派博傳思國際中心

標(biāo)題: Titlebook: Advances in Research on Neurodegeneration; Volume 10 R. Horowski,Y. Mizuno,M. B. H. Youdim Conference proceedings 2003 Springer-Verlag Wien [打印本頁]

作者: JAZZ    時間: 2025-3-21 18:51
書目名稱Advances in Research on Neurodegeneration影響因子(影響力)




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書目名稱Advances in Research on Neurodegeneration網(wǎng)絡(luò)公開度




書目名稱Advances in Research on Neurodegeneration網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Advances in Research on Neurodegeneration被引頻次




書目名稱Advances in Research on Neurodegeneration被引頻次學(xué)科排名




書目名稱Advances in Research on Neurodegeneration年度引用




書目名稱Advances in Research on Neurodegeneration年度引用學(xué)科排名




書目名稱Advances in Research on Neurodegeneration讀者反饋




書目名稱Advances in Research on Neurodegeneration讀者反饋學(xué)科排名





作者: irritation    時間: 2025-3-21 21:29
Jure Stojan,Milan Hodo??ek,Du?anka Jane?i?lls (APCs). Restimulation of invading T cells by such immature APCs (e.g. CD80 negative microglia) may lead to T cell anergy and/or differentiation of regulatory/Th3-like cells due to insufficient costimulation and presence of high levels of TGF-β and IL-10 in the CNS. Thus, T cell -apoptosis, -aner
作者: acrimony    時間: 2025-3-22 04:04

作者: 正面    時間: 2025-3-22 05:38

作者: 同位素    時間: 2025-3-22 10:05
A. Al Mers,A. Mimet,M. Boussouisses. These mechanisms that are usually interrelated in complex vitious circles finally leading to programmed cell death cascades are briefly discussed with reference to their pathogenetic role in many, albeit diverse neurodegenerative diseases, like Alzheimer disease, synucleino-pathies, tauopathies
作者: Cupidity    時間: 2025-3-22 15:05

作者: 不適    時間: 2025-3-22 17:32

作者: BADGE    時間: 2025-3-23 00:09
Solution of Linear Equation Systems,nation with psychotic behavior, which might also be associated with dopaminomimetic and/or anticholinergic treatment as well as with cholinergic deficit or dopaminomimetic induced REM sleep disturbances..As these psychiatric symptoms have a substantial negative effect on the patient’s quality of lif
作者: 允許    時間: 2025-3-23 05:02
Solution of the Navier-Stokes Equations,inant negative inhibitor (Apaf-1-DN) using AAV vector. We showed that Apaf-1-DN delivery using an AAV vector system could prevent nigrostriatal degeneration in MPTP mice, suggesting that it might be an antimitochondrial apoptotic gene therapy for PD.
作者: Ingest    時間: 2025-3-23 07:25
Turbulent-Flow Models and Calculationsd 33%, (n = 6), 42% (n = 6), and 48% (n = 3), respectively. No “off-state” dyskinesias have been observed. Based on these preliminary results, Spheramine? appears to show promise in treating late stage PD patients.
作者: 可卡    時間: 2025-3-23 11:40

作者: Anhydrous    時間: 2025-3-23 17:10
Self-tolerance in the immune privileged CNS: lessons from the entorhinal cortex lesion model,lls (APCs). Restimulation of invading T cells by such immature APCs (e.g. CD80 negative microglia) may lead to T cell anergy and/or differentiation of regulatory/Th3-like cells due to insufficient costimulation and presence of high levels of TGF-β and IL-10 in the CNS. Thus, T cell -apoptosis, -aner
作者: OUTRE    時間: 2025-3-23 18:11

作者: 竊喜    時間: 2025-3-23 23:54

作者: 共同給與    時間: 2025-3-24 05:16
General aspects of neurodegeneration,ses. These mechanisms that are usually interrelated in complex vitious circles finally leading to programmed cell death cascades are briefly discussed with reference to their pathogenetic role in many, albeit diverse neurodegenerative diseases, like Alzheimer disease, synucleino-pathies, tauopathies
作者: 同步信息    時間: 2025-3-24 06:47

