標題: Titlebook: Adhesion?GPCRs; Structure to Functio Simon Yona,Martin Stacey Book 2010 The Editor(s) (if applicable) and The Author(s), under exclusive li [打印本頁] 作者: minutia 時間: 2025-3-21 19:58
書目名稱Adhesion?GPCRs影響因子(影響力)
書目名稱Adhesion?GPCRs影響因子(影響力)學(xué)科排名
書目名稱Adhesion?GPCRs網(wǎng)絡(luò)公開度
書目名稱Adhesion?GPCRs網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Adhesion?GPCRs被引頻次
書目名稱Adhesion?GPCRs被引頻次學(xué)科排名
書目名稱Adhesion?GPCRs年度引用
書目名稱Adhesion?GPCRs年度引用學(xué)科排名
書目名稱Adhesion?GPCRs讀者反饋
書目名稱Adhesion?GPCRs讀者反饋學(xué)科排名
作者: hurricane 時間: 2025-3-22 00:18 作者: 多嘴多舌 時間: 2025-3-22 01:08 作者: FIR 時間: 2025-3-22 06:51
Front Matterues can be successfully extended for nonlinear processes. A more detailed treatment of linear system identification can be found in [40, 81, 171, 172, 193, 233, 360]. Practical experience can be easily gathered by playing around with the MATLAB system identification toolbox [234].作者: CAPE 時間: 2025-3-22 12:02 作者: 無底 時間: 2025-3-22 16:47
GPS Proteolytic Cleavage of Adhesion-GPCRs,oundary value problems of fractional differential systems,?it is expected that it will attract the attention of researchers, modelers and?graduate students who are interested in doing their research on fractional?differential systems.??..?..?.978-3-031-62515-2978-3-031-62513-8作者: 磨坊 時間: 2025-3-22 17:44 作者: Concomitant 時間: 2025-3-22 21:30
Studies on the Very Large G Protein-Coupled Receptor: From Initial Discovery to Determining its Rolignored in conventional ultrasonic NDE methods. Hence, in this chapter, it is hypothesized that such nonlinear effects at the early stage of damage at the lower scale are actually sensed by the ultrasonic NDE probes/sensors and hidden in the ultrasonic signals. Such hidden features are required to b作者: SYN 時間: 2025-3-23 04:06
Adhesion-GPCRs in the CNS,others are unstable but evolve slowly in time into periodically modulated waves. Although the occurrence of multiple families of free waves is interesting for theoretical reasons, practical calculations of waves near resonance should include both forcing and damping. For any given depth ratio, forci作者: Calibrate 時間: 2025-3-23 05:57
Adhesion-GPCRs in Tumorigenesis,urring in “smart” brain-like media that are characterized by highly nontrivial local dynamics and complex networks with nonlocal interactions. Scientists have addressed the dynamics and studied the general principles of learning and memory, and so forth. The ideas and principles also are used in mod作者: 光明正大 時間: 2025-3-23 11:32 作者: 殘忍 時間: 2025-3-23 16:00 作者: CRAFT 時間: 2025-3-23 21:07
Signal Transduction Mediated through Adhesion-GPCRs,echanical energy, which for ideal fluids is the Bernoulli equation (Batchelor 1967; Tritton 1988, etc.). Furthermore, it is also well known that for spatially 1-. models this equation may replace the momentum equation as an equivalent dynamical equation. If this equation is subjected to a turbulence作者: Indecisive 時間: 2025-3-24 01:49 作者: 流行 時間: 2025-3-24 04:22 作者: 2否定 時間: 2025-3-24 08:34 作者: 膠水 時間: 2025-3-24 13:10 作者: anthropologist 時間: 2025-3-24 15:16
Emerging Roles of Brain-Specific Angiogenesis Inhibitor 1,978-1-349-12227-1作者: 強制令 時間: 2025-3-24 19:08 作者: 造反,叛亂 時間: 2025-3-25 02:12 作者: 平躺 時間: 2025-3-25 06:02
Baukonstruktion - vom Prinzip zum Detailatrophilin 1 may control synapse maturation and exocytosis, whereas latrophilin 2 may be involved in breast cancer. Latrophilins may play different roles during development and in adult animals: thus, LAT-1 determines cell fate in early embryogenesis in . and controls neurotransmitter release in adu作者: arboretum 時間: 2025-3-25 10:34
Baukonstruktion - vom Prinzip zum Detailpairment. Although its ligand is currently unknown, VLGR1 is one of the few adhesion-GPCR family members in which mutations have been shown to be responsible for a human malady. Mutations in VLGR1 in humans result in one form (2C) of Usher syndrome, the most common genetic cause of combined blindnes作者: chondromalacia 時間: 2025-3-25 13:57 作者: 放牧 時間: 2025-3-25 18:29
Lutz Franke,Gernod Deckelmann,Holger Stehrtis (CIA), a model for rheumatoid arthritis, was significantly ameliorated suggesting therapeutic value of CD97 targeting. CD97-deficient mice are essentially normal at steady state except for a mild granulocytosis, which increases under inflammatory conditions. Comparison of the consequences of ant作者: alcohol-abuse 時間: 2025-3-25 23:20 作者: anus928 時間: 2025-3-26 01:36 作者: 背信 時間: 2025-3-26 04:26
Baukonstruktion - vom Prinzip zum Detaillopmental biology. We have recently demonstrated that the putative neurotoxin receptor . defines a mechanism required for the alignment of cell division planes in the early embryo of the nematode .. Our analysis suggests that . is required for the propagation rather than the initial establishment of作者: cancer 時間: 2025-3-26 10:09
Baukonstruktion - vom Prinzip zum Detaillysis site (GPS) has been identified as an intrinsic protein modification process of many adhesion-GPCRs. In recent years, the conserved cleavage site, molecular mechanism and the potential functional implication of the GPS proteolysis have been gradually unveiled. However, many aspects of this uniq作者: 佛刊 時間: 2025-3-26 16:19
