581.jpg作者: senile-dementia 時(shí)間: 2025-3-28 11:33 作者: 犬儒主義者 時(shí)間: 2025-3-28 14:42 作者: Volatile-Oils 時(shí)間: 2025-3-28 20:12
Nanostructure Science and Technologysible for the pathogenesis of autosomal recessive juvenile parkinsonism (AR-JP). Various mutations were found in AR-JP patients of Japanese and other ethnic origins, providing a definitive evidence for the Parkin to be a causative gene for AR-JP. The predicted structure of Parkin protein and its mut作者: AV-node 時(shí)間: 2025-3-29 00:59 作者: 富足女人 時(shí)間: 2025-3-29 06:23
https://doi.org/10.1007/978-0-387-74778-1sis, cell turn over and removal of harmful cells. Disruption of the regulation of apoptosis may result in various diseases, including cancer and autoimmune diseases both associated with inhibition of apoptosis and various degenerative disorders associated with enhancement of apoptosis, and therefore作者: Acetaminophen 時(shí)間: 2025-3-29 08:43 作者: extract 時(shí)間: 2025-3-29 12:59
Optimal Disturbances in Boundary Layers,production of Aβ and the development of the disease..Thus, Aβ may not be the sole active fragment of β-amyloid precursor protein (βAPP) in the neurotoxicity assiciated with AD..We focused on the amyloidegenic carboxyl terminal fragments of βAPP containing the full length of Aβ (CT.). We synthesized 作者: 衣服 時(shí)間: 2025-3-29 17:38 作者: 膽小鬼 時(shí)間: 2025-3-29 22:46
Statistical Analysis and Modeling of Data,us human motor disorders. This paper reviews evidence from various sources which suggests that this standard model only incompletely accounts for aspects of basal ganglia function, and thus requires modification.作者: 卜聞 時(shí)間: 2025-3-30 01:25
David T. Melnekoff,Alessandro Laganàase, the level of N-methyl(R)salsolinol increased in cerebrospinal fluid and the high activity of a synthesizing enzyme, (R)salsolinol Nmethyltransferase, was detected in lymphocytes. This isoquinoline induced apoptotic DNA damage in human dopaminergic neuroblastoma SH-SY5Y cells. Among catechol iso作者: coalition 時(shí)間: 2025-3-30 07:31 作者: cajole 時(shí)間: 2025-3-30 09:25 作者: aptitude 時(shí)間: 2025-3-30 15:49 作者: 步履蹣跚 時(shí)間: 2025-3-30 19:23 作者: 吊胃口 時(shí)間: 2025-3-31 00:33
SpringerBriefs in Computer Sciencelutamine stretch. The prominent anticipation and broad spectrum in the clinical presentations of DRPLA have been demonstrated to be tightly correlated with the instability of CAG repeats in the DRPLA gene. Discovery of the causative gene for DRPLA has made it possible to investigate molecular mechan作者: FLAX 時(shí)間: 2025-3-31 03:07 作者: parsimony 時(shí)間: 2025-3-31 08:32
https://doi.org/10.1007/978-0-387-68825-1cules are able to induce relatively selective injury of oligodendrocytes (OLs) and their myelin membranes. The selectivity of this injury could be conferred either by the properties of the effectors or the targets. The former would involve antigen specific recognition by either antibody or T cell re作者: hematuria 時(shí)間: 2025-3-31 09:57 作者: VERT 時(shí)間: 2025-3-31 17:14
Statistical Analysis and Modeling of Data,us human motor disorders. This paper reviews evidence from various sources which suggests that this standard model only incompletely accounts for aspects of basal ganglia function, and thus requires modification.作者: GRUEL 時(shí)間: 2025-3-31 21:26
Huntington disease: new insights on the role of huntingtin cleavage,ransgenic mice expressing normal (YAC18) and mutant (YAC46 and YAC72) human huntingtin in a developmentally appropriate and tissue-specific manner identical to the pattern of expression of endogenous huntingtin. YAC46 and YAC72 mice show early electrophysiological abnormalities indicating neuronal c作者: acrimony 時(shí)間: 2025-3-31 23:12
PARKIN as a pathogenic gene for autosomal recessive juvenile parkinsonism,sible for the pathogenesis of autosomal recessive juvenile parkinsonism (AR-JP). Various mutations were found in AR-JP patients of Japanese and other ethnic origins, providing a definitive evidence for the Parkin to be a causative gene for AR-JP. The predicted structure of Parkin protein and its mut作者: 變態(tài) 時(shí)間: 2025-4-1 03:52