作者: AROMA    時間: 2025-3-24 14:06
,Arguments for the use of dopamine receptor agonists in clinical and preclinical Parkinson’s diseasebenefit late in the therapy of the disorder. In addition, the pharmacological profile of dopamine receptor agonists suggests a possible neuroprotective effect. This paper reviews briefly the pharmacology of dopamine receptor agonists and basic knowledge concerning the dopamine receptor stimulation w
作者: Keratin    時間: 2025-3-24 15:39

作者: Headstrong    時間: 2025-3-24 22:24

作者: 柔軟    時間: 2025-3-25 00:02

作者: FOLLY    時間: 2025-3-25 05:02

作者: 不真    時間: 2025-3-25 07:59

作者: 使堅硬    時間: 2025-3-25 14:29
Computational Methods for Fluid Dynamicsride to D2 receptors is subject to competition from endogenous dopamine, this can be exploited to estimate changes in synaptic levels of dopamine. Assessment of processes downstream to the receptor will require the development of new approaches.
作者: 吞吞吐吐    時間: 2025-3-25 19:39
,Effect of advanced glycation endproducts on cell cycle and their relevance for Alzheimer’s disease,ry tangles), which time-dependently become modified by “advanced glycation endproducts (AGEs)”. Since AGEs activate both mitogenic and redox-sensitive pathways, they might be involved both in cell cycle re-entry and arrest.
作者: Countermand    時間: 2025-3-25 21:23
New concepts and tools in imaging for the study of neurodegenerative disease,ride to D2 receptors is subject to competition from endogenous dopamine, this can be exploited to estimate changes in synaptic levels of dopamine. Assessment of processes downstream to the receptor will require the development of new approaches.
作者: EVADE    時間: 2025-3-26 03:51

作者: 果仁    時間: 2025-3-26 04:28
,Growth/differentiation factor-15 (GDF-15), a novel member of the TGF-β superfamily, promotes survivmolecule can penetrate through the ependymal layer into the parenchyma. Analysis of mouse mutants lacking GDF-15 will reveal whether the endogenous factor also has a role in promoting embryonic and protecting lesioned nigral dopamine neurons.
作者: 轉(zhuǎn)向    時間: 2025-3-26 09:02
Progressive neurodegeneration in Drosophila: a model system,apoptosis or necrosis. In one case, mutant flies show abnormal behavior prior to obvious degeneration while most other mutants reveal such defects only in later stages. These mutants offer a new approach to study basic mechanisms of neurodegeneration and for developing fly models for human diseases.
作者: 上釉彩    時間: 2025-3-26 15:26

作者: Haphazard    時間: 2025-3-26 19:04
978-3-211-83907-2Springer-Verlag Wien 2003
作者: abduction    時間: 2025-3-26 23:41

作者: Herd-Immunity    時間: 2025-3-27 01:49
Electron Collisions with the , Cationl a molecular mechanism that can contribute to change synaptic weights may be represented by multiple interactions between membrane receptors forming aggregates (receptor mosaics) via oligomerization at both pre- and post-synaptic level..These assemblies of receptors together with inter alia single
作者: Charitable    時間: 2025-3-27 08:06

作者: 特征    時間: 2025-3-27 09:27

作者: infatuation    時間: 2025-3-27 13:39
J. Heredia,A. Medina Sierra,J. Carreraase with resulting cell death, predominantly by apoptosis. Further hallmarks of AD are crosslinked protein deposits (amyloid plaques and neurofibrillary tangles), which time-dependently become modified by “advanced glycation endproducts (AGEs)”. Since AGEs activate both mitogenic and redox-sensitive
作者: 惰性女人    時間: 2025-3-27 18:02