Baukonstruktion - vom Prinzip zum Detailes strong neuronal exocytosis in vertebrates. Latrophilin has an unusual structure consisting of two fragments that are produced by the proteolytic cleavage of the parental molecule and that behave independently in the plasma membrane. On binding an agonist, the fragments reassociate and send an int作者: exquisite 時間: 2025-3-26 18:18 作者: 維持 時間: 2025-3-26 21:48
Baukonstruktion - vom Prinzip zum Detaileptors are expressed in the central nervous system (CNS), indicating their possible roles in the development and function of the CNS. Indeed, it has been shown that adhesion-GPCRs are involved in the regulation of neurulation, cortical development and neurite growth. Among the few adhesion-GPCRs bei作者: 休息 時間: 2025-3-27 03:06 作者: 規(guī)范要多 時間: 2025-3-27 06:53
https://doi.org/10.1007/978-3-662-64827-8s and extracellular matrix turn-over. Each of the steps is influenced by tumor cell interaction and interaction of the tumor cell with its microenvironment that consists of different cell types as tumor-associated fibroblasts, endothelial cells and leukocytes as well as the extracellular matrix prod作者: 全等 時間: 2025-3-27 12:40 作者: 注意到 時間: 2025-3-27 15:34 作者: accessory 時間: 2025-3-27 18:42
https://doi.org/10.1007/978-3-663-09351-0a role in T-cell function. It was subsequently identified as a ligand for CD55, which has been previously identified as an innate regulator of complement. The interaction of this receptor-ligand pair has been shown to provide a potent costimulatory signal to human T cells, despite their modest affin作者: Jingoism 時間: 2025-3-27 22:42 作者: EXCEL 時間: 2025-3-28 06:05
Baukonstruktion – vom Prinzip zum Detailue structure containing a cleaved long extracellular domain (ECD) and a seven-transmembrane domain (7TM). In this chapter, we review previous reports which suggest G protein-dependent and -independent signaling pathways of adhesion-GPCRs and present our approach to investigate the signal transductio作者: Alienated 時間: 2025-3-28 10:06
Baukonstruktion – vom Prinzip zum Detailmed for the ability of its extracellular region to inhibit angiogenesis in tumor models, its function in physiological contexts was elusive and remained an orphan receptor until recently..,.,.–. BAI1 is now considered a phagocytic receptor that can recognize phosphatidylserine exposed on apoptotic c作者: Eructation 時間: 2025-3-28 10:45
Baukonstruktion – vom Prinzip zum Detailxpression, little is known about the function of this receptor family in male reproductive physiology. Insights into function are beginning to emerge with the increasing availability of genetically modified mice harbouring mutations in these genes. Gpr64 is the best characterised of the adhesion-GPC作者: Heresy 時間: 2025-3-28 15:45
Simon Yona,Martin StaceyIncludes evolution of the adhesion-GPCR genes in several species.Explores how this proteolytic cleavage was identified as an intrinsic protein modification process in the majority adhesion-GPCRs.Descr作者: Ankylo- 時間: 2025-3-28 20:36
Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/a/image/144923.jpg作者: maculated 時間: 2025-3-29 01:25 作者: 怕失去錢 時間: 2025-3-29 03:56
Baukonstruktion – vom Prinzip zum Detailue structure containing a cleaved long extracellular domain (ECD) and a seven-transmembrane domain (7TM). In this chapter, we review previous reports which suggest G protein-dependent and -independent signaling pathways of adhesion-GPCRs and present our approach to investigate the signal transduction of the adhesion-GPCR, GPR56.作者: PANG 時間: 2025-3-29 10:30
https://doi.org/10.1007/978-1-4419-7913-1Adhesion; Function; GPCR; Stacey; Structure; Yona; leukocyte; tissue作者: V切開 時間: 2025-3-29 12:58
The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines作者: ARK 時間: 2025-3-29 16:42 作者: Genteel 時間: 2025-3-29 21:20
The , GPCRs; Gene Repertoire, Phylogeny and Evolution,ary and the fuzzy logic interpretation vanishes in the background. A couple of attractive key features of these model architectures are analyzed in this chapter. Two of them shall be highlighted here: The separation of the inputs for rule premises and for the rule consequents (local models) allows t作者: 使迷醉 時間: 2025-3-30 01:30 作者: 認為 時間: 2025-3-30 05:33 作者: LEVY 時間: 2025-3-30 10:13 作者: bifurcate 時間: 2025-3-30 16:13
Adhesion-GPCRs in the CNS,ssing around a circular basin of finite uniform depth, with particular attention being given to those waves near resonance. Multiple families of free steady waves are associated with each of the depths at which resonance occurs, with different families having different orderings of the wave componen作者: NAVEN 時間: 2025-3-30 19:35
GPR56 Interacts with Extracellular Matrix and Regulates Cancer Progression,solitons of Bose–Einstein condensates.Studies the dynamics oThis book highlights the methods to engineer dissipative and magnetic nonlinear waves propagating in nonlinear systems. In the first part of the book, the authors present methodologically mathematical models of nonlinear waves propagating i作者: Flatter 時間: 2025-3-31 00:12