作者: 起草    時間: 2025-3-27 23:20
A. Al Mers,A. Mimet,M. Boussouison’s disease. Such models should reproduce the main characteristics of the disease, such as a selective lesion of dopaminergic neurons that evolves over time and the presence of neuronal inclusions known as Lewy bodies. Optimally, such models should also reproduce the lesion of non-dopaminergic neur
作者: Curmudgeon    時間: 2025-3-28 02:27
A. Al Mers,A. Mimet,M. Boussouisstem, often associated with cytoskeletal protein aggregates forming intracytoplasmic and/or intranuclear inclusions in neurons and/or glial cells. Most neurodegenerative disorders are now classified either according to the hitherto known genetic mechanisms or to the major components of their cellula
作者: fodlder    時間: 2025-3-28 09:41
Solution of Linear Equation Systems,rability and the presence of neuromelanin (NM) has not been elucidated. Early histological studies revealed the presence of increasing amounts of NM in the SN with aging in higher mammals, showed that NM granules are surrounded by membrane, and comparatively evaluated the pigmentation of SN in diffe
作者: AWE    時間: 2025-3-28 10:34

作者: AER    時間: 2025-3-28 14:54
Introduction to Numerical Methods, been suggested that .-DOPA itself may contribute to the progression of Parkinson’s disease. This hypothesis is, for many clinicians, the rationale for postponing the employment of and reducing the applied dosage of .-DOPA and for beginning therapy with dopamine receptor agonists or the monoamine ox
作者: Emasculate    時間: 2025-3-28 19:13

作者: cauda-equina    時間: 2025-3-29 00:42
Solution of the Navier-Stokes Equations,s, both in vitro and in vivo. Specifically, GDF-15 promotes survival and differentiation of embryonic rat dopaminergic neurons, but not of other neuron populations, with the exception of serotonergic raphe neurons. The neurotrophic effect of GDF-15 seems to be direct and not mediated through glial c
作者: 未完成    時間: 2025-3-29 03:22

作者: Sigmoidoscopy    時間: 2025-3-29 07:36
Turbulent-Flow Models and Calculationss attached to gelatin microcarriers (Spheramine?) in rodent and non-human primate models of Parkinson’s disease (PD) produces long term amelioration of motor and behavioral deficits, with histological and PET evidence of cell survival without immunosuppression. Long-term safety in cynomologous monke
作者: appall    時間: 2025-3-29 14:48
Advances in Research on Neurodegeneration978-3-7091-0643-3Series ISSN 0303-6995
作者: Arrhythmia    時間: 2025-3-29 18:42
0303-6995 Overview: 978-3-211-83907-2978-3-7091-0643-3Series ISSN 0303-6995
作者: 實現(xiàn)    時間: 2025-3-29 20:45
Possible role of intramembrane receptor-receptor interactions in memory and learning via formation l a molecular mechanism that can contribute to change synaptic weights may be represented by multiple interactions between membrane receptors forming aggregates (receptor mosaics) via oligomerization at both pre- and post-synaptic level..These assemblies of receptors together with inter alia single
作者: 極微小    時間: 2025-3-30 01:18

作者: explicit    時間: 2025-3-30 04:46

作者: 使成波狀    時間: 2025-3-30 11:25

作者: CHIP    時間: 2025-3-30 15:25
What have we learnt from CDNA microarray gene expression studies about the role of iron in MPTP inds animal models, MPTP (N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine) and 6-hydroxydopamine. The advent of cDNA microarray gene expression where expression of thousands of genes can be globally assessed has indicated that mechanism of neurodegeneration by MPTP is a complex cascade of vicious circles.
作者: FIR    時間: 2025-3-30 20:22
,Animal models of Parkinson’s disease in rodents induced by toxins: an update,on’s disease. Such models should reproduce the main characteristics of the disease, such as a selective lesion of dopaminergic neurons that evolves over time and the presence of neuronal inclusions known as Lewy bodies. Optimally, such models should also reproduce the lesion of non-dopaminergic neur
作者: Medley    時間: 2025-3-30 22:56





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